{"title":"更正：MitoNEET通过调节线粒体铁防止铁超载诱导的H9c2细胞胰岛素抵抗。","authors":"","doi":"10.1002/jcp.31456","DOIUrl":null,"url":null,"abstract":"<p>This article corrects the following:</p><p><b>MitoNEET prevents iron overload-induced insulin resistance in H9c2 cells through regulation of mitochondrial iron.</b></p><p>Tam, Eddie, Hye K. Sung, and Gary Sweeney</p><p>Volume 238, Issue 8, Journal of Cellular Physiology</p><p>https://doi.org/10.1002/jcp.31044</p><p>First published online: 03 June 2023</p><p>In the original version of the article, the Mfn2 Western blot image in Figure 3g was not correctly displayed.</p><p>Please find corrected Figure 3g with correct display of Mfn2 Western blot image below.</p>","PeriodicalId":15220,"journal":{"name":"Journal of Cellular Physiology","volume":"239 12","pages":""},"PeriodicalIF":4.5000,"publicationDate":"2024-09-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1002/jcp.31456","citationCount":"0","resultStr":"{\"title\":\"Correction: MitoNEET prevents iron overload-induced insulin resistance in H9c2 cells through regulation of mitochondrial iron\",\"authors\":\"\",\"doi\":\"10.1002/jcp.31456\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>This article corrects the following:</p><p><b>MitoNEET prevents iron overload-induced insulin resistance in H9c2 cells through regulation of mitochondrial iron.</b></p><p>Tam, Eddie, Hye K. Sung, and Gary Sweeney</p><p>Volume 238, Issue 8, Journal of Cellular Physiology</p><p>https://doi.org/10.1002/jcp.31044</p><p>First published online: 03 June 2023</p><p>In the original version of the article, the Mfn2 Western blot image in Figure 3g was not correctly displayed.</p><p>Please find corrected Figure 3g with correct display of Mfn2 Western blot image below.</p>\",\"PeriodicalId\":15220,\"journal\":{\"name\":\"Journal of Cellular Physiology\",\"volume\":\"239 12\",\"pages\":\"\"},\"PeriodicalIF\":4.5000,\"publicationDate\":\"2024-09-30\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://onlinelibrary.wiley.com/doi/epdf/10.1002/jcp.31456\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Journal of Cellular Physiology\",\"FirstCategoryId\":\"99\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1002/jcp.31456\",\"RegionNum\":2,\"RegionCategory\":\"生物学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q2\",\"JCRName\":\"CELL BIOLOGY\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of Cellular Physiology","FirstCategoryId":"99","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1002/jcp.31456","RegionNum":2,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q2","JCRName":"CELL BIOLOGY","Score":null,"Total":0}
引用次数: 0
摘要
MitoNEET通过调节线粒体铁来防止H9c2细胞铁超载引起的胰岛素抵抗。Tam, Eddie, Hye K. Sung, and Gary SweeneyVolume 238, Issue 8, Journal of Cellular Physiologyhttps://doi.org/10.1002/jcp.31044First在线发表：2023年6月3日在原文中，图3g中的Mfn2 Western blot图像显示不正确。请查看更正后的图3g，下图显示了正确的Mfn2 Western blot图像。
期刊介绍：
The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.
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