复发性持续性室性心动过速:以他束分支再入为机制1例报告。

European journal of cardiology Pub Date : 1980-01-01
C P Reddy, J D Slack
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引用次数: 0

摘要

一位57岁男性冠心病患者以复发性持续性室性心动过速为主要表现。在电生理学研究中,以下观察结果表明,他束分支(His- bb)再入是室性心动过速的潜在机制:(1)室性心动过速由室性早搏引起的起始与逆行的他—浦肯野传导(V2H2)延迟有关;(2)心动过速复合体QRS构型与V1、V2相似;(3)每次心动过速QRS复合体发生前均有His束偏转,H-V间隔等于或长于窦性心动过速;(4)心动过速时存在房室分离;(5)普鲁卡因胺后,需要更大的V2H2延迟才能启动心动过速。鉴别和区分His-BB(宏观)再入和微再入的治疗意义进行了讨论。
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Recurrent sustained ventricular tachycardia: report of a case with His-bundle branches reentry as the mechanism.

A 57-yr-old man with coronary artery disease presented with recurrent sustained ventricular tachycardia. During electrophysiologic study, the following observations suggested His-bundle branches (His-BB) reentry as the underlying mechanism of ventricular tachycardia: (1) the initiation of ventricular tachycardia by a premature ventricular beat was related to the presence of retrograde His--Purkinje conduction (V2H2) delay; (2) the QRS configuration of tachycardia complexes was similar to that of V1 and V2; (3) each QRS complex of tachycardia was consistently preceded by a His bundle deflection with an H-V interval which was equal to or longer than that of sinus beats; (4) atrioventricular dissociation was present during tachycardia; and (5) after procainamide a greater V2H2 delay was required to initiate the tachycardia. The therapeutic implications of identifying and differentiating the His-BB (macro) reentry from micro reentry are discussed.

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