{"title":"Update on Antiarrhythmic Therapy: Dofetilide","authors":"Q. Wang, H. Tse, C. Lau","doi":"10.55503/2790-6744.1466","DOIUrl":"https://doi.org/10.55503/2790-6744.1466","url":null,"abstract":"","PeriodicalId":53534,"journal":{"name":"Journal of the Hong Kong College of Cardiology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2022-07-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"47047606","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
K. Moroe, Tadayuki Hiroki, K. Miyoshi, T. Wakiyama
{"title":"Electrophysiologic Mechanism of Conversion of Atrial Fibrillation to Sinus Rhythm","authors":"K. Moroe, Tadayuki Hiroki, K. Miyoshi, T. Wakiyama","doi":"10.55503/2790-6744.1385","DOIUrl":"https://doi.org/10.55503/2790-6744.1385","url":null,"abstract":"","PeriodicalId":53534,"journal":{"name":"Journal of the Hong Kong College of Cardiology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2022-07-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"43360290","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"3rd Annual Scientific Meeting Abstracts for Oral Presentation","authors":"","doi":"10.55503/2790-6744.1470","DOIUrl":"https://doi.org/10.55503/2790-6744.1470","url":null,"abstract":"","PeriodicalId":53534,"journal":{"name":"Journal of the Hong Kong College of Cardiology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2022-07-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"46852060","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Cardiovascular Disease Prevention: What Makes an Intervention\u0000Worthwhile?","authors":"Cyrus R. Kumana, B. M. Cheung","doi":"10.55503/2790-6744.1463","DOIUrl":"https://doi.org/10.55503/2790-6744.1463","url":null,"abstract":"","PeriodicalId":53534,"journal":{"name":"Journal of the Hong Kong College of Cardiology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2022-07-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"42537711","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
LEUNG, ET AL : Drainage of Pericardial Effusions: Percutaneous or Surgical? Background: There are 2 modes of drainage of pericardial effusions: percutaneous pericardiocentesis (PP) and surgical pericardiotomy (SP). Certain features, which are indicative of cardiac tamponade, should guide us to the choice of drainage of pericardial effusions. It is unclear if this is followed in clinical practice. Objective: To review patients with pericardial drainage done for a period of 36 months, and to correlate the features with the choice of drainage mode. Results: 39 patients and 47 procedures (20 PP and 27 SP) were reviewed. Several clinical features were correlated with the use of PP: dyspnoea on exertion, dyspnoea at rest, orthopnoea, tachycardia, elevated jugular venous pressure and hypotension. Pulsus paradoxus was not adequately checked. Electrocardiographic and radiographic findings were similar between the 2 modes. 91% of the patients had large effusions by echocardiography. Right atrial collapse and right ventricular collapse occurred more frequently in the group of PP. Conclusion: Some features were more relied by us to indicate the presence of cardiac tamponade. Their presence should lead us more to the use of percutaneous drainage and they should be checked thoroughly. The decision to drain and the selection should be assessed after an integration of all these features. (J HK Coll Cardiol 1999;7:104-108)
{"title":"Drainage of Pericardial Effusions: Percutaneous or Surgical?","authors":"Albert Wai-Suen Leung, N. Chan, L. Ho, WAI-CHO Yu","doi":"10.55503/2790-6744.1389","DOIUrl":"https://doi.org/10.55503/2790-6744.1389","url":null,"abstract":"LEUNG, ET AL : Drainage of Pericardial Effusions: Percutaneous or Surgical? Background: There are 2 modes of drainage of pericardial effusions: percutaneous pericardiocentesis (PP) and surgical pericardiotomy (SP). Certain features, which are indicative of cardiac tamponade, should guide us to the choice of drainage of pericardial effusions. It is unclear if this is followed in clinical practice. Objective: To review patients with pericardial drainage done for a period of 36 months, and to correlate the features with the choice of drainage mode. Results: 39 patients and 47 procedures (20 PP and 27 SP) were reviewed. Several clinical features were correlated with the use of PP: dyspnoea on exertion, dyspnoea at rest, orthopnoea, tachycardia, elevated jugular venous pressure and hypotension. Pulsus paradoxus was not adequately checked. Electrocardiographic and radiographic findings were similar between the 2 modes. 91% of the patients had large effusions by echocardiography. Right atrial collapse and right ventricular collapse occurred more frequently in the group of PP. Conclusion: Some features were more relied by us to indicate the presence of cardiac tamponade. Their presence should lead us more to the use of percutaneous drainage and they should be checked thoroughly. The decision to drain and the selection should be assessed after an integration of all these features. (J HK Coll Cardiol 1999;7:104-108)","PeriodicalId":53534,"journal":{"name":"Journal of the Hong Kong College of Cardiology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2022-07-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"45928055","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
ET AL.: Role of Angiotensin II Subtype 1 Receptor in Smooth Muscle Cells Apoptosis after Balloon Injury to Vessel. In order to investigate the mechanism of smooth muscle cells apoptosis after balloon injury to vessel, terminal deoxynucleotidyl transferase-mediated dUTP-biotin nick end labeling (TUNEL) and immunohistochemistry were adopted to measure the percentage of vascular smooth muscle cell (VSMC) apoptosis and the expression of angiotensin II subtype 1 receptor (AT 1 R). Compared with sham's, the expression of AT 1 R protein in vascular media was significantly increased at 3 days after balloon injury (P<0.05), and then did not change significantly . At 7 days, it was approximately twice in intima as that in media. At 28 days it reached its peak. VSMCs apoptosis occurred in vascular media at 3 days after balloon injury and reached a peak in media and intima where apoptosis was mainly present at 7 days, then decreased. At 28 days after balloon injury, only a few apoptotic cells were in intima. AT 1 R antagonist (irbesartan) significantly increased VSMCs apoptosis. We thus conclude that AT 1 R is upregulated after balloon injury and angiotensin II inhibits VSMC apoptosis by binding to AT 1 R. (J HK Coll Cardiol 2000;8:49-57) Angiotensin II, receptor, vascular smooth muscle, apoptosis
血管紧张素II亚型1受体在血管球囊损伤后平滑肌细胞凋亡中的作用。为了探讨血管球囊损伤后平滑肌细胞凋亡的机制，采用末端脱氧核苷酸转移酶介导的dUTP生物素缺口末端标记（TUNEL）和免疫组织化学方法检测血管平滑肌细胞（VSMC）凋亡百分比和血管紧张素II亚型1受体（AT1R）的表达。与假手术组相比，球囊损伤后3d血管介质中AT1R蛋白的表达显著增加（P＜0.05），但无明显变化。在第7天，内膜中的表达量大约是中膜中表达量的两倍。第28天达到高峰。球囊损伤后第3天，VSMCs在血管介质中出现凋亡，第7天，VSMC在以凋亡为主的介质和内膜中达到峰值，然后逐渐减少。球囊损伤后28天，内膜中仅有少量凋亡细胞。AT1R拮抗剂（厄贝沙坦）显著增加VSMCs凋亡。因此，我们得出结论，球囊损伤后AT1R上调，血管紧张素II通过与AT1R结合抑制VSMC凋亡。（J HK Coll Cardiol 2000；8:49-57）血管紧张素Ⅱ，受体，血管平滑肌，细胞凋亡
{"title":"Role of Angiotensin II Subtype 1 Receptor in Smooth Muscle Cells Apoptosis after Balloon Injury to Vessel","authors":"Geng M. Wang, G. He, Xiao-hui Xu, GUO-CHAO Wang","doi":"10.55503/2790-6744.1465","DOIUrl":"https://doi.org/10.55503/2790-6744.1465","url":null,"abstract":"ET AL.: Role of Angiotensin II Subtype 1 Receptor in Smooth Muscle Cells Apoptosis after Balloon Injury to Vessel. In order to investigate the mechanism of smooth muscle cells apoptosis after balloon injury to vessel, terminal deoxynucleotidyl transferase-mediated dUTP-biotin nick end labeling (TUNEL) and immunohistochemistry were adopted to measure the percentage of vascular smooth muscle cell (VSMC) apoptosis and the expression of angiotensin II subtype 1 receptor (AT 1 R). Compared with sham's, the expression of AT 1 R protein in vascular media was significantly increased at 3 days after balloon injury (P<0.05), and then did not change significantly . At 7 days, it was approximately twice in intima as that in media. At 28 days it reached its peak. VSMCs apoptosis occurred in vascular media at 3 days after balloon injury and reached a peak in media and intima where apoptosis was mainly present at 7 days, then decreased. At 28 days after balloon injury, only a few apoptotic cells were in intima. AT 1 R antagonist (irbesartan) significantly increased VSMCs apoptosis. We thus conclude that AT 1 R is upregulated after balloon injury and angiotensin II inhibits VSMC apoptosis by binding to AT 1 R. (J HK Coll Cardiol 2000;8:49-57) Angiotensin II, receptor, vascular smooth muscle, apoptosis","PeriodicalId":53534,"journal":{"name":"Journal of the Hong Kong College of Cardiology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2022-07-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"49560844","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Implantable Cardioverter Defibrillator: Clinical Performance of Dual Chamber versus Single Chamber Devices","authors":"Katherine Fan, Kathy Lee, E. Chau, A. Yip","doi":"10.55503/2790-6744.1387","DOIUrl":"https://doi.org/10.55503/2790-6744.1387","url":null,"abstract":"","PeriodicalId":53534,"journal":{"name":"Journal of the Hong Kong College of Cardiology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2022-07-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"43300002","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Cardiac Rehabilitation Service in Hong Kong","authors":"S. Lau, Leonard S. W. Li, Hang-sau Ng, C. Lau","doi":"10.55503/2790-6744.1404","DOIUrl":"https://doi.org/10.55503/2790-6744.1404","url":null,"abstract":"","PeriodicalId":53534,"journal":{"name":"Journal of the Hong Kong College of Cardiology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2022-07-26","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"42389834","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Vol 8 Issue 4 Front Matter","authors":"","doi":"10.55503/2790-6744.1381","DOIUrl":"https://doi.org/10.55503/2790-6744.1381","url":null,"abstract":"","PeriodicalId":53534,"journal":{"name":"Journal of the Hong Kong College of Cardiology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2022-07-26","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"46786125","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Eighth Annual Scientific Congress Front Matter","authors":"","doi":"10.55503/2790-6744.1418","DOIUrl":"https://doi.org/10.55503/2790-6744.1418","url":null,"abstract":"","PeriodicalId":53534,"journal":{"name":"Journal of the Hong Kong College of Cardiology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"2022-07-26","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"44852824","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
京公网安备 11010802042870号
京ICP备2023020795号-1
扫码关注我们
求助内容：
应助结果提醒方式：