Medical Veritas: The Journal of Medical Truth最新文献

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Shoulder Dystocia (SD) and Brachial Plexus Palsy (BPP): cause and prevention 肩难产(SD)和臂丛神经麻痹(BPP):原因和预防

Medical Veritas: The Journal of Medical Truth

Pub Date : 2005-11-01 DOI: 10.1588/MEDVER.2005.02.00073

G. Morley

The current definition of shoulder dystocia is inconsistent. The American College of Obstetricians and Gynecologists’ (ACOG’s) Practice Bulletin Number 40 on shoulder dystocia has two pseudo definitions: (1) failure of the shoulders to deliver spontaneously, putting both mother and fetus at risk for injury; and (2) failure of the delivery attendant to deliver the anterior shoulder by gentle downward traction, thus requiring additional obstetric maneuvers. Shoulder dystocia is caused by impaction of the anterior shoulder behind the pubic symphysis. ACOG does not define any cause for brachial plexus palsy; however, moderate or severe downward head traction is implied to be injurious. A downward tilted pelvis is the major cause of anterior shoulder arrest; it is usually relieved or prevented by McRoberts’ position. The mother then spontaneously delivers the shoulders. This is postural shoulder arrest and is not true shoulder dystocia (SD). Failure of the mother in full hip flexion to deliver the shoulders spontaneously is true shoulder dystocia. Various maneuvers are available to correct this situation; all supplement physiological delivery forces and movements that do not increase traction on the brachial plexus. Resuscitation of the child must be pre-planned. Brachial plexus injury is a traction injury caused by pulling the head and neck down and away from the shoulder. Nerves may be bruised, stretched, torn or ruptured; nerve roots may be avulsed from the spinal cord. SD is largely preventable by delivering all patients in McRoberts’ or equivalent position. Brachial Plexus Palsy (BPP) is avoidable by never applying head traction at any delivery and using maneuvers to deliver the shoulders that avoid any tension on the brachial plexus. © Copyright 2005 Pearblossom Private School, Inc.–Printing Division. All rights reserved.

目前肩部难产的定义是不一致的。美国妇产科医师学会(ACOG)关于肩关节难产的第40号实践公报有两个假定义:(1)肩关节不能自然分娩，使母亲和胎儿都有受伤的危险;(2)接生人员不能通过轻柔的向下牵引来分娩前肩，因此需要额外的产科操作。肩难产是由耻骨联合后面的前肩嵌塞引起的。ACOG没有定义臂丛神经麻痹的任何原因;然而，中度或重度的头部向下牵引是有害的。骨盆向下倾斜是导致前肩停搏的主要原因;它通常被麦克罗伯茨的立场所缓解或阻止。然后母亲自然地生出肩膀。这是体位性肩停搏，不是真正的肩难产(SD)。产妇髋完全屈曲不能自发分娩是真正的肩部难产。可以使用各种机动来纠正这种情况;所有这些都补充了不增加臂丛牵引力的生理输送力和运动。孩子的复苏必须事先计划好。臂丛损伤是一种牵引性损伤，由头部和颈部向下拉并远离肩部引起。神经可能会受伤、拉伸、撕裂或破裂;神经根可能从脊髓上被撕脱。通过让所有患者都处于麦克罗伯茨或同等位置，SD在很大程度上是可以预防的。臂丛神经麻痹(BPP)是可以避免的，在任何分娩时都不要使用头部牵引，使用动作来分娩肩部，避免臂丛神经的紧张。©版权所有2005梨花私立学校有限公司-印刷部。版权所有。

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引用次数: 2

Detoxification: The link to life 排毒:与生命的联系

Medical Veritas: The Journal of Medical Truth

Pub Date : 2005-11-01 DOI: 10.1588/MEDVER.2005.02.00076

J. Hicks

As the American population becomes sicker, detoxification is a missing link to improved health. Metabolic dysfunctions and environmental insults present a challenge to detoxification. The prevalent practice in medicine today is to offer medications that address symptoms but not root causes. Symptoms may give ambiguous information as to cause of disease. Without addressing root causes and imbalances, further imbalances may be caused by the measures given to address symptoms. Our present “medication-for-symptoms medical system” is no longer acceptable. As it has been said that genes only create possible predisposing issues, and that if the “right” stimuli is never encountered then the problem is only theoretical, then, too, we must apply this idea of environmental insult to the medical treatments we employ: Any detoxification protocol must be undertaken with due diligence, ensuring that the patient's body will be able to handle the possible consequential effects of a given detoxifying agent's action upon other physiological pathways and systems. © Copyright 2005 Pearblossom Private School, Inc.–Publishing Division. All rights reserved.

随着美国人的病情加重，戒毒是改善健康的缺失环节。代谢功能障碍和环境损害对解毒提出了挑战。当今医学界普遍的做法是提供治标不治本的药物。症状可能提供有关病因的模糊信息。如果不解决根本原因和不平衡，为解决症状而采取的措施可能会造成进一步的不平衡。我们目前的“治标不治本”的医疗体系已经不能接受了。正如已经说过的那样，基因只会产生可能的易感问题，如果从来没有遇到“正确的”刺激，那么问题就只是理论上的，那么，我们也必须将这种环境侮辱的想法应用于我们所采用的医疗:任何解毒方案都必须尽职尽责地进行，确保患者的身体能够处理特定解毒剂对其他生理途径和系统的作用可能产生的后果。©版权所有2005梨花私立学校股份有限公司-出版部。版权所有。

引用次数: 0

Hypothesis--"The Connection" Subluxation and Autism 假设——半脱位与自闭症的“联系”

Medical Veritas: The Journal of Medical Truth

Pub Date : 2005-11-01 DOI: 10.1588/MEDVER.2005.02.00086

Matthew Gianforte

Today, autism rates are higher then ever before. Children all across the United States are suffering with the symptoms of autism, ADD, and many other “spectrum disorders.” While parents are doing everything they can to get their children the help that they need, very few are aware of the need for good spinal health. Spinal health is critical. The nerve center that controls the entire body is located in the spine. It is where interaction between the brain and all the organs takes place. If there is interference, referred to as subluxation, due to birth trauma, or some other injury, the brain cannot communicate with the body correctly. When a baby has undetected nerve interference, followed by toxins received through vaccines and the environment, the brain simply cannot communicate with the organs in the body to tell them what to do with those toxins. The buildup of these toxins exemplifies the varying symptoms of Autistic Spectrum Disorders, depending on the degree to which a child’s nervous system is functioning. © Copyright 2005 Pearblossom Private School, Inc.–Publishing Division. All rights reserved.

如今，自闭症的发病率比以往任何时候都要高。美国各地的儿童都患有自闭症、注意力缺陷多动症和许多其他“谱系障碍”的症状。当父母们尽其所能让他们的孩子得到他们需要的帮助时，很少有人意识到良好的脊柱健康的必要性。脊柱健康至关重要。控制整个身体的神经中枢位于脊柱。它是大脑和所有器官相互作用的地方。如果由于出生创伤或其他损伤而出现干扰，即半脱位，大脑就不能与身体正确沟通。当婴儿的神经受到未被察觉的干扰，接着是通过疫苗和环境受到的毒素，大脑根本无法与身体的器官沟通，告诉它们如何处理这些毒素。这些毒素的积累体现了自闭症谱系障碍的不同症状，这取决于儿童神经系统功能的程度。©版权所有2005梨花私立学校股份有限公司-出版部。版权所有。

引用次数: 0

Editorial: Reformulating the principles of Hippocrates 社论:重新制定希波克拉底的原则

Medical Veritas: The Journal of Medical Truth

Pub Date : 2005-11-01 DOI: 10.1588/MEDVER.2005.02.00091

M. Girard

引用次数: 1

Review Birth brain injury: etiology and prevention— Part I: Hypoxic-ischemic encephalopathy and cerebral palsy 出生脑损伤:病因学和预防-第一部分:缺氧缺血性脑病和脑瘫

Medical Veritas: The Journal of Medical Truth

Pub Date : 2005-11-01 DOI: 10.1588/MEDVER.2005.02.00064

G. Morley

Hypoxic-Ischemic Encephalopathy (HIE) is a birth brain injury that precedes the development of Cerebral Palsy (CP). Visualized on MRI scanning, the lesions of HIE are infarcts resulting from deficient generalized perfusion of brain tissue. Birth asphyxia/hypoxia is the widely assumed cause of this brain injury, however, HIE may occur without significant hypoxia. Hypovolemia/hypovolemic shock, is evident in the great majority of HIE newborns. Cord compression prior to birth pools fetal blood in the placenta and immediate cord clamping finalizes the hypovolemia. Heart failure (hypovolemic shock) follows. Retraction respiration—pulses of negative intra-thoracic pressure—pulls venous blood into the heart and, with hypotension, pulls arterial blood into the thoracic aorta from peripheral arteries. Circulation in the heart and lungs is maintained at the expense of perfusion of all peripheral organs, including the brain. Prolonged deficient perfusion of the actively metabolizing areas of the brain (basal ganglia and cerebral cortex) results in infarction.

缺氧缺血性脑病(HIE)是一种发生在脑瘫(CP)之前的先天性脑损伤。在MRI扫描上，HIE的病变是由于脑组织全面灌注不足引起的梗死。出生窒息/缺氧被广泛认为是这种脑损伤的原因，然而，HIE可能在没有明显缺氧的情况下发生。低血容量/低血容量性休克，在绝大多数HIE新生儿中是明显的。分娩前脐带压迫使胎儿血液聚集在胎盘中，立即夹紧脐带最终导致低血容量。随后是心力衰竭(低血容量性休克)。收缩呼吸——胸腔内负压脉冲——将静脉血拉入心脏，在低血压时，将动脉血从外周动脉拉入胸主动脉。心脏和肺部的循环是以牺牲包括大脑在内的所有周围器官的灌注为代价的。大脑活跃代谢区域(基底节区和大脑皮层)长期灌注不足导致梗死。

引用次数: 3

Critical assessment of CDC's "Questions and answers: Thimerosal-containing influenza vaccine" 对疾控中心“问答:含硫柳汞流感疫苗”的批判性评估

Medical Veritas: The Journal of Medical Truth

Pub Date : 2005-11-01 DOI: 10.1588/MEDVER.2005.02.00090

P. King

The review that follows is a critical assessment of the information published by the Centers for Disease Control and Prevention (CDC) entitled, Questions & Answers: Thimerosal-containing influenza vaccine, published at http://www.cdc.gov/flu/about/qa/thimerosal .htm on September 22, 2005, (and included in Appendix C) which I visited as a part of my research in this area on September 25, 2005. In general, to clearly differentiate between my assessment comments and those of the author, the author’s printed statements are quoted in a bold or regular Arial font followed by this reviewer’s remarks in a Times New Roman font. Should anyone find any factual misrepresentations in this reviewer’s remarks, then this reviewer requests that the factual error along with the scientifically sound and appropriate facts that prove your point to this reviewer so that this reviewer can learn from you, incorporate that new knowledge into his understanding, and, where indicated, correct the draft. © Copyright 2005 Pearblossom Private School, Inc.–Publishing Division. All rights reserved.

接下来的审查是对疾病控制和预防中心(CDC)发表的题为“问答:含硫柳汞的流感疫苗”的信息的重要评估，该信息于2005年9月22日在http://www.cdc.gov/flu/about/qa/thimerosal .htm上发表(并包括在附录C中)，我于2005年9月25日访问了该信息，作为我在该领域研究的一部分。一般来说，为了清楚地区分我的评估意见和作者的意见，作者的印刷声明用粗体或常规Arial字体引用，然后是评论家的评论，用Times New Roman字体。如果有人在审稿人的评论中发现任何事实上的错误陈述，那么审稿人就会要求审稿人将事实错误与科学合理的事实一起向审稿人证明你的观点，这样审稿人就可以向你学习，将新的知识纳入他的理解中，并在指出的地方纠正草稿。©版权所有2005梨花私立学校股份有限公司-出版部。版权所有。

引用次数: 0

Opening address: Power of Parents Rally, Oct. 8, 2005 at Washington, D.C. 2005年10月8日，华盛顿，“父母的力量”大会开幕致辞

Medical Veritas: The Journal of Medical Truth

Pub Date : 2005-11-01 DOI: 10.1588/MEDVER.2005.02.00078

B. Fisher

引用次数: 0

Analysis of causes that led to Eliza Jane Scovill's cardiac arrest and death 分析导致伊莉莎·简·斯科维尔心脏骤停和死亡的原因

Medical Veritas: The Journal of Medical Truth

Pub Date : 2005-11-01 DOI: 10.1588/MEDVER.2005.02.00075

M. Al-Bayati

My review of the medical evidence presented in this case and the pertinent medical literature clearly shows that Eliza Jane‘s death was not caused by Pneumocystis carinii Pneumonia (PCP) as alleged by the medical examiner, or any other type of pneumonia. Eliza Jane’s lungs did not show an inflammatory response to medically justify a diagnosis of pneumonia. Pneumonia is a term that refers to inflammation and consolidation of the pulmonary parenchyma. Eliza Jane’s death resulted from an acute allergic reaction to amoxicillin, which caused severe hypotension (due to the leakage of significant amount of fluid outside the blood vessels), shock, and cardiac arrest. The autopsy revealed that she had pericardial and pleural effusion and ascites. In addition, her organ weights (lungs, heart, liver, and kidneys) were increased significantly. The weight of Eliza Jane’s lungs, heart, liver, and kidneys were 184%, 131%, 121%, and 146% of the expected average normal weight for her age, respectively. Also, her liver was significantly enlarged and the hepatocytes show micro-and macrovesicular steatosis. Amoxicillin has been known to induce immune mediated toxic changes in the liver. Eliza Jane suffered from an upper respiratory tract infection for about three weeks prior to her death on May 16, 2005. My investigation indicates that her respiratory infection was probably caused by Human Parvovirus B19 (HPVB19) infection. HPVB19 has been known to cause upper respiratory tract infection, encephalitis, and aplastic anemia in children and adults. Eliza Jane had non-specific microscopic lesions in the brain consisting of microglia and multineucleated giant cells. These lesions could be caused by HPVB19.

我对本案中提供的医学证据和相关医学文献的审查清楚地表明，Eliza Jane的死亡不是由法医所说的卡氏肺囊虫肺炎(PCP)引起的，也不是任何其他类型的肺炎。简的肺部没有出现炎症反应，医学上不能诊断为肺炎。肺炎是指肺实质的炎症和实变。伊丽莎·简的死亡是由于对阿莫西林的急性过敏反应，导致严重低血压(由于血管外大量液体泄漏)、休克和心脏骤停。尸检显示她有心包、胸膜积液和腹水。此外，她的器官重量(肺、心、肝、肾)明显增加。Eliza Jane的肺、心、肝和肾的重量分别为预期正常体重的184%、131%、121%和146%。肝脏明显增大，肝细胞呈小泡性和大泡性脂肪变性。已知阿莫西林可诱导免疫介导的肝脏毒性变化。简在2005年5月16日去世前的大约三周时间里一直患有上呼吸道感染。我的调查表明，她的呼吸道感染可能是由人细小病毒B19 (HPVB19)感染引起的。已知HPVB19可引起儿童和成人的上呼吸道感染、脑炎和再生障碍性贫血。伊丽莎·简在大脑中有非特异性的显微镜病变，由小胶质细胞和多核巨细胞组成。这些病变可能是由HPVB19引起的。

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引用次数: 4

Reflections of brain-body functioning 脑-体功能的反映

Medical Veritas: The Journal of Medical Truth

Pub Date : 2005-11-01 DOI: 10.1588/MEDVER.2005.02.00074

Sally Goddard Blythe

The presence of primitive and postural reflexes at key stages in development provide reliable indicators of maturity in the functioning of the Central Nervous System. Primitive reflexes are tested routinely at birth and in the first few weeks of postnatal life but are not re-assessed at a later age as a matter of course. This paper examines the functions and effects of three primitive reflexes, explaining how abnormal primitive reflexes can predispose a child to allergy, anxiety, coordination problems and subsequent learning and behavioral challenges including inattention. © Copyright 2005 Pearblossom Private School, Inc.–Publishing Division. All rights reserved.

引用次数: 0

Interview with Dr. Mary Megson: diagnosing and treating developmentally delayed children 玛丽·梅格森医生访谈:诊断和治疗发育迟缓儿童

Medical Veritas: The Journal of Medical Truth

Pub Date : 2005-11-01 DOI: 10.1588/MEDVER.2005.02.00080

M. Megson, T. Small

Autism may be a disorder linked to the disruption of the G-alpha protein, affecting retinoid receptors in the brain. There are retinoid receptors in cells all over the body. They are in the nucleus of cells; many times a hormone or other messenger will give a signal to a receptor in the cell wall, which then conveys it through another protein called a G-protein; it is then carried out in the center of the cell. So they help translate, in genetic terms, the message that’s given to the cell at the edge of the cell—at the cell wall. Autism may be caused by inserting a G-alpha protein defect, the pertussis toxin found in the DTP vaccine, into genetically atrisk children. This toxin separates the G-alpha protein from retinoid receptors. Those most at risk report a family history of at least one parent with a pre-existing G-alpha protein defect, including night blindness. Natural Vitamin A may reconnect the retinoid receptors critical for vision, sensory perception, language processing and attention. Bethanechol mimics acetylcholine. It gives secretory function back to the gut in that it stimulates muscles around the pancreas and the gall bladder to get everything in the gastrointestinal tract like enzymes to improve food digestion. It gives normal movement of the gut wall, which helps in peristalsis especially if the individual is constipated. © Copyright 2005 Pearblossom Private School, Inc.–Publishing Division. All rights reserved.

自闭症可能是一种与g - α蛋白破坏有关的疾病，会影响大脑中的类视黄醛受体。全身的细胞中都有类视黄酮受体。它们在细胞核中;很多时候，激素或其他信使会向细胞壁的受体发出信号，然后受体通过另一种叫做g蛋白的蛋白质传递信号;然后在细胞中心进行。所以它们帮助翻译，从遗传角度来说，传递给细胞边缘细胞壁的信息。自闭症可能是由于将g - α蛋白缺陷，即百日咳疫苗中发现的百日咳毒素，植入有遗传风险的儿童体内而引起的。这种毒素将g - α蛋白与类视黄醛受体分离。那些最危险的人报告说，他们的家族史中至少有一位父母已经存在g - α蛋白缺陷，包括夜盲症。天然维生素A可以重新连接对视觉、感官知觉、语言处理和注意力至关重要的类视黄醛受体。乙二酚类似于乙酰胆碱。它将分泌功能还给肠道，因为它刺激胰腺和胆囊周围的肌肉，将酶等一切物质输送到胃肠道，以改善食物消化。它使肠壁正常运动，这有助于蠕动，特别是如果个人便秘。©版权所有2005梨花私立学校股份有限公司-出版部。版权所有。

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