Plasma pancreatic polypeptide and gastrin in the assessment of autonomic activity in acute myocardial infarction.

European journal of cardiology Pub Date : 1981-01-01
M J Tansey, L H Opie, A Vinik, B M Kennelly
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Abstract

Measurements of plasma pancreatic polypeptide and gastrin are reported for the first time in patients with acute myocardial infarction and compared with clinical signs of vagal or sympathetic overactivity. Pancreatic polypeptide concentrations were assessed as an index of vagal activity, but elevated values of pancreatic polypeptide found in 7 of the 13 patients on admission did not correlate with clinical evidence of vagal overactivity. The mean pancreatic polypeptide concentrations were not higher in patients with clinical vagal overactivity than in patients with clinical sympathetic overactivity during the 12 h after the onset of symptoms of acute myocardial infarction. Mean gastrin levels were significantly higher on admission and at 4, 5, 6 and 8 h after the onset of infarction in the patients with clinical features of sympathetic overactivity than in the patients with clinical vagal overactivity. Thus plasma gastrin warrants further assessment as an index of sympathetic overactivity in acute myocardial infarction.

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血浆胰多肽和胃泌素对急性心肌梗死患者自主神经活动的评价。
首次报道了急性心肌梗死患者血浆胰多肽和胃泌素的测定,并将其与迷走神经或交感神经过度活动的临床体征进行了比较。胰多肽浓度被评估为迷走神经活动的指标,但入院时13例患者中有7例胰多肽升高与迷走神经过度活动的临床证据无关。在急性心肌梗死症状出现后12小时内,临床迷走神经过度活跃患者的平均胰多肽浓度不高于临床交感神经过度活跃患者。以交感神经过度活跃为临床特征的患者入院时及梗死发生后4、5、6、8 h的平均胃泌素水平明显高于临床迷走神经过度活跃的患者。因此,血浆胃泌素作为急性心肌梗死交感神经过度活跃的指标值得进一步评估。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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