Sri Yenni Trisnawati, Purwa Samatra Dewa Putu Gde, Lely Setyawati Kurniawan, Astari Arum Cendani Goller, Valentina Tjandra Dewi
Pendahuluan: Penyakit Parkinson (PP) memiliki dimensi gejala sangat luas yang secara langsung dan tidak langsung mempengaruhi kualitas hidup penderita maupun keluarga. Gejala non motorik PP diantaranya adalah gangguan neuropsikiatri seperti depresi, psikosis, ansietas. Kami melaporkan sebuah kasus PP dengan gejala psikotik berupa halusinasi yang jarang dijumpai. Laporan Kasus: Pasien perempuan berusia 74 tahun terdiagnosis Parkinson sejak lebih dari 20 tahun, dibawa ke rumah sakit dengan bicara tidak nyambung, adanya halusinasi visual, dan emosi yang tidak stabil sejak 1 bulan. Keluarga pasien juga merasa sejak 6 bulan terakhir pasien menjadi sering lupa. Pasien dengan riwayat PP tidak rutin mengkonsumsi obat parkinson di bawah supervisi dokter. Keluhan halusinasi pernah dirasakan 2 tahun lalu namun membaik setelah perawatan. Diskusi: Gejala non motorik dapat dijumpai seiring perkembangan penyakit Parkinson dandapat diidentifikasi sebelum sindrom motor muncul. Adanya gejala psikosis berupa halusinasi dalam kasus yang dipaparkan terhadi setelah 18 tahun sejak pasien terdiagnosis, dimana berdasarkan hasil studi terdahulu dilaporkan 60% penderita PP berkembang adanya halusinasi atau delusi setelah 12 tahun perawatan dan pada usia lebih tua saat onset PP. Gangguan kognitif pada PP juga dicurigai dalam kasus walau belum sepenuhnya dapat ditegakan serta adanya gangguan tidur pada PP dengan prevalensi 40-90% dijumpai pada kasus yang kami laporkan. Kesimpulan: Manifestasi non motorik PP yang mencakup gejala neuropsikiatri sangat mempengaruhi kualitas hidup pasien maupun keluarga dan perawat pasien. Klinisi harus meningkatkan kewaspadaan terhadap gejala non motorik PP dan pemberian tatalaksana farmakologi dan non farmakologi harus dilakukan secara terintegratif bersama dengan bidang-bidang terkait sesuai dengan kondisi pasien. Kata kunci: Parkinson, non-motor, psikotik, neuropsikiatri
{"title":"GEJALA PSIKOTIK PADA PENYAKIT PARKINSON: SEBUAH LAPORAN KASUS DAN TINJAUAN PUSTAKA","authors":"Sri Yenni Trisnawati, Purwa Samatra Dewa Putu Gde, Lely Setyawati Kurniawan, Astari Arum Cendani Goller, Valentina Tjandra Dewi","doi":"10.29342/cnj.v4i2.152","DOIUrl":"https://doi.org/10.29342/cnj.v4i2.152","url":null,"abstract":"Pendahuluan: Penyakit Parkinson (PP) memiliki dimensi gejala sangat luas yang secara langsung dan tidak langsung mempengaruhi kualitas hidup penderita maupun keluarga. Gejala non motorik PP diantaranya adalah gangguan neuropsikiatri seperti depresi, psikosis, ansietas. Kami melaporkan sebuah kasus PP dengan gejala psikotik berupa halusinasi yang jarang dijumpai. \u0000Laporan Kasus: Pasien perempuan berusia 74 tahun terdiagnosis Parkinson sejak lebih dari 20 tahun, dibawa ke rumah sakit dengan bicara tidak nyambung, adanya halusinasi visual, dan emosi yang tidak stabil sejak 1 bulan. Keluarga pasien juga merasa sejak 6 bulan terakhir pasien menjadi sering lupa. Pasien dengan riwayat PP tidak rutin mengkonsumsi obat parkinson di bawah supervisi dokter. Keluhan halusinasi pernah dirasakan 2 tahun lalu namun membaik setelah perawatan. \u0000Diskusi: Gejala non motorik dapat dijumpai seiring perkembangan penyakit Parkinson dandapat diidentifikasi sebelum sindrom motor muncul. Adanya gejala psikosis berupa halusinasi dalam kasus yang dipaparkan terhadi setelah 18 tahun sejak pasien terdiagnosis, dimana berdasarkan hasil studi terdahulu dilaporkan 60% penderita PP berkembang adanya halusinasi atau delusi setelah 12 tahun perawatan dan pada usia lebih tua saat onset PP. Gangguan kognitif pada PP juga dicurigai dalam kasus walau belum sepenuhnya dapat ditegakan serta adanya gangguan tidur pada PP dengan prevalensi 40-90% dijumpai pada kasus yang kami laporkan. \u0000Kesimpulan: Manifestasi non motorik PP yang mencakup gejala neuropsikiatri sangat mempengaruhi kualitas hidup pasien maupun keluarga dan perawat pasien. Klinisi harus meningkatkan kewaspadaan terhadap gejala non motorik PP dan pemberian tatalaksana farmakologi dan non farmakologi harus dilakukan secara terintegratif bersama dengan bidang-bidang terkait sesuai dengan kondisi pasien. \u0000Kata kunci: Parkinson, non-motor, psikotik, neuropsikiatri","PeriodicalId":339514,"journal":{"name":"Callosum Neurology","volume":"79 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-09-08","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"126181727","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Ida Ayu Sri Indrayani, Andre Dharmawan Wijono, Aurelia Vania, Edwin Pranata Laban, Johan Andrasilli, Muhammad Arismunandar, Fuji Restu Firma
Latar Belakang: COVID-19 sering dihubungkan dengan terjadinya hiperkoabilitas yang menyebabkan komplikasi pada sistem neurovascular, salah satunya adalah stroke. D-dimer merupakan salah satu pemeriksaan yang dapat digunakan untuk melihat adanya koagulopati. Tinjauan ini bertujuan untuk melihat pentingnya pemeriksaan d-dimer pada pasien COVID-19 yang mengalami stroke. Kasus: Dari data yang diambil di RSUP Sanglah bulan Juni sampai dengan Agustus 2020, didapatkan 4 pasien stroke non hemoragik yang terkonfirmasi COVID-19. Keempat pasien tersebut berusia 80, 75, 59, dan 76 tahun dimana semua pasien berjenis kelamin laki-laki. Klinis neurologis yang didapatkan pada keempat pasien tersebut yaitu paresis nervus cranialis VII, paresis nervus cranialis XII dan hemiparesis flaksid disertai dengan adanya refleks patologis. Dari keempat pasien tersebut, hanya ada satu pasien yang tidak mengalami sesak napas. Tiga pasien mengalami pneumonia pada pemeriksaan rontgen thorax. Setelah dilakukan pemeriksaan d-dimer, semua pasien menunjukkan peningkatan angka D-dimer dengan angka masing-masing 0.73, 1.03, 7.03, dan 1.51. Pasien dengan D-dimer 7.03 memiliki skor NIHSS tertinggi yaitu 8. Diskusi: Proses apoptosis sel-sel endotel dari struktur vaskular mengakibatkan terjadinya koagulopati dan peningkatan D-dimer. Studi melaporkan hasil laboratorium D-dimer yang lebih tinggi pada kondisi COVID-19 berat atau COVID-19 dengan gangguan serebrovaskular. Mekanisme gangguan serebrovaskular tanpa faktor risiko vaskular sebelumnya diduga berasal dari kondisi hiperkoagulasi yang menyebabkan pembentukan trombus dalam pembuluh darah. D-dimer meningkat pada 36% pasien dengan COVID-19 di Wuhan, yang dikaitkan dengan risiko kematian yang lebih tinggi. Kesimpulan: Kadar D-dimer berkorelasi dengan keparahan penyakit dan merupakan suatu penanda prognostik tingkat keparahan pada pasien yang dirawat karena COVID-19. Kata kunci: D-dimer, Covid-19, stroke, luaran klinis
{"title":"PENINGKATAN D-DIMER DAN LUARAN KLINIS PASIEN TERKONFIRMASI COVID-19 YANG MENGALAMI STROKE DI RSUP SANGLAH : LAPORAN KASUS","authors":"Ida Ayu Sri Indrayani, Andre Dharmawan Wijono, Aurelia Vania, Edwin Pranata Laban, Johan Andrasilli, Muhammad Arismunandar, Fuji Restu Firma","doi":"10.29342/cnj.v4i2.164","DOIUrl":"https://doi.org/10.29342/cnj.v4i2.164","url":null,"abstract":"Latar Belakang: COVID-19 sering dihubungkan dengan terjadinya hiperkoabilitas yang menyebabkan komplikasi pada sistem neurovascular, salah satunya adalah stroke. D-dimer merupakan salah satu pemeriksaan yang dapat digunakan untuk melihat adanya koagulopati. Tinjauan ini bertujuan untuk melihat pentingnya pemeriksaan d-dimer pada pasien COVID-19 yang mengalami stroke. \u0000Kasus: Dari data yang diambil di RSUP Sanglah bulan Juni sampai dengan Agustus 2020, didapatkan 4 pasien stroke non hemoragik yang terkonfirmasi COVID-19. Keempat pasien tersebut berusia 80, 75, 59, dan 76 tahun dimana semua pasien berjenis kelamin laki-laki. Klinis neurologis yang didapatkan pada keempat pasien tersebut yaitu paresis nervus cranialis VII, paresis nervus cranialis XII dan hemiparesis flaksid disertai dengan adanya refleks patologis. Dari keempat pasien tersebut, hanya ada satu pasien yang tidak mengalami sesak napas. Tiga pasien mengalami pneumonia pada pemeriksaan rontgen thorax. Setelah dilakukan pemeriksaan d-dimer, semua pasien menunjukkan peningkatan angka D-dimer dengan angka masing-masing 0.73, 1.03, 7.03, dan 1.51. Pasien dengan D-dimer 7.03 memiliki skor NIHSS tertinggi yaitu 8. \u0000Diskusi: Proses apoptosis sel-sel endotel dari struktur vaskular mengakibatkan terjadinya koagulopati dan peningkatan D-dimer. Studi melaporkan hasil laboratorium D-dimer yang lebih tinggi pada kondisi COVID-19 berat atau COVID-19 dengan gangguan serebrovaskular. Mekanisme gangguan serebrovaskular tanpa faktor risiko vaskular sebelumnya diduga berasal dari kondisi hiperkoagulasi yang menyebabkan pembentukan trombus dalam pembuluh darah. D-dimer meningkat pada 36% pasien dengan COVID-19 di Wuhan, yang dikaitkan dengan risiko kematian yang lebih tinggi. \u0000Kesimpulan: Kadar D-dimer berkorelasi dengan keparahan penyakit dan merupakan suatu penanda prognostik tingkat keparahan pada pasien yang dirawat karena COVID-19. \u0000Kata kunci: D-dimer, Covid-19, stroke, luaran klinis","PeriodicalId":339514,"journal":{"name":"Callosum Neurology","volume":"35 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-09-08","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"125825563","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
I. K. Aryawan, A. Triningrat, Made Paramita Wijayanti, I. Indrayani, Pande Ketut Kurniari
Introduction: Oculomotor nerve palsy is an pathological condition caused by microvascular injury, head injury, compression due to neoplasm or aneurysm, and also oculomotor nerve palsy can be caused by autoimmune prosses. Peripheral neuropathy is one of the clinical manifestations in patient with SLE, Oculomotor nerve palsy is one type of cranial neuropathy seen with SLE patient. Patient with SLE have a higher risk of serebrovascular event than general population. Case Illustration: Female 34 years old complained drop of the eyelid on the left eye and double vision when see with both eyes since 1 mounth before examination. Patient with history of headace and diagnosed with SLE since 2006 with regular treatment. From the examination, pupil anisocor, on the right eye pupil was 3 mm in diameter with positif direct and indirect reflex. On the left eye pupil was 6 mm with negative direct and indirect reflex. Extraocular movement on the left eye was limited except abduction movement. CT-Scan examination shows bilateral subarachnoid hemorrhage and from CT-Angiography shows dilatation of the left siphon carotid artery. Patient was diagnosed with oculomotor nerve palsy involving pupil caused by aneurysm with SAH and SLE. Discussion: Oculomotor nerve palsy mostly caused by aneurysm compression in posterior communicating artery (PCoA) and internal carotid artery (ICA). Cerebrovascular imaging, MRA and CTAngiography, can showing the aneurysm and its location. Risk of cerebrovascular event increased in patient with SLE than general population. Management patient with oculomotor nerve palsy with SLE nowadays is with pulse dose corticosteroid. Conclusion: SLE with oculomotor nerve palsy will increase risk of cerebrovascular event. Key Words : Oulomotor Nerve Palsy, Subarachnoid Hemorrhage, Systemic Lupus Erithematosus
{"title":"LESI NERVUS OKULOMOTOR INVOLVED PUPIL ET CAUSA ANEURISMA PADA PASIEN DENGAN STROKE SUBARACHNOID HEMORRHAGIC DAN SYSTEMIC LUPUS ERITHEMATOSUS","authors":"I. K. Aryawan, A. Triningrat, Made Paramita Wijayanti, I. Indrayani, Pande Ketut Kurniari","doi":"10.29342/cnj.v4i2.163","DOIUrl":"https://doi.org/10.29342/cnj.v4i2.163","url":null,"abstract":"Introduction: Oculomotor nerve palsy is an pathological condition caused by microvascular injury, head injury, compression due to neoplasm or aneurysm, and also oculomotor nerve palsy can be caused by autoimmune prosses. Peripheral neuropathy is one of the clinical manifestations in patient with SLE, Oculomotor nerve palsy is one type of cranial neuropathy seen with SLE patient. Patient with SLE have a higher risk of serebrovascular event than general population.\u0000Case Illustration: Female 34 years old complained drop of the eyelid on the left eye and double vision when see with both eyes since 1 mounth before examination. Patient with history of headace and diagnosed with SLE since 2006 with regular treatment. From the examination, pupil anisocor, on the right eye pupil was 3 mm in diameter with positif direct and indirect reflex. On the left eye pupil was 6 mm with negative direct and indirect reflex. Extraocular movement on the left eye was limited except abduction movement. CT-Scan examination shows bilateral subarachnoid hemorrhage and from CT-Angiography shows dilatation of the left siphon carotid artery. Patient was diagnosed with oculomotor nerve palsy involving pupil caused by aneurysm with SAH and SLE.\u0000Discussion: Oculomotor nerve palsy mostly caused by aneurysm compression in posterior communicating artery (PCoA) and internal carotid artery (ICA). Cerebrovascular imaging, MRA and CTAngiography, can showing the aneurysm and its location. Risk of cerebrovascular event increased in patient with SLE than general population. Management patient with oculomotor nerve palsy with SLE nowadays is with pulse dose corticosteroid.\u0000Conclusion: SLE with oculomotor nerve palsy will increase risk of cerebrovascular event.\u0000Key Words : Oulomotor Nerve Palsy, Subarachnoid Hemorrhage, Systemic Lupus Erithematosus","PeriodicalId":339514,"journal":{"name":"Callosum Neurology","volume":"41 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-09-08","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"116471216","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Orlando Pikatan, Desie Yuliani, I. Sumada, Ni Ketut Candra Wiratmi
Background: Corona virus disease 2019 (COVID-19) is a pandemic disease with a wide spectrum of symptoms. Neurological symptoms are often found in this disease. Stroke on the other hand is a multifactorial disease that can be caused by a lot of underlying problems. Case: We report a case of a 67 years old man with a weakness on the right side that was worsened one day before arrival in hospital and a shortness of breath. the patient had history of stroke before, and controlled hypertension. Head Ct-scan showed a stroke infarct, and Swab RT-PCR showed + SARS-CoV-2. Discussion: Infection may become the risk factor of stroke. inflammatory mediators such as TNF-a , and CRP support the procoagulant state which leads to stroke. Conclusion: COVID-19 may increase the risk of stroke due to the inflammation state that leads to procoagulant state
{"title":"COVID-19 AS A POTENTIAL RISK FACTOR FOR ISCHEMIC STROKE: A CASE REPORT","authors":"Orlando Pikatan, Desie Yuliani, I. Sumada, Ni Ketut Candra Wiratmi","doi":"10.29342/cnj.v4i2.168","DOIUrl":"https://doi.org/10.29342/cnj.v4i2.168","url":null,"abstract":"Background: Corona virus disease 2019 (COVID-19) is a pandemic disease with a wide spectrum of symptoms. Neurological symptoms are often found in this disease. Stroke on the other hand is a multifactorial disease that can be caused by a lot of underlying problems.\u0000Case: We report a case of a 67 years old man with a weakness on the right side that was worsened one day before arrival in hospital and a shortness of breath. the patient had history of stroke before, and controlled hypertension. Head Ct-scan showed a stroke infarct, and Swab RT-PCR showed + SARS-CoV-2.\u0000Discussion: Infection may become the risk factor of stroke. inflammatory mediators such as TNF-a , and CRP support the procoagulant state which leads to stroke.\u0000Conclusion: COVID-19 may increase the risk of stroke due to the inflammation state that leads to procoagulant state\u0000 ","PeriodicalId":339514,"journal":{"name":"Callosum Neurology","volume":"29 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-09-08","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"114223320","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Rima Febry Lesmana, A. Triningrat, Made Paramita Wijayanti, I. Kusumadjaja, I. Indrayani, Gede Kambayana
Background: Neuromyelitis optica (NMO) is an in?ammatory demyelinating autoimmune disease of the central nervous system that most commonly affects the optic nerves and spinal cord. Seropositive antiAQP4 differentiates NMO from MS and the presence of manifestation in the postrema, brainsteam or diencephalic areas extend to NMO Spectrum Disorder (NMOSD). Case Description: A 18 years old male complain sudden vision loss on his left eye since 2 weeks ago. The examination show the visual acuity on the right eye was 6/6 and LPBP on the left eye. Positive RAPD on the left eye, funduscopy and the OCT RNFL within normal limits. Head MRI focus orbita with contrast show optic neuritis. Patient was diagnosed with left eye retrobulbar optic neuritis and ONTT therapy was given. The visual acuity improved to 1/60 then therapy change to oral steroid. Four months later, the patient suddenly got vision loss on the right eye accompanied by paraparesis. The visual acuity on the right eye was NLP and the left eye was 1/300, with mid-dilated papil. The results of the OCT RNFL show on the right eye edema papil and left eye atrophy papil. An MRI of the head focus orbital and whole spain was reexamined followed by VEP examination and an AntiAQP4 which showed an NMOSD. He was given ONTT then continued with immunosuppressants. The visual acuity of the right eye improved to 3/60 and the left eye remained 1/300. Discussion: This patient first presented with complain on the left eye with clinical and supporting symptoms suggesting an optic neuritis. The presence of a new attack on the right eye with paraparesis is a clinical feature of NMO supported by MRI results and seropositive AQP4 indicates an NMOSD. Conclusion: Establishment of diagnosis and administration of therapy quickly and precisely can reduce the severity and risk of recurrence which leads to greater disability and blindness. Key Words: Neuromyelitis Optica, Neuromyelitis Optica Spectrum Disorder, AQP4
{"title":"NEUROMYELITIS OPTICA SPECTRUM DISORDER (NMOSD) DENGAN ANTIBODI AQP4 POSITIF","authors":"Rima Febry Lesmana, A. Triningrat, Made Paramita Wijayanti, I. Kusumadjaja, I. Indrayani, Gede Kambayana","doi":"10.29342/cnj.v4i2.138","DOIUrl":"https://doi.org/10.29342/cnj.v4i2.138","url":null,"abstract":"Background: Neuromyelitis optica (NMO) is an in?ammatory demyelinating autoimmune disease of the central nervous system that most commonly affects the optic nerves and spinal cord. Seropositive antiAQP4 differentiates NMO from MS and the presence of manifestation in the postrema, brainsteam or diencephalic areas extend to NMO Spectrum Disorder (NMOSD).\u0000Case Description: A 18 years old male complain sudden vision loss on his left eye since 2 weeks ago. The examination show the visual acuity on the right eye was 6/6 and LPBP on the left eye. Positive RAPD on the left eye, funduscopy and the OCT RNFL within normal limits. Head MRI focus orbita with contrast show optic neuritis. Patient was diagnosed with left eye retrobulbar optic neuritis and ONTT therapy was given. The visual acuity improved to 1/60 then therapy change to oral steroid. Four months later, the patient suddenly got vision loss on the right eye accompanied by paraparesis. The visual acuity on the right eye was NLP and the left eye was 1/300, with mid-dilated papil. The results of the OCT RNFL show on the right eye edema papil and left eye atrophy papil. An MRI of the head focus orbital and whole spain was reexamined followed by VEP examination and an AntiAQP4 which showed an NMOSD. He was given ONTT then continued with immunosuppressants. The visual acuity of the right eye improved to 3/60 and the left eye remained 1/300.\u0000Discussion: This patient first presented with complain on the left eye with clinical and supporting symptoms suggesting an optic neuritis. The presence of a new attack on the right eye with paraparesis is a clinical feature of NMO supported by MRI results and seropositive AQP4 indicates an NMOSD.\u0000Conclusion: Establishment of diagnosis and administration of therapy quickly and precisely can reduce the severity and risk of recurrence which leads to greater disability and blindness.\u0000Key Words: Neuromyelitis Optica, Neuromyelitis Optica Spectrum Disorder, AQP4","PeriodicalId":339514,"journal":{"name":"Callosum Neurology","volume":"95 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-09-08","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"124365800","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Latar Belakang: Profil lipid dalam darah diketahui berpengaruh terhadap stroke, terutama stroke iskemik akut. Walaupun masih kontroversial, kadar trigliserida dalam darah diduga turut berpengaruh terhadap luaran klinis maupun mortalitas stroke iskemik akut. Penelitian ini bertujuan untuk mengetahui adanya hubungan kadar trigliserida dalam darah terhadap luaran klinis troke iskemik akut. Metode Penelitian: Penelitian ini merupakan penelitian deskriptif analitik dengan metode potong lintang. Semua pasien stroke iskemik akut serangan pertama yang dibuktikan dengan Computed Topography Scan (CT-Scan) pertama diikutkan sebagai subyek. Data dermografi pasien dan hasil laboratorium didapatkan dari rekam medis. Luaran klinis diukur dengan modified Rankin Scale (mRS). Nilai p<0.05 dianggap bermakna secara statistik. Hasil: Dari 73 subyek penelitian yang disertakan pada penelitian ini, sebanyak 41 subyek (56,2%) adalah laki-laki dengan rerata usia sebesar 60.73 tahun. Sebanyak 16 subyek (32,7%) didapatkan hiperglikemia saat masuk, 42 subyek (57,5%) didapatkan hipertensi saat masuk, 22 subyek (30,1%) didapatkan hiperkolesterolemia saat masuk, dan 24 subyek (32,9%) didapatkan hipertrigliseridemia saat masuk. Terdapat korelasi yang bermakna secara statistic antara kadar trigliserida dalam darah terhadap luaran klinis stroke iskemik akut pada subyek wanita (koefisien r=-0,358, nilai p=0,044) dan tanpa peningkatan kadar gula darah puasa/non hiperglikemia (koefisien r= -0,320, nilai p=0,028). Kesimpulan: Terdapat hubungan terbalik antara kadar trigliserida dalam darah penderita stroke iskemik akut wanita atau tanpa kenaikan kadar gula darah puasa saat masuk terhadap luaran klinis stroke iskemik akut. Kata Kunci: Luaran Klinis, Prognostik, Stroke iskemik, Trigliserida
{"title":"HUBUNGAN KADAR TRIGLISERIDA DALAM DARAH TERHADAP LUARAN KLINIS PENDERITA STROKE ISKEMIK AKUT","authors":"Daniel Mahendrakrisnna, Aria Chandra GTS","doi":"10.29342/cnj.v4i2.143","DOIUrl":"https://doi.org/10.29342/cnj.v4i2.143","url":null,"abstract":"Latar Belakang: Profil lipid dalam darah diketahui berpengaruh terhadap stroke, terutama stroke iskemik akut. Walaupun masih kontroversial, kadar trigliserida dalam darah diduga turut berpengaruh terhadap luaran klinis maupun mortalitas stroke iskemik akut. Penelitian ini bertujuan untuk mengetahui adanya hubungan kadar trigliserida dalam darah terhadap luaran klinis troke iskemik akut.\u0000Metode Penelitian: Penelitian ini merupakan penelitian deskriptif analitik dengan metode potong lintang. Semua pasien stroke iskemik akut serangan pertama yang dibuktikan dengan Computed Topography Scan (CT-Scan) pertama diikutkan sebagai subyek. Data dermografi pasien dan hasil laboratorium didapatkan dari rekam medis. Luaran klinis diukur dengan modified Rankin Scale (mRS). Nilai p<0.05 dianggap bermakna secara statistik.\u0000Hasil: Dari 73 subyek penelitian yang disertakan pada penelitian ini, sebanyak 41 subyek (56,2%) adalah laki-laki dengan rerata usia sebesar 60.73 tahun. Sebanyak 16 subyek (32,7%) didapatkan hiperglikemia saat masuk, 42 subyek (57,5%) didapatkan hipertensi saat masuk, 22 subyek (30,1%) didapatkan hiperkolesterolemia saat masuk, dan 24 subyek (32,9%) didapatkan hipertrigliseridemia saat masuk. Terdapat korelasi yang bermakna secara statistic antara kadar trigliserida dalam darah terhadap luaran klinis stroke iskemik akut pada subyek wanita (koefisien r=-0,358, nilai p=0,044) dan tanpa peningkatan kadar gula darah puasa/non hiperglikemia (koefisien r= -0,320, nilai p=0,028).\u0000Kesimpulan: Terdapat hubungan terbalik antara kadar trigliserida dalam darah penderita stroke iskemik akut wanita atau tanpa kenaikan kadar gula darah puasa saat masuk terhadap luaran klinis stroke iskemik akut.\u0000Kata Kunci: Luaran Klinis, Prognostik, Stroke iskemik, Trigliserida","PeriodicalId":339514,"journal":{"name":"Callosum Neurology","volume":"26 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-09-08","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"125276880","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Background: Herpes zoster (HZ) is caused by reactivation of Varicella Zoster Virus (VZV). Segmental zoster paresis (SZP) is a rare neurological complication of HZ. Case: A 58-year-old woman came to our clinic with a complaint of a post HZ lesion accompanied by weakness, burning sensation, and paresthesia of the left lower limb. These complaints were felt suddenly about 2 weeks after the vesicles appeared. The patient had a history of diabetes mellitus (DM) and hypertension. From physical examination, there was a lesions on the L2-L3 dermatome which had dried out with hyperpigmentation and crusted. Motoric examination of the left lower limb showed weakness of the hip flexors (MRC grade 3/5). While the power of the other muscles in right lower limb and in all muscles groups was MRC 5/5. Tonus and trophic was normal. There was hyperaesthesia noted in the left thigh in the L2-L3 dermatome. Discussion: Neurological complications in HZ can occur in the central nervous system or peripheral nervous system. In this case the patient had SZP as a complication of HZ that involving the motor system. This complication was characterized by paresis of the left lower limb after developing HZ infection. The pathogenesis of SZP remains unclear. Having a history of DM increases the incidence of neurological complications in HZ. This is because the effectiveness of cellular immunity against VZV in DM patients decreases. Conclusion: Segmental zoster paresis (SZP) is a rare neurological complication of HZ. SZP involves the motor nervous system. There are several risk factors that increase the incidence of neurological complications in HZ infection. Key words: Herpes Zoster, Segmental Zoster Paresis
{"title":"MONOPARESIS INFERIOR POST HERPES ZOSTER","authors":"Pande Komang Novi Dyantari, I. Sumada","doi":"10.29342/cnj.v4i2.149","DOIUrl":"https://doi.org/10.29342/cnj.v4i2.149","url":null,"abstract":"Background: Herpes zoster (HZ) is caused by reactivation of Varicella Zoster Virus (VZV). Segmental zoster paresis (SZP) is a rare neurological complication of HZ.\u0000Case: A 58-year-old woman came to our clinic with a complaint of a post HZ lesion accompanied by weakness, burning sensation, and paresthesia of the left lower limb. These complaints were felt suddenly about 2 weeks after the vesicles appeared. The patient had a history of diabetes mellitus (DM) and hypertension. From physical examination, there was a lesions on the L2-L3 dermatome which had dried out with hyperpigmentation and crusted. Motoric examination of the left lower limb showed weakness of the hip flexors (MRC grade 3/5). While the power of the other muscles in right lower limb and in all muscles groups was MRC 5/5. Tonus and trophic was normal. There was hyperaesthesia noted in the left thigh in the L2-L3 dermatome.\u0000Discussion: Neurological complications in HZ can occur in the central nervous system or peripheral nervous system. In this case the patient had SZP as a complication of HZ that involving the motor system. This complication was characterized by paresis of the left lower limb after developing HZ infection. The pathogenesis of SZP remains unclear. Having a history of DM increases the incidence of neurological complications in HZ. This is because the effectiveness of cellular immunity against VZV in DM patients decreases.\u0000Conclusion: Segmental zoster paresis (SZP) is a rare neurological complication of HZ. SZP involves the motor nervous system. There are several risk factors that increase the incidence of neurological complications in HZ infection.\u0000Key words: Herpes Zoster, Segmental Zoster Paresis\u0000 ","PeriodicalId":339514,"journal":{"name":"Callosum Neurology","volume":"35 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-09-08","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"129485985","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Cardiac cephalalgia merupakan nyeri kepala seperti migraine, umumnya namun tidak selalu diperberat dengan aktivitas fisik, timbul selama peristiwa myocardial ischaemia, membaik dengan pemberian nitroglycerine. Cardiac cephalalgia merupakan kasus nyeri kepala yang jarang namun dapat mempengaruhi aspek biopsikososial dan kualitas hidup penderita. Berdasarkan The Internasional Classification of Headache Disorder (ICHD), cardiac cephalalgia digolongkan ke dalam nyeri kepala sekunder yang terkait dengan kelainan homeostasis. Artikel ini membahas pengetahuan terbaru mengenai cardiac cephalalgia terkait definisi, epidemiologi, patofisiologi, gambaran klinis, penegakan diagnosis, diagnosis banding, serta pilihan terapinya dengan melakukan pencarian, review dan telaah serta menyimpulkan berbagai literatur terbaru terkait hal tersebut. Cardiac cephalalgia sering ditemukan pada usia dekade lima. Penegakkan diagnosis berdasarkan nyeri kepala yang berkaiatan dengan adanya myocardial ischaemia, dan berespon baik dengan pemberian nitroglycerine. Penatalaksanaan cardiac cephalalgia menitikberatkan pada penanganan myocardial ischaemia. Nitroglycerine merupakan pilihan utama untuk nyeri kepala Kata Kunci : Cardiac Cephalalgia, Nyeri Kepala Sekunder, Myocardial Ischaemia
{"title":"CARDIAC CEPHALALGIA","authors":"A. Yolanda, I. Adnyana","doi":"10.29342/cnj.v3i2.108","DOIUrl":"https://doi.org/10.29342/cnj.v3i2.108","url":null,"abstract":"Cardiac cephalalgia merupakan nyeri kepala seperti migraine, umumnya namun tidak selalu diperberat dengan aktivitas fisik, timbul selama peristiwa myocardial ischaemia, membaik dengan pemberian nitroglycerine. Cardiac cephalalgia merupakan kasus nyeri kepala yang jarang namun dapat mempengaruhi aspek biopsikososial dan kualitas hidup penderita. Berdasarkan The Internasional Classification of Headache Disorder (ICHD), cardiac cephalalgia digolongkan ke dalam nyeri kepala sekunder yang terkait dengan kelainan homeostasis. Artikel ini membahas pengetahuan terbaru mengenai cardiac cephalalgia terkait definisi, epidemiologi, patofisiologi, gambaran klinis, penegakan diagnosis, diagnosis banding, serta pilihan terapinya dengan melakukan pencarian, review dan telaah serta menyimpulkan berbagai literatur terbaru terkait hal tersebut. Cardiac cephalalgia sering ditemukan pada usia dekade lima. Penegakkan diagnosis berdasarkan nyeri kepala yang berkaiatan dengan adanya myocardial ischaemia, dan berespon baik dengan pemberian nitroglycerine. Penatalaksanaan cardiac cephalalgia menitikberatkan pada penanganan myocardial ischaemia. Nitroglycerine merupakan pilihan utama untuk nyeri kepala \u0000Kata Kunci : Cardiac Cephalalgia, Nyeri Kepala Sekunder, Myocardial Ischaemia","PeriodicalId":339514,"journal":{"name":"Callosum Neurology","volume":"59 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2020-05-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"122040587","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Winda Haeriyoko, Purwa Samatra, S. Trisnawati, I. Budiarsa, A.A.Ayu Suryapraba
Abstrak Latar Belakang : Jumlah kasus Penyakit Parkinson di Indonesia cukup tinggi dengan prevalensi per penduduk dan insiden per penduduk. Gangguan tidur ddapatkan pada penderita Penyakit Parkinson. Data demografi dapat digunakan sebagai pertimbangan klinisi dalam mendiagnosis serta menentukan penanganan lanjutan yang optimal. Tujuan : Mengetahui karakteristik klinis pasien Penyakit Parkinson dengan gangguan tidur di Poliklinik Saraf Rumah Sakit Umum Pusat Sanglah (RSUP) Sanglah dan Rumah Sakit Umum Daerah (RSUD) Wangaya periode bulan 2018. Metode Penelitian : Penelitian deskriptif observasional menggunakan kuesioner pasien Penyakit Parkinson yang berobat di Poliklinik Saraf RSUP Sanglah dan RSUD Wangaya bulan hingga 2018. Hasil : Sebanyak pasien dari 47 pasien Penyakit Parkinson dengan rerata usia 61 – 70 tahun sebanyak dengan laki – laki sebanyak 34 orang (72,3%). Pasien dominan berobat ke RSUP Sanglah sebanyak 30 orang (63,8%) dengan pekerjaan terbanyak adalah petani/buruh sebanyak 13 orang (27,7%). Awitan penyakit rata – rata 1 – 5 tahun (39%). Penderita Penyakit Parkinson mengalami gangguan tidur sebanyak 24 orang (51,1%). Profil gangguan tidur dengan rerata kualitas tidur buruk 55,3%; mengalami latensi tidur 1x seminggu 40,4%. Simpulan : Penyakit Parkinson didominasi oleh pasien laki – laki dengan rerata usia 61 – 70 tahun dengan awitan peyakit rata – rata 1 – 5 tahun yang mengalami gangguan tidur. Gangguan tidur yang banyak diemukan berupa terjadinya latensi tidur sebanyak 1 kali seminggu. Kata kunci : Penyakit Parkinson, gangguan tidur, karakteristik Abstract Background: The number of cases of Parkinson's disease in Indonesia is quite high with prevalence per population and incidence per population. Sleep disorders can be found in people with Parkinson's Disease. Demographic data can be used as a clinician's consideration in diagnosing and determining optimal follow-up treatment. Objective: To determine the clinical characteristics of Parkinson's disease patients with sleep disorders in the Neurology Polyclinic at Sanglah Central Hospital (RSUP) and Sanglah Regional General Hospital (RSUD) Wangaya for the period of 2018. Research Methods: An observational descriptive study using a questionnaire for Parkinson's disease patients seeking treatment at the Neurology Polyclinic at Sanglah Hospital and Wangaya District Hospital until 2018. Results: A total of 47 patients from Parkinson's disease with a mean age of 61-70 years were 34 men (72.3%). Most of the patients went to Sanglah Hospital as many as 30 people (63.8%) with the most work being farmers as many as 13 people (27.7%). The average onset of disease is 1 - 5 years (39%). Patients with Parkinson's Disease experience sleep disorders as many as 24 people (51.1%). Profile of sleep disorders with an average of poor sleep quality 55.3%; sleep latency 1 time a week 40.4%. Conclusion: Parkinson's disease is dominated by male patients with an average age of 61 - 70 years with
抽象背景:印度尼西亚帕金森氏症发病率很高，每个公民普遍存在和事件发生率很高。帕金森患者患有多发性硬化症。人口统计数据可以作为诊断和确定最佳持续治疗的临床考虑因素。目的:了解帕金森病病人的临床特征与神经在诊所公立医院睡眠障碍中心Sanglah (RSUP) Sanglah和公立医院(县)地区Wangaya 2018月时期。研究方法:观察描述性研究使用神经治疗帕金森病的病人在诊所问卷RSUP Sanglah Wangaya月县,直到2018年。结果:帕金森病病人从47多与61—70岁平均多达多达34人(72,3%)——人。病人看病去主导RSUP Sanglah多达30人(63,8%)最多的工作是农民-工人多达13人(27,7%)。平均1 - 5年的病例(39%)。帕金森氏症患者有多达24人(51,1%)睡眠障碍。睡眠障碍的资料质量平均睡眠不好55,3%;睡眠有延时×40,4%一周。结:帕金森病由病人主导——人与平均61—70岁awitan peyakit——平1—5年经历的睡眠障碍。睡眠障碍发生的许多diemukan睡一周1次延时。关键词:帕金森氏症,睡眠障碍,特征抽象背景:帕金森症疾病的案子当家》在印尼是弄得高和prevalence每人口和incidence人口。睡眠障碍可以发现人们在一起帕金森症疾病。Demographic数据可以成为过去美国在diagnosing clinician的对价和determining follow-up最佳治疗。客观临床characteristics》:说到个重大《Neurology帕金森症和睡眠障碍疾病病人Polyclinic at Sanglah中央医院(RSUP)和综合医院(县)地区Sanglah Wangaya为2018年之期。研究方法:An材料。用a questionnaire descriptive study for帕金森病病人寻找治疗at the Neurology Polyclinic at Sanglah医院和Wangaya区医院,直到2018年。Results: A总共47从帕金森病人的疾病和A的意思是时代61-70 72年在34分钟(3%)。美国大多数病人去医院Sanglah之多美国30。63人(8%)和《美国头号工作存在许多美国农民27 13人(7%)。疾病之平均首字母是1 - 5年(39%)。美国和帕金森症病人疾病体验睡眠障碍多美国51 24人(1%)。低调的睡眠障碍与可怜的平均的睡眠质量55。3%;睡眠latency 1时代甲周40 4%。历史性:帕金森症疾病控制是由男性病人的平均年龄和疾病的首字母在61 - 70年的平均15年世卫组织体验睡眠障碍。睡眠障碍是经常发现in the form of美国美国睡眠latency多1时间a week。安装:帕金森症疾病睡眠障碍,characteristics
{"title":"Profil Gangguan Tidur Penderita Parkinson di Rumah Sakit Rujukan di Kota Denpasar Tahun 2018","authors":"Winda Haeriyoko, Purwa Samatra, S. Trisnawati, I. Budiarsa, A.A.Ayu Suryapraba","doi":"10.29342/cnj.v3i1.111","DOIUrl":"https://doi.org/10.29342/cnj.v3i1.111","url":null,"abstract":"Abstrak \u0000Latar Belakang : Jumlah kasus Penyakit Parkinson di Indonesia cukup tinggi dengan prevalensi per penduduk dan insiden per penduduk. Gangguan tidur ddapatkan pada penderita Penyakit Parkinson. Data demografi dapat digunakan sebagai pertimbangan klinisi dalam mendiagnosis serta menentukan penanganan lanjutan yang optimal. \u0000Tujuan : Mengetahui karakteristik klinis pasien Penyakit Parkinson dengan gangguan tidur di Poliklinik Saraf Rumah Sakit Umum Pusat Sanglah (RSUP) Sanglah dan Rumah Sakit Umum Daerah (RSUD) Wangaya periode bulan 2018. \u0000Metode Penelitian : Penelitian deskriptif observasional menggunakan kuesioner pasien Penyakit Parkinson yang berobat di Poliklinik Saraf RSUP Sanglah dan RSUD Wangaya bulan hingga 2018. \u0000Hasil : Sebanyak pasien dari 47 pasien Penyakit Parkinson dengan rerata usia 61 – 70 tahun sebanyak dengan laki – laki sebanyak 34 orang (72,3%). Pasien dominan berobat ke RSUP Sanglah sebanyak 30 orang (63,8%) dengan pekerjaan terbanyak adalah petani/buruh sebanyak 13 orang (27,7%). Awitan penyakit rata – rata 1 – 5 tahun (39%). Penderita Penyakit Parkinson mengalami gangguan tidur sebanyak 24 orang (51,1%). Profil gangguan tidur dengan rerata kualitas tidur buruk 55,3%; mengalami latensi tidur 1x seminggu 40,4%. \u0000Simpulan : Penyakit Parkinson didominasi oleh pasien laki – laki dengan rerata usia 61 – 70 tahun dengan awitan peyakit rata – rata 1 – 5 tahun yang mengalami gangguan tidur. Gangguan tidur yang banyak diemukan berupa terjadinya latensi tidur sebanyak 1 kali seminggu. \u0000Kata kunci : Penyakit Parkinson, gangguan tidur, karakteristik \u0000 \u0000Abstract \u0000Background: The number of cases of Parkinson's disease in Indonesia is quite high with prevalence per population and incidence per population. Sleep disorders can be found in people with Parkinson's Disease. Demographic data can be used as a clinician's consideration in diagnosing and determining optimal follow-up treatment. \u0000Objective: To determine the clinical characteristics of Parkinson's disease patients with sleep disorders in the Neurology Polyclinic at Sanglah Central Hospital (RSUP) and Sanglah Regional General Hospital (RSUD) Wangaya for the period of 2018. \u0000Research Methods: An observational descriptive study using a questionnaire for Parkinson's disease patients seeking treatment at the Neurology Polyclinic at Sanglah Hospital and Wangaya District Hospital until 2018. \u0000Results: A total of 47 patients from Parkinson's disease with a mean age of 61-70 years were 34 men (72.3%). Most of the patients went to Sanglah Hospital as many as 30 people (63.8%) with the most work being farmers as many as 13 people (27.7%). The average onset of disease is 1 - 5 years (39%). Patients with Parkinson's Disease experience sleep disorders as many as 24 people (51.1%). Profile of sleep disorders with an average of poor sleep quality 55.3%; sleep latency 1 time a week 40.4%. \u0000Conclusion: Parkinson's disease is dominated by male patients with an average age of 61 - 70 years with ","PeriodicalId":339514,"journal":{"name":"Callosum Neurology","volume":"5 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2020-01-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"128420780","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Michael Poryono, I. Sumada, Ni Ketut Candra Wiratmi
Background: Tolosa Hunt syndrome (THS) is an uncommon case defined as unilateral orbital or periorbital pain associated with weakness of one or more of the third, fourth and/or sixth cranial nerves, the etiology is still unknown. Pain and paresis Tolosa-Hunt syndrome is cured when adequately treated with corticosteroids Case: We present a case of young female patient with reccurent painful opthalmoplegia since 2 years ago. She also had ptosis and protopsis on her left eye. Contrast Ct scan showed thick lateral rectus muscle and superior rectus muscle. The diagnosis of Tolosa Hunt Syndrome was made according to clinical features, contrast Ct scan of the head and a positive response to corticosteroid treatment. Conclusions: Tolosa Hunt Syndrome is a diagnosis of exclusion. Clinical Features, Neuroimaging, and positive response to corticosteroid treatment are very helpful in determining the diagnosis. Patient must be told that the disease can recur.
{"title":"Tolosa Hunt Syndrome","authors":"Michael Poryono, I. Sumada, Ni Ketut Candra Wiratmi","doi":"10.29342/cnj.v3i1.58","DOIUrl":"https://doi.org/10.29342/cnj.v3i1.58","url":null,"abstract":"Background: Tolosa Hunt syndrome (THS) is an uncommon case defined as unilateral orbital or periorbital pain associated with weakness of one or more of the third, fourth and/or sixth cranial nerves, the etiology is still unknown. Pain and paresis Tolosa-Hunt syndrome is cured when adequately treated with corticosteroids \u0000Case: We present a case of young female patient with reccurent painful opthalmoplegia since 2 years ago. She also had ptosis and protopsis on her left eye. Contrast Ct scan showed thick lateral rectus muscle and superior rectus muscle. The diagnosis of Tolosa Hunt Syndrome was made according to clinical features, contrast Ct scan of the head and a positive response to corticosteroid treatment. \u0000Conclusions: Tolosa Hunt Syndrome is a diagnosis of exclusion. Clinical Features, Neuroimaging, and positive response to corticosteroid treatment are very helpful in determining the diagnosis. Patient must be told that the disease can recur.","PeriodicalId":339514,"journal":{"name":"Callosum Neurology","volume":"85 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2020-01-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"127866440","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
京公网安备 11010802042870号
京ICP备2023020795号-1
扫码关注我们
求助内容：
应助结果提醒方式：