Amygdala Activation and Its Neuropsychological Correlations

Kodhidarla Divya
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Abstract

Early research on primates gave clarifications with respect to the elements of the amygdala, just as a reason for additional exploration. As ahead of schedule as 1888, rhesus monkeys with a lesioned worldly cortex (counting the amygdala) were seen to have critical social and enthusiastic shortfalls. Heinrich Kluver and Paul Bucy later developed this equivalent perception by showing that huge injuries to the front transient projection delivered recognizable changes, including eruption to all items, hypoemotionality, loss of dread, hypersexuality, and hyperorality, a condition wherein unseemly articles are set in the mouth. Some monkeys likewise showed a failure to perceive natural items and would move toward vivify and lifeless things aimlessly, displaying a deficiency of dread towards the experimenters [1]. This conduct issue was subsequently named Kluver-Bucy disorder in like manner, and later examination demonstrated it was explicitly because of amygdala sores. Monkey moms who had amygdala harm showed a decrease in maternal practices towards their newborn children, frequently truly mishandling or dismissing them. In 1981, specialists tracked down that particular radio recurrence injuries of the entire amygdala caused Kluver-Bucy condition.
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杏仁核激活及其神经心理学相关性
早期对灵长类动物的研究澄清了杏仁核的成分,只是作为进一步探索的理由。早在1888年,研究人员就发现,大脑皮层受损的恒河猴(包括杏仁核)在社交和热情方面存在严重缺陷。海因里希·克鲁弗和保罗·布西后来发展出了同样的看法,他们表明,前部短暂突起的巨大损伤会带来可识别的变化,包括所有物品的爆发、情绪低落、恐惧丧失、性欲亢进和道德亢进,这是一种不合适的物品被放在嘴里的情况。一些猴子同样表现出无法感知自然事物的能力,它们会漫无目的地朝有生命的和没有生命的事物移动,这表明它们对实验者缺乏恐惧感[1]。这种行为问题随后以类似的方式被命名为Kluver-Bucy障碍,后来的检查表明,这显然是由于杏仁核溃疡。杏仁核受损的猴子妈妈对新生儿的母性行为有所减少,经常真的对他们处理不当或忽视他们。1981年,专家们追踪到整个杏仁核的特殊放射复发性损伤导致了Kluver-Bucy病。
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