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Experimental Gerontology最新文献

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Proceedings of the 2nd International Conference on Longevity and Aging: Environmental and Nutritional Influences on Aging and Cancer. 第二届长寿与衰老国际会议论文集:环境和营养对衰老和癌症的影响。
IF 3.9 3区 医学 Q2 Biochemistry, Genetics and Molecular Biology
Experimental Gerontology
Pub Date : 1992-09-01
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引用次数: 0
Human Diploid Fibroblast-Like Cells as a Model System for the Study of Senescence. Conference proceedings. Ardmore, Oklahoma, December 15-18, 1991. 人类二倍体成纤维细胞样细胞作为衰老研究的模型系统。会议论文集。1991年12月15日至18日，俄克拉何马州的阿德莫尔。
IF 3.9 3区 医学 Q2 Biochemistry, Genetics and Molecular Biology
Experimental Gerontology
Pub Date : 1992-07-01
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引用次数: 0
Comparative aspects of age-related plasticity in the adult nervous system. 成人神经系统中年龄相关可塑性的比较方面。
IF 3.9 3区 医学 Q2 Biochemistry, Genetics and Molecular Biology
Experimental Gerontology
Pub Date : 1992-01-01
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引用次数: 0
Regulation of neuroendocrine aging. Vol. 24. Interdisciplinary topics in gerontology by A.V. Everitt and J.R. Walton (Eds.), H.P. von Hahn (Series Ed.), Karger, Basel, Switzerland, 1988, ISBN 3-8055-4770-6, 155 pp 神经内分泌衰老的调节。第24卷。A.V.Everitt和J.R.Walton（编辑），H.P.von Hahn（系列编辑），Karger，巴塞尔，瑞士，1988年，ISBN 3-8055-4770-6155页
IF 3.9 3区 医学 Q2 Biochemistry, Genetics and Molecular Biology
Experimental Gerontology
Pub Date : 1991-12-31 DOI: 10.1016/0531-5565(91)90080-6
W. Dean
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引用次数: 0
Review of Biological research in aging, volume 4 by Morton Rothstein (Ed.), Wiley-Liss, New York, 1990, ISBN 0-471-56697-7, 387 pp., $120.00 《衰老生物学研究综述》，Morton Rothstein主编，Wiley-Liss出版社，纽约，1990,ISBN 0-471-56697- 7,387页，120.00美元
IF 3.9 3区 医学 Q2 Biochemistry, Genetics and Molecular Biology
Experimental Gerontology
Pub Date : 1991-12-31 DOI: 10.1016/0531-5565(91)90081-V
W. Dean
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引用次数: 0
Proceedings of the 1st International Symposium on Calcium, Molecular Biology and Aging. Siena, Italy, October 17-19, 1988. 第一届钙、分子生物学与衰老国际学术研讨会论文集。1988年10月17日至19日，意大利锡耶纳。
IF 3.9 3区 医学 Q2 Biochemistry, Genetics and Molecular Biology
Experimental Gerontology
Pub Date : 1990-01-01
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引用次数: 0
Control of cell proliferation in senescent cells. 衰老细胞增殖的控制。
IF 3.9 3区 医学 Q2 Biochemistry, Genetics and Molecular Biology
Experimental Gerontology
Pub Date : 1989-01-01
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引用次数: 0
Biomarkers of aging. Proceedings of a conference. Chicago, Illinois, November 18 and 19, 1986. 衰老的生物标志物。会议记录。芝加哥，伊利诺斯州，1986年11月18日和19日。
IF 3.9 3区 医学 Q2 Biochemistry, Genetics and Molecular Biology
Experimental Gerontology
Pub Date : 1988-01-01
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引用次数: 0
In celebration of the 80th birthday of Nathan W. Shock, Ph.D. 25 December 1986. 1986年12月25日，为庆祝Nathan W. Shock博士80岁生日。
IF 3.9 3区 医学 Q2 Biochemistry, Genetics and Molecular Biology
Experimental Gerontology
Pub Date : 1986-01-01
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引用次数: 0
Age-related differential effects of zinc on concanavalin A-induced capping of human lymphocytes. 锌对豆豆蛋白a诱导的人淋巴细胞封顶的年龄相关性差异效应。
IF 3.9 3区 医学 Q2 Biochemistry, Genetics and Molecular Biology
Experimental Gerontology
Pub Date : 1982-01-01
K M Rao

Con A-induced capping of lymphocytes is altered by a number of cytoskeletal modulating agents. Zinc, in the presence of colchicine, demonstrated an age-dependent differential effect on con A-induced capping of human peripheral blood lymphocytes. In general, zinc enhanced capping of lymphocytes from young individuals and suppressed from those of old individuals. The effect of zinc on the cap formation adds further support to the evidence that zinc might be acting on the microfilaments. However, cytochalasin B, another microfilament modulating agent, does not show a differential effect similar to zinc. This suggests that zinc might modify microfilament formation or function in a manner different from cytochalasin B. The proposal that the effects of zinc might be due to its action on calmodulin suggests that calmodulin, the calcium regulating protein, may have a role in the physiological processes associated with aging. Further studies in this area might yield fruitful results in our understanding of the molecular mechanism of senescence.

Con - a诱导的淋巴细胞封顶被一些细胞骨架调节剂所改变。在秋水仙碱存在的情况下，锌对con - a诱导的人外周血淋巴细胞的覆盖表现出年龄依赖性差异效应。总的来说，锌增加了年轻个体淋巴细胞的封顶，抑制了老年个体的淋巴细胞封顶。锌对帽形成的影响进一步支持了锌可能作用于微丝的证据。然而，细胞松弛素B，另一种微丝调节剂，没有表现出类似锌的差异效应。这表明锌可能以不同于细胞松弛素b的方式改变微丝的形成或功能。锌的作用可能是由于它对钙调蛋白的作用，这表明钙调蛋白，钙调节蛋白，可能在与衰老相关的生理过程中起作用。在这方面的进一步研究可能会在我们对衰老的分子机制的理解上取得丰硕的成果。
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引用次数: 0
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