Atg39 selectively captures inner nuclear membrane into lumenal vesicles for delivery to the autophagosome

Sunandini Chandra, Philip J. Mannino, David J. Thaller, Nicholas R. Ader, M. King, T. Melia, C. Lusk
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引用次数: 14

Abstract

Mechanisms that turnover components of the nucleus and inner nuclear membrane (INM) remain to be fully defined. We explore how components of the INM are selected by a cytosolic autophagy apparatus through a transmembrane nuclear envelope-localized cargo adaptor, Atg39. A split-GFP reporter shows that Atg39 localizes to the outer nuclear membrane (ONM) and thus targets the INM across the nuclear envelope lumen. Consistent with this, sequence elements that confer both nuclear envelope localization and a membrane remodeling activity are mapped to the Atg39 lumenal domain; these lumenal motifs are required for the autophagy-mediated degradation of an integral INM protein. Interestingly, correlative light and electron tomography shows that the overexpression of Atg39 leads to the expansion of the ONM and the enclosure of a network of INM-derived vesicles in the nuclear envelope lumen. Thus, we propose an outside-in model of nucleophagy where INM is delivered into vesicles in the nuclear envelope lumen, which can be targeted by the autophagosome.
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Atg39选择性地捕获核膜进入管腔小泡,并将其传递给自噬体
细胞核和内核膜(INM)转换成分的机制仍未完全确定。我们探索了胞质自噬装置如何通过跨膜核包膜定位的货物适配器Atg39选择INM的成分。分裂- gfp报告显示,Atg39定位于外核膜(ONM),从而穿过核膜腔靶向INM。与此一致的是,赋予核膜定位和膜重塑活性的序列元件被映射到Atg39管腔结构域;这些管腔基序是自噬介导的整体INM蛋白降解所必需的。有趣的是,相关的光和电子断层扫描显示,Atg39的过表达导致ONM的扩张和核膜腔内inm衍生囊泡网络的封闭。因此,我们提出了一种由外而内的核自噬模型,其中INM被递送到核包膜腔中的囊泡中,这可以被自噬体靶向。
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