肝素联合胰岛素治疗高甘油三酯血症引起的急性胰腺炎的失败

Mounir Khalil, Chouikh Chaki, E. Zakarya, Mattous Mohamed
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引用次数: 0

摘要

在未触及脾脏或任何肿块的上腹部,敏感性显著。毛细葡萄糖4.47g/l,尿酮阳性。立即开始了强化管理。生理盐水0.9%经2根外周静脉导管(16G)输注。在依托米他和琥珀胆碱诱导挤压后,气管插管使有创通气在60%的氧气下达到98%的饱和度。芬太尼一开始才起到镇静作用。然后,股静脉导管允许肾上腺素逐步给药至0.5μg/kg/min,以获得稳定的血流动力学状态。她的第一个实验室的血脂酶609 UI/L显著。白细胞计数为17100/mm3,血清甘油三酯水平升高至11g/l。血清阴离子间隙正常。胸片检查正常。腹部CT扫描显示胰腺实质和胰腺周围组织坏死,无积液。
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Failure of heparin associated to insulin in treatment of a hypertriglyceridemia-induced acute pancreatitis
was significant for sensibility in the epigastric region without palpable spleen or any mass. Capillary dextrose was 4.47g/l and urines ketones were positives. Intensive management was started immediately. Physiologic saline serum 0.9% was administered by two peripheral venous catheters (16G). A tracheal tube after a crush induction by Etomidat and succinylcholine allowed invasive ventilation under 60% of oxygen to reach 98% of saturation. Sedation was realized with fentanyl only at first. Then, femoral venous catheter allows adrenalin administration at progressively dose to 0.5μg/kg/min for obtaining stabilized hemodynamic status. Her first labs were significant for blood lipase of 609 UI/L. White blood cells count was 17100/mm3 and serum triglyceride level was elevated at 11g/l. Serum anion gap was normal. Chest X-ray was normal. Abdominal CT scan showed a necrosis of the pancreatic parenchyma and peri pancreatic tissues without fluid collection.
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