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[Abstracts of papers at XXXI annual meeting of Japan Society of Smooth Muscle Research. October 3,4, 1990, Fukuoka City]. 日本平滑肌研究学会第31届年会论文摘要。1990年10月3日至4日,福冈市。
Pub Date : 1990-04-01
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引用次数: 0
[Clinical effects of gastrointestinal-motility stimulating agents on non-ulcer dyspepsia]. [胃肠动力刺激剂治疗非溃疡性消化不良的临床疗效]。
Pub Date : 1990-04-01
S Harasawa, H Takayasu
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引用次数: 0
[Comparative study of effects of gastrointestinal-motility control agents on rats and human colon]. [胃肠动力控制剂对大鼠和人结肠影响的比较研究]。
Pub Date : 1990-04-01
D Sasaki, S Kawakami, Y Yoshida
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引用次数: 0
[Mechanism of action of trimebutine maleate on gastro-colic reflex in patients with irritable bowel syndrome]. [马来酸曲美布汀对肠易激综合征患者胃绞痛反射的作用机制]。
Pub Date : 1990-04-01
T Matsue, S Yamato, N Umeda
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引用次数: 0
[Effects of cisapride on upper-gastrointestinal motility and digestive hormones]. 西沙必利对上消化道运动和消化激素的影响。
Pub Date : 1990-04-01
S Abe, Y Furukawa, K Nakata, M Morita, Y Ohira, Y Haneu, T Aoki
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引用次数: 0
[Experimental investigations of the propagation mechanism and the nerve regulatory mechanism of the interdigestive migrating electric complex in intestinal movement]. [肠道运动中消化间迁移电复合体的传播机制和神经调控机制的实验研究]。
Pub Date : 1990-02-01 DOI: 10.1540/jsmr1965.26.9
T Ozawa, K Kawamura, S Kitahara, H Murakuni, H Deguchi, N Shimada, Y Tsugu

Segments and Thirty-Vella type loops of the small intestine of dogs were prepared, and the excitation propagation pattern of the interdigestive migrating electric complex (IMEC) was observed by electromyography. The nerve regulatory mechanism in intestinal movement was investigated. The results indicated that first, the frequency of the basic electric rhythm (BER) is controlled so that a downward gradient is formed from the upper to the lower intestine, but this gradient continues to be controlled by a two-dimensional control mechanism involving parietal factors and extrinsic nerves due to transection of the intestines and damage to the extrinsic nerves in the dominant region. However, propagation of the IMEC in the small intestine appeared to be regulated by a combination of the two-dimensional control mechanism involving extrinsic and parietal nerves corresponding to the BER control mechanism. It was clear that the propagation mechanism of IMEC is related to the BER frequency, and propagation of the IMEC is downward in accordance with the frequency gradient of BER.

制备犬小肠段和30 - vella型环,用肌电图观察消化间迁移电复合体(IMEC)的兴奋传播模式。研究了肠道运动的神经调节机制。结果表明,首先,基本电节律(BER)的频率受到控制,由上至下形成一个向下的梯度,但由于肠的横断和优势区外源性神经的损伤,这种梯度继续受到一个涉及壁因子和外源性神经的二维控制机制的控制。然而,IMEC在小肠内的繁殖似乎受到与BER控制机制相对应的涉及外源性神经和顶神经的二维控制机制的组合调节。可见,IMEC的传播机制与误码率频率有关,IMEC的传播沿误码率的频率梯度向下。
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引用次数: 2
Inhibition by beta-CCM of cholecystokinin-induced release of acetylcholine from the longitudinal muscle-myenteric plexus preparation in the guinea-pig ileum. β - ccm对豚鼠回肠纵肌肌丛制剂中缩胆囊素诱导的乙酰胆碱释放的抑制作用。
Pub Date : 1990-02-01 DOI: 10.1540/jsmr1965.26.1
M Itonaga, N Sunagane, T Uruno, K Kubota

The antagonism between cholecystokinin (CCK) and methyl beta-carboline-3-carboxylate (beta-CCM) in the nervous system was studied by measuring the release of acetylcholine (ACh) from the longitudinal muscle-myenteric plexus preparation of guinea-pig. The ACh release was assessed by measuring [3H] output from the preparation preincubated with [3H] choline. Thirty mM of KCl caused a pronounced release of [3H] ACh from the preparation. Sulfated cholecystokinin octapeptide (CCK8) also increased the release of [3H] ACh in a dose-dependent manner at concentrations ranging from 10(-10)M to 10(-8)M. CCK8 at a concentration of 10(-8)M released [3H] ACh by about 300% of the 30 mM KCl-induced [3H] ACh release. In the presence of beta-CCM (10(-8)M to 10(-4)M), the release of [3H] ACh by KCl was not affected, but that by CCK8 was significantly inhibited depending on the concentrations of beta-CCM. These results show that the action of CCK8 to stimulate neurons in the myenteric plexus can be selectively antagonized by beta-CCM.

之间的对抗缩胆囊素(CCK)和甲基beta-carboline-3-carboxylate (beta-CCM)神经系统通过测量研究了乙酰胆碱的释放(ACh)纵向muscle-myenteric丛实验材料的准备。通过测定与[3H]胆碱预孵育的[3H]制剂的输出量来评估乙酰胆碱释放。30mm的KCl使[3H]乙酰胆碱从制剂中明显释放。硫酸化缩胆素八肽(CCK8)在10(-10)M至10(-8)M的浓度范围内也以剂量依赖性的方式增加[3H]乙酰胆碱的释放。浓度为10(-8)M的CCK8释放[3H]乙酰胆碱,释放量约为30 mM kcl诱导[3H]乙酰胆碱释放量的300%。在β - ccm (10(-8)M ~ 10(-4)M)存在时,KCl对[3H]乙酰胆碱的释放不受影响,但CCK8对[3H]乙酰胆碱的释放受β - ccm浓度的显著抑制。这些结果表明,CCK8对肌肠丛神经元的刺激作用可被β - ccm选择性拮抗。
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引用次数: 0
[Studies on function of the upper urinary tract. XXIX. Canine ureteral electromyogram isolated from "so-called Pacemaker"]. 上尿路功能的研究。制造。犬输尿管肌电图从“所谓的起搏器”中分离出来。
Pub Date : 1990-02-01 DOI: 10.1540/jsmr1965.26.21
K Suga, T Tsuzuki, S Kawamura, K Seo, T Kubo

Many workers have reported that ureteral peristaltic movement is controlled by the "so-called Pacemaker". But, in our recent studies, it was revealed that the existence of pacemaker is not always necessary for the peristaltic movement. In this study, we made isolated and non-isolated prototype models, using 18 mongrel dogs, to explore the influential factors on ureteral peristaltic discharge. Bilateral kidney and ureter were exposed transperitoneally. Unilateral upper urinary tract was prepared to preserve the pacemaker without renal blood supply, and contralateral one was prepared not to preserve the pacemaker by cutting at the proximal portion of the ureter. Vesico-ureteral reflux (VUR) was caused on these two models. A luminal pressure and ureteral electromyogram was recorded. In the result, there is spontaneous peristaltic discharges of the ureter which had the tendency to increase peristaltic frequency according to the increase of the luminal pressure. It was suggested that adequate expanding stimulation is the factor of peristaltic discharge to increase, and the peristaltic discharge under this condition propagates from upper to lower portion of the ureter.

许多工人报告说,输尿管蠕动运动是由“所谓的起搏器”控制的。但是,在我们最近的研究中发现,起搏器的存在并不总是蠕动运动所必需的。本研究采用18只杂种犬分别制作孤立和非孤立原型模型,探讨影响输尿管蠕动排出的因素。经腹膜暴露双侧肾脏和输尿管。制备单侧上尿路保留起搏器,无肾血供;制备对侧上尿路不保留起搏器,在输尿管近端切开。两种模型均发生膀胱输尿管反流(VUR)。记录管腔压和输尿管肌电图。结果表明,输尿管有自发性的蠕动放电,且随腔压的升高,其蠕动频率有增加的趋势。提示充分的扩张刺激是促使蠕动流量增加的因素,这种情况下的蠕动流量由输尿管上段向下段传播。
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引用次数: 0
[The effects of various gastrokinetic drugs on gastric emptying]. [各种胃动力药物对胃排空的影响]。
Pub Date : 1990-02-01 DOI: 10.1540/jsmr1965.26.31
H Tatsumi

In order to elucidate the effects of various gastrokinetic drugs on gastric emptying and the sites of their action in the stomach, changes in gastric emptying after administration of these drugs were determined in healthy adults by means of radioisotopic technique, by setting 3 regions of interest, i.e., the whole stomach, the proximal area and the antral area. Following results were obtained. 1. With metoclopramide administration, no particular movement of gastric contents was found for several minutes after ingestion. Once the movement of emptying was initiated, the gastric contents were transferred more efficiently from the proximal area to the antral area in comparison with the corresponding movement observed in persons given no metoclopramide. On the other hand, the outflow from the antral area to the duodenum exceeded the inflow from the proximal area to the antral area. 2. With domperidon administration, transfer of gastric contents was markedly increased, but the outflow from the antral area did not exceed the inflow. Domperidone caused overall facilitation of gastric emptying, mainly by enhancing the emptying movement in the proximal area. 3. With aclatonium napadisilate administration, marked transfer of the gastric contents from the proximal area to the antral area was noted, and the outflow from the antral area to the duodenum was equal to the inflow within 10 min, then exceeded the inflow. 4. With trimebutine maleate administration, transfer of gastric contents from the proximal area to the antral area was conspicuous, and the outflow from the antral area exceeded the increased inflow, resulting in overall faciliation of gastric emptying.

为了阐明各种胃动力药物对胃排空的影响及其在胃中的作用部位,采用放射性同位素技术测定了健康成人服用这些药物后胃排空的变化,并设置了3个感兴趣的区域,即整个胃、近端胃和胃窦区胃。得到以下结果:1. 服用甲氧氯普胺后几分钟内胃内容物未见特别运动。一旦排空运动开始,胃内容物从近端区转移到中端区比未给予甲氧氯普胺的人更有效。另一方面,胃窦区向十二指肠的流出量超过近端区向胃窦区的流入量。2. 在给药过程中,胃内容物的转移明显增加,但胃窦区流出量不超过流入量。多潘立酮对胃排空有全面促进作用,主要是通过增强胃近端排空运动。3.应用napadisate aclatonium后,胃内容物明显由近端向胃窦区转移,胃窦区向十二指肠的流出量在10 min内与流入量相等,然后超过流入量。4. 使用马来酸曲美布汀后,胃内容物从近端区向胃窦区转移明显,胃窦区流出量超过流入量的增加,导致胃排空整体加速。
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引用次数: 3
[Rhythmic contraction of smooth muscle in the pulmonary veins of dogs and monkeys]. [狗和猴子肺静脉平滑肌的节律性收缩]。
Pub Date : 1989-12-01
T Horiuchi, T Matsuki, T Ohashi
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引用次数: 0
期刊
Nihon Heikatsukin Gakkai zasshi
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