甲状旁腺切除术后的饥饿骨综合征:发病率和预测因素

Clinical case reports and reviews Pub Date : 2019-01-01 DOI:10.15761/ccrr.1000458

S. Mezri, S. Sayhi, Hadhemi Ben chikha, R. Ben M'hamed, K. Akkari

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摘要

饥饿骨综合征(HBS)是主要发生在原发性甲状旁腺功能亢进(PHP)或继发性甲状旁腺功能亢进(SHPT)甲状旁腺切除术后的重要并发症，通常未被诊断出来。它被定义为长期低钙血症伴低磷血症，并因抑制甲状旁腺激素(PTH)水平而加重[1,2]。事实上，在HBS b[3]期间，密集的骨转换和对钙的过度贪婪得到了发展。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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Hungry bone syndrome after parathyroidectomy: Incidence and predictive factors

The hungry bone syndrome (HBS) is an important complication often under diagnosed that occurs essentially after parathyroidectomy for primary hyperparathyroidism (PHP) or secondary hyperparathyroidism (SHPT). It is defined as a prolonged hypocalcaemia with hypophosphataemia hat exacerbated by suppressed parathyroid hormone (PTH) levels [1,2]. Indeed, intensive bone turnover and excessive greed for calcium is developed during the HBS [3].

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Clinical case reports and reviews

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