Pub Date : 2003-06-01DOI: 10.1177/0533316404047796
Samir Amin
AUTHOR INDEX Alderdice, Lord.‘Response to Lecture by Ivan Urlić’, 472–476. Arnott, B. Book Review, ‘Inconceivable Conceptions. Psychological Aspects of Infertility and Reproductive Technology’, J. Haynes and J. Miller (eds), 567–568. Barwick, N. ‘Bearing Witness: Group Analysis as Witness Training in Action’, 121–136. Behr, H. ‘Commentary on “Drawing the Isolate into the Group Flow” by Louis Ormont’, 76–81. Behr, H. ‘Commentary on Article by Hinshelwood’, 333–338. Billow, R. M. ‘Working Relationally with the Adolescent in Group’, 187–200. Blackwell, R.D. ‘Freedom and Autonomy in Mind, Commentary on “A case for Mind” by Patrick de Maré and Robert Schöllberger, and “Mind” by S. H. Foulkes’, 353–357. Bledin, K. ‘What’s in a Name? Foulkes, Identity and the Social Unconscious’, 477–489. Brunori, L. (see also Gibin, A.M., Miglioli, M. and Bussandri, M.) ‘Analysis of the Therapeutic Course of an Eating Disorders Group’, 387–399; ‘28th S.H. Foulkes Annual Lecture: President’s Introduction’, 451–452. Burman, E. ‘Boundary Objects and Group Analysis: Between Psychoanalysis and Social Theory’, 361–379. Burman, E. ‘Organising for Change? Group-analytic Perspectives on A Feminist Action Research Project’, 91–108. Bush, M (see also Wennberg, P., Weinryb, R.M., Saxon, L., Göransson, S. and Skarbrandt, E.) ‘Personality Levels of Psychological Distress and Premature Termination of Psychodynamic Group Therapy: Results from a Prospective Longitudinal Study’, 179–185. Bussandri, M. (see also Brunori, L., Gibin, A.M., and Miglioli, M.) ‘Analysis of the Therapeutic Course of an Eating Disorders Group’, 387–399. de Maré, P. (see also Schöllberger, R.) ‘A Case For Mind’, 339–352. Diamond, N. Obituary, Hans Cohn, 564–566. Eszik, J. (see also Kis, G. and Terenyi, Z.) ‘Hierarchy And/Or Reflectivity: The Role of Groups in the Function of Psychiatric Wards’, 45–63. Foulkes, E. ‘Response to “What’s in a Name? Foulkes, Identity and the Social Unconscious” by Kenneth Bledin’, 490–491. Friedman, R. ‘Dream-telling as a Request for Containment – Reconsidering the Groupanalytic Approach to the Work with Dreams’, 508–524. Gibin, A.M., (see also Brunori, L. Miglioli, M. and Bussandri, M.) ‘Analysis of the Therapeutic Course of an Eating Disorders Group’, 387–399. Göransson, S. (see also Wennberg, P., Weinryb, R.M., Saxon, L., Bush, M and Skarbrandt, E.) ‘Personality Levels of Psychological Distress and Premature Termination Of Psychodynamic Group Therapy: Results From a Prospective Longitudinal Study’, 179–185.
作者索引奥尔德代斯，勋爵。《对Ivan urliki演讲的回应》，472-476页。阿诺特，B.书评，《不可思议的概念》。不孕不育和生殖技术的心理方面'，J. Haynes和J. Miller(主编)，567-568。巴威克，N.“作证:作为行动中的证人训练的群体分析”，121-136页。评论路易斯·奥蒙特的《将孤立者引入群体流》，第76-81页。Behr, H. '对Hinshelwood文章的评论'，333-338。比洛，r.m.，《与群体中的青少年的工作关系》，187-200。Blackwell, R.D.，《心灵中的自由与自主》，对Patrick de mar和Robert Schöllberger的《心灵案例》和S. H. Foulkes的《心灵》的评论，353-357页。该死，k，名字有什么用?《身份与社会无意识》，第477-489页。Brunori, L.(也见Gibin, a.m.， Miglioli, M.和Bussandri, M.)，《饮食失调组的治疗过程分析》，387-399;第28届S.H. Foulkes年度演讲:校长介绍，第451-452页。边界对象和群体分析:在精神分析和社会理论之间，第361-379页。E. Burman，《组织变革?》《一个女性主义行动研究项目的群体分析视角》，第91-108期。Bush, M(也见Wennberg, P.， Weinryb, r.m.， Saxon, L.， Göransson, S.和Skarbrandt, E.)，“心理困扰的人格水平和心理动力团体治疗的过早终止:来自前瞻性纵向研究的结果”，179-185。Bussandri, M.(另见Brunori, L.， Gibin, a.m.和Miglioli, M.)，《饮食失调组的治疗过程分析》，第387-399页。de mardeur, P.(另见Schöllberger, R.)，《A Case For Mind》，339-352页。讣告，汉斯·科恩，564-566。Eszik, J.(另见Kis, G.和Terenyi, Z.)，“层级和/或反射率:群体在精神科病房功能中的作用”，第45-63页。E. Foulkes对“名字里有什么?”《身份与社会无意识》，Kenneth Bledin著，490-491页。Friedman, R.，《作为遏制要求的梦境讲述——重新考虑与梦一起工作的群体分析方法》，第508-524页。Gibin, a.m.(另见Brunori, L. Miglioli, M.和Bussandri, M.)，《饮食失调组的治疗过程分析》，第387-399页。Göransson, S.(另见Wennberg, P.， Weinryb, r.m.， Saxon, L.， Bush, M.和Skarbrandt, E.)“心理困扰的人格水平和心理动力团体治疗的过早终止:来自前瞻性纵向研究的结果”，179-185。
{"title":"Volume Index","authors":"Samir Amin","doi":"10.1177/0533316404047796","DOIUrl":"https://doi.org/10.1177/0533316404047796","url":null,"abstract":"AUTHOR INDEX Alderdice, Lord.‘Response to Lecture by Ivan Urlić’, 472–476. Arnott, B. Book Review, ‘Inconceivable Conceptions. Psychological Aspects of Infertility and Reproductive Technology’, J. Haynes and J. Miller (eds), 567–568. Barwick, N. ‘Bearing Witness: Group Analysis as Witness Training in Action’, 121–136. Behr, H. ‘Commentary on “Drawing the Isolate into the Group Flow” by Louis Ormont’, 76–81. Behr, H. ‘Commentary on Article by Hinshelwood’, 333–338. Billow, R. M. ‘Working Relationally with the Adolescent in Group’, 187–200. Blackwell, R.D. ‘Freedom and Autonomy in Mind, Commentary on “A case for Mind” by Patrick de Maré and Robert Schöllberger, and “Mind” by S. H. Foulkes’, 353–357. Bledin, K. ‘What’s in a Name? Foulkes, Identity and the Social Unconscious’, 477–489. Brunori, L. (see also Gibin, A.M., Miglioli, M. and Bussandri, M.) ‘Analysis of the Therapeutic Course of an Eating Disorders Group’, 387–399; ‘28th S.H. Foulkes Annual Lecture: President’s Introduction’, 451–452. Burman, E. ‘Boundary Objects and Group Analysis: Between Psychoanalysis and Social Theory’, 361–379. Burman, E. ‘Organising for Change? Group-analytic Perspectives on A Feminist Action Research Project’, 91–108. Bush, M (see also Wennberg, P., Weinryb, R.M., Saxon, L., Göransson, S. and Skarbrandt, E.) ‘Personality Levels of Psychological Distress and Premature Termination of Psychodynamic Group Therapy: Results from a Prospective Longitudinal Study’, 179–185. Bussandri, M. (see also Brunori, L., Gibin, A.M., and Miglioli, M.) ‘Analysis of the Therapeutic Course of an Eating Disorders Group’, 387–399. de Maré, P. (see also Schöllberger, R.) ‘A Case For Mind’, 339–352. Diamond, N. Obituary, Hans Cohn, 564–566. Eszik, J. (see also Kis, G. and Terenyi, Z.) ‘Hierarchy And/Or Reflectivity: The Role of Groups in the Function of Psychiatric Wards’, 45–63. Foulkes, E. ‘Response to “What’s in a Name? Foulkes, Identity and the Social Unconscious” by Kenneth Bledin’, 490–491. Friedman, R. ‘Dream-telling as a Request for Containment – Reconsidering the Groupanalytic Approach to the Work with Dreams’, 508–524. Gibin, A.M., (see also Brunori, L. Miglioli, M. and Bussandri, M.) ‘Analysis of the Therapeutic Course of an Eating Disorders Group’, 387–399. Göransson, S. (see also Wennberg, P., Weinryb, R.M., Saxon, L., Bush, M and Skarbrandt, E.) ‘Personality Levels of Psychological Distress and Premature Termination Of Psychodynamic Group Therapy: Results From a Prospective Longitudinal Study’, 179–185.","PeriodicalId":12073,"journal":{"name":"Experimental Gerontology","volume":"42 1","pages":"1176-1184"},"PeriodicalIF":3.9,"publicationDate":"2003-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1177/0533316404047796","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"65674571","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
The heart faces a high risk of free radical injury owing to a slow generation of antioxidant (AO) enzymes by its cells. A general decline in this system may be another reason for the development of age-related diseases. Although the correlation between aging and exercise has been studied extensively, these studies have produced conflicting data on the effects of vitamin E on the aging heart, when it is introduced as an intervening factor. To investigate these effects, we determined the activities of antioxidant enzymes (AOEs) such as superoxide dismutase (SOD) and catalase (CAT), lipid peroxidation (LP), lipofuscin (LF)-like autofluorescent substances and vitamin E content in the left and right ventricles (LV and RV) of the heart in male Wistar albino rats of 4-(young adults), 8-(old adults), 12-(middle-age) and 22-mos(old) of age. Animals were orally supplemented with vitamin E and allowed to swim for 30 min/day, 5 days/week and for a total period of 60 days. Exercise training in all the age groups except the old was effective in upregulating the SOD activity. Old trainees showed an increase in SOD activity when supplemented with vitamin E. In the 22-mo-olds, a remarkable decrease in CAT activity was seen. Exercise by itself upregulated the CAT as well as SOD activity in all age groups except the old wherein vitamin E was effective in increasing the activities of AOEs. Supplementation significantly reduced LP as evidenced by lowered malondialdehyde (MDA) and LF-like autofluorescent substances in the trained as well as sedentary rats. Tissue vitamin E content was low in the swim trainees that were not supplemented. This change, well emphasized in the trainee groups of 22-mo-old suggests the probable utilization of vitamin E in keeping free radicals at bay. Our results suggest that vitamin E can stand out as a significant tool in ameliorating the declining AO defense in the old rats.
{"title":"Dietary vitamin E and physical exercise: II. Antioxidant status and lipofuscin-like substances in aging rat heart.","authors":"S Asha Devi, S Prathima, M V V Subramanyam","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>The heart faces a high risk of free radical injury owing to a slow generation of antioxidant (AO) enzymes by its cells. A general decline in this system may be another reason for the development of age-related diseases. Although the correlation between aging and exercise has been studied extensively, these studies have produced conflicting data on the effects of vitamin E on the aging heart, when it is introduced as an intervening factor. To investigate these effects, we determined the activities of antioxidant enzymes (AOEs) such as superoxide dismutase (SOD) and catalase (CAT), lipid peroxidation (LP), lipofuscin (LF)-like autofluorescent substances and vitamin E content in the left and right ventricles (LV and RV) of the heart in male Wistar albino rats of 4-(young adults), 8-(old adults), 12-(middle-age) and 22-mos(old) of age. Animals were orally supplemented with vitamin E and allowed to swim for 30 min/day, 5 days/week and for a total period of 60 days. Exercise training in all the age groups except the old was effective in upregulating the SOD activity. Old trainees showed an increase in SOD activity when supplemented with vitamin E. In the 22-mo-olds, a remarkable decrease in CAT activity was seen. Exercise by itself upregulated the CAT as well as SOD activity in all age groups except the old wherein vitamin E was effective in increasing the activities of AOEs. Supplementation significantly reduced LP as evidenced by lowered malondialdehyde (MDA) and LF-like autofluorescent substances in the trained as well as sedentary rats. Tissue vitamin E content was low in the swim trainees that were not supplemented. This change, well emphasized in the trainee groups of 22-mo-old suggests the probable utilization of vitamin E in keeping free radicals at bay. Our results suggest that vitamin E can stand out as a significant tool in ameliorating the declining AO defense in the old rats.</p>","PeriodicalId":12073,"journal":{"name":"Experimental Gerontology","volume":"38 3","pages":"291-7"},"PeriodicalIF":3.9,"publicationDate":"2003-03-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"22241108","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2000-02-01DOI: 10.1016/S0531-5565(99)00086-8
H. Warner
{"title":"Time of our Lives: Tom Kirkwood; New York, Oxford University Press, 1999, 277 pages, ISBN 0-19-512824-9 (US$ 27.50)","authors":"H. Warner","doi":"10.1016/S0531-5565(99)00086-8","DOIUrl":"https://doi.org/10.1016/S0531-5565(99)00086-8","url":null,"abstract":"","PeriodicalId":12073,"journal":{"name":"Experimental Gerontology","volume":"35 1","pages":"117-118"},"PeriodicalIF":3.9,"publicationDate":"2000-02-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/S0531-5565(99)00086-8","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"56239055","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Atherosclerosis is a paradigmatic disease of the elderly, the roots of which are laid in youth, whereas the clinically manifested consequences become evident at old age.","authors":"G Wick, Q Xu","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":12073,"journal":{"name":"Experimental Gerontology","volume":"34 4","pages":"481-2"},"PeriodicalIF":3.9,"publicationDate":"1999-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"21845765","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Immune-inflammatory processes are increasingly discussed as possible pathogenetic factors involved in the development of atherosclerosis. Here, we summarize data on which we have built our "immunological" hypothesis of atherogenesis. This concept is based on the observation that nearly everybody shows protective cellular and humoral immune reactions against microbial heat shock protein 60 (HSP 60). Because a high degree of antigenic homology exists between microbial (viral, bacterial, parasitic) and human HSP 60, this protective immunity may have to be "paid for" by the danger of cross-reactivity with human HSP 60 that is expressed by endothelial cells of stressed arteries. Arterial endothelial cells are more prone to produce HSP 60 and various adhesion molecules upon exposure to stress factors, including classical risk factors for atherosclerosis, due to their life-long exposure to the high arterial as compared to venous blood pressure. Also, endothelial cells are the first potential targets encountered by circulating HSP 60-specific T cells or antibodies. This concept not only opens new avenues for diagnostic approaches, but also may form the basis for new ways of therapeutic intervention.
{"title":"Atherosclerosis--an autoimmune disease.","authors":"G Wick, Q Xu","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Immune-inflammatory processes are increasingly discussed as possible pathogenetic factors involved in the development of atherosclerosis. Here, we summarize data on which we have built our \"immunological\" hypothesis of atherogenesis. This concept is based on the observation that nearly everybody shows protective cellular and humoral immune reactions against microbial heat shock protein 60 (HSP 60). Because a high degree of antigenic homology exists between microbial (viral, bacterial, parasitic) and human HSP 60, this protective immunity may have to be \"paid for\" by the danger of cross-reactivity with human HSP 60 that is expressed by endothelial cells of stressed arteries. Arterial endothelial cells are more prone to produce HSP 60 and various adhesion molecules upon exposure to stress factors, including classical risk factors for atherosclerosis, due to their life-long exposure to the high arterial as compared to venous blood pressure. Also, endothelial cells are the first potential targets encountered by circulating HSP 60-specific T cells or antibodies. This concept not only opens new avenues for diagnostic approaches, but also may form the basis for new ways of therapeutic intervention.</p>","PeriodicalId":12073,"journal":{"name":"Experimental Gerontology","volume":"34 4","pages":"559-66"},"PeriodicalIF":3.9,"publicationDate":"1999-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"21664126","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Proceedings of the 4th International Symposium on the Neurobiology and Neuroendocrinology of Aging. Bregenz, Austria, July 26-31, 1998.","authors":"","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":12073,"journal":{"name":"Experimental Gerontology","volume":"33 7-8","pages":"635-920"},"PeriodicalIF":3.9,"publicationDate":"1998-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"20856830","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Special issue: A tribute to Alex Comfort.","authors":"","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":12073,"journal":{"name":"Experimental Gerontology","volume":"33 1-2","pages":"1-207"},"PeriodicalIF":3.9,"publicationDate":"1998-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"20396138","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Proceedings of the 3rd International Symposium on The Neurobiology and Neuroendocrinology of Aging. Bregenz, Austria, July 21-26, 1996.","authors":"","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":12073,"journal":{"name":"Experimental Gerontology","volume":"32 4-5","pages":"355-614"},"PeriodicalIF":3.9,"publicationDate":"1997-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"20306813","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Proceedings of the 2nd International Conference on Animal Models for Aging Research. Kyoto, Japan, May 12-19, 1995.","authors":"","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":12073,"journal":{"name":"Experimental Gerontology","volume":"32 1-2","pages":"1-242"},"PeriodicalIF":3.9,"publicationDate":"1997-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"20164074","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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