Pub Date : 2015-09-23DOI: 10.1309/YVAD9JBUFGVGAAVP
Ya-ni Feng, T. Bai, Hong Ma, Jun-ke Wang
Background We undertook a study to determine whether propofol pretreatment may protect human proximal renal tubular epithelial (HK-2) cells against anoxia-reoxygenation injury and the role of p38 mitogen-activited protein kinase (p38MAPK). Methods HK-2 cells were randomly divided into 5 groups: a control group (Con); a group treated for 1 hour, cultured with with 300 μM CoCl2 normal medium for 24 hours, then stimulated with no serum media (group CoCl2); a group where the HK-2 cells were pretreated with 5μg/mL−1 propofol for 1 hour, then were treated as group (group Pro); a group where we pretreated CoCl2 the HK-2 cells with 10% intralipid 90 μL for 1 (group Int); hour, then treated as group CoCl2 and a group where we pretreated the HK-2 cells with propofol for 1 hour, 10 μmol/L−1SB203580 were (inhibitor of p38MAPK), and 300 μM CoCl2 added at the same time, then cultured with normal medium for 24 hours (group SB). Using MTT method and flow cytometry to detect the proliferation and apoptosis of HK-2 cells, the reverse transcriptase-polymerase chain reaction method was used to investigate the regulation of Bcl-2 and caspase-3 mRNA; the expression of p38MAPK was measured by Western blot. Results After pretreated with 5 μg/mL−1 propofol, HK-2 cell proliferation increased and apoptosis decreased ( P <0.05 or 0.01). Intralipid did not effect HK-2 cells, the expression of Bcl-2 gene was upregulated, and caspase-3 gene was down-regulated. Meanwhile, the expression of p38MAPK was upregulated after propofol pretreament. These regulations can be reversed by SB203580 ( P <0.05 or 0.01) with no difference between group Int and group CoCl2. Conclusion Pretreatment with 5μg/mL−1 propofol protected HK-2 cells against anoxia-reoxygenation injury at clinically relevant concentrations by regulating the expression of apoptosis related genes. P38MAPK plays an important role in propofol protective effect.
{"title":"Propofol Attenuates Human Proximal Renal Tubular Epithelial Cell Injury Induced by Anoxia-Reoxygenation","authors":"Ya-ni Feng, T. Bai, Hong Ma, Jun-ke Wang","doi":"10.1309/YVAD9JBUFGVGAAVP","DOIUrl":"https://doi.org/10.1309/YVAD9JBUFGVGAAVP","url":null,"abstract":"Background We undertook a study to determine whether propofol pretreatment may protect human proximal renal tubular epithelial (HK-2) cells against anoxia-reoxygenation injury and the role of p38 mitogen-activited protein kinase (p38MAPK).\u0000\u0000Methods HK-2 cells were randomly divided into 5 groups: a control group (Con); a group treated for 1 hour, cultured with with 300 μM CoCl2 normal medium for 24 hours, then stimulated with no serum media (group CoCl2); a group where the HK-2 cells were pretreated with 5μg/mL−1 propofol for 1 hour, then were treated as group (group Pro); a group where we pretreated CoCl2 the HK-2 cells with 10% intralipid 90 μL for 1 (group Int); hour, then treated as group CoCl2 and a group where we pretreated the HK-2 cells with propofol for 1 hour, 10 μmol/L−1SB203580 were (inhibitor of p38MAPK), and 300 μM CoCl2 added at the same time, then cultured with normal medium for 24 hours (group SB). Using MTT method and flow cytometry to detect the proliferation and apoptosis of HK-2 cells, the reverse transcriptase-polymerase chain reaction method was used to investigate the regulation of Bcl-2 and caspase-3 mRNA; the expression of p38MAPK was measured by Western blot.\u0000\u0000Results After pretreated with 5 μg/mL−1 propofol, HK-2 cell proliferation increased and apoptosis decreased ( P <0.05 or 0.01). Intralipid did not effect HK-2 cells, the expression of Bcl-2 gene was upregulated, and caspase-3 gene was down-regulated. Meanwhile, the expression of p38MAPK was upregulated after propofol pretreament. These regulations can be reversed by SB203580 ( P <0.05 or 0.01) with no difference between group Int and group CoCl2.\u0000\u0000Conclusion Pretreatment with 5μg/mL−1 propofol protected HK-2 cells against anoxia-reoxygenation injury at clinically relevant concentrations by regulating the expression of apoptosis related genes. P38MAPK plays an important role in propofol protective effect.","PeriodicalId":54328,"journal":{"name":"Labmedicine","volume":"104 1","pages":"356-360"},"PeriodicalIF":0.0,"publicationDate":"2015-09-23","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"74286253","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-09-23DOI: 10.1309/1L5W3YXVTT5L98W9
M. Pérez, L. Bush, S. Rodriguez
1. Important clinical information includes neck pain, the absence of fever or focal neurologic signs, and syncopal episode. The most striking laboratory findings include the abnormal chemical and cytologic analysis of the cerebrospinal fluid (CSF) sample (hypoglycorrhachia, proteinorrhachia, and pleocytosis) and mild hyperglycemia. 2. Hypoglycorrhachia: The CSF is normally a clear, colorless, and virtually acellular fluid that is actively produced by the choroid plexus and ependymal cells lining the central nervous system (CNS) ventricles and subarachnoidal space through a process of ultrafiltration of plasma. Glucose levels in the CSF are 60% to 70% those of the serum; therefore, comparison between both values is paramount in interpreting abnormal results. Increased CSF glucose (hyperglycorrhachia) may be seen in the context of hyperglycemia or be due to a traumatic tap. Conversely, decreased CSF glucose (hypoglycorrhachia) is commonly found in untreated bacterial, fungal, tuberculous, or amoebic meningitis, and rarely in some viral CNS infections (particulary those caused by mumps and lymphocytic choriomeningitis viruses). Other causes of hypoglycorrhachia include CNS sarcoidosis and neoplasms (primary or metastatic). Proteinorrhachia: The normal CSF protein level is about 1% of the serum protein level. Increased levels are seen with most infections involving the meninges and the CNS, but are Clinical History
{"title":"Head and Neck Pain, Syncopal Episode, Abnormal Cerebrospinal Fluid, and Hyponatremia in a Middle-Aged Woman","authors":"M. Pérez, L. Bush, S. Rodriguez","doi":"10.1309/1L5W3YXVTT5L98W9","DOIUrl":"https://doi.org/10.1309/1L5W3YXVTT5L98W9","url":null,"abstract":"1. Important clinical information includes neck pain, the absence of fever or focal neurologic signs, and syncopal episode. The most striking laboratory findings include the abnormal chemical and cytologic analysis of the cerebrospinal fluid (CSF) sample (hypoglycorrhachia, proteinorrhachia, and pleocytosis) and mild hyperglycemia. 2. Hypoglycorrhachia: The CSF is normally a clear, colorless, and virtually acellular fluid that is actively produced by the choroid plexus and ependymal cells lining the central nervous system (CNS) ventricles and subarachnoidal space through a process of ultrafiltration of plasma. Glucose levels in the CSF are 60% to 70% those of the serum; therefore, comparison between both values is paramount in interpreting abnormal results. Increased CSF glucose (hyperglycorrhachia) may be seen in the context of hyperglycemia or be due to a traumatic tap. Conversely, decreased CSF glucose (hypoglycorrhachia) is commonly found in untreated bacterial, fungal, tuberculous, or amoebic meningitis, and rarely in some viral CNS infections (particulary those caused by mumps and lymphocytic choriomeningitis viruses). Other causes of hypoglycorrhachia include CNS sarcoidosis and neoplasms (primary or metastatic). Proteinorrhachia: The normal CSF protein level is about 1% of the serum protein level. Increased levels are seen with most infections involving the meninges and the CNS, but are Clinical History","PeriodicalId":54328,"journal":{"name":"Labmedicine","volume":"15 1","pages":"337-340"},"PeriodicalIF":0.0,"publicationDate":"2015-09-23","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"87632522","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-09-23DOI: 10.1309/0TQB1HAUX9JFU1B0
C. Storm, O. Danne, C. Lueders, U. Frei, M. Möckel
Background Whole blood choline (WBCHO) concentrations reflect phospholipase D activation pathways involved in coronary plaque vulnerability and platelet activation suppressed by glycoprotein IIb/IIIa receptor (GP IIb/IIIa) inhibitors, but it is unknown whether this treatment affects serial WBCHO levels. Methods We performed a retrospective matched pairs analysis of 32 patients with acute coronary syndrome treated either with standard therapy alone or with tirofiban plus standard therapy with serial measurements of WBCHO. Results The median level of choline decreased after 4 to 6 hours in the tirofiban group (21.2 μmol/L) while the median level in the group without tirofiban increased (29.3 μmol/L) and WBCHO was significantly lower in tirofiban treated patients ( P =0.039). Over time, the decrease of WBCHO was significant in the tirofiban group only ( P =0.006). Conclusions Our results suggest that in patients with acute coronary syndrome, tirofiban treatment is associated with a significant reduction of WBCHO concentration.
{"title":"Effect of the Glycoprotein IIb/IIIa Inhibitor Tirofiban on Concentrations of Whole Blood Choline in Acute Coronary Syndromes","authors":"C. Storm, O. Danne, C. Lueders, U. Frei, M. Möckel","doi":"10.1309/0TQB1HAUX9JFU1B0","DOIUrl":"https://doi.org/10.1309/0TQB1HAUX9JFU1B0","url":null,"abstract":"Background Whole blood choline (WBCHO) concentrations reflect phospholipase D activation pathways involved in coronary plaque vulnerability and platelet activation suppressed by glycoprotein IIb/IIIa receptor (GP IIb/IIIa) inhibitors, but it is unknown whether this treatment affects serial WBCHO levels.\u0000\u0000Methods We performed a retrospective matched pairs analysis of 32 patients with acute coronary syndrome treated either with standard therapy alone or with tirofiban plus standard therapy with serial measurements of WBCHO.\u0000\u0000Results The median level of choline decreased after 4 to 6 hours in the tirofiban group (21.2 μmol/L) while the median level in the group without tirofiban increased (29.3 μmol/L) and WBCHO was significantly lower in tirofiban treated patients ( P =0.039). Over time, the decrease of WBCHO was significant in the tirofiban group only ( P =0.006).\u0000\u0000Conclusions Our results suggest that in patients with acute coronary syndrome, tirofiban treatment is associated with a significant reduction of WBCHO concentration.","PeriodicalId":54328,"journal":{"name":"Labmedicine","volume":"48 1","pages":"349-355"},"PeriodicalIF":0.0,"publicationDate":"2015-09-23","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"87978173","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-09-23DOI: 10.1309/QDNQCF9JP22WK96E
M. Sealfon, Seth Franklin
{"title":"Insidious Onset of Visual Disturbances in a Healthy 56-Year-Old Man","authors":"M. Sealfon, Seth Franklin","doi":"10.1309/QDNQCF9JP22WK96E","DOIUrl":"https://doi.org/10.1309/QDNQCF9JP22WK96E","url":null,"abstract":"","PeriodicalId":54328,"journal":{"name":"Labmedicine","volume":"17 1","pages":"343-346"},"PeriodicalIF":0.0,"publicationDate":"2015-09-23","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"77163163","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-09-23DOI: 10.1309/UQP7HTR8EATFQHVM
K. Ward, Leigh Ann McInnis
{"title":"Managing Workforce Health and Wellness","authors":"K. Ward, Leigh Ann McInnis","doi":"10.1309/UQP7HTR8EATFQHVM","DOIUrl":"https://doi.org/10.1309/UQP7HTR8EATFQHVM","url":null,"abstract":"","PeriodicalId":54328,"journal":{"name":"Labmedicine","volume":"37 1","pages":"325-331"},"PeriodicalIF":0.0,"publicationDate":"2015-09-23","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"72978088","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-09-23DOI: 10.1309/EWKEBC48R8KYETR3
A. Haddad, J. Maciejewski, M. Kalaycio
The myelodysplastic syndromes (MDSs) are a heterogeneous group of diseases characterized by hemopoietic insufficiency associated with cytopenias due to dysfunctional hematopoietic stem cells. Myelodysplastic syndromes sometimes progress to acute myelogenous leukemia. The cytopenias caused by MDS lead to serious complications such as bleeding, infections, and symptomatic anemia. Cytogenetic abnormalities in MDS patients have an important role in categorizing these syndromes and are important in predicting prognosis and treatment response.
{"title":"Clinical Significance of Cytogenetics in Myelodysplastic Syndromes","authors":"A. Haddad, J. Maciejewski, M. Kalaycio","doi":"10.1309/EWKEBC48R8KYETR3","DOIUrl":"https://doi.org/10.1309/EWKEBC48R8KYETR3","url":null,"abstract":"The myelodysplastic syndromes (MDSs) are a heterogeneous group of diseases characterized by hemopoietic insufficiency associated with cytopenias due to dysfunctional hematopoietic stem cells. Myelodysplastic syndromes sometimes progress to acute myelogenous leukemia. The cytopenias caused by MDS lead to serious complications such as bleeding, infections, and symptomatic anemia. Cytogenetic abnormalities in MDS patients have an important role in categorizing these syndromes and are important in predicting prognosis and treatment response.","PeriodicalId":54328,"journal":{"name":"Labmedicine","volume":"87 1","pages":"367-370"},"PeriodicalIF":0.0,"publicationDate":"2015-09-23","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"74434278","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-09-23DOI: 10.1309/NHKBJ8UFM8HKYJRX
Amanda Hernandez, Jacqueline Emmons, Daniel B. Wimmer, D. Payne
1. What is CMV, and why is CMV infection significant in transplant recipients? 2. What laboratory tests are used for the detection of active CMV infection? 3. What is pp65 antigenemia? 4. What is real-time PCR? 5. What are the advantages of using real-time PCR over other methods for detecting (eg, viral culture) and/or quantifying viral DNA? 6. How do you explain the patient’s test results? 7. What assay(s) would you use to definitively explain the test results?
{"title":"Cytomegalovirus-Antigenemia-Positive and Polymerase-Chain-Reaction-Negative Transplant Patient","authors":"Amanda Hernandez, Jacqueline Emmons, Daniel B. Wimmer, D. Payne","doi":"10.1309/NHKBJ8UFM8HKYJRX","DOIUrl":"https://doi.org/10.1309/NHKBJ8UFM8HKYJRX","url":null,"abstract":"1. What is CMV, and why is CMV infection significant in transplant recipients? 2. What laboratory tests are used for the detection of active CMV infection? 3. What is pp65 antigenemia? 4. What is real-time PCR? 5. What are the advantages of using real-time PCR over other methods for detecting (eg, viral culture) and/or quantifying viral DNA? 6. How do you explain the patient’s test results? 7. What assay(s) would you use to definitively explain the test results?","PeriodicalId":54328,"journal":{"name":"Labmedicine","volume":"40 1","pages":"341-342"},"PeriodicalIF":0.0,"publicationDate":"2015-09-23","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"73553665","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-08-01DOI: 10.1309/LMD9PAMUNKPA7DVN
G. Reisfield
Written by Amitava Dasgupta 300 pages. San Diego, Elsevier, 2015. $99.95. ISBN: 9780128003398. Alcohol is one of the most widely used and abused drugs in the United States and the world. According to the Substance Abuse and Mental Health Services Administration, 134 million Americans—more than half of the adult population— report being current drinkers, with nearly 18 million, or approximately 1 in every 12 adults, meeting the criteria for an alcohol use disorder.1 Nearly 90,000 alcohol-attributable deaths occur annually in the United States, making excessive alcohol use the fourth leading preventable cause of death.2 The annual economic costs of alcohol consumption were $223.5 billion in 2006, the last year for which there were comprehensive national estimates.3 As a result, Alcohol and Its …
{"title":"Alcohol and Its Biomarkers: Clinical Aspects and Laboratory Determination","authors":"G. Reisfield","doi":"10.1309/LMD9PAMUNKPA7DVN","DOIUrl":"https://doi.org/10.1309/LMD9PAMUNKPA7DVN","url":null,"abstract":"Written by Amitava Dasgupta 300 pages. San Diego, Elsevier, 2015. $99.95. ISBN: 9780128003398.\u0000\u0000\u0000\u0000Alcohol is one of the most widely used and abused drugs in the United States and the world. According to the Substance Abuse and Mental Health Services Administration, 134 million Americans—more than half of the adult population— report being current drinkers, with nearly 18 million, or approximately 1 in every 12 adults, meeting the criteria for an alcohol use disorder.1 Nearly 90,000 alcohol-attributable deaths occur annually in the United States, making excessive alcohol use the fourth leading preventable cause of death.2 The annual economic costs of alcohol consumption were $223.5 billion in 2006, the last year for which there were comprehensive national estimates.3 As a result, Alcohol and Its …","PeriodicalId":54328,"journal":{"name":"Labmedicine","volume":"1 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2015-08-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"79570602","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-05-01DOI: 10.1309/LMUUIWNKI9U2ZXBA
Bhavna Mehta
Written by Denise M. Harmening and Kathleen Finnegan, 218 pages. Philadelphia, PA: F. A. Davis Company. $28.95. ISBN: 978-0803619029 I enjoyed reading this concise pocket atlas by Denise Harmening and Kathleen Finnegan. I find it to be a well-written book that any medical technologist or physician would love to have in her or his pocket for quick reference. The atlas covers practical theory of normal and abnormal peripheral blood and bone marrow cells. Each cell line is explained completely, with …
{"title":"Heme Notes: A Pocket Atlas of Cell Morphology","authors":"Bhavna Mehta","doi":"10.1309/LMUUIWNKI9U2ZXBA","DOIUrl":"https://doi.org/10.1309/LMUUIWNKI9U2ZXBA","url":null,"abstract":"Written by Denise M. Harmening and Kathleen Finnegan, 218 pages. Philadelphia, PA: F. A. Davis Company. $28.95. ISBN: 978-0803619029\u0000\u0000\u0000\u0000I enjoyed reading this concise pocket atlas by Denise Harmening and Kathleen Finnegan. I find it to be a well-written book that any medical technologist or physician would love to have in her or his pocket for quick reference. The atlas covers practical theory of normal and abnormal peripheral blood and bone marrow cells. Each cell line is explained completely, with …","PeriodicalId":54328,"journal":{"name":"Labmedicine","volume":"53 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2015-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"73590248","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-05-01DOI: 10.1309/LMZE9PXB0A1OJLHD
K. Finnegan
Written by Susan King Strasinger, DA MTASCP), and Marjorie Schaub Di Lorenzo, MT(ASCP)SH, 336 pages. Philadelphia: F.A. Davis Company, 2014. $64.95. ISBN: 9780803639201. I find the textbook Urinalysis and Body Fluids to be well written and of excellent quality. The format of each chapter is clear, concise, and easy to follow. The strengths of each chapter are the understandable learning objectives, key terms, case studies, clinical situations depicted, and technical tips. The historical note with added interesting facts given is a nice touch. Highlighting the key …
作者:Susan King Strasinger, DA MTASCP)和Marjorie Schaub Di Lorenzo, MT(ASCP)SH, 336页。费城:F.A. Davis Company, 2014。64.95美元。ISBN: 9780803639201。我觉得《尿液分析与体液》这本教材写得很好，质量也很好。每章的格式清晰，简洁，易于理解。每章的优势在于可理解的学习目标、关键术语、案例研究、描述的临床情况和技术提示。历史笔记加上有趣的事实是一个很好的手法。突出重点…
{"title":"Urinalysis and Body Fluids, 6th Edition","authors":"K. Finnegan","doi":"10.1309/LMZE9PXB0A1OJLHD","DOIUrl":"https://doi.org/10.1309/LMZE9PXB0A1OJLHD","url":null,"abstract":"Written by Susan King Strasinger, DA MTASCP), and Marjorie Schaub Di Lorenzo, MT(ASCP)SH, 336 pages. Philadelphia: F.A. Davis Company, 2014. $64.95. ISBN: 9780803639201.\u0000\u0000\u0000\u0000I find the textbook Urinalysis and Body Fluids to be well written and of excellent quality. The format of each chapter is clear, concise, and easy to follow. The strengths of each chapter are the understandable learning objectives, key terms, case studies, clinical situations depicted, and technical tips. The historical note with added interesting facts given is a nice touch. Highlighting the key …","PeriodicalId":54328,"journal":{"name":"Labmedicine","volume":"23 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2015-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"86633168","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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