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Handbook of behavioral neuroscience最新文献

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Disease-Associated Changes in the Striosome and Matrix Compartments of the Dorsal Striatum 与疾病相关的纹状体和纹状体基质区的变化
Pub Date : 2017-01-01 DOI: 10.1016/B978-0-12-802206-1.00039-8
J. Crittenden, A. Graybiel
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引用次数: 20
Alterations of Synaptic Function in Huntington's Disease 亨廷顿氏病突触功能的改变
Pub Date : 2017-01-01 DOI: 10.1016/B978-0-12-802206-1.00046-5
Anna Parievsky, C. Cepeda, M. Levine
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引用次数: 1
Receptors and Second Messengers in the Basal Ganglia 基底神经节中的受体和第二信使
Pub Date : 2017-01-01 DOI: 10.1016/B978-0-12-802206-1.00028-3
E. B. Warren, S. Sillivan, C. Konradi
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引用次数: 2
Phasic Dopamine Signaling in Action Selection and Reinforcement Learning 动作选择和强化学习中的阶段性多巴胺信号
Pub Date : 2017-01-01 DOI: 10.1016/B978-0-12-802206-1.00034-9
P. Redgrave, N. Vautrelle, P. Overton, J. Reynolds
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引用次数: 5
Molecular Mechanisms of l-DOPA-Induced Dyskinesia 左旋多巴诱导运动障碍的分子机制
Pub Date : 2017-01-01 DOI: 10.1016/B978-0-12-802206-1.00043-X
A. Nilsson
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引用次数: 2
Toward a Diathesis-Stress Model of Schizophrenia in a Neurodevelopmental Perspective 从神经发育的角度探讨精神分裂症的素质-应激模型
Pub Date : 2016-01-01 DOI: 10.1016/B978-0-12-800981-9.00013-4
A. Berry, F. Cirulli
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引用次数: 1
Dysregulation of Dopamine Systems in a Developmental Disruption Model of Schizophrenia: Implications for Pathophysiology, Treatment, and Prevention 精神分裂症发育中断模型中多巴胺系统的失调:病理生理学、治疗和预防的意义
Pub Date : 2016-01-01 DOI: 10.1016/B978-0-12-800981-9.00008-0
Sofia Dibble, Yijuan Du, A. Grace
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引用次数: 3
Alternative Human Cell Models for Neuropsychiatric Research: Induced Neuronal Cells, Olfactory Epithelium, and Related Resources 神经精神病学研究的替代人类细胞模型:诱导神经元细胞、嗅上皮和相关资源
Pub Date : 2016-01-01 DOI: 10.1016/B978-0-12-800981-9.00024-9
Brian D. Lo, Daniel J Chang, S. Kano
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引用次数: 0
Role of Immune and Autoimmune Dysfunction in Schizophrenia. 免疫和自身免疫功能障碍在精神分裂症中的作用。
Pub Date : 2016-01-01 Epub Date: 2015-11-10 DOI: 10.1016/B978-0-12-800981-9.00029-8
Emily G Severance, Robert H Yolken

In this chapter, we review data in support of the concept that immune system dysregulation is the most plausible explanation that reconciles gene by environmental interactions in schizophrenia. Early investigations of this topic demonstrated aspects of aberrant activation of humoral immunity, including autoimmunity, associated with schizophrenia, whereas current research efforts have expanded this theme to include elements of innate immunity. Advances in our understanding of inflammation and molecules of both the adaptive and innate immune system and their functional roles in standard brain physiology provide an important context by which schizophrenia might arise as the result of the coupling of immune and neurodevelopmental dysregulation.

在本章中,我们回顾了支持免疫系统失调是精神分裂症中通过环境相互作用调和基因的最合理解释这一概念的数据。该主题的早期研究表明,包括自身免疫在内的体液免疫的异常激活与精神分裂症有关,而目前的研究工作已将这一主题扩展到包括先天免疫的要素。我们对炎症和适应性免疫和先天免疫系统分子及其在标准脑生理学中的功能作用的理解的进展,为精神分裂症可能作为免疫和神经发育失调耦合的结果而出现提供了重要的背景。
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引用次数: 2
Determinants of Selective Vulnerability of Dopamine Neurons in Parkinson's Disease 帕金森病多巴胺神经元选择性易感性的决定因素
Pub Date : 2016-01-01 DOI: 10.1016/B978-0-12-802206-1.00041-6
E. Zampese, Daniel J. Galtieri, P. Schumacker, D. Surmeier
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引用次数: 1
期刊
Handbook of behavioral neuroscience
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