C. Egelhoff, R. Budwig, J. K. Foster, B. L. Hansen
{"title":"冠状动脉狭窄血流动力学的实验和计算模型研究:流量、大小、对称性和壁粗糙度的影响","authors":"C. Egelhoff, R. Budwig, J. K. Foster, B. L. Hansen","doi":"10.1115/imece2001/bed-23135","DOIUrl":null,"url":null,"abstract":"\n The motivation for the present study is to better understand the hemodynamics that may be involved with the onset of Myocardial Infarction (MI). We know the sequence of events is plaque rupture followed by thrombogenesis and then MI. There are several theories about the cause of rupture and fluid mechanics is cited as a possible theory.","PeriodicalId":7238,"journal":{"name":"Advances in Bioengineering","volume":null,"pages":null},"PeriodicalIF":0.0000,"publicationDate":"2001-11-11","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"2","resultStr":"{\"title\":\"Investigation of Coronary Artery Stenosis Hemodynamics Using Experimental and Computational Models: Influence of Flowrate, Size, Symmetry and Wall Roughness\",\"authors\":\"C. Egelhoff, R. Budwig, J. K. Foster, B. L. Hansen\",\"doi\":\"10.1115/imece2001/bed-23135\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"\\n The motivation for the present study is to better understand the hemodynamics that may be involved with the onset of Myocardial Infarction (MI). We know the sequence of events is plaque rupture followed by thrombogenesis and then MI. There are several theories about the cause of rupture and fluid mechanics is cited as a possible theory.\",\"PeriodicalId\":7238,\"journal\":{\"name\":\"Advances in Bioengineering\",\"volume\":null,\"pages\":null},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2001-11-11\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"2\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Advances in Bioengineering\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.1115/imece2001/bed-23135\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Advances in Bioengineering","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.1115/imece2001/bed-23135","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Investigation of Coronary Artery Stenosis Hemodynamics Using Experimental and Computational Models: Influence of Flowrate, Size, Symmetry and Wall Roughness
The motivation for the present study is to better understand the hemodynamics that may be involved with the onset of Myocardial Infarction (MI). We know the sequence of events is plaque rupture followed by thrombogenesis and then MI. There are several theories about the cause of rupture and fluid mechanics is cited as a possible theory.