Sinking Skin Flap Syndrome: Cause of Secondary Neurological Deterioration

IF 0.1 Q4 ANESTHESIOLOGY Pediatric Anesthesia and Critical Care Journal Pub Date : 2021-01-01 DOI:10.26502/acc.020
Touab Rida, Rabii Andaloussi Mohamed, Mohsani Mohamed, Mounir Khalil, Bensghir Mustapha, Balkhi Hicham
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Abstract

Introduction: Sinking skin flap syndrome is a rare complication of craniectomy, which is performed as a treatment of severe intracranial hypertension. Cases Reports: The first case is a 55 year old man. Admitted with Glascow score of 13/15, rapid neurological deterioration was noted with a GCS of 9/15, and then anisocoria. CT scan had objectified hemorrhagic contusions, subdural hematoma measured 11 mm and deviation of the median line. The patient was operated with evacuation of the subdural hematoma through a large decompressive craniectomy. In the second month, he presented a cranial deformation with a deepening of the cutaneous plane, with deterioration of the neurological status and a generalized convulsive crisis. The second case is that of a 32 year old man, admitted to the emergency room with 8/15 of GCS with anisocoria. CT scan was showing an 11 mm right subdural hematoma with a hemorrhagic contusion opposite, a 12 mm midline deviation and diffuse cerebral edema. The patient was operated with Anesth Crit Care 2021; 3 (1): 001-009 DOI: 10.26502/acc.020 Anesthesia and Critical Care 2 evacuation of the subdural hematoma through a large craniectomy. The neurological examination after the extubation showed a GCS of 14. Two days later, the patient presented a depression of the right scalp with an aspect of skin flap syndrome on CT scan without significant neurological deterioration. Conclusion: The role of decompressive craniectomy in neurological improvement in still uncertain, and timing of cranioplasty is more debate: early with unclear neurologic status and preventing the skin flap syndrome or delete after final outcome.
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皮瓣下沉综合征:继发性神经功能恶化的原因
引言:皮瓣下沉综合征是颅骨切除术中一种罕见的并发症,用于治疗严重颅内高压。病例报告:第一位病例为55岁男性。入院时Glascow评分为13/15,GCS为9/15,神经系统迅速恶化,然后是异色。CT扫描显示出血性挫伤,硬膜下血肿11mm,中线偏离。患者通过大减压颅骨切除术清除硬膜下血肿。第二个月,患者出现颅骨变形,皮肤平面加深,神经系统状况恶化,出现全身性抽搐危象。第二个病例是一名32岁的男性,因8/15的GCS和异眼而入院急诊室。CT扫描显示右侧硬膜下11mm血肿伴对面出血性挫伤,中线偏移12mm,弥漫性脑水肿。患者采用Anesth Crit Care 2021进行手术;3 (1): 001-009 DOI: 10.26502/acc.020麻醉与重症监护2大颅骨切除术后硬膜下血肿的清除。拔管后的神经学检查显示GCS为14。两天后,患者在CT扫描上表现为右头皮凹陷,伴有皮瓣综合征,无明显神经功能恶化。结论:减压颅骨切除术在神经系统改善中的作用仍不确定,而颅骨成形术的时机更有争议:早期神经系统状况不清,预防皮瓣综合征或最终结局后的皮瓣删除。
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