POSTCOVID-19 WAR era, remarkable accelerated hemato-immunologic processes affecting patients disease progression toward excess mortality

Bahram Alamdary Badlou
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Abstract

Background: The speed of the COVID-19 pandemic, and its potential threat to society, achieved through strange global collaborations and innovation between clinical specialties, different elite research organizations, academic institutions, and governments, recently. Cancer clinical trials have developed into a diverse and sophisticated array of designs suited to differing purposes especially focused on excess accelerated mortality rates between patients. Understanding the mechanism of bidirectional interaction between different angles of the death triangle is a lifesaving novel idea that I conceived from 2018. On one hand, platelet dysfunction after certain pharmaco-toxicological approaches in diabetes, and cancer patients; and on the other hand intervention of different COVID-19 variants, mutating each month, caused (hypothetically) remarkable acceleration of introduced angels of death triangle machinery. A considerable concern is modification of medical sciences using AI-related data processing and manipulations into substantial changes of background information presentation. Discussion: Different study groups describe how different microorganisms might be involved in accelerated mortality and morbidity rate among diabetes and cancer patients, in this postcovid-19 era. There are different theories about cancerogenic processes and associated accelerating factors. Simultaneously, restrictions by certain main Policymakers made mission impossible to tackle excess accelerated increase in mortality and morbidity rates, between2021-2024. How death receptors become activated and could accelerate harmful processes is not completely elucidated yet. Based on recent studies diabetic and cancerogenic processes can initiate susceptibility to getting infected, however. Lack of a golden standard protocol to prevent blood transfusion-based infection and Transplantation-based transfections, also brought certain (re-)actions of Basic Scientists entirely in a cloudy atmosphere, where more questions than answers appeared to need being answered from 2019. In this paper is tried to highlight more about consequent (re-)action and (ab)using diagnostics that can support using a certain type of therapeutics, which they might cause excess accelerated mortality rates, in these cloudy uncertain post-Covid-19 era.
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19 战后,血液免疫过程明显加快,影响了患者的病情发展,导致死亡率过高
背景:最近,COVID-19 的流行速度及其对社会的潜在威胁通过临床专科、不同的精英研究组织、学术机构和政府之间奇特的全球合作与创新得以实现。癌症临床试验已发展成为一个多样化、复杂的设计阵列,适用于不同的目的,尤其侧重于患者之间过高的加速死亡率。了解死亡三角不同角度之间的双向互动机制,是我从2018年开始构想的救命新思路。一方面,糖尿病和癌症患者在采用某些药物毒性方法后出现血小板功能障碍;另一方面,每月变异的不同 COVID-19 变体的干预导致(假设)死亡三角机制中引入的天使显著加速。一个值得关注的问题是,利用人工智能相关数据处理和操作对医学科学进行修改,使背景信息的呈现方式发生重大变化。讨论:不同的研究小组描述了在 "后科维德-19 "时代,不同的微生物是如何参与加速糖尿病和癌症患者死亡率和发病率的。关于致癌过程和相关加速因素有不同的理论。与此同时,某些主要政策制定者的限制使得在 2021-2024 年间无法解决死亡率和发病率过度加速增长的问题。死亡受体是如何被激活并加速有害过程的,目前尚未完全阐明。不过,根据最近的研究,糖尿病和致癌过程可能会引发感染。由于缺乏预防输血感染和移植感染的黄金标准协议,基础科学家的某些(重新)行动也完全处于混沌状态,从 2019 年开始,需要回答的问题似乎比答案更多。在本文中,我们试图强调更多有关随之而来的(重新)行动和(滥用)诊断方法的信息,这些诊断方法可以支持使用某种类型的治疗药物,而这些药物可能会在这个阴云密布、充满不确定性的 "后科维德-19 "时代导致过快的死亡率。
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