The results of a three-year study of 120 patients with Meniere's disease has revealed multiple but specific etiology for 48 percent of the patients: (1) Allergy-14 percent (2) Congenital or acquired syphilis-7 percent (3) Adrenal pituitary insufficiency-6 percent (4) Hypothyroidism-3 percent (5) Stenosis of the internal auditory canal-3 percent (6) Trauma, acoustic or physical-3 percent (7) Combination of the above-12 percent. Emotional or psychiatric factors are not involved in the etiology of this disease. The vestibular and cochlear aqueduct on both temporal bones of patients with Meniere's disease could not be visualized in a significantly greater number than a normal control group suggesting an anatomic predisposition to the development of Meniere's disease. Preliminary immunologic viral investigation suggests the possibility of viral etiology in the remaining group considered idiopathic. It is hoped that this study will encourage other investigators to criticize and help intensify our efforts to solve the questions remaining about Meniere's disease.
{"title":"Meniere's disease study: three-year progress report.","authors":"L Pulec, W F House","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>The results of a three-year study of 120 patients with Meniere's disease has revealed multiple but specific etiology for 48 percent of the patients: (1) Allergy-14 percent (2) Congenital or acquired syphilis-7 percent (3) Adrenal pituitary insufficiency-6 percent (4) Hypothyroidism-3 percent (5) Stenosis of the internal auditory canal-3 percent (6) Trauma, acoustic or physical-3 percent (7) Combination of the above-12 percent. Emotional or psychiatric factors are not involved in the etiology of this disease. The vestibular and cochlear aqueduct on both temporal bones of patients with Meniere's disease could not be visualized in a significantly greater number than a normal control group suggesting an anatomic predisposition to the development of Meniere's disease. Preliminary immunologic viral investigation suggests the possibility of viral etiology in the remaining group considered idiopathic. It is hoped that this study will encourage other investigators to criticize and help intensify our efforts to solve the questions remaining about Meniere's disease.</p>","PeriodicalId":78320,"journal":{"name":"International journal of equilibrium research","volume":"3 1","pages":"156-65"},"PeriodicalIF":0.0,"publicationDate":"1973-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"15904676","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
We draw the following conclusions from clinical observations on traumatized patients with vertigo and lumbar pain. 1) Hypertonicity of the lumbar soft supporting tissues, and especially the lumbar erector muscles (i.e., over-excitment of the lumbar proprioceptors) is a cause of vertigo due to whiplash injury. Equilibrium tests after fixing the waist with a corset are useful for detecting ataxia of lumbar origin. 2) Over-excitement of the lumbar porpiroceptors and dysfunction of the brain stem and the cerebellum are closely related in bringing about vertigo due to whiplash injury. The lumbar proprioceptors in particular may be important, in close correlation with the cerebellum in producing vertigo of this type. 3) Infiltration of procaine into tender spots in the lumbar soft supporting tissues is helpful in therapy of disequilibrium of the eyes and body and of cerebellar ataxia.
{"title":"Studies on ataxia of lumbar origin in cases of vertigo due to whiplash injury.","authors":"M Hinoki, S Hine, N Ushio, Y Ishida, S Koike","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>We draw the following conclusions from clinical observations on traumatized patients with vertigo and lumbar pain. 1) Hypertonicity of the lumbar soft supporting tissues, and especially the lumbar erector muscles (i.e., over-excitment of the lumbar proprioceptors) is a cause of vertigo due to whiplash injury. Equilibrium tests after fixing the waist with a corset are useful for detecting ataxia of lumbar origin. 2) Over-excitement of the lumbar porpiroceptors and dysfunction of the brain stem and the cerebellum are closely related in bringing about vertigo due to whiplash injury. The lumbar proprioceptors in particular may be important, in close correlation with the cerebellum in producing vertigo of this type. 3) Infiltration of procaine into tender spots in the lumbar soft supporting tissues is helpful in therapy of disequilibrium of the eyes and body and of cerebellar ataxia.</p>","PeriodicalId":78320,"journal":{"name":"International journal of equilibrium research","volume":"3 1","pages":"141-52"},"PeriodicalIF":0.0,"publicationDate":"1973-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"15904973","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Response patterns of vestibular thalamic neurons in the rhesus monkey.","authors":"L Deecke, D W Schwarz, J M Fredrickson","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":78320,"journal":{"name":"International journal of equilibrium research","volume":"3 1","pages":"4-7"},"PeriodicalIF":0.0,"publicationDate":"1973-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"15325459","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Studies on optokinetic habituation.","authors":"T Miyoshi, C R Pfaltz","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":78320,"journal":{"name":"International journal of equilibrium research","volume":"3 1","pages":"94"},"PeriodicalIF":0.0,"publicationDate":"1973-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"15904683","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
The question whether optokinetic nystagmus in humans can be habituated or not, has never been answered conclusively. In order to investigate this problem more closely, 15 healthy individuals, divided into 3 groups, have been exposed to repeated optokinetic stimulations of different direction and varying intensity. The velocity of the slow phase has been used as a parameter for the evaluation of the optokinetic response. Habituation of optokinetically induced nystagmus is not characterized by a response decline phenomenon, but on the contrary by an increase of the optokinetic response. Retention of this response increment has been confirmed as well as transfer from one side to the other. Repeated optokinetic stimulation has no influence upon the pattern of the caloric, galvanic and vestibulospinal responses of a test subject, i.e. there is no transfer from optic to vestibular responses. The increase of the optokinetic responses, following repeated optic stimulation, is not interpreted as a true habituation phenomenon, i.e. the simplest type of negative learning, but as the resultant of a process of positive learning.
{"title":"Studies on optokinetic habituation.","authors":"T. Miyoshi, C. R. Pfaltz","doi":"10.1159/000275071","DOIUrl":"https://doi.org/10.1159/000275071","url":null,"abstract":"The question whether optokinetic nystagmus in humans can be habituated or not, has never been answered conclusively. In order to investigate this problem more closely, 15 healthy individuals, divided into 3 groups, have been exposed to repeated optokinetic stimulations of different direction and varying intensity. The velocity of the slow phase has been used as a parameter for the evaluation of the optokinetic response. Habituation of optokinetically induced nystagmus is not characterized by a response decline phenomenon, but on the contrary by an increase of the optokinetic response. Retention of this response increment has been confirmed as well as transfer from one side to the other. Repeated optokinetic stimulation has no influence upon the pattern of the caloric, galvanic and vestibulospinal responses of a test subject, i.e. there is no transfer from optic to vestibular responses. The increase of the optokinetic responses, following repeated optic stimulation, is not interpreted as a true habituation phenomenon, i.e. the simplest type of negative learning, but as the resultant of a process of positive learning.","PeriodicalId":78320,"journal":{"name":"International journal of equilibrium research","volume":"3 1 1","pages":"94"},"PeriodicalIF":0.0,"publicationDate":"1972-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1159/000275071","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"64988054","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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