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Japanese journal of medical science & biology最新文献

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The spread of Shigella dysenteriae type I in Africa. I型志贺氏痢疾菌在非洲的传播。
Pub Date : 1998-01-01 DOI: 10.7883/yoken1952.51.supplement1_s36
D F Wittenberg
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引用次数: 3
N-WASP is an important protein for the actin-based motility of Shigella flexneri in the infected epithelial cells. N-WASP是受感染的上皮细胞中福氏志贺氏菌肌动蛋白运动的重要蛋白。
Pub Date : 1998-01-01 DOI: 10.7883/yoken1952.51.supplement1_s63
T Suzuki, C Sasakawa
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引用次数: 11
Salmonella typhi entry into intestinal epithelial cells. 伤寒沙门氏菌进入肠上皮细胞。
Pub Date : 1998-01-01 DOI: 10.7883/yoken1952.51.supplement1_s90
X Z Huang, B Tall, W Schwan, D J Kopecko
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引用次数: 4
Strategy to prevent the progression of enterohemorrhagic Escherichia coli O157 infection to hemolytic uremic syndrome. 预防肠出血性大肠杆菌O157感染发展为溶血性尿毒症综合征的策略。
Pub Date : 1998-01-01 DOI: 10.7883/yoken1952.51.supplement1_s124
T Takeda
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引用次数: 5
The rediscovery of Shiga toxin and its role in clinical disease. 志贺毒素的重新发现及其在临床疾病中的作用。
Pub Date : 1998-01-01 DOI: 10.7883/yoken1952.51.supplement1_s5
G T Keusch
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引用次数: 5
Clinical aspects of shigellosis in reference of sigmoidoscopic findings and histopathological changes of the colon. 参考乙状结肠镜检查结果和结肠组织病理学改变的志贺氏菌病的临床方面。
Pub Date : 1998-01-01 DOI: 10.7883/yoken1952.51.supplement1_s23
K Kobari
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引用次数: 1
The locus of enterocyte effacement pathogenicity island of Shiga toxin-producing Escherichia coli O157:H7 and other attaching and effacing E. coli. 产志贺毒素大肠埃希菌O157:H7及其他附着和消隐性大肠埃希菌肠细胞消隐致病性岛位点。
Pub Date : 1998-01-01 DOI: 10.7883/yoken1952.51.supplement1_s101
J B Kaper
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引用次数: 34
Virulence of Shiga toxin-producing Escherichia coli (STEC) in orally-infected mice correlates with the type of toxin produced by the infecting strain. 产志贺毒素大肠杆菌(STEC)在口腔感染小鼠中的毒力与感染菌株产生的毒素类型相关。
Pub Date : 1998-01-01 DOI: 10.7883/yoken1952.51.supplement1_s108
A R Melton-Celsa, J E Rogers, C K Schmitt, S C Darnell, A D O'Brien
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引用次数: 51
Shigella and Escherichia coli strategies for survival at low pH. 志贺氏菌和大肠杆菌在低pH下的生存策略。
Pub Date : 1998-01-01 DOI: 10.7883/yoken1952.51.supplement1_s81
P L Small
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引用次数: 6
Enteropathogenic E. coli interactions with host cells. 致病性大肠杆菌与宿主细胞的相互作用。
Pub Date : 1998-01-01 DOI: 10.7883/yoken1952.51.supplement1_s91
B B Finlay, A Abe

Enteropathogenic E. coli (EPEC) interacts with intestinal epithelial cells, causing diarrhea and associated diseases. This pathogen binds to epithelial cells using sophisticated mechanisms that exploit existing epithelial signal transduction pathways and host cytoskeletal components, ultimately resulting in the bacterium resting upon a pedestal on host cell surfaces. Recent data indicates that similar mechanisms occur in vivo. EPEC interactions with host cells illustrate several principles of pathogenesis that are used by bacteria that interact with mammalian host cells.

肠致病性大肠杆菌(EPEC)与肠上皮细胞相互作用,引起腹泻和相关疾病。这种病原体利用复杂的机制与上皮细胞结合,利用现有的上皮信号转导途径和宿主细胞骨架成分,最终导致细菌在宿主细胞表面的基座上休息。最近的数据表明,类似的机制也发生在体内。EPEC与宿主细胞的相互作用说明了细菌与哺乳动物宿主细胞相互作用的几个发病原理。
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引用次数: 3
期刊
Japanese journal of medical science & biology
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