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Endothelium-dependent effects of beta-adrenergic blockers. 肾上腺素能阻滞剂的内皮依赖性作用。
Pub Date : 1990-01-01 DOI: 10.1159/000158822
P M Vanhoutte

Nonselective beta-adrenergic blockers have been reported to affect endothelium-dependent responses in isolated blood vessels in the following ways: (a) cause endothelium-augmented direct relaxations; (b) facilitate the endothelium-dependent relaxations evoked by alpha 2-adrenergic activation, or by acetylcholine; (c) augment the intraluminal release of vasodilator prostanoids, and (d) inhibit endothelium-dependent contractions to anoxia. Important species differences exist in terms of the endothelium-dependent effects of the compounds. If they were to occur in the intact organism, the endothelium-dependent effects of the beta-adrenergic blockers could help to explain their vasodilator properties.

据报道,非选择性β -肾上腺素能阻滞剂通过以下方式影响离体血管的内皮依赖性反应:(a)引起内皮增强的直接松弛;(b)促进由α 2-肾上腺素能激活或乙酰胆碱引起的内皮依赖性松弛;(c)增加血管扩张剂前列腺素的腔内释放,(d)抑制内皮依赖性的缺氧收缩。就这些化合物的内皮依赖作用而言,存在重要的物种差异。如果它们发生在完整的生物体中,β -肾上腺素能阻滞剂的内皮依赖性作用可以帮助解释它们的血管扩张特性。
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引用次数: 5
Endothelins. 内皮素。
Pub Date : 1990-01-01
E E Anggård, R M Botting, J R Vane

The endothelins (ETs) are a family of newly discovered peptides with potent vasoconstrictor properties. They were first discovered in cultured endothelial cells but ET expression has since been found in many other tissues such as brain and kidney. They are peptides with 21 amino acids formed by hydrolytic cleavage of a larger peptide, big ET. Release of ETs from cultured endothelial cells is modulated by a variety of chemical and physical stimuli and as no storage sites have been identified it is suggested that endothelin release is regulated at the level of transcription or translation. Both big ET and ET-1 are found circulating in the blood. The levels are elevated in shock, myocardial infarction and kidney failure indicative of enhanced formation in these diseases. The literature now abounds with reports on actions of the ETs in vitro and in vivo. The vasoconstrictor properties are powerful and long-lasting. Several studies also show a mitogenic effect, indicating a possible trophic role. It is likely that in the next few years the development of inhibitors of endothelin synthesis and/or action will be of importance in unravelling the role of the ETs.

内皮素(ETs)是一类新发现的具有强效血管收缩特性的肽。它们最初是在培养的内皮细胞中发现的,但后来在许多其他组织如脑和肾中也发现了ET的表达。它们是由21个氨基酸组成的肽,由一个更大的肽(大ET)水解裂解形成。培养的内皮细胞的ET释放受多种化学和物理刺激调节,由于没有确定储存位点,因此内皮素的释放在转录或翻译水平上受到调节。大ET和ET-1都存在于血液循环中。在休克、心肌梗死和肾衰竭时,这些水平升高,表明这些疾病的形成增强。现在的文献大量报道了et在体外和体内的作用。血管收缩剂的特性是强大和持久的。几项研究也显示有丝分裂作用,表明可能的营养作用。很可能在未来几年内,内皮素合成和/或作用抑制剂的发展将对揭示内皮素的作用具有重要意义。
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引用次数: 0
Modulation of the action of calcium antagonists in arteries. 动脉中钙拮抗剂作用的调节。
Pub Date : 1990-01-01 DOI: 10.1159/000158809
T Godfraind, N Morel, M Wibo

This paper is a review of the experimental evidence showing that specific binding sites for dihydropyridine Ca antagonists are involved in inhibition of stimulus-dependent Ca entry into arterial cells and thereby in inhibition of the contractile response. The apparent affinity of the dihydropyridine binding site is related to the proportion of a high- and a low-affinity state which is regulated by membrane potential but could also be dependent upon other factors such as G proteins. Among Ca antagonists, a subgroup of agents exhibiting voltage dependence may be identified. Their apparent pharmacological potency is highly dependent on resting membrane potential and on the duration of the depolarizing stimulus.

本文综述了实验证据,表明二氢吡啶钙拮抗剂的特定结合位点参与抑制刺激依赖性钙进入动脉细胞,从而抑制收缩反应。二氢吡啶结合位点的表观亲和力与高亲和力和低亲和力状态的比例有关,这种亲和力由膜电位调节,但也可能取决于其他因素,如G蛋白。在钙拮抗剂中,可以确定一组表现出电压依赖性的药物。它们明显的药理学效力高度依赖于静息膜电位和去极化刺激的持续时间。
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引用次数: 16
Adenosine A2 receptors mediate cerebral vasodilation in the conscious goat. 腺苷A2受体介导有意识山羊的脑血管舒张。
Pub Date : 1990-01-01 DOI: 10.1159/000158793
G Torregrosa, J B Salom, F J Miranda, J A Alabadí, C Alvarez, E Alborch
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引用次数: 6
Mechanoreception by the endothelium: mediators and mechanisms of pressure- and flow-induced vascular responses. 内皮细胞的机械接受:压力和血流诱导的血管反应的介质和机制。
Pub Date : 1990-01-01 DOI: 10.1159/000158816
G M Rubanyi, A D Freay, K Kauser, A Johns, D R Harder

Mechanoreception, a widely distributed sensory modality, has been shown to be present in certain blood vessels. Changes in physical forces, like sudden increase of transmural pressure or flow velocity (shear stress), trigger changes in blood vessel diameter; the former reduces it while the latter increases vessel caliber. These changes in diameter, which are the result of contraction and relaxation of vascular smooth muscle in the blood vessel media, can serve the purpose of physiological regulation of blood flow (autoregulation) and protection of the intima against damages from high shear forces. The precise location of mechanosensor(s) and the mechanism of mechanoreception and signal transduction are poorly understood. Accumulating evidence suggests that the endothelium may be a site of mechanoreception and that changes in the synthesis/release of endothelium-derived relaxing (EDRF, EDHF, PGI2) and contracting factors (EDCF) result in altered vascular smooth muscle tone and vessel caliber. Increased shear stress stimulates the release of EDRF and PGI2 probably via activation of a K+ channel (inward rectifier) in endothelial cell membrane. Endothelium-dependent vascular contraction evoked by increased transmural pressure may be the result of (1) reduced release of EDRF (canine carotid artery) and (2) stimulation of the release of a still unidentified EDCF(s) (feline cerebral artery). Thus the endothelium can serve as pressure and flow sensor and is capable of transducing changes in mechanical forces into changes of vascular smooth muscle tone by modulating the release of endothelium-derived vasoactive factors. The physiological importance of the mechanoreception by endothelial cells in the intact circulation remains to be determined.

机械感受是一种广泛分布的感觉方式,已被证明存在于某些血管中。物理力的变化,如跨壁压力或流速(剪切应力)的突然增加,引发血管直径的变化;前者减小了压力,后者增大了容器口径。这些直径的变化是血管介质中血管平滑肌收缩和松弛的结果,可以达到血液流动的生理调节(自动调节)和保护内膜免受高剪切力损伤的目的。机械传感器的精确位置以及机械接收和信号转导的机制尚不清楚。越来越多的证据表明,内皮可能是机械接受的一个部位,内皮源性舒张因子(EDRF, EDHF, PGI2)和收缩因子(EDCF)的合成/释放的变化导致血管平滑肌张力和血管直径的改变。增加的剪切应力刺激EDRF和PGI2的释放可能是通过激活内皮细胞膜上的K+通道(向内整流)。经壁压力增加引起的内皮依赖性血管收缩可能是(1)犬颈动脉EDRF释放减少和(2)刺激尚未识别的猫脑动脉EDCF释放的结果。因此,内皮可以作为压力和流量传感器,并能够通过调节内皮源性血管活性因子的释放,将机械力的变化转化为血管平滑肌张力的变化。内皮细胞在完整循环中机械接受的生理重要性仍有待确定。
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引用次数: 164
Inhibitor of ATP-sensitive K+ channel alters neither hypoxic contraction nor relaxation of rat aorta. atp敏感的K+通道抑制剂既不改变大鼠主动脉的缺氧收缩也不改变主动脉的舒张。
Pub Date : 1990-01-01 DOI: 10.1159/000158830
D M Rodman, K Hasunuma, J L Peach, I F McMurtry
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引用次数: 7
7th international symposium on vascular neuroeffector mechanisms. A satellite symposium of the 11th International Congress of Pharmacology, Amsterdam 1990. Bonn, FRG, July 8-11, 1990. Abstracts. 第七届血管神经效应机制国际研讨会。第11届国际药理学大会卫星专题讨论会,阿姆斯特丹,1990。波恩,德国,1990年7月8-11日。摘要。
Pub Date : 1990-01-01
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引用次数: 0
Protein kinase C as a modulator of response amplification in vascular smooth muscle. 蛋白激酶C在血管平滑肌反应放大中的调节作用。
Pub Date : 1990-01-01 DOI: 10.1159/000158827
I Laher, L P Thompson, L Gagne

The amplification of alpha-adrenoceptor-mediated vasoconstriction by angiotensin II was studied in femoral artery rings from rabbits. Threshold concentrations of angiotensin II (0.1 nM) increased the maximal response to clonidine to 139 +/- 8% of control and produced a 3.2-fold increase in sensitivity. These effects of angiotensin II were reversed when tissues were pretreated with staurosporine (50 nM), an inhibitor of protein kinase C. The amplification of the alpha-adrenoceptor-mediated vasoconstrictor effects of thrombin and norepinephrine by angiotensin II were also reversed by pretreatment with staurosporine. Angiotensin II induced a response amplification in vascular smooth muscle known to be a nonspecific phenomenon, implying postreceptor interaction at intracellular transducer systems. Our findings suggest that upon activation of protein kinase C by angiotensin II, arterial responses to alpha-adrenoceptor agonists are amplified. This provides for nonspecific changes in vascular sensitivity by tonic alterations in postsynaptic modulation by enzyme systems known to regulate Ca2(+)-dependent phenomena, e.g. those related to vascular excitation-contraction mechanisms.

在兔股动脉环中研究了α -肾上腺素受体介导的血管紧张素II对血管收缩的扩增作用。血管紧张素II的阈值浓度(0.1 nM)使对可乐定的最大反应增加到对照的139 +/- 8%,敏感性增加3.2倍。当组织用staurosporine(一种蛋白激酶c的抑制剂)预处理时,血管紧张素II的这些作用被逆转。血管紧张素II对α -肾上腺素介导的凝血酶和去甲肾上腺素的血管收缩作用的放大也被staurosporine预处理逆转。血管紧张素II诱导血管平滑肌的反应放大是一种非特异性现象,暗示细胞内换能器系统的受体后相互作用。我们的研究结果表明,当血管紧张素II激活蛋白激酶C时,动脉对α -肾上腺素受体激动剂的反应被放大。这提供了血管敏感性的非特异性变化,通过已知的调节Ca2(+)依赖性现象的酶系统的突触后调节的强直性改变,例如那些与血管兴奋-收缩机制相关的现象。
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引用次数: 24
Noradrenaline induces rhythmic contractions of feline middle cerebral artery at low extracellular magnesium concentration. 低细胞外镁浓度下,去甲肾上腺素诱导猫大脑中动脉节律性收缩。
Pub Date : 1990-01-01 DOI: 10.1159/000158832
C. Szabó, E. Dóra, M. Faragó, I. Horváth, A. Kovách
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引用次数: 4
Mechanisms of vasodilation. Proceedings of the 5th international symposium, Strasbourg, France, July 5-7, 1989. 血管舒张的机制。第五届国际研讨会论文集,斯特拉斯堡,法国,1989年7月5-7日。
Pub Date : 1990-01-01
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引用次数: 0
期刊
Blood vessels
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