THE need for inquiries into the physiological mechanisms underlying the secretion of sebum is indicated, not only by the lack of information concerning this function of the skin, but also by the fact that it has not as yet been determined how far perversions of secretion are the cause of various pathological conditions such as seborrheeic dermatitis. In these circumstances it seemed desirable to ascertain to what extent the sebaceous glands are under the control of the nervous system. The results of an investigation directed to this end will be reported in this paper, but it is first necessary to indicate the background of the study. As is well known the sebaceous glands are holocrine in nature and the secretion is the end product of cellular disruption. On a priori grounds it would seem unlikely that these glands would be activated by nervous influences, but Boeke (1934) has described a plexus of nerves investing the glands. He considered it to be sympathetic in origin and excitor in function though he noted that the individual cells are not innervated. Moreover, many authors such as Stein (1926), Pachur (1931), and Serrati (1938) have reported that sebaceous secretion is disturbed in various affections of the central nervous system, most notably in chronic encephalitis lethargica. Details of unilateral disturbances of secretion are furnished by Serrati. This has led to the tacit assumption that the sebaceous glands are activated directly by nerves. In further support of this position are the observations of Arloing (1891) on the ear of the ass which lead him to believe that the sympathetic fibres exerted both an excitatory and an inhibitory action. Also frequently quoted is the'case of supra-orbital neuralgia reported by Marschalko (1905) in which a seborrhoeic condition existed'in the area supplied by the nerve. Abel (1936) also appears to concur, for he states that sympathectomy abolishes sebaceous secretion. Contrary opinions indicating that the glands are not directly under nervous regulation are expressed in Starling's text book (1936) and by Goldsmith (1936). This would seem to be the view of the majority at the present time, but the literature does not reveal any reason for modifying the statement of Reid (1898) that " Nothing is definitely
{"title":"STUDIES IN DENERVATION: G.-SEBACEOUS SECRETION.","authors":"J Doupe, M E Sharp","doi":"10.1136/jnnp.6.3-4.133","DOIUrl":"https://doi.org/10.1136/jnnp.6.3-4.133","url":null,"abstract":"THE need for inquiries into the physiological mechanisms underlying the secretion of sebum is indicated, not only by the lack of information concerning this function of the skin, but also by the fact that it has not as yet been determined how far perversions of secretion are the cause of various pathological conditions such as seborrheeic dermatitis. In these circumstances it seemed desirable to ascertain to what extent the sebaceous glands are under the control of the nervous system. The results of an investigation directed to this end will be reported in this paper, but it is first necessary to indicate the background of the study. As is well known the sebaceous glands are holocrine in nature and the secretion is the end product of cellular disruption. On a priori grounds it would seem unlikely that these glands would be activated by nervous influences, but Boeke (1934) has described a plexus of nerves investing the glands. He considered it to be sympathetic in origin and excitor in function though he noted that the individual cells are not innervated. Moreover, many authors such as Stein (1926), Pachur (1931), and Serrati (1938) have reported that sebaceous secretion is disturbed in various affections of the central nervous system, most notably in chronic encephalitis lethargica. Details of unilateral disturbances of secretion are furnished by Serrati. This has led to the tacit assumption that the sebaceous glands are activated directly by nerves. In further support of this position are the observations of Arloing (1891) on the ear of the ass which lead him to believe that the sympathetic fibres exerted both an excitatory and an inhibitory action. Also frequently quoted is the'case of supra-orbital neuralgia reported by Marschalko (1905) in which a seborrhoeic condition existed'in the area supplied by the nerve. Abel (1936) also appears to concur, for he states that sympathectomy abolishes sebaceous secretion. Contrary opinions indicating that the glands are not directly under nervous regulation are expressed in Starling's text book (1936) and by Goldsmith (1936). This would seem to be the view of the majority at the present time, but the literature does not reveal any reason for modifying the statement of Reid (1898) that \" Nothing is definitely","PeriodicalId":54783,"journal":{"name":"Journal of Neurology and Psychiatry","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1943-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.6.3-4.133","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"40109757","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
THE electrical reactions of denervated muscles form an integral part of the clinical picture associated with lesions of peripheral nerves. These reactions consist of changes in contractility and changes in excitability, both of which were stressed by Erb (1 883a). He considered, however, that the former as revealed by the slow contraction was the more important indication of denervation, and this opinion was reaffirmed by Athanassio-Benisty (1918) and by Pollock (1920). In recent years greater stress has come to be placed on the changes in excitability, possibly because technical developments have them lead two types muscle.
{"title":"STUDIES IN DENERVATION: I.-THE CONTRACTILITY AND EXCITABILITY OF DENERVATED MUSCLE.","authors":"J Doupe","doi":"10.1136/jnnp.6.3-4.141","DOIUrl":"https://doi.org/10.1136/jnnp.6.3-4.141","url":null,"abstract":"THE electrical reactions of denervated muscles form an integral part of the clinical picture associated with lesions of peripheral nerves. These reactions consist of changes in contractility and changes in excitability, both of which were stressed by Erb (1 883a). He considered, however, that the former as revealed by the slow contraction was the more important indication of denervation, and this opinion was reaffirmed by Athanassio-Benisty (1918) and by Pollock (1920). In recent years greater stress has come to be placed on the changes in excitability, possibly because technical developments have them lead two types muscle.","PeriodicalId":54783,"journal":{"name":"Journal of Neurology and Psychiatry","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1943-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.6.3-4.141","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"40109759","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
PICK (1898) described as apperceptive blindness the inability to see resulting from loss of visual attention. He observed that symptom in cases of circumscribed atrophy of the occipital lobes. According to Pick the loss of visual attention resulted in an inability to focus and in a loss of visual apperception, i.e. of the capacity for conscious perception of visual stimuli. Potzl (1928) who studied the symptom in the same kind of case material expressed the opinion that apperceptive blindness belonged to the visual agnosias, the basic disorder being an inability to synthesize visual impressions. Observations made by Horn and Stengel (1930) in a case of Pick's disease corroborated Potzl's view. The literature does not contain further reports on that symptom which obviously occurs only under a specific set of conditions of which little is known. We have observed a patient who to our knowledge is the first reported case of apperceptive blindness in general paralysis; it allows us to carry the analysis of that symptom further than has been possible hitherto.
{"title":"APPERCEPTIVE BLINDNESS IN LISSAUER'S DEMENTIA PARALYTICA.","authors":"M T Paterson, E Stengel","doi":"10.1136/jnnp.6.3-4.83","DOIUrl":"https://doi.org/10.1136/jnnp.6.3-4.83","url":null,"abstract":"PICK (1898) described as apperceptive blindness the inability to see resulting from loss of visual attention. He observed that symptom in cases of circumscribed atrophy of the occipital lobes. According to Pick the loss of visual attention resulted in an inability to focus and in a loss of visual apperception, i.e. of the capacity for conscious perception of visual stimuli. Potzl (1928) who studied the symptom in the same kind of case material expressed the opinion that apperceptive blindness belonged to the visual agnosias, the basic disorder being an inability to synthesize visual impressions. Observations made by Horn and Stengel (1930) in a case of Pick's disease corroborated Potzl's view. The literature does not contain further reports on that symptom which obviously occurs only under a specific set of conditions of which little is known. We have observed a patient who to our knowledge is the first reported case of apperceptive blindness in general paralysis; it allows us to carry the analysis of that symptom further than has been possible hitherto.","PeriodicalId":54783,"journal":{"name":"Journal of Neurology and Psychiatry","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1943-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.6.3-4.83","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"40109748","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
IN his address, The Relation of Physiology to Medicine, Lewis (1939) has suggested that one of the functions of a good hypothesis is to stimulate further investigations. Such an hypothesis was presented by Lewis and Pickering (1936) in an endeavour to explain why sympathectomized limbs stay warm while denervated digits become cold. The present paper, which was directly stimulated by their hypothesis, may be introduced by a brief survey of the literature to ascertain if any clues to an alternative explanation can be found. Previous to the work of Lewis and Pickering only Goltz (1874) seems to have been impressed by the paradoxical nature of the facts. He found that after sciatic nerve lesions in dogs the denervated limb was first warm but became cold in the course of two to four weeks. Claude Bernard, however, had shown that sympathectomy caused vasodilatation in the rabbit's ear. To explain this Goltz conjectured that the initial vasodilatation in the limbs of his dogs and the vasodilatation in the rabbit's ear might be due to traumatic excitation of sympathetic vasodilator fibres. He therefore designed the crucial experiment of stimulating the sciatic nerve and found that this produced vasodilatation. Bayliss (1901) was later to show that the result was due to anti-dromic impulses but Goltz, because of the unfortunate (but not uncommon) coincidence of a mistaken theory and a misinterpreted crucial experiment, was lead into the error of believing that the coldness of limbs following section of peripheral nerves was due to a paralysis of sympathetic vasodilator fibres. Nevertheless, despite the seventy years that have passed, ready assent may still be given to Goltz's opinion of the problem for he states that it caused him much worry and many sleepless nights. Lewis and Pickering in their paper of 1936 resolved the problem with Euclidian succinctness. They state " . . . while loss of sympathetic supply causes the corresponding fingers to be in general warmer than they otherwise would be, loss of all nerve supply causes the corresponding fingers to be in general colder than they otherwise would be.
{"title":"STUDIES IN DENERVATION: B.-THE CIRCULATION IN DENERVATED DIGITS.","authors":"J Doupe","doi":"10.1136/jnnp.6.3-4.97","DOIUrl":"https://doi.org/10.1136/jnnp.6.3-4.97","url":null,"abstract":"IN his address, The Relation of Physiology to Medicine, Lewis (1939) has suggested that one of the functions of a good hypothesis is to stimulate further investigations. Such an hypothesis was presented by Lewis and Pickering (1936) in an endeavour to explain why sympathectomized limbs stay warm while denervated digits become cold. The present paper, which was directly stimulated by their hypothesis, may be introduced by a brief survey of the literature to ascertain if any clues to an alternative explanation can be found. Previous to the work of Lewis and Pickering only Goltz (1874) seems to have been impressed by the paradoxical nature of the facts. He found that after sciatic nerve lesions in dogs the denervated limb was first warm but became cold in the course of two to four weeks. Claude Bernard, however, had shown that sympathectomy caused vasodilatation in the rabbit's ear. To explain this Goltz conjectured that the initial vasodilatation in the limbs of his dogs and the vasodilatation in the rabbit's ear might be due to traumatic excitation of sympathetic vasodilator fibres. He therefore designed the crucial experiment of stimulating the sciatic nerve and found that this produced vasodilatation. Bayliss (1901) was later to show that the result was due to anti-dromic impulses but Goltz, because of the unfortunate (but not uncommon) coincidence of a mistaken theory and a misinterpreted crucial experiment, was lead into the error of believing that the coldness of limbs following section of peripheral nerves was due to a paralysis of sympathetic vasodilator fibres. Nevertheless, despite the seventy years that have passed, ready assent may still be given to Goltz's opinion of the problem for he states that it caused him much worry and many sleepless nights. Lewis and Pickering in their paper of 1936 resolved the problem with Euclidian succinctness. They state \" . . . while loss of sympathetic supply causes the corresponding fingers to be in general warmer than they otherwise would be, loss of all nerve supply causes the corresponding fingers to be in general colder than they otherwise would be.","PeriodicalId":54783,"journal":{"name":"Journal of Neurology and Psychiatry","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1943-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.6.3-4.97","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"40109753","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
The case to be described is unusual in presenting most of the changes of Lindau's disease without any cerebellar tumour. In addition there is evidence of a hereditary tendency to the disease, as four relations of the patient suffered from progressive visual defect, and one other had an intracranial tumour. The patient herself had an hkmangioblastoma in one retina and two in the other, an hemangioblastoma of the spinal cord with extensive syringomyelia and cystic pancreas and kidneys.
{"title":"A CASE OF ATYPICAL LINDAU'S DISEASE.","authors":"W R Brain, J G Greenfield, D W Northfield","doi":"10.1136/jnnp.6.1-2.32","DOIUrl":"https://doi.org/10.1136/jnnp.6.1-2.32","url":null,"abstract":"The case to be described is unusual in presenting most of the changes of Lindau's disease without any cerebellar tumour. In addition there is evidence of a hereditary tendency to the disease, as four relations of the patient suffered from progressive visual defect, and one other had an intracranial tumour. The patient herself had an hkmangioblastoma in one retina and two in the other, an hemangioblastoma of the spinal cord with extensive syringomyelia and cystic pancreas and kidneys.","PeriodicalId":54783,"journal":{"name":"Journal of Neurology and Psychiatry","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1943-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.6.1-2.32","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"40109744","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Morale. E. A. Strecker and K. E. Appel. 159. Comparative Study of the E.E.G.S. of Schizophrenic and ManicAnalysis of Certain Factors in Histories of Two Hundred Soldiers Depressive Patients. P. A. Davis. 210. Discharged from the Army for Neuropsychiatric Disabilities. S. J. Undirectional Electrostimulated Convulsive Therapy. I. E. FriedRosenberg and R. H. Lambert. 164. man. 218. The Self-Inflicted Injury. D. J. Flicker. 168. Unit Reaction States in Oculogyric Crises. A. A. Rosner. 224. Brain Injury, Drugs, and Environment as Causes of Mental Decay in Tuberculosis in Drug Addicts. M. A. Diamond. 229. Epilepsy. W. G. Lennox. 174. Some Administrative Aspects of Suicide in the Mental Hospital. Evaluation of Continued Therapy with Phenytoin Sodium. L. J. L. S. Lipschutz. 181. Robinson. 231. Manic-Depressive Psychoses among College Students. L. E. Himler Debatable Administrative Features. J. L. van der Mark. 238. and T. Raphael. 188. Hospital Treatment of Patients with Psychoneurotic Disorders. History of the First Psychopathic Institution of the American D. M. Hamilton, H. I. Varney, and J. H. Wall. 243.
士气。e·a·斯特莱克和k·e·阿佩尔，159页。200名军人抑郁症患者精神分裂症与躁狂的脑电图比较研究。P. A.戴维斯。因为神经精神障碍从军队退伍。单向电刺激抽搐疗法。[j]弗里德·罗森伯格，r·h·兰伯特。男人。218. 自残。D. J.弗莱克。生态危机中的单位反应状态。A. A.罗斯纳。脑损伤、药物和环境是吸毒成瘾者肺结核患者智力衰退的原因。M. A. Diamond。癫痫。w·g·伦诺克斯。精神病院自杀管理的若干方面。苯妥英钠继续治疗的评价。[j]李普舒兹。罗宾逊》231。大学生躁狂抑郁性精神病L. E.希姆勒有争议的行政特征。范德马克。238。和T.拉斐尔。精神神经症患者的住院治疗。《美国第一个精神病态机构的历史》，汉密尔顿博士、瓦尼博士和沃尔博士。
{"title":"EPITOME OF CURRENT JOURNALS","authors":"","doi":"10.1136/jnnp.6.1-2.69","DOIUrl":"https://doi.org/10.1136/jnnp.6.1-2.69","url":null,"abstract":"Morale. E. A. Strecker and K. E. Appel. 159. Comparative Study of the E.E.G.S. of Schizophrenic and ManicAnalysis of Certain Factors in Histories of Two Hundred Soldiers Depressive Patients. P. A. Davis. 210. Discharged from the Army for Neuropsychiatric Disabilities. S. J. Undirectional Electrostimulated Convulsive Therapy. I. E. FriedRosenberg and R. H. Lambert. 164. man. 218. The Self-Inflicted Injury. D. J. Flicker. 168. Unit Reaction States in Oculogyric Crises. A. A. Rosner. 224. Brain Injury, Drugs, and Environment as Causes of Mental Decay in Tuberculosis in Drug Addicts. M. A. Diamond. 229. Epilepsy. W. G. Lennox. 174. Some Administrative Aspects of Suicide in the Mental Hospital. Evaluation of Continued Therapy with Phenytoin Sodium. L. J. L. S. Lipschutz. 181. Robinson. 231. Manic-Depressive Psychoses among College Students. L. E. Himler Debatable Administrative Features. J. L. van der Mark. 238. and T. Raphael. 188. Hospital Treatment of Patients with Psychoneurotic Disorders. History of the First Psychopathic Institution of the American D. M. Hamilton, H. I. Varney, and J. H. Wall. 243.","PeriodicalId":54783,"journal":{"name":"Journal of Neurology and Psychiatry","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1943-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.6.1-2.69","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"63908210","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
J. M. Looney, R. G. Hoskins, G. L. Maltby, A. Persky
Some Current Features of Psychiatry in the United States Navy. U. H. Helgesson. 633. The Neuropsychiatric Selection of Recruits. C. L. Wittson, H. I. Harris, W. A. Hunt, P. S. Solomon, and M. M. Jackson. 639. The Psychoneurotic in the Armed Forces. N. Michael. 651. Forensic Issues in the Neuroses of War. A. Kardiner. 654. The Mental Health Program in Tennessee. W. F. Roth and F. H. Luton. 662. Anatomic and Pathologic Considerations in Convulsive Disorder. R. W. Wagoner, K. Lowenberg, and R. W. Howell. 676. Review of Brain Pathology in the Convulsive Disorders. W. W. Dickerson. 679. The Effect of Electrically and Chemically Induced Convulsions on Conditioned Reflexes. M. Kessler and E. Gelhorn. 687. The Organization and Administration of a State Hospital Insulin-
{"title":"EPITOME OF CURRENT JOURNALS","authors":"J. M. Looney, R. G. Hoskins, G. L. Maltby, A. Persky","doi":"10.1136/jnnp.6.3-4.162","DOIUrl":"https://doi.org/10.1136/jnnp.6.3-4.162","url":null,"abstract":"Some Current Features of Psychiatry in the United States Navy. U. H. Helgesson. 633. The Neuropsychiatric Selection of Recruits. C. L. Wittson, H. I. Harris, W. A. Hunt, P. S. Solomon, and M. M. Jackson. 639. The Psychoneurotic in the Armed Forces. N. Michael. 651. Forensic Issues in the Neuroses of War. A. Kardiner. 654. The Mental Health Program in Tennessee. W. F. Roth and F. H. Luton. 662. Anatomic and Pathologic Considerations in Convulsive Disorder. R. W. Wagoner, K. Lowenberg, and R. W. Howell. 676. Review of Brain Pathology in the Convulsive Disorders. W. W. Dickerson. 679. The Effect of Electrically and Chemically Induced Convulsions on Conditioned Reflexes. M. Kessler and E. Gelhorn. 687. The Organization and Administration of a State Hospital Insulin-","PeriodicalId":54783,"journal":{"name":"Journal of Neurology and Psychiatry","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1943-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.6.3-4.162","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"63908294","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Spurious pregnancy (false pregnancy or pseudocyesis) constitutes a subject of considerable practical importance not only for the general practitioner and obstetrician but also for the psychiatrist, as it presents one of the most impressive examples of the influence of emotional mechanisms on physical processes. The occurrence of this condition in humans and animals has attracted attention throughout the ages, one of the earliest descriptions being found in the writings of Hippocrates. Paddock, who in 1928 made a survey of the literature on that subject viewed from the obstetric aspect, quotes various authors, among them James Y. Simpson and Montgomery (1863). The psychological significance of the syndrome, however, has not received adequate attention. While some authors maintain that pseudocyesis is found mainly in women of the 25-35 age period, Simpson and Montgomery emphasize its more frequent association with the climacteric period. Paddock maintains that most of the cases reported were young women. He regards false pregnancy at the climacteric as part of the general menopausal syndrome. Maranon (1929) supports Simpson and Montgomery in their view that the frequency of the condition is highest at the climacteric and classifies the condition as a metabolic disturbance originating from emotional causes. " In women whose desire to remain young makes them forget how old they are
{"title":"THE PSYCHO-SOMATIC SYNDROME OF SPURIOUS PREGNANCY AFTER THE MENOPAUSE.","authors":"B Jacobs","doi":"10.1136/jnnp.6.1-2.17","DOIUrl":"https://doi.org/10.1136/jnnp.6.1-2.17","url":null,"abstract":"Spurious pregnancy (false pregnancy or pseudocyesis) constitutes a subject of considerable practical importance not only for the general practitioner and obstetrician but also for the psychiatrist, as it presents one of the most impressive examples of the influence of emotional mechanisms on physical processes. The occurrence of this condition in humans and animals has attracted attention throughout the ages, one of the earliest descriptions being found in the writings of Hippocrates. Paddock, who in 1928 made a survey of the literature on that subject viewed from the obstetric aspect, quotes various authors, among them James Y. Simpson and Montgomery (1863). The psychological significance of the syndrome, however, has not received adequate attention. While some authors maintain that pseudocyesis is found mainly in women of the 25-35 age period, Simpson and Montgomery emphasize its more frequent association with the climacteric period. Paddock maintains that most of the cases reported were young women. He regards false pregnancy at the climacteric as part of the general menopausal syndrome. Maranon (1929) supports Simpson and Montgomery in their view that the frequency of the condition is highest at the climacteric and classifies the condition as a metabolic disturbance originating from emotional causes. \" In women whose desire to remain young makes them forget how old they are","PeriodicalId":54783,"journal":{"name":"Journal of Neurology and Psychiatry","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1943-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.6.1-2.17","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"40109742","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"EROSION OF THE ALA NASI FOLLOWING TRIGEMINAL DENERVATION.","authors":"J Schorstein","doi":"10.1136/jnnp.6.1-2.46","DOIUrl":"https://doi.org/10.1136/jnnp.6.1-2.46","url":null,"abstract":"","PeriodicalId":54783,"journal":{"name":"Journal of Neurology and Psychiatry","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1943-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.6.1-2.46","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"40109746","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Two Factors in the Prognosis of Alcoholism. J. M. Nagle. 633. On the Fear of Being Buried Alive. S. Feldman. 641. Contrasting Schizophrenic Patterns in the Graphic Rorschach. J. H. Grassi. 646. Ward Therapy-A New Method of Group Psychotherapy. N. Blackman. 660. Prognosis of Carbon Monoxide Poisoning in 15 Patients, previously Mentally Ill. R. J. Van Amberg. 668. The Massive Structure of Delinquency. A. N. Foxe. 681.
{"title":"AIDS TO THE INVESTIGATION OF PERIPHERAL NERVE INJURIES","authors":"April","doi":"10.1136/jnnp.6.1-2.81","DOIUrl":"https://doi.org/10.1136/jnnp.6.1-2.81","url":null,"abstract":"Two Factors in the Prognosis of Alcoholism. J. M. Nagle. 633. On the Fear of Being Buried Alive. S. Feldman. 641. Contrasting Schizophrenic Patterns in the Graphic Rorschach. J. H. Grassi. 646. Ward Therapy-A New Method of Group Psychotherapy. N. Blackman. 660. Prognosis of Carbon Monoxide Poisoning in 15 Patients, previously Mentally Ill. R. J. Van Amberg. 668. The Massive Structure of Delinquency. A. N. Foxe. 681.","PeriodicalId":54783,"journal":{"name":"Journal of Neurology and Psychiatry","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1943-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.6.1-2.81","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"63908247","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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