Pub Date : 2022-01-07DOI: 10.1079/cabicompendium.96368
D. Hamblin, J. Marchand
This datasheet on parathion poisoning covers Identity.
这份关于对硫磷中毒的数据表包括身份。
{"title":"Parathion poisoning.","authors":"D. Hamblin, J. Marchand","doi":"10.1079/cabicompendium.96368","DOIUrl":"https://doi.org/10.1079/cabicompendium.96368","url":null,"abstract":"This datasheet on parathion poisoning covers Identity.","PeriodicalId":7734,"journal":{"name":"American practitioner and digest of treatment","volume":"4 1","pages":"1-12"},"PeriodicalIF":0.0,"publicationDate":"2022-01-07","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"74216149","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Ulcer.","authors":"A. Bock, J. Littlefield","doi":"10.32388/gtxsts","DOIUrl":"https://doi.org/10.32388/gtxsts","url":null,"abstract":"","PeriodicalId":7734,"journal":{"name":"American practitioner and digest of treatment","volume":"21 1","pages":"637-42"},"PeriodicalIF":0.0,"publicationDate":"2020-12-07","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"84860677","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
An autosomal recessive inherited metabolic disorder caused by mutations in the SLC3A1 and SLC7A9 genes. It is characterized by deficient re-absorption of cystine in the proximal tubules of the kidney. It results in the formation of stones in the kidney, ureter, and urinary bladder.
{"title":"Cystinuria.","authors":"S. Ezell","doi":"10.32388/t6zheh","DOIUrl":"https://doi.org/10.32388/t6zheh","url":null,"abstract":"An autosomal recessive inherited metabolic disorder caused by mutations in the SLC3A1 and SLC7A9 genes. It is characterized by deficient re-absorption of cystine in the proximal tubules of the kidney. It results in the formation of stones in the kidney, ureter, and urinary bladder.","PeriodicalId":7734,"journal":{"name":"American practitioner and digest of treatment","volume":"229 1","pages":"748"},"PeriodicalIF":0.0,"publicationDate":"2020-02-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"76101250","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Typhoid.","authors":"D. Farnsworth, R. Bishop","doi":"10.32388/y96q7b","DOIUrl":"https://doi.org/10.32388/y96q7b","url":null,"abstract":"","PeriodicalId":7734,"journal":{"name":"American practitioner and digest of treatment","volume":"7 1","pages":"81-5"},"PeriodicalIF":0.0,"publicationDate":"2020-02-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"87639415","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Murine typhus, flea-borne typhus, and shop fever are other names used for this disease. Endemic typhus fever is caused by bacteria called Rickettsia typhi or another bacteria called Rickettsia felis. Endemic typhus is not directly spread from person to person. People become infected when they come into contact with fleas infected with the bacteria that cause endemic typhus fever. Endemic typhus differs from epidemic or louse-borne typhus, which does not usually occur in this country.
{"title":"Endemic typhus fever.","authors":"J. Chesnut","doi":"10.32388/ahv8tr","DOIUrl":"https://doi.org/10.32388/ahv8tr","url":null,"abstract":"Murine typhus, flea-borne typhus, and shop fever are other names used for this disease. Endemic typhus fever is caused by bacteria called Rickettsia typhi or another bacteria called Rickettsia felis. Endemic typhus is not directly spread from person to person. People become infected when they come into contact with fleas infected with the bacteria that cause endemic typhus fever. Endemic typhus differs from epidemic or louse-borne typhus, which does not usually occur in this country.","PeriodicalId":7734,"journal":{"name":"American practitioner and digest of treatment","volume":"4 1","pages":"141-5"},"PeriodicalIF":0.0,"publicationDate":"2020-02-08","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"73283935","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Orthostatic hypotension.","authors":"A. S. Yuskis, G. Griffith","doi":"10.32388/ql1kht","DOIUrl":"https://doi.org/10.32388/ql1kht","url":null,"abstract":"","PeriodicalId":7734,"journal":{"name":"American practitioner and digest of treatment","volume":"121 1","pages":"736-7"},"PeriodicalIF":0.0,"publicationDate":"2020-02-07","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"72964640","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
This review will discuss generalized myxedema as it develops in hypothyroidism. First, the precipitating conditions (thyroprivic trophoprivic + goitrous forms) and the clinical manifestations of thyroid hormone deficiency are presented. Pathobiochemical and pathophysiological factors that lead to the main manifestations include retention of fluid, retention of sodium and hyponatremia. In particular are primary and direct consequences of reduced thyroid hormone levels, and secondary or indirect consequences, such as cardiovascular and renal derangements. In hypothyroidism many biochemical disturbances result. Most important is the interstitial deposition of hydrophilic mucopolysaccharides, which in turn lead to fluid and Na retention and impairment of blood circulation and lymphatic drainage. Myxedema, therefore, is to a large extent a lymphatic edema. Hyponatremia is an indirect consequence of the lack of T3 and is directly caused by impaired renal Na reabsorption. Renal Na,K-ATPase is reduced in specific segments. The often discussed role of inappropriate elevation of circulating ADH does not seem to be a key factor in myxedema. Impaired capacity of renal water excretion is caused by reduced GFR. We discuss the time dependent development of the derangement of different organ systems, and include recently published biochemical results, according to which the lack of T3 interferes not only with the metabolism of numerous compounds of the interstitial matrix, but also with cell surface proteins and intracellular proteins of microfilaments. Finally, we refer briefly to pretibial myxedema in states of hyperthyroidism, that is, infiltrative dermopathy in Graves' disease, which is caused by poorly understood autoimmune processes.
这篇综述将讨论在甲状腺功能减退症中发生的全面性黏液性水肿。首先介绍了甲状腺激素缺乏症的发病条件(甲状腺营养不良+甲状腺肿形式)和临床表现。导致其主要表现为液体潴留、钠潴留和低钠血症的病理生化和病理生理因素。特别是甲状腺激素水平降低的主要和直接后果，以及继发性或间接后果，如心血管和肾脏紊乱。甲状腺功能减退导致许多生化紊乱。最重要的是亲水性粘多糖的间质沉积，这反过来导致液体和钠潴留，损害血液循环和淋巴引流。因此，黏液性水肿在很大程度上是淋巴水肿。低钠血症是T3缺乏的间接后果，是由肾钠重吸收受损直接引起的。肾Na, k - atp酶在特定节段减少。经常讨论的循环ADH不适当升高的作用似乎不是黏液性水肿的关键因素。肾水排泄能力受损是由GFR降低引起的。我们讨论了不同器官系统紊乱的时间依赖性发展，并包括最近发表的生化结果，根据这些结果，T3的缺乏不仅会干扰间质基质中许多化合物的代谢，还会干扰细胞表面蛋白和微丝细胞内蛋白。最后，我们简要地提到甲状腺功能亢进状态下的胫前黏液水肿，即Graves病的浸润性皮肤病，这是由自身免疫过程引起的。
{"title":"Myxedema.","authors":"G. Fitzhugh, L. Cecil","doi":"10.32388/mnkznb","DOIUrl":"https://doi.org/10.32388/mnkznb","url":null,"abstract":"This review will discuss generalized myxedema as it develops in hypothyroidism. First, the precipitating conditions (thyroprivic trophoprivic + goitrous forms) and the clinical manifestations of thyroid hormone deficiency are presented. Pathobiochemical and pathophysiological factors that lead to the main manifestations include retention of fluid, retention of sodium and hyponatremia. In particular are primary and direct consequences of reduced thyroid hormone levels, and secondary or indirect consequences, such as cardiovascular and renal derangements. In hypothyroidism many biochemical disturbances result. Most important is the interstitial deposition of hydrophilic mucopolysaccharides, which in turn lead to fluid and Na retention and impairment of blood circulation and lymphatic drainage. Myxedema, therefore, is to a large extent a lymphatic edema. Hyponatremia is an indirect consequence of the lack of T3 and is directly caused by impaired renal Na reabsorption. Renal Na,K-ATPase is reduced in specific segments. The often discussed role of inappropriate elevation of circulating ADH does not seem to be a key factor in myxedema. Impaired capacity of renal water excretion is caused by reduced GFR. We discuss the time dependent development of the derangement of different organ systems, and include recently published biochemical results, according to which the lack of T3 interferes not only with the metabolism of numerous compounds of the interstitial matrix, but also with cell surface proteins and intracellular proteins of microfilaments. Finally, we refer briefly to pretibial myxedema in states of hyperthyroidism, that is, infiltrative dermopathy in Graves' disease, which is caused by poorly understood autoimmune processes.","PeriodicalId":7734,"journal":{"name":"American practitioner and digest of treatment","volume":"27 1","pages":"237-9"},"PeriodicalIF":0.0,"publicationDate":"2020-02-07","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"91093649","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
B. Barikbin, A. Ayatollahi, Somayeh Hejazi, Hamid Reza Abaei, Somayeh Hejazi
The answer is that the Intense Pulsed Light (IPL) was used for removing this telangiectasia. Our system is a fourth generation of IPL technology that is a long pulse system. The IPL used in our case has a wavelength of 572 nm and pulse duration of 55 milliseconds. The energy used for this patient was 36J/cm2, and the procedure was performed in 3 consecutive pulses. After one treatment session, the telangiectasia faded away nearly completely. Generally, telangiectasias is classified into four types: linear, arborizing, spider and punctiform, or papular. Linear and arborizing telangiectasias with the size of 0.1-1.0 mm in diameter, frequently occur on the face, particularly the nose, cheeks and the chin. Factors such as chronic sun exposure, hormones (in particular estrogen), pregnancy, physical stress and (rarely) genetic disorders are among the causes of telangiectasia (1). Please cite this article as follows: Barikbin B, Ayatollahi A, Hejazi S, Abaei HR. What is your diagnosis? J Lasers Med Sci. 2011;2 (3):129-30
答案是使用强脉冲光(IPL)去除毛细血管扩张。我们的系统是第四代IPL技术，是一个长脉冲系统。本案例中使用的IPL波长为572纳米，脉冲持续时间为55毫秒。该患者使用的能量为36J/cm2，手术以3个连续脉冲进行。一次治疗后，毛细血管扩张几乎完全消失。一般来说，毛细血管扩张分为四种类型:线状、树状、蜘蛛状和点状或丘疹状。线状和树状毛细血管扩张，直径0.1-1.0 mm，常见于面部，尤其是鼻子、脸颊和下巴。长期日晒、激素(尤其是雌激素)、怀孕、身体压力和(很少)遗传疾病等因素都是毛细血管扩张的原因(1)。请引用以下文章:Barikbin B, Ayatollahi A, Hejazi S, Abaei HR。你的诊断是什么?激光医学科学，2011;2 (3):129-30
{"title":"WHAT'S your diagnosis?","authors":"B. Barikbin, A. Ayatollahi, Somayeh Hejazi, Hamid Reza Abaei, Somayeh Hejazi","doi":"10.22037/2010.V2I3.2298","DOIUrl":"https://doi.org/10.22037/2010.V2I3.2298","url":null,"abstract":"The answer is that the Intense Pulsed Light (IPL) was used for removing this telangiectasia. Our system is a fourth generation of IPL technology that is a long pulse system. The IPL used in our case has a wavelength of 572 nm and pulse duration of 55 milliseconds. The energy used for this patient was 36J/cm2, and the procedure was performed in 3 consecutive pulses. After one treatment session, the telangiectasia faded away nearly completely. Generally, telangiectasias is classified into four types: linear, arborizing, spider and punctiform, or papular. Linear and arborizing telangiectasias with the size of 0.1-1.0 mm in diameter, frequently occur on the face, particularly the nose, cheeks and the chin. Factors such as chronic sun exposure, hormones (in particular estrogen), pregnancy, physical stress and (rarely) genetic disorders are among the causes of telangiectasia (1). Please cite this article as follows: Barikbin B, Ayatollahi A, Hejazi S, Abaei HR. What is your diagnosis? J Lasers Med Sci. 2011;2 (3):129-30","PeriodicalId":7734,"journal":{"name":"American practitioner and digest of treatment","volume":"62 1","pages":"47-8"},"PeriodicalIF":0.0,"publicationDate":"2011-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"73031054","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 1966-09-01DOI: 10.1097/00000441-196609000-00030
G. Harrell, J. Aikawa, W. Kelsey
{"title":"Rocky Mountain spotted fever.","authors":"G. Harrell, J. Aikawa, W. Kelsey","doi":"10.1097/00000441-196609000-00030","DOIUrl":"https://doi.org/10.1097/00000441-196609000-00030","url":null,"abstract":"","PeriodicalId":7734,"journal":{"name":"American practitioner and digest of treatment","volume":"22 1","pages":"425-35"},"PeriodicalIF":0.0,"publicationDate":"1966-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"75124534","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Social nutrition and malnutrition.","authors":"L L HAVENS","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":7734,"journal":{"name":"American practitioner and digest of treatment","volume":"13 ","pages":"570-2"},"PeriodicalIF":0.0,"publicationDate":"1962-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"23496627","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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