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53 Cytoskeleton reorganization induces the uPA gene VIA the Ras/ERK signaling pathway 53细胞骨架重组通过Ras/ERK信号通路诱导uPA基因
Fibrinolysis
Pub Date : 1996-06-01 DOI: 10.1016/S0268-9499(96)80141-X
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引用次数: 0
32 Clearance studies of PAI-1 protease complexes suggest that PAI-1 contains a cryptic LRP binding site 32对PAI-1蛋白酶复合物的清除研究表明，PAI-1含有一个隐性LRP结合位点
Fibrinolysis
Pub Date : 1996-06-01 DOI: 10.1016/S0268-9499(96)80120-2
{"title":"32 Clearance studies of PAI-1 protease complexes suggest that PAI-1 contains a cryptic LRP binding site","authors":"","doi":"10.1016/S0268-9499(96)80120-2","DOIUrl":"https://doi.org/10.1016/S0268-9499(96)80120-2","url":null,"abstract":"","PeriodicalId":84750,"journal":{"name":"Fibrinolysis","volume":"10 ","pages":"Page 11"},"PeriodicalIF":0.0,"publicationDate":"1996-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/S0268-9499(96)80120-2","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"71716026","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
48 Correlation of cardiac valvular vegetations with thromboembolism and plasma D-dimer levels — A prospective echocardiographic study of 200 cancer patients 心脏瓣膜植被与血栓栓塞和血浆d -二聚体水平的相关性——一项对200名癌症患者的前瞻性超声心动图研究
Fibrinolysis
Pub Date : 1996-06-01 DOI: 10.1016/S0268-9499(96)80136-6
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引用次数: 2
43 Basic Fibroblast growth factor, urokinase-type plasminogen activator and urokinase-type plasminogen activator receptor in breast cancer cells 乳腺癌细胞中碱性成纤维细胞生长因子、尿激酶型纤溶酶原激活剂和尿激酶型纤溶酶原激活剂受体的研究
Fibrinolysis
Pub Date : 1996-06-01 DOI: 10.1016/S0268-9499(96)80131-7
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引用次数: 0
19 Functional analysis of the flexible loop of pro-urokinase (pro-UK) by site-directed mutagenesis 19 .位点定向诱变对前尿激酶(pro-UK)柔性环的功能分析
Fibrinolysis
Pub Date : 1996-06-01 DOI: 10.1016/S0268-9499(96)80107-X
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引用次数: 0
88 Ligand binding to the plasminogen kringle 2 domain: Characterization by 1H-NMR spectroscopy 配体与纤溶酶原kringle 2结构域的结合:1H-NMR表征
Fibrinolysis
Pub Date : 1996-06-01 DOI: 10.1016/S0268-9499(96)80175-5
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引用次数: 0
99 Impaired urokinase receptor (u-PAR)-mediated cellular fibrinolysis in patients suffering from paroxysmal nocturnal haemoglobinuria 阵发性夜间血红蛋白尿患者尿激酶受体(u-PAR)介导的细胞纤维蛋白溶解受损
Fibrinolysis
Pub Date : 1996-06-01 DOI: 10.1016/S0268-9499(96)80186-X
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引用次数: 0
101 Demonstration of a novel, ≈70K u-PA binding protein in platelet membranes 一种新型的，≈70K的u-PA结合蛋白在血小板膜中的演示
Fibrinolysis
Pub Date : 1996-06-01 DOI: 10.1016/S0268-9499(96)80188-3
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引用次数: 0
23 Effect of mutagenized NH2-terminal amino acid region on plasminogen activator activity of staphylokinase nh2末端氨基酸区诱变对葡萄激酶纤溶酶原激活剂活性的影响
Fibrinolysis
Pub Date : 1996-06-01 DOI: 10.1016/S0268-9499(96)80111-1
{"title":"23 Effect of mutagenized NH2-terminal amino acid region on plasminogen activator activity of staphylokinase","authors":"","doi":"10.1016/S0268-9499(96)80111-1","DOIUrl":"https://doi.org/10.1016/S0268-9499(96)80111-1","url":null,"abstract":"","PeriodicalId":84750,"journal":{"name":"Fibrinolysis","volume":"10 ","pages":"Page 8"},"PeriodicalIF":0.0,"publicationDate":"1996-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/S0268-9499(96)80111-1","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"71795827","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Coagulation and fibrinolysis in children with APC-resistance: a population study apc耐药儿童的凝血和纤溶:一项人群研究
Fibrinolysis
Pub Date : 1996-06-01 DOI: 10.1016/S0268-9499(96)80040-3
U. Nowak-Göttl , H. Vielhaber , R. Schneppenheim , H.G. Koch

Objective: resistance to activated protein C (APCR) has emerged as the most important hereditary cause of venous thromboembolism. To determine to what extent this relatively common gene mutation influences the overall haemostatic balance, coagulation and fibrinolysis were investigated in a population of APC resistant children (n=46) in comparison to an age-matched healthy control group (n=80).

Results: compared to the sex- and age-matched healthy control thrombomodulin (TM), F1+2, D-Dimer formation, t-PA, u-PA, PAP and PAI 1 were significantly increased in the APCR children. In addition, TM, D-Dimer, t-PA, u-PA, PAI 1 and PAP showed a significantly positive correlation to the amount of thrombin generated. No difference was found between children and patients who had suffered from vascular insults before this study was conducted.

Conclusions: children with APCR showed a combination of activated coagulation and fibrinolysis.

目的：对活化蛋白C（APCR）的耐药性已成为静脉血栓栓塞症最重要的遗传原因。为了确定这种相对常见的基因突变在多大程度上影响总体止血平衡，在APC耐药儿童群体（n=46）中与年龄匹配的健康对照组（n=80）进行了凝血和纤溶研究，PAP和PAI1在APCR患儿中显著升高。此外，TM、D-二聚体、t-PA、u-PA、PAI 1和PAP与凝血酶生成量呈显著正相关。在进行这项研究之前，儿童和患有血管损伤的患者之间没有发现差异。结论：APCR患儿表现为活化凝血和纤溶的结合。
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引用次数: 6
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