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Cerebral Ischemia最新文献

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Hypoxic-Ischemic Brain Injury after Perinatal Asphyxia as a Possible Factor in the Pathology of Alzheimer’s Disease 围产期窒息后缺氧缺血性脑损伤是阿尔茨海默病病理的可能因素
Pub Date : 2021-08-31 DOI: 10.36255/exonpublications.cerebralischemia.2021.perinatalasphyxia
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引用次数: 4
Neurovascular Reactivity in Tissue Scarring Following Cerebral Ischemia 脑缺血后组织瘢痕的神经血管反应性
Pub Date : 2021-08-18 DOI: 10.36255/exonpublications.cerebralischemia.2021.neurovascularreactivity
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引用次数: 2
The Role of Curcumin in Post-Ischemic Brain 姜黄素在脑缺血后的作用
Pub Date : 2021-08-17 DOI: 10.36255/exonpublications.cerebralischemia.2021.curcumin
M. Rusek, S. Czuczwar
Progressive accumulation of misfolded amyloid and tau protein in intracellular and extracellular spaces is the most crucial etiopathological feature of brain ischemia, synaptic damage, or neural communication impairment. Clinical data suggest that dietary intake of curcumin enhances neurogenesis and offers neuroprotection. Curcumin is a natural polyphenolic compound with diverse and attractive biological properties. It may prevent aging-associated changes in cellular proteins, such as β-amyloid peptide and tau protein, that lead to protein insolubility and aggregation after ischemic brain damage. Therefore, curcumin seems to be a promising supplementary agent against neurodegeneration development after brain ischemia. The aim of this chapter is to highlight our current understanding of the neuroprotective role of curcumin in cerebral ischemia-reperfusion injury. The limitations and adverse events of curcumin are also presented.
错误折叠的淀粉样蛋白和tau蛋白在细胞内和细胞外空间的进行性积累是脑缺血、突触损伤或神经通讯障碍的最重要的病因病理学特征。临床数据表明,饮食中摄入姜黄素可促进神经发生并提供神经保护。姜黄素是一种具有多种生物学特性的天然多酚类化合物。它可以防止细胞蛋白(如β-淀粉样肽和tau蛋白)的衰老相关变化,这些变化导致缺血性脑损伤后蛋白质的不溶性和聚集。因此,姜黄素似乎是一种很有前途的抗脑缺血后神经退行性发展的补充剂。本章的目的是强调我们目前对姜黄素在脑缺血再灌注损伤中的神经保护作用的理解。也介绍了姜黄素的局限性和不良事件。
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引用次数: 1
Neuroinflammation in Post-Ischemic Brain 脑缺血后的神经炎症
Pub Date : 2021-08-17 DOI: 10.36255/exonpublications.cerebralischemia.2021.neuroinflammation
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引用次数: 4
期刊
Cerebral Ischemia
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