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Glucose starvation induced upregulation of Prohibitin 1 via ROS generation causes mitochondrial dysfunction and apoptosis in breast cancer cells. 葡萄糖饥饿通过ROS生成诱导prohibition - 1上调,导致乳腺癌细胞线粒体功能障碍和凋亡。
Pub Date : 2019-12-01 DOI: 10.1016/j.freeradbiomed.2019.09.020
Ganesh Kumar Raut, Moumita Chakrabarti, D. Pamarthy, M. Bhadra
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引用次数: 31
Gender differences in albumin and ascorbic acid in the vitreous antioxidant system. 玻璃体抗氧化系统中白蛋白和抗坏血酸的性别差异。
Pub Date : 2019-11-06 DOI: 10.1016/j.freeradbiomed.2019.11.008
Schwab Christoph, Paar Margret, Fengler Vera Heike, Ivastinovic Domagoj, Haas Anton, Seidel Gerald, Glatz Wilfried, Malle Eva-Maria, Weger Martin, Velikay-Parel Michaela, Faustmann Gernot, Wedrich Andreas, Reibnegger Gilbert, Winklhofer-Roob Brigitte, Oettl Karl
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引用次数: 7
A short guide to nomenclature of radicals, radical ions, iron-oxygen complexes and polycyclic aromatic hydrocarbons 自由基、自由基离子、铁氧配合物和多环芳烃的命名指南
Pub Date : 1986-01-01 DOI: 10.1016/S8755-9668(86)80029-1
James G. Traynham

This article summarizes and illustrates by examples the principal official rules for naming some species used by research writers in the field of free radical biology.

本文总结并举例说明了自由基生物学领域研究作者使用的一些物种命名的主要官方规则。
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引用次数: 4
The radical view 激进的观点
Pub Date : 1986-01-01 DOI: 10.1016/S8755-9668(86)80016-3
William A. Pryor (Editor-in-Chief)
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引用次数: 12
Contents of volume 2 第二卷内容
Pub Date : 1986-01-01 DOI: 10.1016/S8755-9668(86)80031-X
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引用次数: 0
Quinones in Biology: Functions in electron transfer and oxygen activation 醌类在生物学中的作用:电子传递和氧活化
Pub Date : 1986-01-01 DOI: 10.1016/S8755-9668(86)80030-8
Hans Nohl, Werner Jordan, Richard J. Youngman

The widespread occurrence and function of quinones in diverse biological systems with particular reference to their role in oxygen activation has been reviewed and discussed. The importance of quinones in such basic metabolic processes as respiration and photosynthesis has been well established. Less clear are the conditions which lead to loss of the normal metabolic functions of quinones, often resulting in the generation of active oxygen species.

Exogenous quinones have been used extensively as tools to investigate physiological processes. In addition, they are increasingly applied to influence such reactions artificially. In particular, the loss of normal quinone function invariably has toxicological consequences, in which the activation of oxyqen hy quinone redox reactions plays a central role.

The present review discusses hiological and toxicological aspects of quinone metabolism. Physical and chemical data are considered in order to attempt to explain the physiological effects.

综述和讨论了醌类化合物在不同生物系统中的广泛存在和功能,特别是它们在氧活化中的作用。醌类化合物在呼吸和光合作用等基本代谢过程中的重要性已得到充分证实。不太清楚的是导致醌正常代谢功能丧失的条件,通常导致活性氧的产生。外源性醌已被广泛用作研究生理过程的工具。此外,它们被越来越多地用于人为地影响这些反应。特别是,正常醌功能的丧失不可避免地具有毒理学后果,其中氧醌氧化还原反应的激活起着核心作用。本文综述了醌代谢的生理和毒理学方面的研究进展。为了试图解释生理效应,考虑了物理和化学数据。
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引用次数: 154
SINGLET O2 (VOLUMES I, II, III, AND IV) 单重态o2(第一、二、三、四卷)
Pub Date : 1986-01-01 DOI: 10.1016/S8755-9668(86)80010-2
K. Davies
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引用次数: 0
A Comment on Free Radical Nomenclature 对自由基命名法的评述
Pub Date : 1986-01-01 DOI: 10.1016/S8755-9668(86)80028-X
Dr. William A. Pryor (Editor-in-Chief)
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引用次数: 0
Superoxide radical: A likely link between reperfusion injury and inflammation 超氧自由基:可能与再灌注损伤和炎症有关
Pub Date : 1986-01-01 DOI: 10.1016/S8755-9668(86)80018-7
Joe M. McCord

The roles of the superoxide free radical (02.-) in the inflammatory process are known to include bactericidal action as well as participation in cell-to- cell communication via the activation of a superoxide-dependent chemo-attractant. A role for the radical is now also understood in post-ischemic injury brought about by the reintroduction of molecular oxygen. In this case the superoxide is produced within the reperfused tissue by the en~me xanthine oxidase. While the mechanisms of free radical production are biochemically distinct in these two pathological conditions, there are logical links apparent whereby an ischemia/reperfusion-induced injury would lead to inflammation, and conversely whereby an inflamed tissue could become vulnerable to ischemic injury. The occasional study notwithstanding, much evidence now supports roles for superoxide and hydrogen peroxide in reperfusion injury to the heart. In the rat and dog, xanthine oxidase appears to be the primary source of these forms of active oxygen. Manifestations of injury include inactivation of specific enzymes, functional impairment of the mitochondria, and conductance abnormalities.

已知超氧化物自由基(02.-)在炎症过程中的作用包括杀菌作用以及通过激活超氧化物依赖的化学引诱剂参与细胞间的通讯。自由基在重新引入分子氧引起的缺血后损伤中的作用现在也被理解。在这种情况下,超氧化物在再灌注的组织中由en~me黄嘌呤氧化酶产生。虽然在这两种病理条件下,自由基产生的机制在生物化学上是不同的,但有明显的逻辑联系,即缺血/再灌注诱导的损伤会导致炎症,反过来,炎症组织可能容易受到缺血性损伤。尽管偶尔有研究,但现在有很多证据支持超氧化物和过氧化氢在心脏再灌注损伤中的作用。在大鼠和狗身上,黄嘌呤氧化酶似乎是这些形式的活性氧的主要来源。损伤的表现包括特定酶的失活、线粒体的功能损伤和传导异常。
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引用次数: 75
The role of antioxidants in the retina and retinal pigment epithelium and the nature of prooxidant-induced damage 抗氧化剂在视网膜和视网膜色素上皮中的作用以及抗氧化剂诱导损伤的性质
Pub Date : 1986-01-01 DOI: 10.1016/S8755-9668(86)80024-2
Garry J. Handelman, Edward A. Dratz

The vertebrate retina has several features that make it vulnerable to damage from autoxidation. The photoreceptor membranes contain high levels of polyunsaturated fatty acids; abundant mitochondria are present which may leak activated oxygen species; and light exposure of the retina may cause photoxidation. These features are analyzed in detail, and the various antioxidant mechanisms of the vertebrate retina are surveyed. The interplay among oxidant stress and antioxidant defenses is illustrated by review of situations where these variables are either artificially manipulated or changed naturally. Vitamin E deficiency damages the retina in a number of well-defined vertebrate model systems, and a lipid autoxidation mechanism for this damage is widely assumed. The retina is quite sensitive to damage by elevated or prolonged light exposure; however, a free-radical role in light damage to the retina has not been established. An alternative mechanism for damage due to vitamin E deficiency and light is considered, which involves elevated vitamin A levels and vitamin A toxicity. Evidence is reviewed that the primate retina requires both vitamin E and selenium. The puzzling role of ocular melanin in light damage and protection is reviewed. Possible contributions of autoxidative damage to aging of the human retina are discussed.

脊椎动物的视网膜有几个特点,使它容易受到自氧化的损害。感光膜含有大量的多不饱和脂肪酸;存在丰富的线粒体,可能会泄漏活性氧;视网膜暴露在光线下可能会引起光氧化。详细分析了这些特征,并对脊椎动物视网膜的各种抗氧化机制进行了综述。氧化应激和抗氧化防御之间的相互作用通过回顾这些变量被人为操纵或自然改变的情况来说明。维生素E缺乏损害视网膜在许多明确定义的脊椎动物模型系统,脂质自氧化机制的这种损害被广泛认为。视网膜对高强度或长时间的光照非常敏感;然而,自由基在视网膜光损伤中的作用尚未确定。维生素E缺乏和光照导致的另一种损伤机制被认为涉及维生素A水平升高和维生素A毒性。有证据表明,灵长类动物的视网膜需要维生素E和硒。本文就眼部黑色素在光损伤及保护中的作用作一综述。讨论了自氧化损伤对人视网膜衰老的可能贡献。
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引用次数: 171
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Advances in Free Radical Biology & Medicine
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