{"title":"甲状旁腺与COVID-19;被忽视的实体","authors":"H. Nasri","doi":"10.34172/jpd.2022.11165","DOIUrl":null,"url":null,"abstract":"Implication for health policy/practice/research/medical education: The mechanisms interacted as the etiologies of hypocalcemia in SARS-CoV-2 are impaired intestinal absorption of calcium, disturbed secretion or impaired response to parathormone due to increased concentration of inflammatory cytokines, vitamin D insufficiency and hypoalbuminemia. Moreover, hypoxia-induce tissue injury with consequent rise in calcium influx are another possible mechanism. However, the effect of SARS-CoV-2 on parathormone secretion could explain the hypocalcemia of COVID-19 patients.","PeriodicalId":16657,"journal":{"name":"Journal of Parathyroid Disease","volume":"278 1","pages":""},"PeriodicalIF":0.0000,"publicationDate":"2022-10-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Parathyroid glands and COVID-19; a neglected entity\",\"authors\":\"H. Nasri\",\"doi\":\"10.34172/jpd.2022.11165\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Implication for health policy/practice/research/medical education: The mechanisms interacted as the etiologies of hypocalcemia in SARS-CoV-2 are impaired intestinal absorption of calcium, disturbed secretion or impaired response to parathormone due to increased concentration of inflammatory cytokines, vitamin D insufficiency and hypoalbuminemia. Moreover, hypoxia-induce tissue injury with consequent rise in calcium influx are another possible mechanism. However, the effect of SARS-CoV-2 on parathormone secretion could explain the hypocalcemia of COVID-19 patients.\",\"PeriodicalId\":16657,\"journal\":{\"name\":\"Journal of Parathyroid Disease\",\"volume\":\"278 1\",\"pages\":\"\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2022-10-19\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Journal of Parathyroid Disease\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.34172/jpd.2022.11165\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of Parathyroid Disease","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.34172/jpd.2022.11165","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Parathyroid glands and COVID-19; a neglected entity
Implication for health policy/practice/research/medical education: The mechanisms interacted as the etiologies of hypocalcemia in SARS-CoV-2 are impaired intestinal absorption of calcium, disturbed secretion or impaired response to parathormone due to increased concentration of inflammatory cytokines, vitamin D insufficiency and hypoalbuminemia. Moreover, hypoxia-induce tissue injury with consequent rise in calcium influx are another possible mechanism. However, the effect of SARS-CoV-2 on parathormone secretion could explain the hypocalcemia of COVID-19 patients.
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