{"title":"更正：\"O-GlcNAcylation通过调节FOXA2的稳定性和转录活性促进肝癌细胞的迁移能力\"。","authors":"","doi":"10.1002/jcp.31381","DOIUrl":null,"url":null,"abstract":"<p>Huang, H., Wu, Q., Guo, X., Huang, T., Xie, X., Wang, L., Liu, Y., Shi, L., Li, W., Zhang, J., & Liu, Y. (2021). O-GlcNAcylation promotes the migratory ability of hepatocellular carcinoma cells via regulating FOXA2 stability and transcriptional activity. <i>J Cell Physiol</i>, 236, 7491–7503. https://doi.org/10.1002/jcp.30385</p><p>In the first published version of this manuscript, a transwell migration microscope image presented in Figure 6b of the HepG2 Mock group was accidentally misused during the assembly of the figures, resulting in image duplication. In the corrected Figure 6b below, the photo corresponding to HepG2 Mock group has been replaced with the original and accurate one.</p><p>The authors regret the initial mistake in manuscript preparation. This error does not affect the scientific validity of the conclusions of this study. The authors apologize for any confusion they may have caused.</p>","PeriodicalId":15220,"journal":{"name":"Journal of Cellular Physiology","volume":null,"pages":null},"PeriodicalIF":4.5000,"publicationDate":"2024-07-18","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1002/jcp.31381","citationCount":"0","resultStr":"{\"title\":\"Correction to: “O-GlcNAcylation promotes the migratory ability of hepatocellular carcinoma cells via regulating FOXA2 stability and transcriptional activity”\",\"authors\":\"\",\"doi\":\"10.1002/jcp.31381\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>Huang, H., Wu, Q., Guo, X., Huang, T., Xie, X., Wang, L., Liu, Y., Shi, L., Li, W., Zhang, J., & Liu, Y. (2021). O-GlcNAcylation promotes the migratory ability of hepatocellular carcinoma cells via regulating FOXA2 stability and transcriptional activity. <i>J Cell Physiol</i>, 236, 7491–7503. https://doi.org/10.1002/jcp.30385</p><p>In the first published version of this manuscript, a transwell migration microscope image presented in Figure 6b of the HepG2 Mock group was accidentally misused during the assembly of the figures, resulting in image duplication. In the corrected Figure 6b below, the photo corresponding to HepG2 Mock group has been replaced with the original and accurate one.</p><p>The authors regret the initial mistake in manuscript preparation. This error does not affect the scientific validity of the conclusions of this study. The authors apologize for any confusion they may have caused.</p>\",\"PeriodicalId\":15220,\"journal\":{\"name\":\"Journal of Cellular Physiology\",\"volume\":null,\"pages\":null},\"PeriodicalIF\":4.5000,\"publicationDate\":\"2024-07-18\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://onlinelibrary.wiley.com/doi/epdf/10.1002/jcp.31381\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Journal of Cellular Physiology\",\"FirstCategoryId\":\"99\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1002/jcp.31381\",\"RegionNum\":2,\"RegionCategory\":\"生物学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q2\",\"JCRName\":\"CELL BIOLOGY\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of Cellular Physiology","FirstCategoryId":"99","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1002/jcp.31381","RegionNum":2,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q2","JCRName":"CELL BIOLOGY","Score":null,"Total":0}
Correction to: “O-GlcNAcylation promotes the migratory ability of hepatocellular carcinoma cells via regulating FOXA2 stability and transcriptional activity”
Huang, H., Wu, Q., Guo, X., Huang, T., Xie, X., Wang, L., Liu, Y., Shi, L., Li, W., Zhang, J., & Liu, Y. (2021). O-GlcNAcylation promotes the migratory ability of hepatocellular carcinoma cells via regulating FOXA2 stability and transcriptional activity. J Cell Physiol, 236, 7491–7503. https://doi.org/10.1002/jcp.30385
In the first published version of this manuscript, a transwell migration microscope image presented in Figure 6b of the HepG2 Mock group was accidentally misused during the assembly of the figures, resulting in image duplication. In the corrected Figure 6b below, the photo corresponding to HepG2 Mock group has been replaced with the original and accurate one.
The authors regret the initial mistake in manuscript preparation. This error does not affect the scientific validity of the conclusions of this study. The authors apologize for any confusion they may have caused.
期刊介绍：
The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.
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