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Apheresis methods in COVID-19 era: What about Long COVID? 新冠肺炎时代的分离方法:长冠肺炎怎么办?
Pub Date : 2023-08-23 DOI: 10.46439/nephrology.4.014
Zikou Xanthi, Polychronidou Vasiliki, Derveni Vaia, Aloizos Stavros
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引用次数: 0
Effect of pentoxifylline on the dose of erythropoietin among hemodialysis patients: A double-blind randomized clinical trial 己酮茶碱对血液透析患者促红细胞生成素剂量的影响:一项双盲随机临床试验
Pub Date : 2023-05-09 DOI: 10.46439/nephrology.4.013
M. Pakfetrat, L. Malekmakan, Mohammad Hosein Rezazadeh, Pegah Aghajanzade
Introduction: It was suggested that pentoxifylline (PTX) might improve the response to recombinant human erythropoietin (rhEPO) in anemic hemodialysis (HD) patients. However, there is no considerable evidence for it. We aimed to evaluate the effect of PTX on anemia and prescription of rhEPO dose in HD patients.Methods: This double-blind randomized clinical trial study was conducted on 57 HD patients (54.1 ± 13.8 years old and 52.6% of them were women). Patients were randomly categorized into 2 groups (27 PTX cases and control group with 30 cases). Hemoglobin (Hb), erythrocyte sedimentation rate (ESR), C-reactive protein (CRP), and albumin (Alb) were measured before and after the study. Data were analyzed using SPSS, and p-value <0.05 was considered significant.Results: Hb levels increased significantly after treatment in both groups (p<0.002). Although the mean Hb change in the PTX group was more but not significant (p=0.195). rhEPO dose decreased significantly after treatment in the PTX groups (11000.0 ± 3140.0 IU vs. 9100.0 ± 3400.0 IU, p=0.018) compared to the control (11130.0 ± 3180.0 IU vs. 10870.0 ± 3900.0 IU, p= 0.690). CRP levels significantly reduced only in the PTX group (22.8 ± 15.3 vs. 16.5 ± 9.5, p=0.005). Also, a significant increase in Alb was observed only in the PTX (3.9 ± 0.4 vs. 4.1 ± 0.3, p=0.031).Conclusion: Using the PTX may reduce the required rhEPO dose, so it could be used in the anemia treatment in HD patients. Although, as a therapeutic strategy in HD patients with anemia it is controversial. Due to the limitations of the studies in this field, further studies with more sample size are recommended.
提示己酮茶碱(PTX)可改善贫血血液透析(HD)患者对重组人促红细胞生成素(rhEPO)的反应。然而,并没有足够的证据证明这一点。我们的目的是评估PTX对HD患者贫血的影响和rhEPO剂量的处方。方法:对57例HD患者(54.1±13.8岁,女性占52.6%)进行双盲随机临床试验研究。患者随机分为2组(PTX组27例,对照组30例)。研究前后分别测定血红蛋白(Hb)、红细胞沉降率(ESR)、c反应蛋白(CRP)、白蛋白(Alb)。数据采用SPSS统计分析,p值<0.05被认为是显著的。结果:两组治疗后Hb水平均显著升高(p<0.002)。PTX组平均Hb变化较多但不显著(p=0.195)。与对照组(11130.0±3180.0 IU对10870.0±3900 IU, p= 0.690)相比,PTX组治疗后rhEPO剂量显著降低(11000.0±3140.0 IU对9100.0±3400.0 IU, p=0.018)。CRP水平仅在PTX组显著降低(22.8±15.3比16.5±9.5,p=0.005)。此外,仅在PTX中观察到Alb的显著增加(3.9±0.4比4.1±0.3,p=0.031)。结论:PTX可降低rhEPO需药量,可用于HD患者贫血的治疗。然而,作为一种治疗HD伴贫血患者的策略仍存在争议。由于本领域研究的局限性,建议进一步开展样本量更大的研究。
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引用次数: 0
Role of vitamin D in diabetic nephropathy 维生素D在糖尿病肾病中的作用
Pub Date : 2022-12-31 DOI: 10.46439/nephrology.3.012
Lei He, Lin Zhou, Tian-ya Zhao, Alexander Temple Witherspoon, Long Ouyang
Diabetic nephropathy (DN) or Diabetic kidney disease (DKD) is one of the major microvascular complications of diabetes mellitus (DM). DN is observed in approximately 20–40% of diabetic patients. DN is also an important risk factor for DM patient’s death. Nowadays, DN has become the leading cause of chronic renal failure (CRF) in most countries without effective therapeutic methods. Recently, the renoprotective effects mediated by vitamin D have been evidenced. Currently available evidence showed that vitamin D is effective in reducing proteinuria in DN patients. A recent meta-analysis demonstrated the therapeutic effect of vitamin D, on urinary albumin excretion, in DN patients. This review summarized the multiple roles of vitamin D in mechanism of renopretective effect of vitamin D to explore much more and effective therapeutic methods for DN.
糖尿病肾病(DN)或糖尿病肾病(DKD)是糖尿病(DM)的主要微血管并发症之一。约20-40%的糖尿病患者存在DN。DN也是DM患者死亡的重要危险因素。目前,在大多数国家,肾病已成为慢性肾功能衰竭(CRF)的主要原因,但缺乏有效的治疗方法。近年来,维生素D介导的肾保护作用已得到证实。目前已有证据表明,维生素D可有效减少肾病患者的蛋白尿。最近的一项荟萃分析证实了维生素D对肾病患者尿白蛋白排泄的治疗作用。本文综述了维生素D在肾保护作用机制中的多重作用,以期探索更多有效的DN治疗方法。
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引用次数: 0
An update on proliferative glomerulonephritis with monoclonal immunoglobulin deposits in pediatric patients 儿童患者伴单克隆免疫球蛋白沉积的增生性肾小球肾炎的最新进展
Pub Date : 2022-12-31 DOI: 10.46439/nephrology.3.11
W. Hiser, Guo-lan Xing, X. Zhou
Proliferative glomerulonephritis with monoclonal immunoglobulin deposits (PGNMID) was first described by Nasr et al. in 2004 with a subsequent study composed of a larger patient cohort a few years later [1,2]. Although more commonly occurring in older adults, PGNMID has since been reported in a wide range of age groups including children [3-5]. PGNMID is categorized as a monoclonal gammopathy of renal significance (MGRS), and is additionally included in the recently expanded concept of monoclonal gammopathy of clinical significance (MGCS), which encompasses disorders of any organ system related to underlying plasma cell or B-cell clones [6]; however, most patients with PGNMID do not show evidence of a circulating monoclonal protein or clonal plasma cell proliferation, and monoclonal gammopathy has not been reported in any pediatric patients with the disease. Herein we briefly discuss the clinical and histopathologic features of PGNMID, as well as advances in our understanding of the pathogenesis and clinical course of the disease.
增殖性肾小球肾炎伴单克隆免疫球蛋白沉积(PGNMID)最早由Nasr等人于2004年报道,几年后又进行了一项由更大患者队列组成的研究[1,2]。虽然更常见于老年人,但PGNMID已在包括儿童在内的广泛年龄组中被报道[3-5]。PGNMID被归类为具有肾脏意义的单克隆γ病(MGRS),最近又被纳入了具有临床意义的单克隆γ病(MGCS)的概念,其中包括与潜在浆细胞或b细胞克隆相关的任何器官系统疾病[6];然而,大多数PGNMID患者没有表现出循环单克隆蛋白或克隆浆细胞增殖的证据,并且在任何患有该疾病的儿科患者中未报道单克隆伽玛病。在此,我们简要讨论PGNMID的临床和组织病理学特征,以及我们对该疾病的发病机制和临床病程的认识的进展。
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引用次数: 0
Evaluation of the pathophysiological mechanisms of salt sensitivity 盐敏感的病理生理机制评价
Pub Date : 2021-06-24 DOI: 10.46439/nephrology.2.010
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引用次数: 0
An overview of non-invasive methods for transcutaneous measurements of glomerular filtration 非侵入性经皮肾小球滤过测量方法综述
Pub Date : 2021-06-24 DOI: 10.46439/nephrology.2.008
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引用次数: 0
Antioxidant vitamins in diabetic kidney disease: The unsettled issues 抗氧化维生素在糖尿病肾病中的作用:尚未解决的问题
Pub Date : 2021-06-24 DOI: 10.46439/nephrology.2.006
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引用次数: 0
Host range of zoonotic hepatitis E viruses 人畜共患戊型肝炎病毒的宿主范围
Pub Date : 2021-06-24 DOI: 10.46439/nephrology.2.009
A. Gröner
HEV is a major cause of acute clinical hepatitis among humans throughout the world. Acute hepatitis E in humans in developing countries is caused by the HEV genotypes 1 (HEV-1; occurrence mainly in Asia) and 2 (HEV-2; occurrence mainly in Africa and Mexico) with a host range restricted to humans [3]. These genotypes are transmitted primarily by the faecal/oral route (primarily by contaminated water). The human associated as well as zoonotic genotypes of HEV are grouped into the species Orthohepevirus A, which includes a total of 8 genotypes, originating mostly from pig, wild boar, rabbit, and camel species. Orthohepevirus B consists of avian hepatitis E virus species, whereas Orthohepevirus C viruses were isolated from rodents (rats, voles, and shrew) and carnivores (such as ferrets, mink and foxes). HEV from bats are classed in the species Orthohepevirus D and fish-related HEV belongs to genus Piscihepevirus [4] (compare Table 1).
戊型肝炎是全世界人类急性临床肝炎的一个主要病因。发展中国家人类急性戊型肝炎是由HEV基因型1 (HEV-1;主要发生在亚洲)和2 (HEV-2;主要发生在非洲和墨西哥),宿主范围仅限于人类[3]。这些基因型主要通过粪/口途径(主要通过受污染的水)传播。HEV的人类相关基因型和人畜共患基因型归为正疱疹病毒A种,共包括8种基因型,主要源自猪、野猪、兔和骆驼。正疱疹病毒B由禽戊型肝炎病毒组成,而正疱疹病毒C是从啮齿动物(大鼠、田鼠和鼩鼱)和食肉动物(如雪貂、水貂和狐狸)中分离出来的。来自蝙蝠的HEV属于正疱疹病毒D种,与鱼相关的HEV属于鱼疱疹病毒属[4](比较表1)。
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引用次数: 0
The beneficial role of vitamin B12 in injury induced by ischemia/reperfusion: Beyond scavenging superoxide? 维生素B12在缺血/再灌注损伤中的有益作用:超越清除超氧化物?
Pub Date : 2021-01-01 DOI: 10.46439/nephrology.2.007
Feng Li

Vitamin B12 (B12) is required for cellular metabolism and DNA synthesis as a co-enzyme; it also possesses anti-reactive oxygen species (ROS) property as a superoxide scavenger. B12 deficiency has been implicated in multiple diseases such as megaloblastic anemia, and this disease can be effectively cured by supplementation of B12. Multiple studies suggest that B12 also benefits the conditions associated with excess ROS. Recently, we have reported that oral high dose B12 decreases superoxide level and renal injury induced by ischemia/reperfusion in mice. Here, we discuss potential mechanism(s) other than decreasing superoxide by which B12 executes its beneficial effects.

维生素B12是细胞代谢和DNA合成所必需的辅酶;它还具有抗活性氧(ROS)的特性,是一种超氧化物清除剂。B12缺乏与巨幼细胞性贫血等多种疾病有关,这种疾病可以通过补充B12有效治愈。多项研究表明,B12也有利于与过量活性氧相关的疾病。最近,我们报道了口服大剂量B12可以降低小鼠的超氧化物水平和缺血再灌注引起的肾损伤。在这里,我们讨论了B12发挥其有益作用的潜在机制,而不是减少超氧化物。
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引用次数: 2
Rickets in renal tubular acidosis: A clinical appraisal 佝偻病肾小管酸中毒:临床评价
Pub Date : 2020-12-31 DOI: 10.46439/nephrology.1.004
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引用次数: 2
期刊
Journal of experimental nephrology
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