Cyrus V. Edelson, S. Edelson, J. Edelson, G. Dooley
Urinothorax (UT) is an uncommon cause of pleural effusion where urine collects in the pleural cavity that is usually caused by trauma or urinary obstruction. Spontaneous bladder rupture is another rare condition that is commonly caused by underlying bladder wall pathology and exacerbated by binge alcohol consumption, malignancy, autoimmune disorders, radiation, and obstructive or neurogenic pathology. We present a case of urinothorax caused by spontaneous bladder rupture in an adult patient successfully recognized and treated.
{"title":"Urinothorax: A rare complication of spontaneous bladder rupture","authors":"Cyrus V. Edelson, S. Edelson, J. Edelson, G. Dooley","doi":"10.5430/crim.v6n4p16","DOIUrl":"https://doi.org/10.5430/crim.v6n4p16","url":null,"abstract":"Urinothorax (UT) is an uncommon cause of pleural effusion where urine collects in the pleural cavity that is usually caused by trauma or urinary obstruction. Spontaneous bladder rupture is another rare condition that is commonly caused by underlying bladder wall pathology and exacerbated by binge alcohol consumption, malignancy, autoimmune disorders, radiation, and obstructive or neurogenic pathology. We present a case of urinothorax caused by spontaneous bladder rupture in an adult patient successfully recognized and treated.","PeriodicalId":72533,"journal":{"name":"Case reports in internal medicine","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2019-09-28","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.5430/crim.v6n4p16","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"49642924","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Daniel C. Alcantar, F. Galeano, Christine C. Junia
Pylephlebitis is a rare complication associated with an intra-abdominal septic process in the portal venous system. It is defined as thrombophlebitis of the portal vein and is often reported in association with appendicitis and diverticulitis. We present a 64-yearold female who presented with fever, chills, myalgia, and loss of appetite. A computerized tomography (CT) chest/abdomen/pelvis was performed and the patient was found to have a low-density lesion within the left lobe of the liver suspicious for a hepatic abscess and a suspected left segmental plyephlebitis. The diagnosis of pylephlebitis can be challenging as there is a broad differential diagnosis to consider. When considering pylephlebitis, empiric antibiotic coverage for poly-microbial infection targeting both gram-negative aerobes and anaerobes should be initiated. Antimicrobial therapy is modified according to blood culture results and treatment can be extended for 4 to 6 weeks. To our knowledge, there are only a few cases identifying liver abscesses as an etiology for pylephlebitis. This case was atypical compared to other cases in that the diagnosis of pylephlebitiswas incidental.
{"title":"Pylephlebitis caused by a liver abscess","authors":"Daniel C. Alcantar, F. Galeano, Christine C. Junia","doi":"10.5430/crim.v6n4p13","DOIUrl":"https://doi.org/10.5430/crim.v6n4p13","url":null,"abstract":"Pylephlebitis is a rare complication associated with an intra-abdominal septic process in the portal venous system. It is defined as thrombophlebitis of the portal vein and is often reported in association with appendicitis and diverticulitis. We present a 64-yearold female who presented with fever, chills, myalgia, and loss of appetite. A computerized tomography (CT) chest/abdomen/pelvis was performed and the patient was found to have a low-density lesion within the left lobe of the liver suspicious for a hepatic abscess and a suspected left segmental plyephlebitis. The diagnosis of pylephlebitis can be challenging as there is a broad differential diagnosis to consider. When considering pylephlebitis, empiric antibiotic coverage for poly-microbial infection targeting both gram-negative aerobes and anaerobes should be initiated. Antimicrobial therapy is modified according to blood culture results and treatment can be extended for 4 to 6 weeks. To our knowledge, there are only a few cases identifying liver abscesses as an etiology for pylephlebitis. This case was atypical compared to other cases in that the diagnosis of pylephlebitiswas incidental.","PeriodicalId":72533,"journal":{"name":"Case reports in internal medicine","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2019-09-26","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.5430/crim.v6n4p13","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"43761983","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Convulsive syncope can be difficult to distinguish from seizures at times. Misdiagnosis can result in unnecessary, and usually unsuccessful, treatment with antiepileptic drugs. A detailed history taking and physical examination along with continuous cardiac monitoring can help in making the correct diagnosis. Our patient was eventually found to have viral myocarditis. Moredetailed studies for the role of noninvasive cardiac testing such as Cardiac MRI (CMR) and newer treatment modalities such as anti-thymocyte immunoglobulin are required for the management of Viral Myocarditis.
{"title":"Intractable stokes adams attack with myriad of ventricular arrythmias masquerading acute myocarditis","authors":"M. Kaur, Prema Bezwada, C. Ayala-Rodriguez","doi":"10.5430/crim.v6n4p9","DOIUrl":"https://doi.org/10.5430/crim.v6n4p9","url":null,"abstract":"Convulsive syncope can be difficult to distinguish from seizures at times. Misdiagnosis can result in unnecessary, and usually unsuccessful, treatment with antiepileptic drugs. A detailed history taking and physical examination along with continuous cardiac monitoring can help in making the correct diagnosis. Our patient was eventually found to have viral myocarditis. Moredetailed studies for the role of noninvasive cardiac testing such as Cardiac MRI (CMR) and newer treatment modalities such as anti-thymocyte immunoglobulin are required for the management of Viral Myocarditis.","PeriodicalId":72533,"journal":{"name":"Case reports in internal medicine","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2019-09-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.5430/crim.v6n4p9","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"41833421","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
G. Rwegerera, Munashe Rimbi, Vimbai Mudhina, M. Simone, Moranodi Sefo, Bofelo Segona
Dolutegravir associated hepatic failure has rarely been reported. Hepatic failure can occur either acutely or after few weeks as it happened in our patient. We report a case of 61 years old HIV-positive treatment naïve patient who started experiencing features of hepatic injury one month after starting Dolutegravir-based regimen. Patient presented late with overt hepatic failure. We emphasize the importance of close monitoring both at initiation and long-term so as identify patients with early hepatic injury and limit fatal outcomes which are potentially avoidable.
{"title":"Dolutegravir induced sub-acute hepatic failure in HIV positive treatment naïve man in Botswana","authors":"G. Rwegerera, Munashe Rimbi, Vimbai Mudhina, M. Simone, Moranodi Sefo, Bofelo Segona","doi":"10.5430/crim.v6n4p5","DOIUrl":"https://doi.org/10.5430/crim.v6n4p5","url":null,"abstract":"Dolutegravir associated hepatic failure has rarely been reported. Hepatic failure can occur either acutely or after few weeks as it happened in our patient. We report a case of 61 years old HIV-positive treatment naïve patient who started experiencing features of hepatic injury one month after starting Dolutegravir-based regimen. Patient presented late with overt hepatic failure. We emphasize the importance of close monitoring both at initiation and long-term so as identify patients with early hepatic injury and limit fatal outcomes which are potentially avoidable.","PeriodicalId":72533,"journal":{"name":"Case reports in internal medicine","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2019-09-24","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.5430/crim.v6n4p5","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"47420201","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
J. Lowenthal, Jack Xu, E. Pagan, Richard Tangel, D. Schaer, Theodore J. Maglione, A. Saluja
The manifestation of atrial flutter, particularly with 1:1 conduction, is rare in patients with ventricular preexcitation secondary to Wolff-Parkinson-White Syndrome (WPW). Very few cases have been reported in the literature. We present a 40-year old male with a history of untreated WPW who presented with severe chest pain and shortness of breath. He was found to have a rapid, regular, wide complex tachycardia. He underwent successful synchronized cardioversion, in which the patient converted tonormal sinus rhythm with classic WPW waveform characteristics, including a shortened PR interval and prolonged QRS complex with a slurred upstroke. Surprisingly, a subsequent electrophysiology study revealed atrial flutter, with bystander conduction of 1:1 atrial flutter being the most likely cause of the patient’s presenting symptoms, and a posteroseptal accessory pathway consistent with the diagnosis of WPW. While considerably rarer than ventricular tachycardia or AVRT, it is nevertheless important for clinicians to consider atrial flutter with 1:1 conduction as a potential diagnosis in patients with WPW presenting with wide complex tachycardia.
{"title":"A case of wide complex tachycardia in wolff-parkinson-white syndrome","authors":"J. Lowenthal, Jack Xu, E. Pagan, Richard Tangel, D. Schaer, Theodore J. Maglione, A. Saluja","doi":"10.5430/crim.v6n4p1","DOIUrl":"https://doi.org/10.5430/crim.v6n4p1","url":null,"abstract":"The manifestation of atrial flutter, particularly with 1:1 conduction, is rare in patients with ventricular preexcitation secondary to Wolff-Parkinson-White Syndrome (WPW). Very few cases have been reported in the literature. We present a 40-year old male with a history of untreated WPW who presented with severe chest pain and shortness of breath. He was found to have a rapid, regular, wide complex tachycardia. He underwent successful synchronized cardioversion, in which the patient converted tonormal sinus rhythm with classic WPW waveform characteristics, including a shortened PR interval and prolonged QRS complex with a slurred upstroke. Surprisingly, a subsequent electrophysiology study revealed atrial flutter, with bystander conduction of 1:1 atrial flutter being the most likely cause of the patient’s presenting symptoms, and a posteroseptal accessory pathway consistent with the diagnosis of WPW. While considerably rarer than ventricular tachycardia or AVRT, it is nevertheless important for clinicians to consider atrial flutter with 1:1 conduction as a potential diagnosis in patients with WPW presenting with wide complex tachycardia.","PeriodicalId":72533,"journal":{"name":"Case reports in internal medicine","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2019-09-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.5430/crim.v6n4p1","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"45277328","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Breast cancer is the most common malignancy in women. Metastatic disease is usually found in the bones, lungs, regional lymph nodes, liver, and brain. Metastases to the gastrointestinal tract occur in less than 1% of cases. We report an unusual case of metastatic invasive lobular carcinoma presenting as gastric outlet obstruction seven years after the initial diagnosis. To our knowledge, this is the first time that endoscopic ultrasound with fine-needle aspiration is used to establish a definite diagnosis of recurrent invasive lobular carcinoma to the gastrointestinal tract causing gastric outlet obstruction.
{"title":"Recurrent invasive lobular breast carcinoma presenting as gastric outlet obstruction","authors":"A. Contreras, A. Robles, A. Mendoza-Ladd","doi":"10.5430/crim.v6n3p26","DOIUrl":"https://doi.org/10.5430/crim.v6n3p26","url":null,"abstract":"Breast cancer is the most common malignancy in women. Metastatic disease is usually found in the bones, lungs, regional lymph nodes, liver, and brain. Metastases to the gastrointestinal tract occur in less than 1% of cases. We report an unusual case of metastatic invasive lobular carcinoma presenting as gastric outlet obstruction seven years after the initial diagnosis. To our knowledge, this is the first time that endoscopic ultrasound with fine-needle aspiration is used to establish a definite diagnosis of recurrent invasive lobular carcinoma to the gastrointestinal tract causing gastric outlet obstruction.","PeriodicalId":72533,"journal":{"name":"Case reports in internal medicine","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2019-08-28","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.5430/crim.v6n3p26","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"46317258","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Coccidioidomycosis is a disease endemic to the southwest United States, as well as Central and South America, and primarily causes pulmonary infection. The cutaneous findings associated with Coccidioides species often present as an exanthema or as dissemination from a primary pulmonary infection. Rarely do individuals present with primary cutaneous coccidioidomycosis due to direct inoculation of the skin by arthroconidia. We report a case in which an immunocompetent oil field worker presented with a severe, fungating nasal lesion, who with culture, serology, histopathology, and a convincing history of exposure revealed a diagnosis of primary cutaneous coccidioidomycosis.
{"title":"Primary cutaneous coccidioidomycosis in an immunocompetent oil field worker in West Texas","authors":"E. Christensen, M. Feldman","doi":"10.5430/CRIM.V6N3P21","DOIUrl":"https://doi.org/10.5430/CRIM.V6N3P21","url":null,"abstract":"Coccidioidomycosis is a disease endemic to the southwest United States, as well as Central and South America, and primarily causes pulmonary infection. The cutaneous findings associated with Coccidioides species often present as an exanthema or as dissemination from a primary pulmonary infection. Rarely do individuals present with primary cutaneous coccidioidomycosis due to direct inoculation of the skin by arthroconidia. We report a case in which an immunocompetent oil field worker presented with a severe, fungating nasal lesion, who with culture, serology, histopathology, and a convincing history of exposure revealed a diagnosis of primary cutaneous coccidioidomycosis.","PeriodicalId":72533,"journal":{"name":"Case reports in internal medicine","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2019-08-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.5430/CRIM.V6N3P21","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"46061173","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Tsung-Han Lin, Tien-Chi Huang, Wen-Hsien Lee, C. Chu, Ho-Ming Su, Tsung-Hsien Lin, P. Hsu
Thoracoacromial artery perforation is a rare complication after coronary angiography. Herein, we reported a 70-year-old male previous post coronary artery bypass surgery being admitted for chest discomfort. Coronary angiography from radial artery approach showed all patent bypassed grafts and the patient was sent back to the ordinary ward. However, enlarging mass over left chest wall as woman’s breast was noted suddenly and we immediately used sandbag and elastic adhesive tape to compress the mass. Emergent chest computed tomography showed a large hematoma at the left chest wall with active bleeding from pectoral branch of thoracoacromial artery. However, left chest wall mass gradually subsided after compression strategy. This patient reminds physicians thoracoacromial artery perforation manifesting as chest wall mass is a rare but possible complication after coronary angiography.
{"title":"Thoracoacromial artery bleeding manifests as an enlarging chest mass: A rare case report","authors":"Tsung-Han Lin, Tien-Chi Huang, Wen-Hsien Lee, C. Chu, Ho-Ming Su, Tsung-Hsien Lin, P. Hsu","doi":"10.5430/CRIM.V6N3P18","DOIUrl":"https://doi.org/10.5430/CRIM.V6N3P18","url":null,"abstract":"Thoracoacromial artery perforation is a rare complication after coronary angiography. Herein, we reported a 70-year-old male previous post coronary artery bypass surgery being admitted for chest discomfort. Coronary angiography from radial artery approach showed all patent bypassed grafts and the patient was sent back to the ordinary ward. However, enlarging mass over left chest wall as woman’s breast was noted suddenly and we immediately used sandbag and elastic adhesive tape to compress the mass. Emergent chest computed tomography showed a large hematoma at the left chest wall with active bleeding from pectoral branch of thoracoacromial artery. However, left chest wall mass gradually subsided after compression strategy. This patient reminds physicians thoracoacromial artery perforation manifesting as chest wall mass is a rare but possible complication after coronary angiography.","PeriodicalId":72533,"journal":{"name":"Case reports in internal medicine","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2019-08-15","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.5430/CRIM.V6N3P18","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"45603644","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Satoko Hijii, T. Kodaka, Takae Goka, Y. Aoyama, Hiroko Tsunemine, Takayuki Takahashi
Acquired von Willebrand (vW) syndrome in Waldenström’s macroglobulinemia (WM) should be differentially diagnosed from hyperviscosity syndrome of WM, which exhibits a bleeding tendency. We report a rare case of acquired vW syndrome secondary to WM. A 62-year-old woman was referred to our hospital because of extensive subcutaneous hemorrhage following a light hit to the left arm. Although the platelet count was normal, APTT was prolonged to 49.4 sec. Furthermore, the serum concentration of IgM was elevated to 7,796 mg/dL, which was revealed to be IgM-κ monoclonal protein, leading to a diagnosis of WM. On ophthalmofundoscopy, mild hemorrhage, but not retinal vein dilatation, was observed. Regarding the abnormal APTT value, we measured coagulation factors in the intrinsic arm, revealing reduced activities of vW factor and factor VIII of 11 and 18%, respectively. Furthermore, the amount of vW protein was decreased to 23%. Multimer analysis of vW factor demonstrated an abnormal pattern lacking high-molecular-weight- bands. Additional diagnosis of acquired vW syndrome secondary to WM was made. The APTT cross-mixing test showed a simple but not inhibitor-related decreasing pattern of vW factor, suggesting the absorption of this factor by abnormal lymphoplasmacytic cells. The patient was treated with bendamustine, leading to reduced IgM, improvement of the APTT value, and the normal multimer pattern of vW factor.
{"title":"Acquired von Willebrand syndrome secondary to Waldenström’s macroglobulinemia","authors":"Satoko Hijii, T. Kodaka, Takae Goka, Y. Aoyama, Hiroko Tsunemine, Takayuki Takahashi","doi":"10.5430/CRIM.V6N3P13","DOIUrl":"https://doi.org/10.5430/CRIM.V6N3P13","url":null,"abstract":"Acquired von Willebrand (vW) syndrome in Waldenström’s macroglobulinemia (WM) should be differentially diagnosed from hyperviscosity syndrome of WM, which exhibits a bleeding tendency. We report a rare case of acquired vW syndrome secondary to WM. A 62-year-old woman was referred to our hospital because of extensive subcutaneous hemorrhage following a light hit to the left arm. Although the platelet count was normal, APTT was prolonged to 49.4 sec. Furthermore, the serum concentration of IgM was elevated to 7,796 mg/dL, which was revealed to be IgM-κ monoclonal protein, leading to a diagnosis of WM. On ophthalmofundoscopy, mild hemorrhage, but not retinal vein dilatation, was observed. Regarding the abnormal APTT value, we measured coagulation factors in the intrinsic arm, revealing reduced activities of vW factor and factor VIII of 11 and 18%, respectively. Furthermore, the amount of vW protein was decreased to 23%. Multimer analysis of vW factor demonstrated an abnormal pattern lacking high-molecular-weight- bands. Additional diagnosis of acquired vW syndrome secondary to WM was made. The APTT cross-mixing test showed a simple but not inhibitor-related decreasing pattern of vW factor, suggesting the absorption of this factor by abnormal lymphoplasmacytic cells. The patient was treated with bendamustine, leading to reduced IgM, improvement of the APTT value, and the normal multimer pattern of vW factor.","PeriodicalId":72533,"journal":{"name":"Case reports in internal medicine","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2019-07-18","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.5430/CRIM.V6N3P13","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"48147286","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
J. Lowenthal, Jessica Kunadia, Vivek Bose, Jack Xu, T. Nahass, I. Iwata
We present a patient with mitochondrial myopathy who developed rhabdomyolysis following treatment with clindamycin. While clindamycin is not yet linked to drug-induced rhabdomyolysis in the literature, other drugs with mechanisms of action similar to clindamycin have been shown to damage human host mitochondria. Given this, we contend that clindamycin may also be capable of causing mitochondrial injury, and that while in otherwise healthy patients it may not produce any negative clinical outcome, it can precipitate rhabdomyolysis in certain patients whose mitochondria are already vulnerable.
{"title":"Clindamycin-Induced Rhabdomyolysis in the Setting of Mitochondrial Myopathy","authors":"J. Lowenthal, Jessica Kunadia, Vivek Bose, Jack Xu, T. Nahass, I. Iwata","doi":"10.5430/CRIM.V6N3P10","DOIUrl":"https://doi.org/10.5430/CRIM.V6N3P10","url":null,"abstract":"We present a patient with mitochondrial myopathy who developed rhabdomyolysis following treatment with clindamycin. While clindamycin is not yet linked to drug-induced rhabdomyolysis in the literature, other drugs with mechanisms of action similar to clindamycin have been shown to damage human host mitochondria. Given this, we contend that clindamycin may also be capable of causing mitochondrial injury, and that while in otherwise healthy patients it may not produce any negative clinical outcome, it can precipitate rhabdomyolysis in certain patients whose mitochondria are already vulnerable.","PeriodicalId":72533,"journal":{"name":"Case reports in internal medicine","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2019-07-15","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.5430/CRIM.V6N3P10","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"46105814","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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