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Investigation of Emotion Characters of Internet Abusers Using Psychophysiological Signals 利用心理生理信号研究网络滥用者的情绪特征
Pub Date : 2018-06-27 DOI: 10.5772/INTECHOPEN.73692
D. Hsieh, T. Hsiao
Because of ubiquitous Internet and devices, the numbers of the Internet users rapidly increase. Internet addiction (IA) is also a fast growing, serious, and unavoidable problem around the world. The fifth version of Diagnostic and Statistical Manual for Mental Disorders suggested that IA should be studied in a scientific manner, and more related data must be acquired. Emotion was one important character of IA, and emotion-related syndrome was also reported in these years. IA contains psychological and physiological features and usually is assessed by IA scales. However, self-reporting scale is a subjective measurement tool that may be biased. Furthermore, scales or questionnaires assess the long-term developed states of IA rather than the developing states of IA or shortterm change. The physiological signals of IA abusers can provide objective, short-term, dynamic change information instead. Therefore, the dynamic physiological regulation and the psychological and physiological responses to emotion of IA abusers, particularly respiration and respiratory sinus arrhythmia, were studied. IA abusers tended to perform thoracic movement for emotion regulation, whereas people without IA tended to perform abdominal movement. IA abusers exhibited stronger RSA reactivity following negative emotion, but exhibited weaker RSA reactivity following positive emotion.
由于无处不在的互联网和设备,互联网用户的数量迅速增加。网络成瘾(IA)也是世界范围内一个快速增长、严重且不可避免的问题。第五版《精神障碍诊断与统计手册》建议对IA进行科学研究,并收集更多相关数据。情绪是IA的一个重要特征,近年来也有情绪相关综合征的报道。IA包含心理和生理特征,通常用IA量表进行评估。然而,自我报告量表是一种主观的测量工具,可能存在偏见。此外,量表或问卷评估的是IA的长期发达状态,而不是IA的发展状态或短期变化。IA滥用者的生理信号反而可以提供客观、短期、动态的变化信息。因此,我们研究了IA滥用者的动态生理调节和对情绪的心理和生理反应,特别是呼吸和呼吸性窦性心律失常。IA滥用者倾向于通过胸部运动来调节情绪,而非IA滥用者倾向于通过腹部运动来调节情绪。IA滥用者负面情绪后的RSA反应较强,积极情绪后的RSA反应较弱。
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引用次数: 0
Methadone Treatment for Heroin Dependence 美沙酮治疗海洛因依赖
Pub Date : 2018-06-27 DOI: 10.5772/INTECHOPEN.78066
Baconi Daniela Luiza, A. Ciobanu, R. Vasile, A. Vlăsceanu, Mirela Nedelescu, M. Stan
In substitution therapy for treatment of heroin addiction, methadone is the synthetic opi- oid agonist of first choice. Methadone doses vary depending on addict profile established by repeated evaluation. It studied a group of 82 patients both male and female, aged between 19 and 47 years, residing in Bucharest, with diagnosis of heroin addiction. They were voluntarily submitted in the methadone substitution treatment at a specialized treatment center for addiction in Bucharest. The study group was characterized in detail, taking into account demographic, comorbid and addiction characteristics, heroin use his- tory, treatment history, and clinical and paraclinical evaluation. The outcomes resulting from the study design on 82 heroin addict patients enrolled into a methadone main - tenance program highlighted: lowering of the onset age of heroin use, HVC infection comorbidity, and the extension of the treatment period due to the relapses. The results obtained by clinical, laboratory, and psychological complex evaluations in a correlative approach is essential both in initiating methadone treatment and monitoring the detox period but also in the supervision of methadone maintenance treatment. of at onset of the is sig nificantly different from the age of the patients at the onset of their drug consumption. The total duration of the consumption is statistically different from the duration of the consumption, previous to the first MMT.
在海洛因成瘾的替代疗法中,美沙酮是合成阿片激动剂的首选。美沙酮的剂量取决于反复评估确定的成瘾者概况。该研究研究了82名年龄在19至47岁之间、居住在布加勒斯特、被诊断为海洛因成瘾的男性和女性患者。他们自愿在布加勒斯特的一个成瘾专门治疗中心接受美沙酮替代治疗。考虑到人口统计学、合并症和成瘾特征、海洛因使用史、治疗史以及临床和临床旁评价,对研究组进行了详细的特征分析。对82名美沙酮维持项目的海洛因成瘾患者的研究设计结果突出显示:海洛因使用起始年龄降低,HVC感染合并症降低,因复发而延长治疗期。临床、实验室和心理综合评估的相关结果对于美沙酮治疗的开始和排毒期的监测以及美沙酮维持治疗的监督都是至关重要的。其发病时间与患者发病年龄有显著差异。消费的总持续时间与第一次MMT之前的消费持续时间有统计学差异。
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引用次数: 0
Effect of Alcohol on Brain Development 酒精对大脑发育的影响
Pub Date : 2018-06-27 DOI: 10.5772/INTECHOPEN.73693
Farhin Patel, P. Mandal
In the world, 3.3 million deaths occur every year due to harmful use of alcohol; this represents 5.9% of all deaths. Ethanol metabolites’ production and their post-translation modification are one of the proposed mechanisms that lead to neuronal toxicity. The projected neurochemical changes in chronic alcohol drinkers may be due to an imbalance between excitatory and inhibitory neurotransmitters. Interaction of alcohol with GABA and glutamate receptors (NMDA and AMPA) resulted in diverse adaptive changes in gene expression through neuronal pathways leading to alcohol toxicity. Alcohol con - sumption in an individual leads to biochemical changes that are correlated with complex inflammatory signaling pathways such as phosphorylation of proteins, synthesis of nitric oxide (NO), NF-kappaB and MAP kinase pathways in certain regions of the brain. Ethanol exposure activates neurons and microglial cells that lead to release of neu - roimmune factors like high-mobility group box 1 (HMGB1), toll-like receptor 4 (TLR4) and certain cytokines involved in immune responses leading to neuroimmune signaling in the brain. Epigenetic modification of DNA and histones may lead to neuronal gene expression, thus regulating ethanol toxicity. Researchers attempt to modulate therapies that can help to foil alcohol toxicity and support the development of original neuronal cells that have been injured or degenerated by alcohol exposure. synaptic plasticity and synaptic formation. Downregulation of genes by ethanol includes protein synthesis, myelination and the ubiquitin-proteasome pathway. Chronic ethanol exposure increases HMGB1–TLR4 and NF-κB signaling which leads to improved NF-κB target genes’ expression. This results in determining neuroimmune responses to ethanol toxicity that releases HMGB1 or directly stimulates TLR and/or NMDA receptors. An epigenetic mecha nism will show potential towards drug dependence by changing the DNA protein structure. Microglial cells will arbitrate the effect of alcohol toxicity on neurogenesis. The progression towards the neurobiologic techniques including micro array, QTL and proteomics will pro vide some anticipation for researching the molecular and cellular mechanisms that act as a keystone for the understanding of neuronal toxicity and enlightening new therapeutic gene targets for this public health burden.
在世界上,每年有330万人死于有害使用酒精;这占死亡总数的5.9%。乙醇代谢物的产生及其翻译后修饰是导致神经元毒性的机制之一。预测慢性饮酒者的神经化学变化可能是由于兴奋性和抑制性神经递质之间的不平衡。酒精与GABA和谷氨酸受体(NMDA和AMPA)的相互作用通过神经通路导致基因表达的各种适应性变化,从而导致酒精毒性。个体饮酒会导致生化变化,这些变化与复杂的炎症信号通路相关,如蛋白质磷酸化、一氧化氮(NO)的合成、大脑某些区域的NF-kappaB和MAP激酶通路。乙醇暴露激活神经元和小胶质细胞,导致释放新的免疫因子,如高迁移率组盒1 (HMGB1)、toll样受体4 (TLR4)和某些细胞因子,这些因子参与免疫反应,导致大脑中的神经免疫信号传导。DNA和组蛋白的表观遗传修饰可能导致神经元基因表达,从而调节乙醇毒性。研究人员试图调节治疗方法,以帮助抑制酒精毒性,并支持因酒精暴露而受伤或退化的原始神经细胞的发育。突触可塑性和突触形成。乙醇下调的基因包括蛋白质合成、髓鞘形成和泛素-蛋白酶体途径。慢性乙醇暴露增加HMGB1-TLR4和NF-κB信号通路,导致NF-κB靶基因表达增加。结果确定乙醇毒性释放HMGB1或直接刺激TLR和/或NMDA受体的神经免疫反应。一种通过改变DNA蛋白结构的表观遗传机制将显示药物依赖的潜力。小胶质细胞将决定酒精毒性对神经发生的影响。包括微阵列、QTL和蛋白质组学在内的神经生物学技术的进展将为研究神经毒性的分子和细胞机制提供一些预期,这些机制将成为理解神经毒性的基石,并为这一公共卫生负担提供新的治疗基因靶点。
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引用次数: 1
Nursing Care for Persons with Drug Addiction 吸毒成瘾者的护理
Pub Date : 2018-06-27 DOI: 10.5772/INTECHOPEN.73334
Ek-uma Imkome
Persons with drug addiction (PDDs) may exhibit symptoms affecting the central nervous system. Multidisciplinary treatment teams may offer the most updated treatment and care. Pharmacotherapy is one standard treatment, effective in managing psychotic symp- toms with supportive psychosocial interventions. As part of the health-care team, nurses deal with PDD on a 24-hour basis. Quality nursing care is essential for improving quality of life, health status, and continued abuse-free status of PDD. Weak serotonin 2A antagonism
吸毒成瘾的人可能会出现影响中枢神经系统的症状。多学科治疗团队可以提供最新的治疗和护理。药物治疗是一种标准治疗方法,通过支持性心理社会干预有效地控制精神病症状。作为保健小组的一部分,护士24小时处理PDD。高质量的护理对于改善PDD患者的生活质量、健康状况和持续无虐待状态至关重要。弱血清素2A拮抗剂
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引用次数: 2
Molecular-Cellular Targets of the Pathogenetic Action of Ethanol in the Human Brain in Ontogenesis and the Possibility of Targeted Therapy Aimed at Correcting the Effect of Pathogenic Factors 乙醇在人体发育中致病作用的分子-细胞靶点及靶向治疗纠正致病因素作用的可能性
Pub Date : 2018-06-27 DOI: 10.5772/INTECHOPEN.73333
T. Shushpanova, A. Solonskii, O. Shushpanova
Prenatal exposure to ethanol has an impact on angiogenesis and synaptogenesis and forma- tion of neurotransmitter receptors in the brain of the embryo and fetus. Compensatory mechanism of hypoxia in conditions of prenatal exposure to alcohol involves decrease in the perimeter of the vessel and the area of the vessel in the cross section and an increase in the number of vessels in the brain. A significant effect of prenatal exposure to ethanol on the development of synaptic structures in the developing brain of the fetus was expressed in the slowing down of the formation of synaptic contacts and in the reduction of their number in comparison with the norm. Shaping synaptic contact is one of the leading processes during which largely determine the future integrative brain capabilities. The properties of benzodi- azepine receptors in the developing brain of the human ’ s embryo and fetus under prenatal alcohol influence were characterized bya decrease in affinityand an increase in their density as compensatory adaptation of the fetal nervous system to the effects of alcohol. It is reflected on during synaptogenesis in the developing brain and can lay the basis of severe disorders in the unborn child. Alcohol abuse induces neuroadaptive alters of benzodiazepine receptor system in the brain in patients with alcoholism that can modulate GABA A R and mediation of GABA in the brain, which can cause alcohol addiction. The study of the effect of mother ’ s alcoholism on the developing fetal brain (prenatal exposure to alcohol) was carried out in the brain tissue of embryos and human fetuses at the 7 – 15 week of pregnancy in accordance with the requirements of the Ethics Committee and with the consent of patients during abortion procedures under strict medical indications. About 33 embryos and fetuses were obtained from female, suffering from alcoholism and constituted the main study group. The age of women who suffered from alcoholism was 26 – 39 years old, and the duration of the disease was from 3 to 13 years. In all cases, according to ICD-10 criteria, alcoholism of grade II was diagnosed (ICD-10 F10.201, F10.202). The diagnosis of alcoholism was established in the Department of Addictive Conditions, the Institute of Mental Health, Tomsk National Scientific Medical Center Russian Academy of Science (RASci). The control group included samples of the brain tissue of embryos and fetuses obtained from healthy women who do not have a history of neurological or mental diseases comparable in age. Exclusion criteria were cases of adverse effects on brain development of embryos, namely exposure to radiation, chemicals, certain pharmacological agents and maternal diseases during pregnancy: influenza, rubella, toxoplasmosis and others. Ultrastructure of synaptic contacts and vessels of the brain tissue from embryonic and fetal brain were examined under JEM-100B and JEM-100CX electron microscopes. Electron microscopy studies addressed the intermediate layer of the
产前暴露于乙醇对胚胎和胎儿大脑血管生成和突触发生以及神经递质受体的形成有影响。产前酒精暴露条件下缺氧的代偿机制涉及血管周长和横截面血管面积的减少以及大脑血管数量的增加。产前暴露于乙醇对胎儿发育中的大脑突触结构发育的显著影响表现为突触接触形成的减慢以及与正常情况相比突触接触数量的减少。形成突触接触是主要的过程之一,在很大程度上决定了未来的综合脑能力。胎儿胎儿神经系统对酒精的代偿性适应作用下,苯并二-氮卓受体在胚胎和胎儿发育中的大脑中的特性表现为亲和性降低和密度增加。它反映在发育中的大脑突触发生过程中,并可能为未出生的孩子的严重疾病奠定基础。酒精滥用导致酒精中毒患者大脑苯二氮卓受体系统的神经适应性改变,这种改变可以调节GABA A R并介导GABA在大脑中的作用,从而导致酒精成瘾。根据伦理委员会的要求,并在严格的医学指示下进行堕胎手术期间征得患者的同意,对怀孕7 - 15周的胚胎和人类胎儿的脑组织进行了母亲酗酒对发育中的胎儿大脑的影响(产前接触酒精)的研究。从酗酒的女性身上获得约33个胚胎和胎儿,构成主要研究组。酗酒妇女的年龄为26 - 39岁,病程为3 - 13年。根据ICD-10标准,所有病例均诊断为II级酒精中毒(ICD-10 F10.201, F10.202)。酒精中毒的诊断是在俄罗斯科学院托木斯克国家科学医学中心心理健康研究所成瘾病症部确定的。对照组包括胚胎和胎儿的脑组织样本,这些样本来自没有年龄相当的神经或精神疾病史的健康妇女。排除标准是对胚胎大脑发育有不利影响的情况,即在怀孕期间暴露于辐射、化学品、某些药理学制剂和产妇疾病:流感、风疹、弓形虫病等。在JEM-100B和JEM-100CX电镜下观察胚胎和胎脑脑组织突触接触和血管超微结构。电镜研究发现了前脑壁的中间层,这是神经母细胞和胶质母细胞(包括小胶质细胞)的积累,血管在其之间开始生长。使用从电子显微镜获得的6至9厘米底片的照片进行形态计量学分析。有些底片是用扫描仪数字化的,没有中间的纸张打印。由Scion公司在美国国立卫生研究院开发的Scion Image for Windows用于评估不同人类成熟大脑中的突触前受体区域,这些酗酒者使用紧急尸检获得的尸检材料(死后)进行检测。在紧急尸检期间(不迟于死亡后6小时)获得人脑尸检材料样本。长期酗酒者(基于记忆)和对照组的大脑前额叶皮层、小脑皮层和尾状核头部的组织样本是死后采集的。大脑样本被冷冻并储存在装有液氮的热水瓶中。从人类大脑的不同区域共获得126个样本用于无线电受体结合的研究,包括基础组和参考对照组。在回顾性资料的基础上,采用男性慢性酒精化的客观生物学标准(脂肪肝、肝硬化等)组成主组。对照组包括没有神经和精神疾病的患者。尸检材料仅来自男性,年龄范围为33 - 54岁。酗酒患者由精神卫生研究所的精神科医生监督,并根据ICD-10: F10.232进行诊断;F10.302。病人
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引用次数: 1
Deep Brain Stimulation in Treatment-Refractory Addiction 脑深部电刺激治疗难治性成瘾
Pub Date : 2018-06-27 DOI: 10.5772/INTECHOPEN.73694
A. Lavano, G. Guzzi, A. Torre, Donatella Gabriele, D. Chirchiglia, C. Stroscio, GiorgioVolpentesta
Surgical treatment for addiction has been proposed after the successful efficacy of deep brain stimulation (DBS) for the treatment of neurological movement disorders such as Parkinson’s disease (PD). In the field of psychiatric diseases, DBS has been used firstly for obsessive compulsive disorder (OCD) and treatment-resistant depression. The role in addiction has been proposed only recently. The target areas for DBS in treatment-refractory addiction are nucleus accumbens (NAcc), lateral hypothalamus (LH), amigdala, lateral habenula (LHb), dorsal striatum, prefrontal cortex (PFC) and subthalamic nucleus (STN). A well-documented rationale for the choice of the target is required in order to investigate the effectiveness, safety and feasibility. NAcc appears to be the most effective and safe target for DBS followed by STN; PFC is another promising target but needs further exploration to establish its suitability for clinical purposes. DBS is not free of risks, so every patient has to be carefully evaluated and precise ethical standards must be defined in the form of inclusion and exclusion criteria.
在深部脑刺激(DBS)成功治疗神经运动障碍如帕金森病(PD)后,手术治疗成瘾被提出。在精神疾病领域,DBS首先用于强迫症(OCD)和难治性抑郁症。它在成瘾中的作用直到最近才被提出。DBS治疗难治性成瘾的靶区为伏隔核(NAcc)、下丘脑外侧(LH)、杏仁核、外侧束(LHb)、背纹状体、前额叶皮层(PFC)和丘脑下核(STN)。为了调查目标的有效性、安全性和可行性,需要有充分证据证明选择目标的理由。NAcc似乎是DBS最有效和安全的靶标,其次是STN;PFC是另一个有希望的靶点,但需要进一步探索以确定其临床用途的适用性。DBS并非没有风险,因此必须对每位患者进行仔细评估,并以纳入和排除标准的形式确定精确的道德标准。
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引用次数: 0
Bereavement and Substance Use Disorder 丧亲之痛和物质使用障碍
Pub Date : 2018-02-07 DOI: 10.5772/INTECHOPEN.73125
L. Masferrer, B. Caparrós
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引用次数: 1
DRINKING PATTERNS AND DSM-IV ALCOHOL USE DISORDERS' CRITERIA IN ARGENTINEAN EMERGENCY DEPARTMENT PATIENTS. 阿根廷急诊科患者的饮酒模式和dsm-iv酒精使用障碍标准
Mariana Cremonte, Cheryl J Cherpitel, Guilherme Borges, Raquel I Peltzer, Pablo R Santángelo

BACKGROUND: Previous studies have shown cultural variations in normative drinking and furthermore, in the quantity and frequency of drinking related to alcohol use disorders. AIM: The main goal of this study is to characterize alcohol drinking patterns in Argentinean Emergency Department patients, and secondly, to explore the association between those drinking patterns and DSM-IV alcohol use disorders. METHOD: Data were collected from a probability sample of patients admitted to the Emergency Department of a large public hospital in Mar del Plata, Argentina. Data analyzed here pertain to those who reported consuming at least one drink during the last twelve months (n=529). A factor analysis of multiple correspondences and a hierarchic classification were performed. For the factor analysis, usual quantity and frequency of drinking (for the last 12 months) were considered active variables; number of DSM-IV dependence criteria met, positive or negative diagnostic status for abuse, positive or negative diagnostic status for dependence (both for the last 12 months), and socio-demographic characteristics (age, gender and economic level) were considered illustrative variables. RESULTS: The first five factorial axes were retained, accounting for 88% of the total variance. Hierarchic classification resulted in six distinctive classes of drinking patterns. Two patterns were associated with a positive diagnosis of abuse and dependence, respectively. One, drinking between 4 and 6 drinks per occasion mostly on a weekly basis, was associated with a diagnosis of abuse; this pattern was also associated with meeting one or two dependence criteria (dependence orphans). The other, drinking 7 or more drinks per occasion, was associated with a diagnosis of dependence, and also with a diagnostic orphan condition. This class, composed primarily of males, was not characterized by any particular frequency of drinking. The other four drinking patterns were not associated with a positive diagnosis of an alcohol use disorder. Two of them were characterized by drinking low quantities with a low frequency (either monthly or yearly). Participants in both of these classes tended to be female. The other two patterns were characterized by drinking less than 3 drinks per occasion, either daily or weekly: the former associated with being older than 35 years, and the later with no distinctive socio-demographic characteristics. CONCLUSIONS: Results demonstrated six distinct drinking patterns, two of them related to a positive diagnosis of an alcohol use disorder. Our findings support previous research indicating that dependence orphans share some characteristics with abuse and dependence cases. Given the lack of similar studies in the region, these findings, although descriptive, enrich the knowledge of alcohol use disorders in the regional context. Furthermore, they may contribute to the development of local drinking guidelines and prevention strategies.

背景:先前的研究表明,在规范饮酒方面存在文化差异,此外,饮酒的数量和频率与酒精使用障碍有关。目的:本研究的主要目的是表征阿根廷急诊科患者的饮酒模式,其次,探讨这些饮酒模式与DSM-IV酒精使用障碍之间的关系。方法:从阿根廷马德普拉塔一家大型公立医院急诊科收治的患者概率样本中收集数据。这里分析的数据与那些报告在过去12个月内至少喝过一杯酒的人有关(n=529)。进行了多重对应的因子分析和层次分类。在因子分析中,通常饮酒量和饮酒频率(过去12个月)被认为是积极变量;符合DSM-IV依赖标准的数量、滥用的阳性或阴性诊断状态、依赖的阳性或阴性诊断状态(过去12个月)以及社会人口特征(年龄、性别和经济水平)被认为是说明性变量。结果:前5个因子轴保留,占总方差的88%。等级分类导致了六种不同的饮酒模式。两种模式分别与滥用和依赖的阳性诊断相关。第一,每周每次饮酒4到6杯,与被诊断为滥用有关;这种模式还与满足一个或两个依赖标准(依赖孤儿)有关。另一种,每次喝7杯或更多的酒,被诊断为依赖,也被诊断为孤儿。这个班级主要由男性组成,没有任何饮酒频率的特征。其他四种饮酒模式与酒精使用障碍的阳性诊断无关。其中两组的特点是饮酒量少,频率低(每月或每年)。这两门课的参与者都是女性。另外两种模式的特征是每次饮酒少于3杯,无论是每天还是每周:前者与年龄超过35岁有关,后者没有明显的社会人口统计学特征。结论:结果显示了六种不同的饮酒模式,其中两种与酒精使用障碍的阳性诊断有关。我们的发现支持了先前的研究,即依赖孤儿与虐待和依赖案例有一些共同的特征。鉴于该区域缺乏类似的研究,这些发现虽然是描述性的,但丰富了该区域背景下对酒精使用障碍的认识。此外,它们可能有助于制定当地饮酒指南和预防战略。
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引用次数: 0
Addressing Trauma Among Women With Serious Addictive Disorders: Treatment Models, Program Factors, And Potential Mediators. 解决严重成瘾障碍妇女的创伤:治疗模式,项目因素和潜在的中介。
Douglas L Polcin, Madhabika B Nayak, Susan Blacksher

A large majority of women entering addiction treatment present significant symptoms of trauma related to physical or sexual abuse. Despite research indicating that trauma interventions are integral to women's successful recovery from addiction, many programs do not adequately address violence-related trauma. This chapter provides a review of the literature on trauma among women with addictive disorders and several manual based interventions developed to address co-occurring addiction and trauma-related disorders. One intervention, "Beyond Trauma," which has become increasingly popular among community based programs is described in detail. Beyond Trauma appears to have several advantages over other therapies for treating trauma and addiction in women, including 1) a theoretical foundation that draws on relational theory as a guide to the intervention, 2) a broad based approach that can be utilized by a variety of professional and paraprofessional staff members, 3) a focus that goes beyond treating women with a formal diagnosis of post traumatic stress disorder to include treatment for an array of symptoms and problems associated with trauma, and 4) gender-appropriate use of expressive arts in its curriculum. The chapter also discusses treatment program environment factors that may be critically important to treatment outcome for women: 1) whether the program is gender specific, 2) the degree of emphasis on peer involvement in recovery, 3) program recognition of the value of knowledge-based recovery experience, 4) program facilitation of cohesion, 5) the empowerment of clients in decisions affecting the program and 6) skills training relevant to managing moods, relationships and a variety of problems that women face during recovery. Possible mechanisms of change for Beyond Trauma are explored with particular emphasis on the variety of ways the intervention attempts to impact problem areas experienced by women (e.g., mental health functioning self esteem and social support). Recommendations for future research in the treatment of trauma and addiction-related disorders in women are outlined.

绝大多数接受成瘾治疗的妇女都表现出与身体或性虐待有关的严重创伤症状。尽管有研究表明,创伤干预是女性成功戒除毒瘾不可或缺的一部分,但许多项目并没有充分解决与暴力有关的创伤。本章回顾了有关成瘾障碍女性创伤的文献,以及为解决同时发生的成瘾和创伤相关障碍而开发的几种基于手册的干预措施。一种干预,“超越创伤”,已成为越来越流行的社区为基础的项目详细描述。与其他治疗女性创伤和成瘾的疗法相比,“超越创伤”似乎有几个优势,包括:1)利用关系理论作为干预指导的理论基础;2)广泛的方法,可以被各种专业和准专业人员使用;3)将重点从对患有创伤后应激障碍的女性的正式诊断扩展到对与创伤相关的一系列症状和问题的治疗;4)在课程中适当地使用表现艺术。本章还讨论了可能对女性治疗结果至关重要的治疗方案环境因素:1)该项目是否有性别针对性,2)康复过程中同伴参与的强调程度,3)该项目对基于知识的康复经验价值的认可,4)该项目促进凝聚力,5)在影响该项目的决策中赋予客户权力,6)与管理情绪、关系和女性在康复过程中面临的各种问题相关的技能培训。探讨了超越创伤的可能改变机制,特别强调了干预措施试图影响妇女所经历的问题领域的各种方式(例如,心理健康、功能、自尊和社会支持)。对今后在治疗妇女创伤和成瘾相关疾病方面的研究提出了建议。
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Journal of drug addiction, education, and eradication
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