神经生物学中线粒体与血清素的相互作用及其在阿尔茨海默病中的意义。

IF 2.8 Q2 NEUROSCIENCES Journal of Alzheimer's disease reports Pub Date : 2023-11-01 eCollection Date: 2023-01-01 DOI:10.3233/ADR-230070
Jing Tian, Eric Du, Lan Guo
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引用次数: 0

摘要

阿尔茨海默病(AD)是一种致命的神经退行性疾病,其特征是严重的脑部病变和进行性认知能力下降。虽然这种疾病的确切原因尚不清楚,但新出现的证据表明,神经递质失调有助于AD病理和症状的发展。血清素是大脑中一种重要的神经递质,在调节各种大脑过程中起着关键作用,并与包括阿尔茨海默病在内的神经和精神疾病有关。最近的研究揭示了线粒体功能和脑生理学中血清素调节之间的相互作用。在阿尔茨海默病中,血清素缺乏,同时线粒体功能受损,特别是血清素能神经元受损。此外,线粒体酶活性的改变,如单胺氧化酶,可能导致AD患者血清素失调。了解线粒体和5 -羟色胺之间的复杂关系,为了解AD的潜在机制提供了有价值的见解,并确定了恢复5 -羟色胺稳态和缓解AD症状的潜在治疗靶点。本文综述了脑线粒体与血清素之间联系的最新进展,强调了它们在AD发病机制中的意义,并强调了该领域进一步研究的重要性。阐明线粒体在血清素功能障碍中的作用将促进治疗和预防这种神经退行性疾病的治疗策略的发展。
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Mitochondrial Interaction with Serotonin in Neurobiology and Its Implication in Alzheimer's Disease.

Alzheimer's disease (AD) is a lethal neurodegenerative disorder characterized by severe brain pathologies and progressive cognitive decline. While the exact cause of this disease remains unknown, emerging evidence suggests that dysregulation of neurotransmitters contributes to the development of AD pathology and symptoms. Serotonin, a critical neurotransmitter in the brain, plays a pivotal role in regulating various brain processes and is implicated in neurological and psychiatric disorders, including AD. Recent studies have shed light on the interplay between mitochondrial function and serotonin regulation in brain physiology. In AD, there is a deficiency of serotonin, along with impairments in mitochondrial function, particularly in serotoninergic neurons. Additionally, altered activity of mitochondrial enzymes, such as monoamine oxidase, may contribute to serotonin dysregulation in AD. Understanding the intricate relationship between mitochondria and serotonin provides valuable insights into the underlying mechanisms of AD and identifies potential therapeutic targets to restore serotonin homeostasis and alleviate AD symptoms. This review summarizes the recent advancements in unraveling the connection between brain mitochondria and serotonin, emphasizing their significance in AD pathogenesis and underscoring the importance of further research in this area. Elucidating the role of mitochondria in serotonin dysfunction will promote the development of therapeutic strategies for the treatment and prevention of this neurodegenerative disorder.

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