Pub Date : 2011-12-01DOI: 10.1177/204946371100500404
Donald Nixdorf, Estephan Moana-Filho
• New terminology, persistent dento-alveolar pain disorder (PDAP), and diagnostic criteria have been put forward to address the shortcomings of existing nomenclature, which are associated with unclear criteria. • Arriving at an accurate diagnosis of PDAP is based on excluding other possible aetiologies, and may involve different care providers. • Synthesis of published data suggests that PDAP has a frequency of occurrence following root canal therapy of around 1.6%. • The putative risk factors involved in PDAP are largely unknown, but seem to be similar to those being identified with other post-surgical chronic pain disorders. • The underlying mechanisms involved in the development of and/or perpetuating PDAP are unknown and the approach to treatment remains empiric in nature.
{"title":"Persistent dento-alveolar pain disorder (PDAP): Working towards a better understanding.","authors":"Donald Nixdorf, Estephan Moana-Filho","doi":"10.1177/204946371100500404","DOIUrl":"https://doi.org/10.1177/204946371100500404","url":null,"abstract":"• New terminology, persistent dento-alveolar pain disorder (PDAP), and diagnostic criteria have been put forward to address the shortcomings of existing nomenclature, which are associated with unclear criteria. • Arriving at an accurate diagnosis of PDAP is based on excluding other possible aetiologies, and may involve different care providers. • Synthesis of published data suggests that PDAP has a frequency of occurrence following root canal therapy of around 1.6%. • The putative risk factors involved in PDAP are largely unknown, but seem to be similar to those being identified with other post-surgical chronic pain disorders. • The underlying mechanisms involved in the development of and/or perpetuating PDAP are unknown and the approach to treatment remains empiric in nature.","PeriodicalId":90719,"journal":{"name":"Reviews in pain","volume":"5 4","pages":"18-27"},"PeriodicalIF":0.0,"publicationDate":"2011-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1177/204946371100500404","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"32742402","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2002-07-01DOI: 10.1191/0968130202PR190RA
K. Wynne
{"title":"Facet joint injections in the management of chronic low back pain: a review","authors":"K. Wynne","doi":"10.1191/0968130202PR190RA","DOIUrl":"https://doi.org/10.1191/0968130202PR190RA","url":null,"abstract":"","PeriodicalId":90719,"journal":{"name":"Reviews in pain","volume":"87 1","pages":"81-86"},"PeriodicalIF":0.0,"publicationDate":"2002-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"79154843","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2002-04-01DOI: 10.1191/0968130202PR187RA
J. Roelofs, M. D. Boissevain, M. Peters, J. Jong, J. Vlaeyen
{"title":"Psychological treatments for chronic low back pain: Past, present, and beyond","authors":"J. Roelofs, M. D. Boissevain, M. Peters, J. Jong, J. Vlaeyen","doi":"10.1191/0968130202PR187RA","DOIUrl":"https://doi.org/10.1191/0968130202PR187RA","url":null,"abstract":"","PeriodicalId":90719,"journal":{"name":"Reviews in pain","volume":"14 1","pages":"29-40"},"PeriodicalIF":0.0,"publicationDate":"2002-04-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"75288906","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2002-04-01DOI: 10.1191/0968130202PR186RA
T. Newton-John
This article offers a critical review of the literature examining spouse responses to the pain behaviour of chronic pain patients. An overview is given of 27 studies that have explored patient‐ spouse interactions in chronic pain, together with a summary of the various e ndings. It is concluded that the body of research is broadly supportive of the operant behavioural paradigm on which it has developed. Patients’ coping with chronic pain is signie cantly ine uenced by the ways in which those closest to them respond to their expressions of discomfort. However, it is argued that the behavioural model alone is insufe cient when accounting for the complexity of pain couples’ interactions. The impact of the spouse’ s response is mediated by a range of cognitive and affective variables that have yet to be fully recognized in the research literature. It is also argued that the operationalization of the construct of solicitousness, which is central to research on chronic pain couples, is e awed. A number of suggestions for future theoretical and empirical developments in this area are made.
{"title":"Solicitousness and chronic pain: a critical review","authors":"T. Newton-John","doi":"10.1191/0968130202PR186RA","DOIUrl":"https://doi.org/10.1191/0968130202PR186RA","url":null,"abstract":"This article offers a critical review of the literature examining spouse responses to the pain behaviour of chronic pain patients. An overview is given of 27 studies that have explored patient‐ spouse interactions in chronic pain, together with a summary of the various e ndings. It is concluded that the body of research is broadly supportive of the operant behavioural paradigm on which it has developed. Patients’ coping with chronic pain is signie cantly ine uenced by the ways in which those closest to them respond to their expressions of discomfort. However, it is argued that the behavioural model alone is insufe cient when accounting for the complexity of pain couples’ interactions. The impact of the spouse’ s response is mediated by a range of cognitive and affective variables that have yet to be fully recognized in the research literature. It is also argued that the operationalization of the construct of solicitousness, which is central to research on chronic pain couples, is e awed. A number of suggestions for future theoretical and empirical developments in this area are made.","PeriodicalId":90719,"journal":{"name":"Reviews in pain","volume":"42 1","pages":"7-27"},"PeriodicalIF":0.0,"publicationDate":"2002-04-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"74201889","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2002-04-01DOI: 10.1191/0968130202PR188RA
W. Farquhar-Smith
Pain and cannabinoids: science and evidence. The endogenous cannabinoid system is widespread and responsible for a number of modulatory functions, including analgesia. Akin to the endogenous opioid system, the analgesic actions of cannabinoids are mediated by the neuronal receptor ( CB1) and immune cell- expressed receptor ( CB2). Animal data have shown cannabinoids to be effective in many models of inflammatory and neuropathic pain. Less evidence is available from human studies, although investigation is under way. Ultimately, an effective cannabinoid analgesic should aim to be therapeutic but without the 'high' of unwanted central side effects.
{"title":"Pain and cannabinoids: science and evidence.","authors":"W. Farquhar-Smith","doi":"10.1191/0968130202PR188RA","DOIUrl":"https://doi.org/10.1191/0968130202PR188RA","url":null,"abstract":"Pain and cannabinoids: science and evidence. The endogenous cannabinoid system is widespread and responsible for a number of modulatory functions, including analgesia. Akin to the endogenous opioid system, the analgesic actions of cannabinoids are mediated by the neuronal receptor ( CB1) and immune cell- expressed receptor ( CB2). Animal data have shown cannabinoids to be effective in many models of inflammatory and neuropathic pain. Less evidence is available from human studies, although investigation is under way. Ultimately, an effective cannabinoid analgesic should aim to be therapeutic but without the 'high' of unwanted central side effects.","PeriodicalId":90719,"journal":{"name":"Reviews in pain","volume":"13 1","pages":"41-67"},"PeriodicalIF":0.0,"publicationDate":"2002-04-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"82027849","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2002-04-01DOI: 10.1191/0968130202PR185RA
Blake, N. Shenker, P. Mapp, R. Haigh, N. Harris
{"title":"Musgrave–Mitchell–Charcot neuroarthropathy: a review and hypothesis","authors":"Blake, N. Shenker, P. Mapp, R. Haigh, N. Harris","doi":"10.1191/0968130202PR185RA","DOIUrl":"https://doi.org/10.1191/0968130202PR185RA","url":null,"abstract":"","PeriodicalId":90719,"journal":{"name":"Reviews in pain","volume":"5 1","pages":"1-6"},"PeriodicalIF":0.0,"publicationDate":"2002-04-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"78163950","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2001-10-01DOI: 10.1191/0968130201PR182RA
Mark I. Johnson
{"title":"Transcutaneous Electrical Nerve Stimulation (TENS) and TENS-like devices: do they provide pain relief?","authors":"Mark I. Johnson","doi":"10.1191/0968130201PR182RA","DOIUrl":"https://doi.org/10.1191/0968130201PR182RA","url":null,"abstract":"","PeriodicalId":90719,"journal":{"name":"Reviews in pain","volume":"142 1","pages":"121-158"},"PeriodicalIF":0.0,"publicationDate":"2001-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"89221434","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2001-10-01DOI: 10.1191/0968130201PR181RA
J. Walker
Therapy with opioids is an exciting new development for arthritis, especially since there is the potential for fewer side effects from molecules that act outside the central nervous system. We have found k-opioid drugs to be powerfully anti-in eammatory, reducing disease severity by as much as 80% and attenuating arthritis in a dose-dependent, stereoselective, antagonist-reversible manner. In contrast, opioids acting at other receptors were therapeutic only at near toxic doses. Currently, however, no pure k-opioids are available for clinical use. The hypothalamic-pituitary-adrenal axis was found to be only partially involved; thus we investigated other neural and immune mechanisms. The results showed that the k-opioid anti-in eammatory actions were exerted via: (1) reduced adhesion molecule expression; (2) inhibition of cell trafecking; (3) reduced tumour necrosis factor release; and (4) alterations in mRNA expression and substance P (SP) and calcitonin gene-related peptide protein in joint tissue. The ability of k-opioids to act at multiple sites in the in eammatory cascade, as suggested by the presence of opioid receptors at various locations throughout the cascade, may explain their powerful actions. k-Opioids are, however, most therapeutic during disease onset; thus it is likely they exert their anti-in eammatory effects predominantly via changes in cellular activation and cytokine expression rather than via the nervous system. The involvement of SP and the efecacy of neurokinin 1 (NK1) antagonists predicts that combined opioid ‐NK1 regimens have therapeutic promise. Peripherally acting opioids may prove to be a potent new treatment for rheumatoid arthritis sufferers in the future.
{"title":"Anti-inè ammatory effects of k-opioids: relevance to rheumatoid arthritis","authors":"J. Walker","doi":"10.1191/0968130201PR181RA","DOIUrl":"https://doi.org/10.1191/0968130201PR181RA","url":null,"abstract":"Therapy with opioids is an exciting new development for arthritis, especially since there is the potential for fewer side effects from molecules that act outside the central nervous system. We have found k-opioid drugs to be powerfully anti-in eammatory, reducing disease severity by as much as 80% and attenuating arthritis in a dose-dependent, stereoselective, antagonist-reversible manner. In contrast, opioids acting at other receptors were therapeutic only at near toxic doses. Currently, however, no pure k-opioids are available for clinical use. The hypothalamic-pituitary-adrenal axis was found to be only partially involved; thus we investigated other neural and immune mechanisms. The results showed that the k-opioid anti-in eammatory actions were exerted via: (1) reduced adhesion molecule expression; (2) inhibition of cell trafecking; (3) reduced tumour necrosis factor release; and (4) alterations in mRNA expression and substance P (SP) and calcitonin gene-related peptide protein in joint tissue. The ability of k-opioids to act at multiple sites in the in eammatory cascade, as suggested by the presence of opioid receptors at various locations throughout the cascade, may explain their powerful actions. k-Opioids are, however, most therapeutic during disease onset; thus it is likely they exert their anti-in eammatory effects predominantly via changes in cellular activation and cytokine expression rather than via the nervous system. The involvement of SP and the efecacy of neurokinin 1 (NK1) antagonists predicts that combined opioid ‐NK1 regimens have therapeutic promise. Peripherally acting opioids may prove to be a potent new treatment for rheumatoid arthritis sufferers in the future.","PeriodicalId":90719,"journal":{"name":"Reviews in pain","volume":"51 1","pages":"113-119"},"PeriodicalIF":0.0,"publicationDate":"2001-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"86953413","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2001-06-01DOI: 10.1191/0968130201PR178RA
S. Grond, T. Meert, H. Noorduin
{"title":"Safety and tolerability of opioid analgesia: a comparable class profile?","authors":"S. Grond, T. Meert, H. Noorduin","doi":"10.1191/0968130201PR178RA","DOIUrl":"https://doi.org/10.1191/0968130201PR178RA","url":null,"abstract":"","PeriodicalId":90719,"journal":{"name":"Reviews in pain","volume":"6 1","pages":"103-111"},"PeriodicalIF":0.0,"publicationDate":"2001-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"82826702","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2001-06-01DOI: 10.1191/0968130201PR176RA
M. Börjesson, Henrik Norssell
{"title":"Visceral chest pain: the role of neurostimulation","authors":"M. Börjesson, Henrik Norssell","doi":"10.1191/0968130201PR176RA","DOIUrl":"https://doi.org/10.1191/0968130201PR176RA","url":null,"abstract":"","PeriodicalId":90719,"journal":{"name":"Reviews in pain","volume":"76 1","pages":"75-94"},"PeriodicalIF":0.0,"publicationDate":"2001-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"81038980","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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