Ralf Dittrich, Martin A. Ritter, Erich B. Ringelstein
{"title":"超声在自发性颈动脉夹层中的应用","authors":"Ralf Dittrich, Martin A. Ritter, Erich B. Ringelstein","doi":"10.1016/j.permed.2012.02.024","DOIUrl":null,"url":null,"abstract":"<div><p>Spontaneous cervical artery dissection is caused by a hematoma in the arterial wall. Recent research revealed that the most likely pathophysiological key mechanism is rupture of a vas vasorum resulting in a bleeding into the medio-adventitial borderzone <span>[1]</span>. The expansion of the hematoma into the arterial lumen can secondarily lead to a rupture of the tunica intima with a high risk of thrombus formation and embolic cerebral infarction <span>[2]</span>. Moreover the expansion of the hematoma causes an arterial stenosis or arterial occlusion with the risk of hemodynamic impairment. The risk of an ischemic stroke in the course of a dissection is thought to be about 70% for dissections of the internal carotid artery (ICA) <span>[3]</span> and about 80% for dissections of the vertebral artery (VA) <span>[4]</span>. The annual incidence of dissections of the ICA has been estimated to be 2.5–3/100,000 and for the VA 0.97–1.5/100,000 <span>[5]</span>, <span>[6]</span>. Although dissections as such are rare they are a frequent etiology of stroke in children and young adults. Approximately 25% of the strokes in patients younger than 50 are caused by dissections with a peak age between 40 and 45 years <span>[7]</span>, <span>[8]</span>, <span>[9]</span>, <span>[10]</span>, <span>[11]</span>, <span>[12]</span>, <span>[13]</span>, <span>[14]</span>, <span>[15]</span>, <span>[16]</span>.</p></div>","PeriodicalId":101010,"journal":{"name":"Perspectives in Medicine","volume":"1 1","pages":"Pages 250-254"},"PeriodicalIF":0.0000,"publicationDate":"2012-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/j.permed.2012.02.024","citationCount":"9","resultStr":"{\"title\":\"Ultrasound in spontaneous cervical artery dissection\",\"authors\":\"Ralf Dittrich, Martin A. Ritter, Erich B. Ringelstein\",\"doi\":\"10.1016/j.permed.2012.02.024\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<div><p>Spontaneous cervical artery dissection is caused by a hematoma in the arterial wall. Recent research revealed that the most likely pathophysiological key mechanism is rupture of a vas vasorum resulting in a bleeding into the medio-adventitial borderzone <span>[1]</span>. The expansion of the hematoma into the arterial lumen can secondarily lead to a rupture of the tunica intima with a high risk of thrombus formation and embolic cerebral infarction <span>[2]</span>. Moreover the expansion of the hematoma causes an arterial stenosis or arterial occlusion with the risk of hemodynamic impairment. The risk of an ischemic stroke in the course of a dissection is thought to be about 70% for dissections of the internal carotid artery (ICA) <span>[3]</span> and about 80% for dissections of the vertebral artery (VA) <span>[4]</span>. The annual incidence of dissections of the ICA has been estimated to be 2.5–3/100,000 and for the VA 0.97–1.5/100,000 <span>[5]</span>, <span>[6]</span>. Although dissections as such are rare they are a frequent etiology of stroke in children and young adults. Approximately 25% of the strokes in patients younger than 50 are caused by dissections with a peak age between 40 and 45 years <span>[7]</span>, <span>[8]</span>, <span>[9]</span>, <span>[10]</span>, <span>[11]</span>, <span>[12]</span>, <span>[13]</span>, <span>[14]</span>, <span>[15]</span>, <span>[16]</span>.</p></div>\",\"PeriodicalId\":101010,\"journal\":{\"name\":\"Perspectives in Medicine\",\"volume\":\"1 1\",\"pages\":\"Pages 250-254\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2012-09-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://sci-hub-pdf.com/10.1016/j.permed.2012.02.024\",\"citationCount\":\"9\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Perspectives in Medicine\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://www.sciencedirect.com/science/article/pii/S2211968X12000319\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Perspectives in Medicine","FirstCategoryId":"1085","ListUrlMain":"https://www.sciencedirect.com/science/article/pii/S2211968X12000319","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Ultrasound in spontaneous cervical artery dissection
Spontaneous cervical artery dissection is caused by a hematoma in the arterial wall. Recent research revealed that the most likely pathophysiological key mechanism is rupture of a vas vasorum resulting in a bleeding into the medio-adventitial borderzone [1]. The expansion of the hematoma into the arterial lumen can secondarily lead to a rupture of the tunica intima with a high risk of thrombus formation and embolic cerebral infarction [2]. Moreover the expansion of the hematoma causes an arterial stenosis or arterial occlusion with the risk of hemodynamic impairment. The risk of an ischemic stroke in the course of a dissection is thought to be about 70% for dissections of the internal carotid artery (ICA) [3] and about 80% for dissections of the vertebral artery (VA) [4]. The annual incidence of dissections of the ICA has been estimated to be 2.5–3/100,000 and for the VA 0.97–1.5/100,000 [5], [6]. Although dissections as such are rare they are a frequent etiology of stroke in children and young adults. Approximately 25% of the strokes in patients younger than 50 are caused by dissections with a peak age between 40 and 45 years [7], [8], [9], [10], [11], [12], [13], [14], [15], [16].