World Allergy Organization Journal最新文献

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Cutaneous Manifestations in Primary Immunodeficiency Diseases: A Comprehensive Review 原发性免疫缺陷疾病的皮肤表现：综述

IF 4.3 2区医学 Q2 ALLERGY

World Allergy Organization Journal

Pub Date : 2026-02-01 Epub Date: 2026-03-05 DOI: 10.1016/j.waojou.2026.101282

Samin Sharafian , Zahra Chavoshzadeh

引用次数: 0

Biologic Therapies in ENT: A Systematic Review of Efficacy, Safety, and Outcomes 耳鼻喉科生物疗法：疗效、安全性和结果的系统综述

IF 4.3 2区医学 Q2 ALLERGY

World Allergy Organization Journal

Pub Date : 2026-02-01 Epub Date: 2026-03-05 DOI: 10.1016/j.waojou.2026.101326

Zainab Alsalloom, Zahraa Adel Mohamed, Esra Qadami, Alaa Mandeel, Mahmood Alowainati

引用次数: 0

Increased glucose transporter 1 contributes to epithelial barrier dysfunction in allergic rhinitis 葡萄糖转运蛋白1升高与变应性鼻炎上皮屏障功能障碍有关

IF 4.3 2区医学 Q2 ALLERGY

World Allergy Organization Journal

Pub Date : 2026-01-01 Epub Date: 2026-01-05 DOI: 10.1016/j.waojou.2025.101158

Cui Xia PhD , Kang Zhu MD , Chao Yu MD , Jingguo Chen MD , Tianxi Gao MD , Yanni Zhang MD

Background

Epithelial barrier impairment is a characteristic pathological hallmark of allergic rhinitis (AR), yet the mechanisms contributing to this dysfunction remain incompletely understood. Our aim is to assess the impact of glucose transporter 1 (GLUT1) on epithelial barrier function in AR.

Methods

We performed proteomics analysis on nasal mucosa from AR patients and healthy controls (HCs) to identify differential proteins, which were validated using immunofluorescence, western blotting, and RT-qPCR. Nasal epithelial cells from HCs were isolated to evaluate house dust mite (HDM)-induced changes in GLUT1 expression and regulatory mechanisms. An AR mouse model was constructed to examine the effects of GLUT1 inhibition on nasal inflammation and barrier function.

Results

Tissue proteomics analysis revealed a distinct protein expression profile in AR patients compared with HCs, with GLUT1 identified as the most upregulated protein. Cohort validation demonstrated significantly elevated GLUT1 expression in the AR group, predominantly localized in epithelial cells. Moreover, GLUT1 mRNA levels showed a positive correlation with visual analogue scales and total nasal symptom scores, and a negative correlation with tissue ZO-1 and occludin expressions. In vitro studies indicated that HDM stimulation enhanced GLUT1 expression and reduced ZO-1 and occludin levels in nasal epithelial cells in a dose-dependent manner. Treatment with a GLUT1 inhibitor effectively restored ZO-1 and occludin expressions. Animal model experiments further confirmed that GLUT1 inhibition alleviated nasal inflammation and improved mucosal barrier function.

Conclusion

AR displays a distinct tissue-specific protein expression profile, with enhanced GLUT1 levels associated with disease severity and epithelial barrier dysfunction. Inhibition of GLUT1 has been shown to reduce nasal inflammation and improve epithelial barrier function in AR.

上皮屏障损伤是变应性鼻炎（AR）的一个特征性病理标志，但导致这种功能障碍的机制仍不完全清楚。我们的目的是评估葡萄糖转运蛋白1 （GLUT1）对AR上皮屏障功能的影响。方法我们对AR患者和健康对照（hc）的鼻黏膜进行蛋白质组学分析，以鉴定差异蛋白，并使用免疫荧光、western blotting和RT-qPCR验证。分离hc鼻上皮细胞，研究屋尘螨（HDM）诱导的GLUT1表达变化及其调控机制。建立AR小鼠模型，观察GLUT1抑制对鼻部炎症和屏障功能的影响。结果组织蛋白质组学分析显示，与hcc相比，AR患者的蛋白表达谱明显不同，其中GLUT1被确定为上调最多的蛋白。队列验证表明，在AR组中，GLUT1的表达显著升高，主要集中在上皮细胞中。GLUT1 mRNA水平与视觉模拟量表和鼻症状总分呈正相关，与组织ZO-1和occludin表达呈负相关。体外研究表明，HDM刺激增强了鼻上皮细胞中GLUT1的表达，降低了ZO-1和occludin的水平，并呈剂量依赖性。用GLUT1抑制剂治疗可有效恢复ZO-1和occludin的表达。动物模型实验进一步证实，抑制GLUT1可减轻鼻部炎症，改善粘膜屏障功能。结论ar表现出独特的组织特异性蛋白表达谱，GLUT1水平升高与疾病严重程度和上皮屏障功能障碍相关。抑制GLUT1已被证明可减少鼻炎并改善AR的上皮屏障功能。

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引用次数: 0

IF 4.3 2区医学 Q2 ALLERGY

World Allergy Organization Journal

Pub Date : 2026-01-01

引用次数: 0

Characterization of Pana g 1, an important cause of pollen-food allergy syndrome from Korean ginseng, Panax ginseng 人参花粉食物过敏综合征的重要致病因子Pana g1的鉴定

IF 4.3 2区医学 Q2 ALLERGY

World Allergy Organization Journal

Pub Date : 2026-01-01 Epub Date: 2026-01-03 DOI: 10.1016/j.waojou.2025.101164

Kyoung Yong Jeong PhD , Yoon Ji Shin BSc , Haeun Kim BSc , Yong Seok Lee PhD , Minkyu Sang PhD , Hyun Kyung Oh MD , Kyung Hee Park MD, PhD , Jae-Hyun Lee MD, PhD , Jung-Won Park MD, PhD

Background

Ginseng is a widely consumed herbal supplement. However, ginseng, especially raw ginseng, can cause allergic reactions, including pollen-food allergy syndrome (PFAS). This study aimed to identify the PFAS-causative allergen in Korean ginseng and to establish methods for its quantification.

Methods

Candidate allergens were screened using genomic and transcriptomic analyses. Proteomic profiling with patient sera was performed to identify the clinically relevant allergen. A recombinant protein was generated, and its allergenicity compared with the primary sensitizer, Que ac 1, by ELISA. A two-site ELISA was developed for the quantification of the ginseng allergen using monoclonal antibodies against recombinant protein. Multiple reaction monitoring (MRM) mass spectrometry was applied for validation.

Results

Genome and transcriptome analysis identified 4 candidate allergens: pathogenesis-related 10 (PR-10) protein, profilin, non-specific lipid transfer protein (nsLTP), and thaumatin-like protein. Among these, PR-10 (designated Pana g 1) was the sole allergen detected by proteomic analysis. Recombinant Pana g 1 was recognized by 4 of 5 patients. Inhibition ELISA showed stronger IgE reactivity to Que ac 1 than to Pana g 1, with marked cross-reactivity between the 2. Pana g 1 levels in ginseng extract were quantified as 4.26 μg/mg of protein by ELISA and 4.54 μg/mg by MRM in the ginseng extract.

Conclusion

Pana g 1 is the major PFAS-causative allergen in Korean ginseng. Recombinant Pana g 1 shows promise as a diagnostic tool for ginseng-induced PFAS. The quantification systems established here may also support standardization of ginseng extracts and allergen monitoring.

人参是一种被广泛食用的草药补充剂。然而，人参，尤其是生人参，会引起过敏反应，包括花粉食物过敏综合征（PFAS）。本研究旨在鉴定红参中诱发pfas的过敏原，并建立其定量方法。方法采用基因组学和转录组学分析筛选候选过敏原。对患者血清进行蛋白质组学分析，以确定临床相关的过敏原。制备了重组蛋白，并通过ELISA对其与原致敏剂Que ac 1的致敏性进行了比较。利用重组蛋白单克隆抗体，建立了人参过敏原的双位点ELISA定量方法。采用多反应监测（MRM）质谱法进行验证。结果基因组和转录组分析鉴定出4种候选过敏原：发病相关10蛋白（PR-10）、profilin、非特异性脂质转移蛋白（nsLTP）和thaumatin样蛋白。其中PR-10（命名为Pana g1）是唯一通过蛋白质组学分析检测到的过敏原。重组Pana g1在5例患者中被4例识别。抑制酶联免疫吸附试验显示，IgE对Que ac 1的反应性强于对Pana g1的反应性，且两者之间存在明显的交叉反应。ELISA法测定人参提取物中Pana g 1蛋白含量为4.26 μg/mg， MRM法测定人参提取物中Pana g 1蛋白含量为4.54 μg/mg。结论panag1是红参中主要的致pfas变应原。重组panag1有望作为人参诱导的PFAS的诊断工具。这里建立的定量系统也可以支持人参提取物和过敏原监测的标准化。

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引用次数: 0

IF 4.3 2区医学 Q2 ALLERGY

World Allergy Organization Journal

Pub Date : 2026-01-01

引用次数: 0

IF 4.3 2区医学 Q2 ALLERGY

World Allergy Organization Journal

Pub Date : 2026-01-01

引用次数: 0

The exploration of immunological landscape and drug targets in chronic rhinosinusitis with nasal polyps 慢性鼻窦炎伴鼻息肉的免疫景观及药物靶点探讨

IF 4.3 2区医学 Q2 ALLERGY

World Allergy Organization Journal

Pub Date : 2026-01-01 Epub Date: 2025-12-15 DOI: 10.1016/j.waojou.2025.101140

Ruxiang Zhang PhD, Peipei Fu PhD, Hao Tian PhD

Background

Chronic rhinosinusitis with nasal polyps (CRSwNP) is a prevalent inflammatory disorder characterized by nasal obstruction and polyp formation. Despite its prevalence, the complex pathogenesis of CRSwNP remains not fully understood, hindering the development of effective treatments. This study aims to delineate the immunological landscape of CRSwNP by integrating single-cell RNA sequencing (scRNA-seq) and Mendelian randomization (MR) approaches.

Methods

We conducted a systematic MR analysis using summary statistics from genome-wide association studies (GWAS) and expression quantitative trait loci (eQTL) data. The identified genes were further scrutinized through scRNA-seq analysis of CRSwNP tissues to assess cell-specific expression patterns. Pathway enrichment and protein-protein interaction (PPI) network analyses were performed to explore the biological mechanisms underlying CRSwNP.

Results

The MR analysis identified several genes, including HLA-DRB1, HLA-DQA1, and HLA-DQB1, as significantly associated with CRSwNP. The scRNA-seq analysis validated these findings, revealing cell-specific enrichment in basal cells. Notably, these genes were found to be involved in immune cell recruitment and the reshaping of the immune microenvironment. Furthermore, the study highlighted the role of genes like TCF7L1, KANSL1-AS1, and POLR2J3, which showed contrasting expression patterns and potential regulatory roles in CRSwNP.

Conclusion

This integrative study provides novel insights into the molecular and cellular underpinnings of CRSwNP. The identified genes and their role in immunopathogenesis offer potential therapeutic targets and highlight the importance of cell-specific gene expression in disease mechanisms. The combination of MR with scRNA-seq represents a powerful approach to elucidate complex traits and may pave the way for precision medicine in CRSwNP management.

慢性鼻窦炎伴鼻息肉（CRSwNP）是一种以鼻塞和息肉形成为特征的常见炎症性疾病。尽管CRSwNP普遍存在，但其复杂的发病机制仍未完全了解，这阻碍了有效治疗方法的发展。本研究旨在通过整合单细胞RNA测序（scRNA-seq）和孟德尔随机化（MR）方法来描绘CRSwNP的免疫学景观。方法利用全基因组关联研究（GWAS）和表达数量性状位点（eQTL）数据的汇总统计数据进行系统的MR分析。通过CRSwNP组织的scRNA-seq分析进一步仔细检查鉴定的基因，以评估细胞特异性表达模式。通过通路富集和蛋白-蛋白相互作用（PPI）网络分析来探索CRSwNP的生物学机制。结果MR分析确定了几个基因，包括HLA-DRB1、HLA-DQA1和HLA-DQB1，与CRSwNP显著相关。scRNA-seq分析证实了这些发现，揭示了基底细胞中的细胞特异性富集。值得注意的是，这些基因被发现参与免疫细胞募集和免疫微环境的重塑。此外，该研究强调了TCF7L1、KANSL1-AS1和POLR2J3等基因在CRSwNP中的作用，这些基因在CRSwNP中表现出不同的表达模式和潜在的调节作用。这项综合研究为CRSwNP的分子和细胞基础提供了新的见解。已鉴定的基因及其在免疫发病机制中的作用提供了潜在的治疗靶点，并突出了细胞特异性基因表达在疾病机制中的重要性。MR与scRNA-seq的结合代表了一种阐明复杂性状的有力方法，并可能为CRSwNP管理的精准医学铺平道路。

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引用次数: 0

IF 4.3 2区医学 Q2 ALLERGY

World Allergy Organization Journal

Pub Date : 2026-01-01

引用次数: 0

IF 4.3 2区医学 Q2 ALLERGY

World Allergy Organization Journal

Pub Date : 2026-01-01

引用次数: 0

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