睾丸病理的表观遗传学转基因遗传和支持细胞表观突变:男性不育的世代起源。

IF 4.8 Q1 GENETICS & HEREDITY Environmental Epigenetics Pub Date : 2019-08-29 eCollection Date: 2019-07-01 DOI:10.1093/eep/dvz013
Ingrid Sadler-Riggleman, Rachel Klukovich, Eric Nilsson, Daniel Beck, Yeming Xie, Wei Yan, Michael K Skinner
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引用次数: 0

摘要

几十年来,男性生殖健康一直在下降,精子数量下降,不孕不育增加,这造成了巨大的社会和经济负担。从20世纪70年代到现在,人群中的精子浓度普遍下降了50%以上。环境毒物诱导的表观遗传已被证明会影响睾丸病理和精子数量。支持细胞通过为发育中的生殖细胞提供物理和营养支持,在精子发生中发挥着重要作用。目前的研究旨在进一步研究与睾丸疾病发作相关的大鼠支持细胞表观基因组和转录组的转基因表观遗传学变化。妊娠雌性F0代大鼠在胎儿性腺性别测定期间短暂暴露于环境毒物,如二氯二苯基三氯乙烷(DDT)或长春唑啉。对F1代后代进行繁殖(即在谱系内进行杂交)以产生F2代大后代,然后对其进行繁殖以产生转基因F3代(即大后代),而不使用兄弟姐妹或表亲繁殖。本研究的重点是研究转基因睾丸疾病的病因,因此使用F3代大鼠。DNA和RNA是从F3代大鼠出生后20天大的雄性睾丸中分离的纯化Sertoli细胞中获得的。与对照(载体暴露)谱系相比,在长春花唑啉和滴滴涕谱系的支持细胞中观察到DNA甲基化、非编码RNA和基因表达的跨代变化。鉴定了与支持细胞功能异常和睾丸病理相关的基因,并测定了长春花唑啉和滴滴涕对转基因的影响。关键基因途径的改变,如丙酮酸代谢途径,被鉴定。观察结果表明,祖先暴露于环境毒物会促进支持细胞的表观遗传和转录组改变,这些改变与睾丸异常有关。这些表观遗传学改变似乎是睾丸病理和男性不育的发育和世代起源的关键因素。
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Epigenetic transgenerational inheritance of testis pathology and Sertoli cell epimutations: generational origins of male infertility.

Male reproductive health has been in decline for decades with dropping sperm counts and increasing infertility, which has created a significant societal and economic burden. Between the 1970s and now, a general decline of over 50% in sperm concentration has been observed in the population. Environmental toxicant-induced epigenetic transgenerational inheritance has been shown to affect testis pathology and sperm count. Sertoli cells have an essential role in spermatogenesis by providing physical and nutritional support for developing germ cells. The current study was designed to further investigate the transgenerational epigenetic changes in the rat Sertoli cell epigenome and transcriptome that are associated with the onset of testis disease. Gestating female F0 generation rats were transiently exposed during the period of fetal gonadal sex determination to the environmental toxicants, such as dichlorodiphenyltrichloroethane (DDT) or vinclozolin. The F1 generation offspring were bred (i.e. intercross within the lineage) to produce the F2 generation grand-offspring that were then bred to produce the transgenerational F3 generation (i.e. great-grand-offspring) with no sibling or cousin breeding used. The focus of the current study was to investigate the transgenerational testis disease etiology, so F3 generation rats were utilized. The DNA and RNA were obtained from purified Sertoli cells isolated from postnatal 20-day-old male testis of F3 generation rats. Transgenerational alterations in DNA methylation, noncoding RNA, and gene expression were observed in the Sertoli cells from vinclozolin and DDT lineages when compared to the control (vehicle exposed) lineage. Genes associated with abnormal Sertoli cell function and testis pathology were identified, and the transgenerational impacts of vinclozolin and DDT were determined. Alterations in critical gene pathways, such as the pyruvate metabolism pathway, were identified. Observations suggest that ancestral exposures to environmental toxicants promote the epigenetic transgenerational inheritance of Sertoli cell epigenetic and transcriptome alterations that associate with testis abnormalities. These epigenetic alterations appear to be critical factors in the developmental and generational origins of testis pathologies and male infertility.

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来源期刊
Environmental Epigenetics
Environmental Epigenetics GENETICS & HEREDITY-
CiteScore
6.50
自引率
5.30%
发文量
0
审稿时长
17 weeks
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