对一名患有性别焦虑和先天性肾上腺增生的青春期女孩进行性虐待和剥削

Hakan Ogutlu, İbrahim Tiryaki, I. S. Esin, O. Dursun
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摘要

性别焦虑症是一种性心理障碍,其特征是强烈的跨性别认同和对自己生理性别角色的持续不满。先天性肾上腺增生症(CAH)是一种由先天性皮质醇合成酶缺乏和功能障碍引起的性发育障碍。5.2%的CAH患者可出现性别焦虑。在我们的案例中,将讨论对患有性别不安和CAH的青春期女孩的性虐待和剥削。这名17岁的女孩因对女孩的性欲增加、对身为女孩的不满、渴望成为男性、自残、逾越个人界限而入院。她违背女孩的意愿与她们发生性接触,试图亲吻她们,渴望与她们发生性关系,当她们拒绝性交时,如果她的要求得不到满足,她就用刀威胁她们;她袭击了该机构的工作人员,并割伤了自己的手臂。在她的病史中,基于阴唇融合和1个月时出现单一泌尿生殖器开口,诊断为46XX单纯性男性化CAH。在患者1.5岁时,手术将其作为女孩抚养,并完成手术重建。由于父母照顾不足,她在4岁时被送到机构照料。我们了解到,在该机构待到17岁的病人经常从机构逃跑,曾经遭受过性虐待。我们还了解到她在上一个机构威胁过一个女孩并性虐待了她三个月。患者存在性别焦虑和人格障碍特征。遗传和环境因素可能导致性别焦虑。就像我们的情况一样,对同性伴侣的兴趣和强迫性接触都是性别焦虑症可能出现的问题。需要强调的是,对于存在性别焦虑症遗传或环境风险因素的个体,需要考虑发生性别焦虑症的风险和性虐待的风险。
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Sexual abuse and exploitation of an adolescent girl with gender dysphoria and congenital adrenal hyperplasia
Sexual abuse and exploitation of an adolescent girl with gender dysphoria and congenital adrenal hyperplasia Gender dysphoria is a psychosexual disorder characterized by powerful cross-gender identification and a continuous dissatisfaction with one’s biological gender role. Congenital Adrenal Hyperplasia (CAH) is a sexual developmental disorder resulting from congenital deficiency and functional impairment of enzymes enabling cortisol synthesis. Gender dysphoria may be developed in 5.2% of CAH patients. In our case, sexual abuse and exploitation of an adolescent girl with gender dysphoria and CAH will be discussed. The 17-year-old girl was admitted to our clinic with complaints of increasing sexual desire for girls, dissatisfaction with being a girl, desire to be male, self-harming, and overstepping personal borders. She sexually touched girls against their will, tried to kiss them, desired to have sex with them, and when they rejected sexual intercourse, she threatened them with a knife when her demands were not met; she attacked the officials at the institution and cut her own arm. In her medical history, a diagnosis of 46XX simple virilizing CAH had been made on the basis of labioscrotal fusion and the presence of a single urogenital opening at 1 month. At the age of 1.5 years, the patient was operated to be raised as a girl and surgical reconstruction was completed. As a result of inadequate parental care, she had been taken into institutional care at the age of 4. We learned that the patient who was in the institution until the age of 17 was frequently running away from the institution and once had been exposed to sexual abuse. We also learned that she had threatened a girl in her last institution and sexually abused her for 3 months. Gender dysphoria and personality disorder traits were detected in the patient. Genetic and environmental causes may lead to gender dysphoria. As in our case, interest in same-sex partners and forced sexual contact are problems that may occur in gender dysphoria. It should be emphasized that in individuals with genetic or environmental risk factors of gender dysphoria, the risk of developing gender dysphoria and the risk of sexual abuse need to be considered.
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