控制肾脏激素调节磷酸盐转运的 NHERF1 PDZ 蛋白的分子社会学。

IF 3.8 3区 生物学 Q2 BIOCHEMISTRY & MOLECULAR BIOLOGY Bioscience Reports Pub Date : 2024-03-27 DOI:10.1042/BSR20231380
Peter A Friedman, Tatyana Mamonova
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引用次数: 0

摘要

甲状旁腺激素(PTH)和成纤维细胞生长因子-23(FGF23)通过调节肾脏 NPT2A 介导的磷酸盐转运来控制细胞外磷酸盐水平,这一过程需要 PDZ 支架蛋白 NHERF1。NHERF1 有两个 PDZ 结构域(PDZ1 和 PDZ2),具有相同的核心结合 GYGF 基序,可明确识别不同的结合伙伴,这些伙伴在激素调控的磷酸盐转运过程中发挥不同的特定作用。PDZ1 与 NPT2A(C-TRL)羧基末端 PDZ 结合基团的相互作用是基础磷酸盐转运所必需的。PDZ2 是一个调控结构域,为多个生物靶标提供支架,包括参与 FGF23 和 PTH 信号转导的激酶和磷酸酶。FGF23 和 PTH 通过可逆的激素刺激磷酸化触发 NHERF1-NPT2A 复合物的解体,从而导致 NPT2A 封存、下调并停止磷酸盐吸收。在缺乏 NHERF1-NPT2A 相互作用的情况下,FGF23 或 PTH 信号的抑制会导致磷酸盐平衡紊乱和磷酸盐消耗。更多的研究对于阐明 NHERF1 如何时空协调细胞伙伴以调节细胞外磷酸盐水平至关重要。
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The molecular sociology of NHERF1 PDZ proteins controlling renal hormone-regulated phosphate transport.

Parathyroid hormone (PTH) and fibroblast growth factor-23 (FGF23) control extracellular phosphate levels by regulating renal NPT2A-mediated phosphate transport by a process requiring the PDZ scaffold protein NHERF1. NHERF1 possesses two PDZ domains, PDZ1 and PDZ2, with identical core-binding GYGF motifs explicitly recognizing distinct binding partners that play different and specific roles in hormone-regulated phosphate transport. The interaction of PDZ1 and the carboxy-terminal PDZ-binding motif of NPT2A (C-TRL) is required for basal phosphate transport. PDZ2 is a regulatory domain that scaffolds multiple biological targets, including kinases and phosphatases involved in FGF23 and PTH signaling. FGF23 and PTH trigger disassembly of the NHERF1-NPT2A complex through reversible hormone-stimulated phosphorylation with ensuing NPT2A sequestration, down-regulation, and cessation of phosphate absorption. In the absence of NHERF1-NPT2A interaction, inhibition of FGF23 or PTH signaling results in disordered phosphate homeostasis and phosphate wasting. Additional studies are crucial to elucidate how NHERF1 spatiotemporally coordinates cellular partners to regulate extracellular phosphate levels.

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来源期刊
Bioscience Reports
Bioscience Reports 生物-细胞生物学
CiteScore
8.50
自引率
0.00%
发文量
380
审稿时长
6-12 weeks
期刊介绍: Bioscience Reports provides a home for sound scientific research in all areas of cell biology and molecular life sciences. Since 2012, Bioscience Reports has been fully Open Access and publishes all papers under the liberal CC BY licence, giving the life science community quality research to share and discuss.Content before 2012 is subscription-only, and is accessible via archive purchase. Articles are assessed on soundness, providing a home for valid findings and data. We welcome papers that span disciplines (e.g. chemistry, medicine), including papers describing: -new methodologies -tools and reagents to probe biological questions -mechanistic details -disease mechanisms -metabolic processes and their regulation -structure and function -bioenergetics
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