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Cracked Tooth Syndrome and Strategies for Restoring 牙裂综合征及修复策略
Q2 Dentistry Pub Date : 2023-09-22 DOI: 10.1007/s40496-023-00352-1
Bora Korkut, Ezgi Tüter Bayraktar, Dilek Tağtekin, Hakan Çolak, Mutlu Özcan
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引用次数: 0
Introduction of a New Classification for Resin Composites with Enhanced Color Adjustment Potential 一种具有增强颜色调节潜力的树脂复合材料的新分类介绍
Q2 Dentistry Pub Date : 2023-09-22 DOI: 10.1007/s40496-023-00351-2
Bora Korkut, Bilge Tarçın, Pınar Yılmaz Atalı, Mutlu Özcan
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引用次数: 0
Relationship of NLRP3 Inflammasome with Oral Health NLRP3炎性体与口腔健康的关系
Q2 Dentistry Pub Date : 2023-09-21 DOI: 10.1007/s40496-023-00350-3
Seyhan Karaaslan
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引用次数: 0
The Impact of the COVID-19 Pandemic on Oral and Oropharyngeal Cancer 新冠肺炎大流行对口腔和口咽癌症的影响
Q2 Dentistry Pub Date : 2023-09-04 DOI: 10.1007/s40496-023-00349-w
Emily McIlvanna, Caroline McCarthy, Katarzyna Gurzawska-Comis
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引用次数: 0
Dentin Degradation: From Tissue Breakdown to Possibilities for Therapeutic Intervention. 牙本质降解:从组织分解到治疗干预的可能性
Q2 Dentistry Pub Date : 2023-09-01 Epub Date: 2023-07-25 DOI: 10.1007/s40496-023-00341-4
Cristina M P Vidal, Marcela R Carrilho

Purpose of the review: Presently, dental materials science is driven by the search for new and improved materials that can trigger specific reactions from the affected tissue to stimulate repair or regeneration while interacting with the oral environment to promote or maintain oral health. In parallel, evidence from the past decades has challenged the exclusive role of bacteria in dentin tissue degradation in caries, questioning our understanding of caries etiopathogenesis. The goal of this review is to recapitulate the current evidence on the host and bacterial contributions to degradation, inflammation, and repair of the dentin-pulp complex in caries.

Recent findings: Contrasting findings attribute dentin breakdown to the activity of endogenous enzymes, such as matrix metalloproteinases (MMPs) and cathepsins, while the role of bacteria and their by-products in the destruction of dentin organic matrix and pulp inflammation has been for decades supported as an incontestable paradigm. Aiming to better understand the mechanisms involved in collagen degradation by host enzymes in caries, studies have showed that these proteinases are expressed in the mature dentin (i.e., after dentin formation) and become activated by the low pH in the acidic environment resulted by bacterial metabolism in caries. However, different host sources other than dentin-bound proteinases seem to also contribute to caries progression, such as saliva and pulp. Interestingly, studies evaluating pulp responses to bacteria invasion and inflammation in caries report higher levels of MMPs and cathepsins in inflamed tissue, but also showed MMP potential to resolve inflammation and stimulate wound healing. Notably, as reported for other tissues, MMPs exert dual roles in the dentin-pulp complex in caries, participating or regulating both degradative and reparative mechanisms.

Summary: The specific roles of host and bacteria and their by-products in caries progression have yet to be clarified. The complex interactions between inflammation and repair in caries pose challenges to a clear understanding of the dentin-pulp complex responses and changes to bacteria invasion. However, it opens new venues for the development of novel therapies and dental biomaterials based on the modulation of specific mechanisms to favor tissue repair and healing.

综述目的:目前,牙科材料科学的发展方向是寻找新的和改进的材料,这些材料可以引发受影响组织的特定反应,刺激修复或再生,同时与口腔环境相互作用,促进或保持口腔健康。与此同时,过去几十年的证据对细菌在龋齿中牙本质组织降解中的独特作用提出了质疑,质疑了我们对龋齿发病机制的理解。这篇综述的目的是概述目前关于宿主和细菌对龋齿中牙本质-牙髓复合体降解、炎症和修复的作用的证据。最近的研究结果:对比研究结果将牙本质破坏归因于内源性酶的活性,如基质金属蛋白酶(MMPs)和组织蛋白酶,而细菌及其副产物在破坏牙本质有机基质和牙髓炎症中的作用几十年来一直被支持为一种无可争议的范式。为了更好地了解宿主酶在龋齿中降解胶原蛋白的机制,研究表明,这些蛋白酶在成熟的牙本质中表达(即在牙本质形成后),并在龋齿中细菌代谢导致的酸性环境中的低pH值下被激活。然而,除牙本质结合蛋白酶外,不同的宿主来源似乎也有助于龋齿的发展,如唾液和牙髓。有趣的是,评估牙髓对细菌入侵和龋齿炎症反应的研究报告称,炎症组织中MMPs和组织蛋白酶水平较高,但也显示MMP有可能解决炎症并刺激伤口愈合。值得注意的是,正如其他组织所报道的那样,MMPs在龋齿的牙本质-牙髓复合体中发挥双重作用,参与或调节降解和修复机制。摘要:宿主和细菌及其副产物在龋齿进展中的具体作用尚待阐明。龋齿中炎症和修复之间的复杂相互作用对清楚理解牙本质-牙髓复合体对细菌入侵的反应和变化提出了挑战。然而,它为开发基于特定机制的新型疗法和牙科生物材料开辟了新的途径,以促进组织修复和愈合。
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引用次数: 0
Hospital Dental Services to Reduce Hospital-Acquired Pneumonia 医院牙科服务减少医院获得性肺炎
Q2 Dentistry Pub Date : 2023-08-22 DOI: 10.1007/s40496-023-00346-z
Kenichiro Ozaki, H. Tohara, M. Baba, Satoru Komatsumoto
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引用次数: 0
Highlights in Contemporary Smart Dental Materials: a Review 当代智能牙科材料研究综述
Q2 Dentistry Pub Date : 2023-08-15 DOI: 10.1007/s40496-023-00348-x
Tamer M. Hamdy
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引用次数: 0
Sensory Systems for Sugar-Induced Cephalic Phase Insulin Release 糖诱导的头期胰岛素释放的感觉系统
Q2 Dentistry Pub Date : 2023-08-12 DOI: 10.1007/s40496-023-00347-y
R. Yoshida
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引用次数: 0
The Association Between Periodontal Disease and Non-Alcoholic Fatty Liver Disease is Linked to Metabolic Disorders 牙周病和非酒精性脂肪性肝病之间的联系与代谢紊乱有关
Q2 Dentistry Pub Date : 2023-08-11 DOI: 10.1007/s40496-023-00342-3
Yuto Kusu, M. Furuta, T. Takeshita
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引用次数: 0
Zirconia: More and More Translucent 氧化锆:越来越半透明
Q2 Dentistry Pub Date : 2023-08-09 DOI: 10.1007/s40496-023-00344-1
J. Hjerppe, M. Özcan
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引用次数: 0
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Current Oral Health Reports
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