{"title":"潜在的广谱抗病毒药物通过触发先天免疫起作用","authors":"D. Holzman","doi":"10.1128/MICROBE.11.100.1","DOIUrl":null,"url":null,"abstract":"Low-molecular-weight compounds that trigger innate immunity response genes might serve as broad-spectrum antiviral agents, acting through the innate immune system to suppress a wide range of RNA viruses, including influenza A and hepatitis C, as well as the emerging dengue, Ebola, Lassa, Nipah, and West Nile viruses, according to Michael Gale, Jr., of the University of Washington, Seattle, and his collaborators. The research appeared 16 December 2015 in the Journal of Virology (doi:10.1128/JVI.02202–15).","PeriodicalId":87479,"journal":{"name":"Microbe (Washington, D.C.)","volume":null,"pages":null},"PeriodicalIF":0.0000,"publicationDate":"2016-03-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Potential Broad-Spectrum Antiviral Agents Act by Triggering Innate Immunity\",\"authors\":\"D. Holzman\",\"doi\":\"10.1128/MICROBE.11.100.1\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Low-molecular-weight compounds that trigger innate immunity response genes might serve as broad-spectrum antiviral agents, acting through the innate immune system to suppress a wide range of RNA viruses, including influenza A and hepatitis C, as well as the emerging dengue, Ebola, Lassa, Nipah, and West Nile viruses, according to Michael Gale, Jr., of the University of Washington, Seattle, and his collaborators. The research appeared 16 December 2015 in the Journal of Virology (doi:10.1128/JVI.02202–15).\",\"PeriodicalId\":87479,\"journal\":{\"name\":\"Microbe (Washington, D.C.)\",\"volume\":null,\"pages\":null},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2016-03-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Microbe (Washington, D.C.)\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.1128/MICROBE.11.100.1\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Microbe (Washington, D.C.)","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.1128/MICROBE.11.100.1","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Potential Broad-Spectrum Antiviral Agents Act by Triggering Innate Immunity
Low-molecular-weight compounds that trigger innate immunity response genes might serve as broad-spectrum antiviral agents, acting through the innate immune system to suppress a wide range of RNA viruses, including influenza A and hepatitis C, as well as the emerging dengue, Ebola, Lassa, Nipah, and West Nile viruses, according to Michael Gale, Jr., of the University of Washington, Seattle, and his collaborators. The research appeared 16 December 2015 in the Journal of Virology (doi:10.1128/JVI.02202–15).
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