人们如何概括物体的因果关系?非参数贝叶斯论述。

Computational brain & behavior Pub Date : 2022-01-01 Epub Date: 2021-11-30 DOI:10.1007/s42113-021-00124-z
Bonan Zhao, Christopher G Lucas, Neil R Bramley
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引用次数: 0

摘要

人们如何根据一个因果关系所适用的实体或情况来决定它的普遍性?人们用什么特征来决定一个新情况是受新因果律还是旧因果律支配?人们如何才能快速、高效地做出这些困难的判断?我们在两个实验中解决了这些问题,这两个实验要求参与者从一对物体之间的一次(实验 1)或多次(实验 2)因果互动中归纳出规律。在每种情况下,参与者都会看到一个代理对象作用于一个接受对象,从而导致接受对象发生一些变化。与人类少量概念学习的能力相一致,我们发现因果概括的系统模式更倾向于较简单的因果律,这些因果律会扩展到类似物体的类别中。在实验 1 中,我们发现参与者的推论受他们被问及的概括问题顺序的影响。在这两个实验中,我们都发现了因果类别形成的不对称性:参与者更倾向于根据代理对象的特征而不是接受者的特征来识别因果律。为了解释这一现象,我们开发了一个计算模型,该模型结合了程序归纳(关于隐藏的因果律)和非参数类别推断(关于其影响领域)。我们证明,我们的建模方法既能解释实验 1 中的顺序效应,也能解释因果不对称现象,其效果优于天真的贝叶斯解释,同时还为现实世界的因果泛化提供了一种计算上合理的机制。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

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How Do People Generalize Causal Relations over Objects? A Non-parametric Bayesian Account.

How do people decide how general a causal relationship is, in terms of the entities or situations it applies to? What features do people use to decide whether a new situation is governed by a new causal law or an old one? How can people make these difficult judgments in a fast, efficient way? We address these questions in two experiments that ask participants to generalize from one (Experiment 1) or several (Experiment 2) causal interactions between pairs of objects. In each case, participants see an agent object act on a recipient object, causing some changes to the recipient. In line with the human capacity for few-shot concept learning, we find systematic patterns of causal generalizations favoring simpler causal laws that extend over categories of similar objects. In Experiment 1, we find that participants' inferences are shaped by the order of the generalization questions they are asked. In both experiments, we find an asymmetry in the formation of causal categories: participants preferentially identify causal laws with features of the agent objects rather than recipients. To explain this, we develop a computational model that combines program induction (about the hidden causal laws) with non-parametric category inference (about their domains of influence). We demonstrate that our modeling approach can both explain the order effect in Experiment 1 and the causal asymmetry, and outperforms a naïve Bayesian account while providing a computationally plausible mechanism for real-world causal generalization.

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