小关节综合征的随机观察

B. Sweetman
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It would take up far too much room here to review all the work in this field that has been accumulating since the syndrome was originally posited a century ago. But whilst out for my walk earlier this morning, I noticed that my right-sided low back pain was worse. I have all along believed that this trouble has been due to a predominantly unilateral right-sided facet joint syndrome, particularly as the diagnostic system developed from our above-mentioned research had scored me for this explanation. As you do when out walking, I decided to check what was happening. Our existing main criterion for the diagnosis was the paradoxical demonstration of the right-sided low back pain being exacerbated by bending sideways or twisting in the opposite direction, i.e. moves towards the left side. The other syndromes do not do this. Any syndrome can hurt if you bend or twist to the side of the pain, but the ‘contralateral’ phenomenon is seemingly restricted to facet joint involvement. Up until now, I had thought that pain exacerbation on lateral flection, as opposed to rotational movement, might distinguish between involvement of the lowest lumbosacral facet joints and that twisting implicated the joints above at L4/5. On testing, this morning I seemed to get different answers on repeating the tests. I then realized that lateral flexion would induce the contralateral pain only if I was standing whilst doing the test when my posture was slightly bent forwards (flexed). In contrast, lateral rotation induced the contralateral pain if my posture standing was slightly bent backwards (extension). For me, this was an ‘Oh wow!’ moment. I thought I should share these ideas just in case any readers were deciding to set up a study to help differentiate the various forms of back pain presentation. Of the vast number of tests that have been proposed, it is difficult to select a shortlist for inclusion at the outset of such clinical research. A recent editorial criticized a lot of cluster analysis studies for involving an insufficient number of cases and carrying out inadequately sophisticated analytic work. But, whilst this morning’s observations were made on only one case (n= 1) and there were no studies of repeatability, reliability, or validity, I do hope that these suggestions are worth considering, and if not, perhaps further discussion or criticism might be thought worthwhile. One can of course now develop and run such a program on the ubiquitous portable lap top computer. Why, I also ask, is it that I seem to have such trouble in the first place? If osteoarthritis (OA) is the likely mechanism for facet joint involvement, then my problems may have been predestined. My mother had some rheumatics and Heberden’s nodes, and seemingly I may have early such changes. OA is very common and I only have one ‘T’ reducing my risk, according to a genetic profile performed on a spit sample recently. 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It would take up far too much room here to review all the work in this field that has been accumulating since the syndrome was originally posited a century ago. But whilst out for my walk earlier this morning, I noticed that my right-sided low back pain was worse. I have all along believed that this trouble has been due to a predominantly unilateral right-sided facet joint syndrome, particularly as the diagnostic system developed from our above-mentioned research had scored me for this explanation. As you do when out walking, I decided to check what was happening. Our existing main criterion for the diagnosis was the paradoxical demonstration of the right-sided low back pain being exacerbated by bending sideways or twisting in the opposite direction, i.e. moves towards the left side. The other syndromes do not do this. Any syndrome can hurt if you bend or twist to the side of the pain, but the ‘contralateral’ phenomenon is seemingly restricted to facet joint involvement. Up until now, I had thought that pain exacerbation on lateral flection, as opposed to rotational movement, might distinguish between involvement of the lowest lumbosacral facet joints and that twisting implicated the joints above at L4/5. On testing, this morning I seemed to get different answers on repeating the tests. I then realized that lateral flexion would induce the contralateral pain only if I was standing whilst doing the test when my posture was slightly bent forwards (flexed). In contrast, lateral rotation induced the contralateral pain if my posture standing was slightly bent backwards (extension). For me, this was an ‘Oh wow!’ moment. I thought I should share these ideas just in case any readers were deciding to set up a study to help differentiate the various forms of back pain presentation. Of the vast number of tests that have been proposed, it is difficult to select a shortlist for inclusion at the outset of such clinical research. A recent editorial criticized a lot of cluster analysis studies for involving an insufficient number of cases and carrying out inadequately sophisticated analytic work. But, whilst this morning’s observations were made on only one case (n= 1) and there were no studies of repeatability, reliability, or validity, I do hope that these suggestions are worth considering, and if not, perhaps further discussion or criticism might be thought worthwhile. One can of course now develop and run such a program on the ubiquitous portable lap top computer. Why, I also ask, is it that I seem to have such trouble in the first place? If osteoarthritis (OA) is the likely mechanism for facet joint involvement, then my problems may have been predestined. My mother had some rheumatics and Heberden’s nodes, and seemingly I may have early such changes. OA is very common and I only have one ‘T’ reducing my risk, according to a genetic profile performed on a spit sample recently. Our industrial studies seemed to suggest that back rotation can in varying degrees induce such problems. Therewas a spread, fromwork being associatedwith frequent twisting, and at the other extreme one or a few such injuries, the latter presumably more forceful on the facet joints. In my teens I may have provoked the latter mechanism with some back strains due to awkward waterentries from high board diving as well as a particular rugby injury. 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引用次数: 0

摘要

在最近的IBM SPSS用户会议上,有人认为这是机器学习的时代,在这个时代,大量数据筛选可以定义用于检查的模型,预测分析可以进一步定义这些概念。我们自己的团队在近三分之一个世纪前就开始了这样的研究,当时世界上只有为数不多的大型计算机能够处理执行这些任务的新程序。我们能够检查常见腰痛形式的复杂性,通常被称为非特异性腰痛,这反过来意味着临床图片是“不可分割的”。“在这种发病率的传播中,我们认为我们可以识别出六种左右不同的图像,其中一种随后被认为是难以捉摸的关节突(颧骨)关节综合征的表现。”在这里,要回顾自一个世纪前该综合征最初被提出以来在这一领域所积累的所有工作,将占用太多的篇幅。但是今天早上早些时候出去散步的时候,我注意到我的右侧腰痛更严重了。我一直认为,这种麻烦主要是由于单侧右侧小关节综合征,特别是从我们上述研究中发展出来的诊断系统已经为我的这种解释加分。就像你出去散步时做的那样,我决定看看发生了什么事。我们现有的主要诊断标准是右侧腰痛的矛盾表现,即侧弯或相反方向的扭转,即向左侧移动,加剧了右侧腰痛。其他综合症不会这样做。如果你弯曲或扭曲到疼痛的一侧,任何综合症都可能疼痛,但“对侧”现象似乎仅限于小关节受累。到目前为止,我一直认为,与旋转运动相反,侧屈引起的疼痛加剧可能会区分腰骶关节关节面和腰4/5关节的扭曲。关于测试,今天早上我似乎在重复测试时得到了不同的答案。然后我意识到,只有当我站着做测试时,我的姿势稍微向前弯曲(弯曲)时,侧屈才会引起对侧疼痛。相反,如果我站姿稍微向后弯曲(伸展),侧旋会引起对侧疼痛。对我来说,这是一个“哦哇!”的时刻。我想我应该分享这些想法,以防有读者决定建立一个研究来帮助区分各种形式的背痛表现。在已经提出的大量测试中,在此类临床研究开始时很难选择一个入围名单。最近的一篇社论批评了许多聚类分析研究,因为它们涉及的病例数量不足,分析工作不够复杂。但是,虽然今天上午的观察只针对一个案例(n= 1),并且没有对可重复性,可靠性或有效性的研究,但我确实希望这些建议值得考虑,如果没有,也许进一步的讨论或批评可能被认为是值得的。当然,现在人们可以在随处可见的便携式笔记本电脑上开发和运行这样的程序。我又问,为什么我一开始就有这样的麻烦?如果骨关节炎(OA)是小关节受累的可能机制,那么我的问题可能是注定的。我母亲有一些风湿病和希伯登淋巴结,看来我可能有早期的这些变化。根据最近对唾液样本进行的基因分析,OA很常见,我只有一个“T”,这降低了我的风险。我们的工业研究似乎表明,背部旋转会在不同程度上引起这些问题。在另一种极端情况下,有一个或几个这样的损伤,后者可能对关节突关节更有力。在我十几岁的时候,我可能因为在高跳板跳水时笨拙的入水以及一次特殊的橄榄球受伤而引发了后一种机制。年龄也在影响我。
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Casual observations on the facet joint syndrome
Recent IBM conference for SPSS users, it was suggested that this was the era of machine learning in which mass data screening could define models for examination and predictive analysis could further define such concepts. Our own team were able to start such studies nearly a third of a century ago with the help of the few existing mainframe computers in the world that could cope with the fledgling programs performing these tasks. We were able to examine the complexity of common forms of low back pain, often referred to as non-specific back pain, which in turn implied that the clinical picture was ‘indivisible.’ Within this spread of morbidity, we thought that we could identify half a dozen or so distinct pictures, one of which was subsequently considered to be the manifestation of the elusive facet (zygapophysial) joint syndrome. It would take up far too much room here to review all the work in this field that has been accumulating since the syndrome was originally posited a century ago. But whilst out for my walk earlier this morning, I noticed that my right-sided low back pain was worse. I have all along believed that this trouble has been due to a predominantly unilateral right-sided facet joint syndrome, particularly as the diagnostic system developed from our above-mentioned research had scored me for this explanation. As you do when out walking, I decided to check what was happening. Our existing main criterion for the diagnosis was the paradoxical demonstration of the right-sided low back pain being exacerbated by bending sideways or twisting in the opposite direction, i.e. moves towards the left side. The other syndromes do not do this. Any syndrome can hurt if you bend or twist to the side of the pain, but the ‘contralateral’ phenomenon is seemingly restricted to facet joint involvement. Up until now, I had thought that pain exacerbation on lateral flection, as opposed to rotational movement, might distinguish between involvement of the lowest lumbosacral facet joints and that twisting implicated the joints above at L4/5. On testing, this morning I seemed to get different answers on repeating the tests. I then realized that lateral flexion would induce the contralateral pain only if I was standing whilst doing the test when my posture was slightly bent forwards (flexed). In contrast, lateral rotation induced the contralateral pain if my posture standing was slightly bent backwards (extension). For me, this was an ‘Oh wow!’ moment. I thought I should share these ideas just in case any readers were deciding to set up a study to help differentiate the various forms of back pain presentation. Of the vast number of tests that have been proposed, it is difficult to select a shortlist for inclusion at the outset of such clinical research. A recent editorial criticized a lot of cluster analysis studies for involving an insufficient number of cases and carrying out inadequately sophisticated analytic work. But, whilst this morning’s observations were made on only one case (n= 1) and there were no studies of repeatability, reliability, or validity, I do hope that these suggestions are worth considering, and if not, perhaps further discussion or criticism might be thought worthwhile. One can of course now develop and run such a program on the ubiquitous portable lap top computer. Why, I also ask, is it that I seem to have such trouble in the first place? If osteoarthritis (OA) is the likely mechanism for facet joint involvement, then my problems may have been predestined. My mother had some rheumatics and Heberden’s nodes, and seemingly I may have early such changes. OA is very common and I only have one ‘T’ reducing my risk, according to a genetic profile performed on a spit sample recently. Our industrial studies seemed to suggest that back rotation can in varying degrees induce such problems. Therewas a spread, fromwork being associatedwith frequent twisting, and at the other extreme one or a few such injuries, the latter presumably more forceful on the facet joints. In my teens I may have provoked the latter mechanism with some back strains due to awkward waterentries from high board diving as well as a particular rugby injury. Age is also catching up with me.
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