Over 400 participants from 55 universities and health centers attended the congress in Iran. The program was chaired and directed by Samad Nourizad, Alireza Ebadi, Mohsen Adib Haajbagheri and Negin Masoudi Alavi. The main goal of the congress was to review the current research findings in the field of geriatric and gerontology in Iran. The congress was divided into five sessions with the following titles: • The epidemiologic status of the elderly in the World and in Iran • Physical and mental health and quality of life in the elderly • Common health-related problems in the elderly • The role of health professionals in elderly care • Nutrition in the elderly
{"title":"Update on geriatric research","authors":"N. Alavi","doi":"10.2217/1745509X.4.1.23","DOIUrl":"https://doi.org/10.2217/1745509X.4.1.23","url":null,"abstract":"Over 400 participants from 55 universities and health centers attended the congress in Iran. The program was chaired and directed by Samad Nourizad, Alireza Ebadi, Mohsen Adib Haajbagheri and Negin Masoudi Alavi. The main goal of the congress was to review the current research findings in the field of geriatric and gerontology in Iran. The congress was divided into five sessions with the following titles: • The epidemiologic status of the elderly in the World and in Iran • Physical and mental health and quality of life in the elderly • Common health-related problems in the elderly • The role of health professionals in elderly care • Nutrition in the elderly","PeriodicalId":88353,"journal":{"name":"Aging health","volume":"4 1","pages":"23-24"},"PeriodicalIF":0.0,"publicationDate":"2008-01-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.2217/1745509X.4.1.23","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"68187825","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Evaluation of: Nichol KL, Nordin JD, Nelson DB, Mullooly JP, Hak E: Effectiveness of influenza vaccine in the community-dwelling elderly. N. Engl. J. Med. 357(14), 1373–1381 (2007). Recent publications have questioned the benefit of influenza vaccination in the older adult population. To address this issue, Nichol et al. reported a study of 713,872 person-seasons over ten influenza seasons, which showed that influenza vaccination was associated with significant reductions in influenza-related hospitalizations and deaths among community-dwelling older adults. The comprehensive database ensured adequate baseline and follow-up data for annually defined influenza seasons. This analytic method reduces the potential of survivor bias when the baseline is established in the summer months preceding the influenza season, as has been used in other studies. The results were further supported by a sensitivity analysis using a hypothetical confounder to detect a healthy vaccinee bias and showed that the hospitalization...
Nichol KL, Nordin JD, Nelson DB, Mullooly JP, Hak E:流感疫苗在社区居住老年人中的有效性评价。心血管病。中华医学杂志，2003,14(4)，344 - 344(2007)。最近的出版物对老年人接种流感疫苗的益处提出了质疑。为了解决这个问题，Nichol等人报告了一项针对10个流感季节713,872人的研究，结果表明，流感疫苗接种与社区居住老年人流感相关住院和死亡的显著减少有关。全面的数据库确保了每年确定的流感季节有足够的基线和后续数据。如在其他研究中所使用的那样，在流感季节之前的夏季建立基线时，这种分析方法减少了幸存者偏差的可能性。使用假设混杂因素检测健康疫苗接种者偏差的敏感性分析进一步支持了该结果，并表明住院治疗…
{"title":"Influenza vaccination in the elderly as a strategy to improve vaccine effectiveness","authors":"J. Mcelhaney","doi":"10.2217/1745509X.4.1.33","DOIUrl":"https://doi.org/10.2217/1745509X.4.1.33","url":null,"abstract":"Evaluation of: Nichol KL, Nordin JD, Nelson DB, Mullooly JP, Hak E: Effectiveness of influenza vaccine in the community-dwelling elderly. N. Engl. J. Med. 357(14), 1373–1381 (2007). Recent publications have questioned the benefit of influenza vaccination in the older adult population. To address this issue, Nichol et al. reported a study of 713,872 person-seasons over ten influenza seasons, which showed that influenza vaccination was associated with significant reductions in influenza-related hospitalizations and deaths among community-dwelling older adults. The comprehensive database ensured adequate baseline and follow-up data for annually defined influenza seasons. This analytic method reduces the potential of survivor bias when the baseline is established in the summer months preceding the influenza season, as has been used in other studies. The results were further supported by a sensitivity analysis using a hypothetical confounder to detect a healthy vaccinee bias and showed that the hospitalization...","PeriodicalId":88353,"journal":{"name":"Aging health","volume":"4 1","pages":"33-35"},"PeriodicalIF":0.0,"publicationDate":"2008-01-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.2217/1745509X.4.1.33","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"68187405","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Delirium is a common problem, mostly affecting older patients in hospital, which results in greater mortality, nursing-home placement and cognitive and functional impairment. Delirium can be triggered by a wide range of conditions, treatments and procedures, as well as by certain environments. Some hospital environments have been causally implicated, but until it was possible to compare treatment in-hospital with treatment in other places, the observation remained at the level of an association. However, the development of ‘Hospital in the Home’ services has allowed clinicians to explore this question scientifically. Recently, a number of studies comparing treatment of acute conditions, both medical and surgical, and rehabilitation in hospital with treatment at home, have found a lower incidence of delirium with home treatment, as well as lower rates of the sequelae of delirium. Since delirium is an indicator of a wide range of subsequent poor outcomes, this information has broad implications for the deli...
{"title":"Does ‘Hospital in the Home’ treatment prevent delirium?","authors":"G. Caplan","doi":"10.2217/1745509X.4.1.69","DOIUrl":"https://doi.org/10.2217/1745509X.4.1.69","url":null,"abstract":"Delirium is a common problem, mostly affecting older patients in hospital, which results in greater mortality, nursing-home placement and cognitive and functional impairment. Delirium can be triggered by a wide range of conditions, treatments and procedures, as well as by certain environments. Some hospital environments have been causally implicated, but until it was possible to compare treatment in-hospital with treatment in other places, the observation remained at the level of an association. However, the development of ‘Hospital in the Home’ services has allowed clinicians to explore this question scientifically. Recently, a number of studies comparing treatment of acute conditions, both medical and surgical, and rehabilitation in hospital with treatment at home, have found a lower incidence of delirium with home treatment, as well as lower rates of the sequelae of delirium. Since delirium is an indicator of a wide range of subsequent poor outcomes, this information has broad implications for the deli...","PeriodicalId":88353,"journal":{"name":"Aging health","volume":"4 1","pages":"69-74"},"PeriodicalIF":0.0,"publicationDate":"2008-01-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.2217/1745509X.4.1.69","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"68187721","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Prolonged-release (PR) melatonin (Circadin ® , Neurim Pharmaceuticals, Tel-Aviv, Israel) is a nonbenzodiazepine licensed to treat primary insomnia in patients aged 55 years and older. It exerts its effects by mimicking endogenous melatonin, a hormone which regulates sleep. PR-melatonin has been shown to be effective in improving quality of sleep and morning alertness. An oral dose of 2 mg once-daily for 3 weeks has generally been well tolerated, and does not cause ‘hangover’ effects or impair memory and psychomotor skills. Insomnia is a common sleep disorder, the diagnosis of which is based on a patient’s complaint of difficulty in initiating or maintaining sleep and/or sleep that is of inadequate quality (nonrestorative sleep). The sleep disturbance should have been present for at least 1 month and be associated with a negative impact on functioning the following day [1,2]. Insomnia occurs in 30–45% of adults [3,4] and is 1.5-times more likely in women than in men [5,6]. The prevalence of insomnia increases with age [7,8]. Insomnia may be secondary – particularly when associated with mental disorders or pain – or primary, that is not attributable to any physical or mental disorder or to any environmental cause. There are widely varying reports of the prevalence of primary insomnia in the general population from 1–10% and ranging up to 25% in the elderly [9,10]. Sleep disorders, including primary insomnia, place a tremendous burden on society due to their association with psychiatric disorders, their negative impact on quality of life, their association with falls and road traffic accidents and the reduced productivity and increased utilization of healthcare services by sufferers [10–16]. There is a poor correlation between the clinical complaints associated with insomnia and the findings at polysomnography that frequently fail to demonstrate any sleep-quality related cause for the problem [17–20]. However, the tenth revision of the International Classification of Diseases (ICD 10) acknowledges that ‘there are people who suffer immensely from the poor quality of their sleep, while sleep in quantity is judged subjectively, and/or objectively as within the normal limits’ [2]. Sleep quality is thus an important parameter of the clinical complaint of insomnia, with the diagnosis and the evaluation of treatment effects based solely on subjective assessments.
{"title":"Prolonged-release melatonin for the treatment of insomnia: targeting quality of sleep and morning alertness","authors":"A. Wade, N. Zisapel, P. Lemoine","doi":"10.2217/1745509X.4.1.11","DOIUrl":"https://doi.org/10.2217/1745509X.4.1.11","url":null,"abstract":"Prolonged-release (PR) melatonin (Circadin ® , Neurim Pharmaceuticals, Tel-Aviv, Israel) is a nonbenzodiazepine licensed to treat primary insomnia in patients aged 55 years and older. It exerts its effects by mimicking endogenous melatonin, a hormone which regulates sleep. PR-melatonin has been shown to be effective in improving quality of sleep and morning alertness. An oral dose of 2 mg once-daily for 3 weeks has generally been well tolerated, and does not cause ‘hangover’ effects or impair memory and psychomotor skills. Insomnia is a common sleep disorder, the diagnosis of which is based on a patient’s complaint of difficulty in initiating or maintaining sleep and/or sleep that is of inadequate quality (nonrestorative sleep). The sleep disturbance should have been present for at least 1 month and be associated with a negative impact on functioning the following day [1,2]. Insomnia occurs in 30–45% of adults [3,4] and is 1.5-times more likely in women than in men [5,6]. The prevalence of insomnia increases with age [7,8]. Insomnia may be secondary – particularly when associated with mental disorders or pain – or primary, that is not attributable to any physical or mental disorder or to any environmental cause. There are widely varying reports of the prevalence of primary insomnia in the general population from 1–10% and ranging up to 25% in the elderly [9,10]. Sleep disorders, including primary insomnia, place a tremendous burden on society due to their association with psychiatric disorders, their negative impact on quality of life, their association with falls and road traffic accidents and the reduced productivity and increased utilization of healthcare services by sufferers [10–16]. There is a poor correlation between the clinical complaints associated with insomnia and the findings at polysomnography that frequently fail to demonstrate any sleep-quality related cause for the problem [17–20]. However, the tenth revision of the International Classification of Diseases (ICD 10) acknowledges that ‘there are people who suffer immensely from the poor quality of their sleep, while sleep in quantity is judged subjectively, and/or objectively as within the normal limits’ [2]. Sleep quality is thus an important parameter of the clinical complaint of insomnia, with the diagnosis and the evaluation of treatment effects based solely on subjective assessments.","PeriodicalId":88353,"journal":{"name":"Aging health","volume":"4 1","pages":"11-21"},"PeriodicalIF":0.0,"publicationDate":"2008-01-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.2217/1745509X.4.1.11","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"68187780","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
There is growing evidence for the association of antiepileptic medications (AEDs) and bone disease. Increased rates of vitamin D deficiency, elevated laboratory markers of bone turnover and increased rates of osteopenia, osteomalacia and fracture have all been reported. Although most commonly attributed to anticonvulsants that induce hepatic cytochrome P450 (CYP) enzymes, recent studies suggest that valproate, a CYP inhibitor, is also problematic. Data specific to other newer AEDs are lacking. Drug-induced inactivation of vitamin D is the most widely proposed mechanism for AED-related bone disease, however, other not yet fully elucidated pathways must also contribute. There are currently no evidence-based guidelines for diagnosis or treatment of bone disease specific to persons on AED therapy. Periodic screening for vitamin D deficiency and with dual-energy x-ray absorptiometry is likely beneficial. Prophylactic supplementation with vitamin D and calcium can be considered for all patients on AEDs, unless ...
{"title":"Bone health and antiseizure drugs: diagnosis and management","authors":"K. Noe, S. Schrader","doi":"10.2217/1745509X.4.1.75","DOIUrl":"https://doi.org/10.2217/1745509X.4.1.75","url":null,"abstract":"There is growing evidence for the association of antiepileptic medications (AEDs) and bone disease. Increased rates of vitamin D deficiency, elevated laboratory markers of bone turnover and increased rates of osteopenia, osteomalacia and fracture have all been reported. Although most commonly attributed to anticonvulsants that induce hepatic cytochrome P450 (CYP) enzymes, recent studies suggest that valproate, a CYP inhibitor, is also problematic. Data specific to other newer AEDs are lacking. Drug-induced inactivation of vitamin D is the most widely proposed mechanism for AED-related bone disease, however, other not yet fully elucidated pathways must also contribute. There are currently no evidence-based guidelines for diagnosis or treatment of bone disease specific to persons on AED therapy. Periodic screening for vitamin D deficiency and with dual-energy x-ray absorptiometry is likely beneficial. Prophylactic supplementation with vitamin D and calcium can be considered for all patients on AEDs, unless ...","PeriodicalId":88353,"journal":{"name":"Aging health","volume":"4 1","pages":"75-84"},"PeriodicalIF":0.0,"publicationDate":"2008-01-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.2217/1745509X.4.1.75","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"68187726","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Alzheimer’s disease (AD) is a growing public health concern with potentially devastating effects. At present, there are no methods of prevention or any known cures for the disease. While genetic factors are relevant in early-onset cases, they are less relevant in late-onset sporadic AD, which is the most common form [1]. Research seeking to find drugs to slow its progression may pay off – someday. However, as the disease typically strikes very late in life, delaying its symptoms could be a good cure for many people. For example, it is now widely accepted that delaying the onset of the disease by even 5 years can cut its incidence in half. Indeed, clinical and epidemiological evidence points to lifestyle factors, including nutrition, as being crucial in delaying the onset of AD. Mounting evidence suggests that brain cells are remarkably responsive to what somebody is doing or eating. Among other nongenetic factors influencing AD, recent studies strongly support the hypothesis that certain dietary factors, such as the Mediterranean diet, may play a beneficial role in the relative risk for AD clinical dementia. Indeed, the effect of diet in AD has been an area of research that has produced enlightening results. More importantly, the evidence supporting a direct link between nutrition and AD neuropathology and dementia continues to grow, as the mechanistic pathways are defined and their biochemical functions scrutinized. Most remarkably, there is also evidence that high caloric intake in the form of saturated fat promotes AD-type neuropathology and, conversely, caloric restriction via reduced carbohydrate intake can prevent it. This evidence is very exciting and is consistent, in part, with current epidemiological studies, which suggest that obesity and diabetes are associated with a more than fourfold increased risk for developing AD. Clarifying the mechanisms through which certain dietary lifestyle factors and caloric intake may ultimately influence AD neuropathology will provide new avenues for the design of ‘therapeutic lifestyle strategies’ to treat AD, and possibly other neurodegenerative disorders.
{"title":"Can diet modifications play a preventative role in the onset of Alzheimer’s disease?","authors":"G. Pasinetti","doi":"10.2217/1745509X.4.1.1","DOIUrl":"https://doi.org/10.2217/1745509X.4.1.1","url":null,"abstract":"Alzheimer’s disease (AD) is a growing public health concern with potentially devastating effects. At present, there are no methods of prevention or any known cures for the disease. While genetic factors are relevant in early-onset cases, they are less relevant in late-onset sporadic AD, which is the most common form [1]. Research seeking to find drugs to slow its progression may pay off – someday. However, as the disease typically strikes very late in life, delaying its symptoms could be a good cure for many people. For example, it is now widely accepted that delaying the onset of the disease by even 5 years can cut its incidence in half. Indeed, clinical and epidemiological evidence points to lifestyle factors, including nutrition, as being crucial in delaying the onset of AD. Mounting evidence suggests that brain cells are remarkably responsive to what somebody is doing or eating. Among other nongenetic factors influencing AD, recent studies strongly support the hypothesis that certain dietary factors, such as the Mediterranean diet, may play a beneficial role in the relative risk for AD clinical dementia. Indeed, the effect of diet in AD has been an area of research that has produced enlightening results. More importantly, the evidence supporting a direct link between nutrition and AD neuropathology and dementia continues to grow, as the mechanistic pathways are defined and their biochemical functions scrutinized. Most remarkably, there is also evidence that high caloric intake in the form of saturated fat promotes AD-type neuropathology and, conversely, caloric restriction via reduced carbohydrate intake can prevent it. This evidence is very exciting and is consistent, in part, with current epidemiological studies, which suggest that obesity and diabetes are associated with a more than fourfold increased risk for developing AD. Clarifying the mechanisms through which certain dietary lifestyle factors and caloric intake may ultimately influence AD neuropathology will provide new avenues for the design of ‘therapeutic lifestyle strategies’ to treat AD, and possibly other neurodegenerative disorders.","PeriodicalId":88353,"journal":{"name":"Aging health","volume":"4 1","pages":"1-3"},"PeriodicalIF":0.0,"publicationDate":"2008-01-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.2217/1745509X.4.1.1","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"68187770","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
J. Kwan, D. Jenkinson, M. Vassallo, N. Englyst, V. Perry
After an acute stroke, systemic infection can complicate the recovery process and lead to a worse clinical outcome, including a higher risk of mortality. Post-stroke infection (PSI) is responsible for the majority of the mortality occurring between 1 week and 1 month after stroke, peaking towards the end of the second week. The effects of PSI on longer-term outcome and other aspects of recovery, such as cognition, mood and quality of life, are largely unknown. The cerebrovascular event itself may result in a systemic immunosuppressed state, hence lowering the threshold for subsequent systemic bacterial infections. Although there have been advances in the basic understanding of the pathophysiological mechanisms of PSI, clinical studies have not provided any clear guidelines on the best methods of managing or preventing PSI. This article provides a review of the current knowledge of the phenomenon of PSI and the possible future developments in the understanding and treatment of PSI.
{"title":"Systemic infections after acute stroke","authors":"J. Kwan, D. Jenkinson, M. Vassallo, N. Englyst, V. Perry","doi":"10.2217/1745509X.4.1.85","DOIUrl":"https://doi.org/10.2217/1745509X.4.1.85","url":null,"abstract":"After an acute stroke, systemic infection can complicate the recovery process and lead to a worse clinical outcome, including a higher risk of mortality. Post-stroke infection (PSI) is responsible for the majority of the mortality occurring between 1 week and 1 month after stroke, peaking towards the end of the second week. The effects of PSI on longer-term outcome and other aspects of recovery, such as cognition, mood and quality of life, are largely unknown. The cerebrovascular event itself may result in a systemic immunosuppressed state, hence lowering the threshold for subsequent systemic bacterial infections. Although there have been advances in the basic understanding of the pathophysiological mechanisms of PSI, clinical studies have not provided any clear guidelines on the best methods of managing or preventing PSI. This article provides a review of the current knowledge of the phenomenon of PSI and the possible future developments in the understanding and treatment of PSI.","PeriodicalId":88353,"journal":{"name":"Aging health","volume":"73 1","pages":"85-94"},"PeriodicalIF":0.0,"publicationDate":"2008-01-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.2217/1745509X.4.1.85","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"68187733","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2007-12-12DOI: 10.2217/1745509X.3.6.735
T. Dharmarajan, David Widjaja
Hypothermia is common in the geriatric population and its significance perhaps under-recognized. Hypothermia is associated with substantial morbidity and fatality, but to some extent is preventable provided caregivers and providers of healthcare to older adults adopt preventive measures to lower its occurrence. Hypothermia occurs in both cold and warm settings; its pathogenesis involves alterations in the age-related physiology of thermoregulation, along with a variable combination of environmental factors, disease processes and medications, many of which are recognizable. Once diagnosed, treatment must be prompt and aggressive, and must consider several options for rewarming. Education of healthcare providers regarding early recognition of hypothermia and a better understanding of preventive and treatment measures will undoubtedly lower complications of hypothermia in affected elderly.
{"title":"Hypothermia in the geriatric population","authors":"T. Dharmarajan, David Widjaja","doi":"10.2217/1745509X.3.6.735","DOIUrl":"https://doi.org/10.2217/1745509X.3.6.735","url":null,"abstract":"Hypothermia is common in the geriatric population and its significance perhaps under-recognized. Hypothermia is associated with substantial morbidity and fatality, but to some extent is preventable provided caregivers and providers of healthcare to older adults adopt preventive measures to lower its occurrence. Hypothermia occurs in both cold and warm settings; its pathogenesis involves alterations in the age-related physiology of thermoregulation, along with a variable combination of environmental factors, disease processes and medications, many of which are recognizable. Once diagnosed, treatment must be prompt and aggressive, and must consider several options for rewarming. Education of healthcare providers regarding early recognition of hypothermia and a better understanding of preventive and treatment measures will undoubtedly lower complications of hypothermia in affected elderly.","PeriodicalId":88353,"journal":{"name":"Aging health","volume":"3 1","pages":"735-741"},"PeriodicalIF":0.0,"publicationDate":"2007-12-12","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.2217/1745509X.3.6.735","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"68187611","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2007-12-12DOI: 10.2217/1745509X.3.6.685
T. Dwolatzky
As a resident in geriatric medicine, I walked past the staff notice board one afternoon on the way to the emergency intake unit of the hospital with the knowledge that a long night was ahead of me. I cast a casual glance at the notices of forthcoming seminars, journal clubs, lectures, conferences – and one caught my eye. The next day at noon, a certain Professor Bernard Isaacs would be delivering a lecture on the topic of incontinence in the elderly. Well, the subject was certainly of interest to me, and we would most definitely be asked about incontinence in the imminent qualifying examination in geriatric medicine. On the other hand, I was sure that my exhaustion after a night in emergency would get the better of me and I would probably go home to sleep and miss the lecture. The night was, needless to say, rather eventful – an 90-year-old man with atrial fibrillation and pulmonary edema, a 76-year-old woman with dementia who fell and fractured her femur, a frail lady in her early 80s with pneumonia who required mechanical ventilation, to mention but a few. The next morning, after handing the patients over to the day staff and updating all the charts, I suddenly recalled that there was a lecture on the topic of incontinence at noon. A few sips of strong coffee and I was on the way to the lecture. The chairman introduced Bernard Isaacs, Professor of Geriatric Medicine at Birmingham University in the UK, as one of the leading geriatricians of this generation. Isaacs had characterized the principal symptoms affecting the elderly as the ‘Geriatric Giants’, these being incontinence, immobility, instability (resulting in falls) and intellectual impairment. He would now enlighten the audience with a talk on one of the Giants – incontinence. Bernard Isaacs, a balding man with an infectious smile, commenced his lecture with the following statement:
{"title":"Geriatric challenge: how to cure incontinence with a screwdriver","authors":"T. Dwolatzky","doi":"10.2217/1745509X.3.6.685","DOIUrl":"https://doi.org/10.2217/1745509X.3.6.685","url":null,"abstract":"As a resident in geriatric medicine, I walked past the staff notice board one afternoon on the way to the emergency intake unit of the hospital with the knowledge that a long night was ahead of me. I cast a casual glance at the notices of forthcoming seminars, journal clubs, lectures, conferences – and one caught my eye. The next day at noon, a certain Professor Bernard Isaacs would be delivering a lecture on the topic of incontinence in the elderly. Well, the subject was certainly of interest to me, and we would most definitely be asked about incontinence in the imminent qualifying examination in geriatric medicine. On the other hand, I was sure that my exhaustion after a night in emergency would get the better of me and I would probably go home to sleep and miss the lecture. The night was, needless to say, rather eventful – an 90-year-old man with atrial fibrillation and pulmonary edema, a 76-year-old woman with dementia who fell and fractured her femur, a frail lady in her early 80s with pneumonia who required mechanical ventilation, to mention but a few. The next morning, after handing the patients over to the day staff and updating all the charts, I suddenly recalled that there was a lecture on the topic of incontinence at noon. A few sips of strong coffee and I was on the way to the lecture. The chairman introduced Bernard Isaacs, Professor of Geriatric Medicine at Birmingham University in the UK, as one of the leading geriatricians of this generation. Isaacs had characterized the principal symptoms affecting the elderly as the ‘Geriatric Giants’, these being incontinence, immobility, instability (resulting in falls) and intellectual impairment. He would now enlighten the audience with a talk on one of the Giants – incontinence. Bernard Isaacs, a balding man with an infectious smile, commenced his lecture with the following statement:","PeriodicalId":88353,"journal":{"name":"Aging health","volume":"3 1","pages":"685-686"},"PeriodicalIF":0.0,"publicationDate":"2007-12-12","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.2217/1745509X.3.6.685","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"68187490","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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