Latrogenic twin anemia: polycythemia sequence post-amnioreduction in twin-twin transfusion syndrome

Guoqing Chen, Jinna Jiang, Qinqin Wu, Jing Lu
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Abstract

We present a case of Iatrogenic twin anemia–polycythemia sequence (TAPS) post-amnioreduction in twin-twin transfusion syndrome at 32 gestational weeks. Slight middle cerebral artery (MCA) peak systolic velocity (PSV) discrepancy was present 3 days after the amnioreduction, with MCA PSV around 1.5 MoM of the doner and 1.0 MoM of the recipient. Eighteen days after the amnioreduction, though MCA PSV remained stable, placental dichotomy, starry-sky liver of the recipient and small amount of right pleural effusion of donor were noted. TAPS was diagnosed and postnatal examination of the twins and the placenta confirmed it. We conjectured that the decompression of the placenta after the sudden reduction of amniotic fluid volume may cause the patency of the tiny anastomoses, resulting in the TAPS.
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潜伏性双胎贫血:双胎输血综合征羊水减少后的多血症序列
我们提出一个病例医源性双胞胎贫血-红细胞增多症序列(TAPS)后羊膜减少在32孕周的双胞胎输血综合征。羊膜还原后3天出现轻微的大脑中动脉(MCA)峰值收缩速度(PSV)差异,供体的MCA峰值收缩速度(PSV)约为1.5 MoM,受体的MCA峰值收缩速度约为1.0 MoM。羊膜切除18天后,虽然MCA PSV保持稳定,但发现受体胎盘二分,肝呈星空状,供体右侧胸腔积液较少。产后检查和胎盘证实了这一诊断。我们推测羊水突然减少后胎盘的减压可能导致微小吻合口的通畅,从而导致TAPS。
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