首页 > 最新文献

Journal of Neuroscience and Neurological Disorders最新文献

英文 中文
The turing machine theory for some spinal cord and brain condition, A toxicological - antidotic depurative approach 图灵机理论对于一些脊髓和脑部疾病,是一种毒理学-解毒-净化的方法
Pub Date : 2019-07-31 DOI: 10.29328/JOURNAL.JNND.1001023
M. Luisetto, Behzad Nili Ahmadabadi, Ahmed Yesvi Rafa, R. Sahu, L. Cabianca, G. Mashori, F. Khan
Aim of this work is to produce a general theory related an new depurative strategy to be devalued for reduce or delay some spinal cord and brain degenerative and infl ammatory chronic disease or acute traumatic condition. It is used and informatics approach in order to set correct the problem and the process. Scope of this project is to submit to the researcher a new therapeutic strategy (under a depurativetoxicological-pharmacological) in this complex kind of disease. A Turing machine theory say us a method to TRASLATE the need of a strategy in a practical hypotesys of work. A global conceptual map can help in this fi eld. More Information *Address for Correspondence: Mauro Luisetto, Applied Pharmacologist, European Specialist Lab, Medicine, Branch General Toxicology, Italy; Email: maurolu65@gmail.com Submitted: 22 July 2019 Approved: 30 July 2019 Published: 31 July 2019 How to cite this article: Luisetto M, Ahmadabadi BN, Rafa AY, Sahu RK, Cabianca L, et al. The turing machine theory for some spinal cord and brain condition, A toxicological antidotic depurative approach. J Neurosci Neurol Disord.
本研究的目的是为减轻或延缓某些脊髓和大脑退行性和炎症性慢性疾病或急性创伤性疾病提供一种新的贬值策略。运用信息学的方法来设定和纠正问题和过程。该项目的范围是向研究人员提交一种新的治疗策略(在净化毒理学-药理学下),以治疗这种复杂的疾病。图灵机理论为我们提供了一种在实际工作假设中翻译策略需求的方法。全球概念图可以在这个领域提供帮助。更多信息*通信地址:Mauro Luisetto,应用药理学家,欧洲专家实验室,医学,一般毒理学分部,意大利;Email: maurolu65@gmail.com提交时间:2019年7月22日批准时间:2019年7月30日发布时间:2019年7月31日文章来源:Luisetto M, Ahmadabadi BN, Rafa AY, Sahu RK, Cabianca L, et al.等。图灵机理论对于一些脊髓和脑部疾病,是一种毒理学解毒净化的方法。神经科学[J]。
{"title":"The turing machine theory for some spinal cord and brain condition, A toxicological - antidotic depurative approach","authors":"M. Luisetto, Behzad Nili Ahmadabadi, Ahmed Yesvi Rafa, R. Sahu, L. Cabianca, G. Mashori, F. Khan","doi":"10.29328/JOURNAL.JNND.1001023","DOIUrl":"https://doi.org/10.29328/JOURNAL.JNND.1001023","url":null,"abstract":"Aim of this work is to produce a general theory related an new depurative strategy to be devalued for reduce or delay some spinal cord and brain degenerative and infl ammatory chronic disease or acute traumatic condition. It is used and informatics approach in order to set correct the problem and the process. Scope of this project is to submit to the researcher a new therapeutic strategy (under a depurativetoxicological-pharmacological) in this complex kind of disease. A Turing machine theory say us a method to TRASLATE the need of a strategy in a practical hypotesys of work. A global conceptual map can help in this fi eld. More Information *Address for Correspondence: Mauro Luisetto, Applied Pharmacologist, European Specialist Lab, Medicine, Branch General Toxicology, Italy; Email: maurolu65@gmail.com Submitted: 22 July 2019 Approved: 30 July 2019 Published: 31 July 2019 How to cite this article: Luisetto M, Ahmadabadi BN, Rafa AY, Sahu RK, Cabianca L, et al. The turing machine theory for some spinal cord and brain condition, A toxicological antidotic depurative approach. J Neurosci Neurol Disord.","PeriodicalId":382788,"journal":{"name":"Journal of Neuroscience and Neurological Disorders","volume":"44 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2019-07-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"132036189","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 5
Comparative study of carboxylate and amide forms of HLDF-6 peptide: Neuroprotective and nootropic effects in animal models of ischemic stroke HLDF-6肽羧酸和酰胺形式的比较研究:对缺血性脑卒中动物模型的神经保护和益智作用
Pub Date : 2019-07-24 DOI: 10.29328/JOURNAL.JNND.1001022
Аnna P Bogachuk, Z. Storozheva, A. Proshin, V. V. Sherstnev, Irina V. Smirnova, T. Shuvaeva, V. Lipkin
Cerebrovascular diseases are among the most urgent medical and social problems of the modern world. The prevalence of nervous system diseases associated with circulatory insuf iciency is up to 20% of the population and is trending upwards. Many nervous system diseases are characterized by a lingering severe course and persistent functional defects resulting in limited or total disability [1]. One of the most severe cerebrovascular diseases is ischaemic stroke (IS). Mechanisms of brain tissue damage development under conditions of cerebral ischaemia necessitate the introduction of a special type of therapy known as neuroprotection. Neuroprotection is aimed at interrupting the delayed mechanisms of cell death—oxidative stress, cytokine imbalance, local in lammation, trophic dysfunction, and apoptosis. A tight interconnection between all mechanisms of nervous tissue death motivates exploitation of the modulating effect that could be achieved through stimulation of the regulatory systems. Peptides are among the key players in regulatory processes [2]. An optimal scenario of therapy for IS and other cerebral disorders should comprise the application of agents simultaneously affecting several key components in the pathogenesis of the disease. Another important requirement for these compounds is the absence of toxic effects when used for prolonged preventive treatment. Due to Abstract
脑血管疾病是现代世界最紧迫的医疗和社会问题之一。与循环功能不全相关的神经系统疾病的患病率高达人口的20%,并且呈上升趋势。许多神经系统疾病的特点是病程持续严重,持续的功能缺陷导致有限或完全残疾。缺血性脑卒中是最严重的脑血管疾病之一。脑缺血条件下脑组织损伤发展的机制需要引入一种称为神经保护的特殊治疗。神经保护旨在阻断细胞死亡的延迟机制——氧化应激、细胞因子失衡、局部层压、营养功能障碍和细胞凋亡。神经组织死亡的所有机制之间的紧密联系激发了调节效应的开发,可以通过刺激调节系统来实现。多肽是调控过程中的关键参与者之一。治疗IS和其他脑部疾病的最佳方案应该包括同时影响疾病发病机制中几个关键组成部分的药物的应用。对这些化合物的另一个重要要求是在用于长期预防性治疗时没有毒性作用。由于摘要
{"title":"Comparative study of carboxylate and amide forms of HLDF-6 peptide: Neuroprotective and nootropic effects in animal models of ischemic stroke","authors":"Аnna P Bogachuk, Z. Storozheva, A. Proshin, V. V. Sherstnev, Irina V. Smirnova, T. Shuvaeva, V. Lipkin","doi":"10.29328/JOURNAL.JNND.1001022","DOIUrl":"https://doi.org/10.29328/JOURNAL.JNND.1001022","url":null,"abstract":"Cerebrovascular diseases are among the most urgent medical and social problems of the modern world. The prevalence of nervous system diseases associated with circulatory insuf iciency is up to 20% of the population and is trending upwards. Many nervous system diseases are characterized by a lingering severe course and persistent functional defects resulting in limited or total disability [1]. One of the most severe cerebrovascular diseases is ischaemic stroke (IS). Mechanisms of brain tissue damage development under conditions of cerebral ischaemia necessitate the introduction of a special type of therapy known as neuroprotection. Neuroprotection is aimed at interrupting the delayed mechanisms of cell death—oxidative stress, cytokine imbalance, local in lammation, trophic dysfunction, and apoptosis. A tight interconnection between all mechanisms of nervous tissue death motivates exploitation of the modulating effect that could be achieved through stimulation of the regulatory systems. Peptides are among the key players in regulatory processes [2]. An optimal scenario of therapy for IS and other cerebral disorders should comprise the application of agents simultaneously affecting several key components in the pathogenesis of the disease. Another important requirement for these compounds is the absence of toxic effects when used for prolonged preventive treatment. Due to Abstract","PeriodicalId":382788,"journal":{"name":"Journal of Neuroscience and Neurological Disorders","volume":"32 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2019-07-24","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"127987125","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 1
Long-term clinical outcome of minimally invasive versus open single-level transforaminal lumbar interbody fusion for degenerative lumbar diseases with a minimum follow-up of 2 years: A meta-analysis protocol 微创与开放单节段经椎间孔腰椎椎间融合术治疗退行性腰椎疾病的长期临床结果,至少随访2年:一项荟萃分析方案
Pub Date : 2019-07-18 DOI: 10.29328/JOURNAL.JNND.1001021
J. Heemskerk, Oluwaseun O. Akinduro, W. Clifton, Alfredo Quiñones-Hinojosa, K. Abode-Iyamah
Introduction: Minimally invasive surgical transforaminal lumbar interbody fusion (MIS-TLIF) is a relatively new approach for fusion of the lumbar spine. It was developed in addition to traditional open transforaminal interbody fusion (OTLIF) to minimize iatrogenic soft tissue damage and prevent denervation and atrophy of the paraspinal trunk musculature. Potential disadvantages of MIS-TLIF are inadequate neural element decompression and higher risk of cage migration, resulting in persistent symptoms and a need for reoperation. Other disadvantages include inadequate visualization of bony landmarks for instrumentation, and less robust postero-lateral arthrodesis than open approaches, which may affect fusion rates. Several studies have reviewed the differences between both treatments for degenerative lumbar disease, but they mainly focused on the perioperative and short-term post-operative outcomes. Since the potential disadvantages of MIS may be expected after a longer follow-up, we performed a meta-analysis comparing MISTLIF with O-TLIF for long-term outcomes.
微创手术经椎间孔腰椎椎体间融合术(MIS-TLIF)是一种相对较新的腰椎融合术。它是在传统的开放式经椎间孔椎间融合(OTLIF)的基础上发展起来的,以减少医源性软组织损伤,防止椎旁干肌肉组织的去神经支配和萎缩。MIS-TLIF的潜在缺点是神经元件减压不足和笼子移位的风险较高,导致症状持续并需要再次手术。其他的缺点包括不能很好地显示内固定的骨标志,并且后外侧关节融合术不如开放入路健壮,这可能会影响融合率。一些研究回顾了两种治疗退行性腰椎疾病的差异,但它们主要集中在围手术期和短期术后结果。由于经过较长时间的随访,MIS可能存在潜在的缺点,因此我们对MISTLIF和O-TLIF的长期结果进行了荟萃分析。
{"title":"Long-term clinical outcome of minimally invasive versus open single-level transforaminal lumbar interbody fusion for degenerative lumbar diseases with a minimum follow-up of 2 years: A meta-analysis protocol","authors":"J. Heemskerk, Oluwaseun O. Akinduro, W. Clifton, Alfredo Quiñones-Hinojosa, K. Abode-Iyamah","doi":"10.29328/JOURNAL.JNND.1001021","DOIUrl":"https://doi.org/10.29328/JOURNAL.JNND.1001021","url":null,"abstract":"Introduction: Minimally invasive surgical transforaminal lumbar interbody fusion (MIS-TLIF) is a relatively new approach for fusion of the lumbar spine. It was developed in addition to traditional open transforaminal interbody fusion (OTLIF) to minimize iatrogenic soft tissue damage and prevent denervation and atrophy of the paraspinal trunk musculature. Potential disadvantages of MIS-TLIF are inadequate neural element decompression and higher risk of cage migration, resulting in persistent symptoms and a need for reoperation. Other disadvantages include inadequate visualization of bony landmarks for instrumentation, and less robust postero-lateral arthrodesis than open approaches, which may affect fusion rates. Several studies have reviewed the differences between both treatments for degenerative lumbar disease, but they mainly focused on the perioperative and short-term post-operative outcomes. Since the potential disadvantages of MIS may be expected after a longer follow-up, we performed a meta-analysis comparing MISTLIF with O-TLIF for long-term outcomes.","PeriodicalId":382788,"journal":{"name":"Journal of Neuroscience and Neurological Disorders","volume":"20 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2019-07-18","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"125944772","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 1
Mimicking multiple sclerosis - Ghost tumor that comes and goes in different parts of the brain without any treatment 模拟多发性硬化症——不经任何治疗就在大脑不同部位来来去去的幽灵肿瘤
Pub Date : 2019-07-09 DOI: 10.29328/JOURNAL.JNND.1001020
Long Ching, M. Yuen, T. Poon, F. Cheung, S. Ting, W. Fong
{"title":"Mimicking multiple sclerosis - Ghost tumor that comes and goes in different parts of the brain without any treatment","authors":"Long Ching, M. Yuen, T. Poon, F. Cheung, S. Ting, W. Fong","doi":"10.29328/JOURNAL.JNND.1001020","DOIUrl":"https://doi.org/10.29328/JOURNAL.JNND.1001020","url":null,"abstract":"","PeriodicalId":382788,"journal":{"name":"Journal of Neuroscience and Neurological Disorders","volume":"88 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2019-07-09","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"133672778","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Role of plants, environmental toxins and physical neurotoxicological factors in Amyotrophic lateral sclerosis, Alzheimer Disease and other Neurodegenerative Diseases 植物、环境毒素和物理神经毒理学因素在肌萎缩侧索硬化症、阿尔茨海默病和其他神经退行性疾病中的作用
Pub Date : 2019-03-04 DOI: 10.29328/JOURNAL.JNND.1001019
M. Luisetto, N. Almukhtar, Ahmed Yesvi Rafa, Behzad Nili Ahmadabadi, G. Mashori, F. Khan, R. Sahu, Gamal Abdul Hamid, K. Edbey
Mauro Luisetto1*, Naseer Almukhtar2, Ahmed Yesvi Rafa3, Behzad Nili Ahmadabadi4, Ghulam Rasool Mashori5, Farhan Ahmad Khan6, Ram Kumar Sahu7, Gamal Abdul Hamid8 and Khaled Edbey9 1Applied Pharmacologist, Independent Researcher, Italy 2Professor, Physiology, College of Medicine, University of Babylon, Hilla, Iraq 3Founder and President, Yugen Research Organization (YRO), Independent Researcher, Bangladesh 4Pharm D/PhD Innovative Pharmaceutical Product Development Specialist, USA 5Professor of Pharmacology, People University of Medical & Health Sciences for Women, Nawabshah, Pakistan 6Associate Professor and Head, Department of Pharmacology, Government Medical College, Shahdol, M.P, India 7Associate Professor, Pt Deendayal Upadhyay Memorial Health Science and Ayush University of Chhattisgarh, Raipur, India 8Professor, Hematology Oncology, University of Aden, Aden, Yemen 9Professor, Department of Chemistry, University of Benghazi, Libya
Mauro Luisetto1*, Naseer Almukhtar2, Ahmed Yesvi raf3, Behzad Nili Ahmadabadi4, Ghulam Rasool Mashori5, Farhan Ahmad Khan6, Ram Kumar Sahu7, Gamal Abdul Hamid8和Khaled Edbey9 1意大利应用药理学家,独立研究员2伊拉克Hilla巴比伦大学医学院生理学教授3 Yugen研究组织(YRO)创始人兼总裁,孟加拉国药学博士,创新药物产品开发专家美国5巴基斯坦纳瓦布沙阿人民妇女医学与健康科学大学药理学教授6印度沙赫尔政府医学院药理学副教授兼系主任7印度莱普尔市Deendayal Upadhyay纪念健康科学博士和恰蒂斯加尔邦阿尤什大学副教授8也门亚丁大学血液肿瘤学教授9利比亚班加西大学化学系教授
{"title":"Role of plants, environmental toxins and physical neurotoxicological factors in Amyotrophic lateral sclerosis, Alzheimer Disease and other Neurodegenerative Diseases","authors":"M. Luisetto, N. Almukhtar, Ahmed Yesvi Rafa, Behzad Nili Ahmadabadi, G. Mashori, F. Khan, R. Sahu, Gamal Abdul Hamid, K. Edbey","doi":"10.29328/JOURNAL.JNND.1001019","DOIUrl":"https://doi.org/10.29328/JOURNAL.JNND.1001019","url":null,"abstract":"Mauro Luisetto1*, Naseer Almukhtar2, Ahmed Yesvi Rafa3, Behzad Nili Ahmadabadi4, Ghulam Rasool Mashori5, Farhan Ahmad Khan6, Ram Kumar Sahu7, Gamal Abdul Hamid8 and Khaled Edbey9 1Applied Pharmacologist, Independent Researcher, Italy 2Professor, Physiology, College of Medicine, University of Babylon, Hilla, Iraq 3Founder and President, Yugen Research Organization (YRO), Independent Researcher, Bangladesh 4Pharm D/PhD Innovative Pharmaceutical Product Development Specialist, USA 5Professor of Pharmacology, People University of Medical & Health Sciences for Women, Nawabshah, Pakistan 6Associate Professor and Head, Department of Pharmacology, Government Medical College, Shahdol, M.P, India 7Associate Professor, Pt Deendayal Upadhyay Memorial Health Science and Ayush University of Chhattisgarh, Raipur, India 8Professor, Hematology Oncology, University of Aden, Aden, Yemen 9Professor, Department of Chemistry, University of Benghazi, Libya","PeriodicalId":382788,"journal":{"name":"Journal of Neuroscience and Neurological Disorders","volume":"1 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2019-03-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"125684783","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 7
Endogenous archeological sciences: Physiology, Neuroscience, Biochemistry, Immunology, Pharmacology, Oncology and Genetics as instrument for a new field of investigation? Modern global aspects for a new discipline 内源性考古科学:生理学、神经科学、生物化学、免疫学、药理学、肿瘤学和遗传学作为一个新的研究领域?现代全球方面为一门新学科
Pub Date : 2018-11-27 DOI: 10.29328/journal.jnnd.1001017
L. M
{"title":"Endogenous archeological sciences: Physiology, Neuroscience, Biochemistry, Immunology, Pharmacology, Oncology and Genetics as instrument for a new field of investigation? Modern global aspects for a new discipline","authors":"L. M","doi":"10.29328/journal.jnnd.1001017","DOIUrl":"https://doi.org/10.29328/journal.jnnd.1001017","url":null,"abstract":"","PeriodicalId":382788,"journal":{"name":"Journal of Neuroscience and Neurological Disorders","volume":"55 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2018-11-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"130909372","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 2
Vigour of CRISPR/Cas9 Gene Editing in Alzheimer’s Disease CRISPR/Cas9基因编辑在阿尔茨海默病中的作用
Pub Date : 2018-10-05 DOI: 10.29328/journal.jnnd.1001014
P. Jes
{"title":"Vigour of CRISPR/Cas9 Gene Editing in Alzheimer’s Disease","authors":"P. Jes","doi":"10.29328/journal.jnnd.1001014","DOIUrl":"https://doi.org/10.29328/journal.jnnd.1001014","url":null,"abstract":"","PeriodicalId":382788,"journal":{"name":"Journal of Neuroscience and Neurological Disorders","volume":"7 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2018-10-05","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"127972031","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
期刊
Journal of Neuroscience and Neurological Disorders
全部 Acc. Chem. Res. ACS Applied Bio Materials ACS Appl. Electron. Mater. ACS Appl. Energy Mater. ACS Appl. Mater. Interfaces ACS Appl. Nano Mater. ACS Appl. Polym. Mater. ACS BIOMATER-SCI ENG ACS Catal. ACS Cent. Sci. ACS Chem. Biol. ACS Chemical Health & Safety ACS Chem. Neurosci. ACS Comb. Sci. ACS Earth Space Chem. ACS Energy Lett. ACS Infect. Dis. ACS Macro Lett. ACS Mater. Lett. ACS Med. Chem. Lett. ACS Nano ACS Omega ACS Photonics ACS Sens. ACS Sustainable Chem. Eng. ACS Synth. Biol. Anal. Chem. BIOCHEMISTRY-US Bioconjugate Chem. BIOMACROMOLECULES Chem. Res. Toxicol. Chem. Rev. Chem. Mater. CRYST GROWTH DES ENERG FUEL Environ. Sci. Technol. Environ. Sci. Technol. Lett. Eur. J. Inorg. Chem. IND ENG CHEM RES Inorg. Chem. J. Agric. Food. Chem. J. Chem. Eng. Data J. Chem. Educ. J. Chem. Inf. Model. J. Chem. Theory Comput. J. Med. Chem. J. Nat. Prod. J PROTEOME RES J. Am. Chem. Soc. LANGMUIR MACROMOLECULES Mol. Pharmaceutics Nano Lett. Org. Lett. ORG PROCESS RES DEV ORGANOMETALLICS J. Org. Chem. J. Phys. Chem. J. Phys. Chem. A J. Phys. Chem. B J. Phys. Chem. C J. Phys. Chem. Lett. Analyst Anal. Methods Biomater. Sci. Catal. Sci. Technol. Chem. Commun. Chem. Soc. Rev. CHEM EDUC RES PRACT CRYSTENGCOMM Dalton Trans. Energy Environ. Sci. ENVIRON SCI-NANO ENVIRON SCI-PROC IMP ENVIRON SCI-WAT RES Faraday Discuss. Food Funct. Green Chem. Inorg. Chem. Front. Integr. Biol. J. Anal. At. Spectrom. J. Mater. Chem. A J. Mater. Chem. B J. Mater. Chem. C Lab Chip Mater. Chem. Front. Mater. Horiz. MEDCHEMCOMM Metallomics Mol. Biosyst. Mol. Syst. Des. Eng. Nanoscale Nanoscale Horiz. Nat. Prod. Rep. New J. Chem. Org. Biomol. Chem. Org. Chem. Front. PHOTOCH PHOTOBIO SCI PCCP Polym. Chem.
×
引用
GB/T 7714-2015
复制
MLA
复制
APA
复制
导出至
BibTeX EndNote RefMan NoteFirst NoteExpress
×
0
微信
客服QQ
Book学术公众号 扫码关注我们
反馈
×
意见反馈
请填写您的意见或建议
请填写您的手机或邮箱
×
提示
您的信息不完整,为了账户安全,请先补充。
现在去补充
×
提示
您因"违规操作"
具体请查看互助需知
我知道了
×
提示
现在去查看 取消
×
提示
确定
Book学术官方微信
Book学术文献互助
Book学术文献互助群
群 号:481959085
Book学术
文献互助 智能选刊 最新文献 互助须知 联系我们:info@booksci.cn
Book学术提供免费学术资源搜索服务,方便国内外学者检索中英文文献。致力于提供最便捷和优质的服务体验。
Copyright © 2023 Book学术 All rights reserved.
ghs 京公网安备 11010802042870号 京ICP备2023020795号-1