Purpose: New symptoms from established idiopathic or senescent pathophysiology are often misinterpreted as an injury (damage to tissues by an external force). Misinterpretation of age-related imaging abnormalities as damage from an external force introduces potential for overdiagnosis, overtreatment, overprotection, and misplacement of a condition under work insurance. Evidence from the shoulder and knee suggests that awareness of the bilateral nature of many idiopathic and senescent pathophysiologies can limit erroneous diagnosis of traumatic pathophysiology.
Methods: Sixty-four scrutinized work injury claims with unilateral wrist symptoms underwent bilateral wrist magnetic resonance imaging (MRI) as part of routine care. The radiologist's interpretation of the MRIs was reviewed. Abnormalities were documented for each side and rated as either corresponding with or incidental to the location of the symptoms. We analyzed factors associated with MRI abnormalities present in the symptomatic wrist alone.
Results: MRI signal abnormalities were detected in 97% (n = 62) of symptomatic wrists and 91% (n = 58) of asymptomatic wrists, with an average of three abnormal findings per wrist. Signal abnormalities of the articular disc and extensor carpi ulnaris tendon were present in 64% and 45% percent of wrists and they were bilateral in 85% and 72% of patients, respectively. MRI findings were considered incidental to the symptoms in 95% (n = 61) of patients. In 55% (n = 35) of the cohort, symptoms were attributed to idiopathic or senescent pathophysiology and in 41% (n = 26) symptoms were considered nonspecific (no pathophysiological explanation). A trip and fall injury mechanism was the only variable associated with the presence of an abnormal MRI signal in the symptomatic wrist alone.
Conclusions: Occupational injury claimants with unilateral wrist symptoms tend to have symmetric MRI signal changes that do not correspond with symptoms, suggesting that new symptoms from idiopathic or degenerative conditions are far more common than traumatic pathophysiology.
Type of study/level of evidence: Diagnostic IV.
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