对非肌层浸润性膀胱癌和肌层浸润性膀胱癌进行的蛋白质组学分析凸显了具有代谢、基质和免疫特征的不同亚组,并强调了基质区的重要性。

IF 5.6 2区 医学 Q1 ONCOLOGY The Journal of Pathology Pub Date : 2024-11-25 DOI:10.1002/path.6367
Thien-Ly Julia Dinh, Manuel Rogg, Miguel Cosenza-Contreras, Mujia Li, Max Zirngibl, Niko Pinter, Konrad Kurowski, Frank Hause, Lena Pauli, Fiona Imberg, Alana Huynh, Marlene Schmid, Ievgen Glavinsky, Luisa Braun, Clara Van Wymersch, Luise Bergmann, Xenia Ungefug, Marion Kunz, Tilman Werner, Patrick Bernhard, Guadalupe Espadas, Eva Brombacher, Julia Schueler, Eduard Sabido, Clemens Kreutz, Christian Gratzke, Martin Werner, Markus Grabbert, Peter Bronsert, Christoph Schell, Oliver Schilling
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引用次数: 0

摘要

我们对 79 例膀胱癌进行了蛋白质组学分析,包括未经治疗的非肌层浸润性膀胱癌(NMIBC,17 例)、肌层浸润性膀胱癌(MIBC,51 例)和新辅助治疗的肌层浸润性膀胱癌(MIBC,11 例)。从福尔马林固定、石蜡包埋的样本中提取蛋白质,并采用数据独立采集方法进行分析,共获得超过8000个定量蛋白质。与NMIBC相比,MIBC显示出细胞外基质(ECM)和免疫反应特征,以及代谢蛋白质组的改变,同时参与细胞-细胞粘附和脂质代谢的蛋白质也随之减少。新辅助治疗并不会持续影响残留肿瘤块的蛋白质组。NMIBC有两个蛋白质组亚群,它们与组织学分级和细胞粘附或脂质/DNA代谢特征相关。治疗无效的 MIBC 有三个蛋白质组亚群,分别与基底鳞状、富含基质或管腔亚型相似,具有新陈代谢、免疫功能或 ECM 特征。新陈代谢亚组呈现出一种免疫贫乏的微环境,而 ECM 和免疫亚组则富含 M2 样肿瘤相关巨噬细胞和树突状细胞的标记物。自然杀伤细胞的标志物是 ECM 亚组独有的,而细胞毒性 T 细胞的标志物则是免疫亚组的特征。MIBC的内源性蛋白分解增加,同时基质金属蛋白酶(包括MMP-14)上调。基因组面板测序结果显示,NMIBC 和 MIBC 的典型特征分别是普遍的 FGRF3 改变和 TP53 改变。通过对源自患者的异种移植物进行蛋白质组分析,研究了MIBC的肿瘤与基质之间的相互作用,突出了肿瘤和基质对基质组和肿瘤诱导的基质蛋白质组表型的特定贡献。© 2024 作者。病理学杂志》由 John Wiley & Sons Ltd 代表大不列颠及爱尔兰病理学会出版。
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Proteomic analysis of non-muscle invasive and muscle invasive bladder cancer highlights distinct subgroups with metabolic, matrisomal, and immune hallmarks and emphasizes importance of the stromal compartment.

We present the proteomic profiling of 79 bladder cancers, including treatment-naïve non-muscle-invasive bladder cancer (NMIBC, n = 17), muscle-invasive bladder cancer (MIBC, n = 51), and neoadjuvant-treated MIBC (n = 11). Proteins were extracted from formalin-fixed, paraffin-embedded samples and analyzed using data-independent acquisition, yielding >8,000 quantified proteins. MIBC, compared to NMIBC, shows an extracellular matrix (ECM) and immune response signature as well as alteration of the metabolic proteome together with concomitant depletion of proteins involved in cell-cell adhesion and lipid metabolism. Neoadjuvant treatment did not consistently impact the proteome of the residual tumor mass. NMIBC presents two proteomic subgroups that correlate with histological grade and feature signatures of cell adhesion or lipid/DNA metabolism. Treatment-naïve MIBC presents three proteomic subgroups with resemblance to the basal-squamous, stroma-rich, or luminal subtypes and signatures of metabolism, immune functionality, or ECM. The metabolic subgroup presents an immune-depleted microenvironment, whereas the ECM and immune subgroups are enriched for markers of M2-like tumor-associated macrophages and dendritic cells. Markers for natural killer cells are exclusive for the ECM subgroup, and markers for cytotoxic T cells are a hallmark of the immune subgroup. Endogenous proteolysis is increased in MIBC alongside upregulation of matrix metalloproteases, including MMP-14. Genomic panel sequencing yielded the prototypical profile of prevalent FGRF3 alterations in NMIBC and TP53 alterations in MIBC. Tumor-stroma interactions of MIBC were investigated by proteomic analysis of patient-derived xenografts, highlighting specific tumor and stroma contributions to the matrisome and tumor-induced stromal proteome phenotypes. © 2024 The Author(s). The Journal of Pathology published by John Wiley & Sons Ltd on behalf of The Pathological Society of Great Britain and Ireland.

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来源期刊
The Journal of Pathology
The Journal of Pathology 医学-病理学
CiteScore
14.10
自引率
1.40%
发文量
144
审稿时长
3-8 weeks
期刊介绍: The Journal of Pathology aims to serve as a translational bridge between basic biomedical science and clinical medicine with particular emphasis on, but not restricted to, tissue based studies. The main interests of the Journal lie in publishing studies that further our understanding the pathophysiological and pathogenetic mechanisms of human disease. The Journal of Pathology welcomes investigative studies on human tissues, in vitro and in vivo experimental studies, and investigations based on animal models with a clear relevance to human disease, including transgenic systems. As well as original research papers, the Journal seeks to provide rapid publication in a variety of other formats, including editorials, review articles, commentaries and perspectives and other features, both contributed and solicited.
期刊最新文献
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