RETRACTION: CP-31398 inhibits the progression of cervical cancer through reversing the epithelial mesenchymal transition via the downregulation of PAX2s
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引用次数: 0
Abstract
RETRACTION: Ling Liu, Tan-Tan Yu, Chen-Chen Ren, Li Yang, Shi-Hong Cui, Xiao-An Zhang, “CP-31398 inhibits the progression of cervical cancer through reversing the epithelial mesenchymal transition via the downregulation of PAX2s,” Journal of Cellular Physiology 234, no. 3 (2019): 2929-2942, https://doi.org/10.1002/jcp.27109.
The above article, published online on 21 August 2018 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the authors; the journal Editor-in-Chief, Alexander Hutchison; and Wiley Periodicals LLC. The retraction has been agreed due to concerns related to the data presented in the article. Several flaws and inconsistencies between results presented and experimental methods described were found. Additionally, duplications affecting Figures 5a and 5c, and Figure 8a and 8c have been detected. Accordingly, the conclusions of this article are considered invalid by the editors.
撤稿：Ling Liu, Tan-Tan Yu, Chen-Chen Ren, Li Yang, Shi-Hong Cui, Xiao-An Zhang, "CP-31398 inhibits progression of cervical cancer through the reversing the epithelial mesenchymal transition via the downregulation of PAX2s," Journal of Cellular Physiology 234, no.3 (2019): 2929-2942, https://doi.org/10.1002/jcp.27109。上述文章于2018年8月21日在线发表于威利在线图书馆（wileyonlinelibrary.com），经作者、期刊主编亚历山大-哈奇森（Alexander Hutchison）和威利期刊有限责任公司（Wiley Periodicals LLC）同意，已被撤回。之所以同意撤稿，是因为文章中提供的数据令人担忧。我们发现文章中介绍的结果与实验方法之间存在若干缺陷和不一致之处。此外，还发现了影响图 5a 和 5c 以及图 8a 和 8c 的重复内容。因此，编辑认为这篇文章的结论无效。
期刊介绍：
The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.
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