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Are your kidneys Ok? Detect early to protect kidney health
IF 14.8 1区 医学 Q1 UROLOGY & NEPHROLOGY
Kidney international
Pub Date : 2025-02-19 DOI: 10.1016/j.kint.2024.12.006
Joseph A. Vassalotti , Anna Francis , Augusto Cesar Soares dos Santos Jr. , Ricardo Correa-Rotter , Dina Abdellatif , Li-Li Hsiao , Stefanos Roumeliotis , Agnes Haris , Latha A. Kumaraswami , Siu-Fai Lui , Alessandro Balducci , Vassilios Liakopoulos , World Kidney Day Joint Steering Committee
Early identification of kidney disease can protect kidney health, prevent kidney disease progression and related complications, reduce cardiovascular disease risk, and decrease mortality. We must ask “Are your kidneys ok?” using serum creatinine to estimate kidney function and urine albumin to assess for kidney and endothelial damage. Evaluation for causes and risk factors for chronic kidney disease (CKD) includes testing for diabetes and measurement of blood pressure and body mass index. This World Kidney Day we assert that case-finding in high-risk populations, or even population level screening, can decrease the burden of kidney disease globally. Early-stage CKD is asymptomatic and simple to test for, and recent paradigm shifting CKD treatments such as sodium glucose co-transporter-2 inhibitors dramatically improve outcomes and favor the cost-benefit analysis for screening or case-finding programs. Despite this, numerous barriers exist, including resource allocation, health care funding, health care infrastructure, and health care professional and population awareness of kidney disease. Coordinated efforts by major kidney nongovernmental organizations to prioritize the kidney health agenda for governments and aligning early detection efforts with other current programs will maximize efficiencies.
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引用次数: 0
Utilization of kidney biopsies in patients with acute kidney injury: a piece of the puzzle in a complex patient setting
IF 14.8 1区 医学 Q1 UROLOGY & NEPHROLOGY
Kidney international
Pub Date : 2025-02-19 DOI: 10.1016/j.kint.2024.12.001
T. Alp Ikizler
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引用次数: 0
The authors reply
IF 14.8 1区 医学 Q1 UROLOGY & NEPHROLOGY
Kidney international
Pub Date : 2025-02-19 DOI: 10.1016/j.kint.2024.12.004
Janina M. Herold , Carsten A. Böger , Iris M. Heid
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引用次数: 0
Glomerular filtration rate equations in kidney transplant recipients: the need for specificity
IF 14.8 1区 医学 Q1 UROLOGY & NEPHROLOGY
Kidney international
Pub Date : 2025-02-19 DOI: 10.1016/j.kint.2024.11.015
Marc Raynaud , Stephan J.L. Bakker , Andrew J. Bentall , Alexandre Loupy
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引用次数: 0
Subscription Information
IF 14.8 1区 医学 Q1 UROLOGY & NEPHROLOGY
Kidney international
Pub Date : 2025-02-19 DOI: 10.1016/S0085-2538(25)00020-1
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引用次数: 0
The combination of SGLT2 inhibitors and glucagon-like peptide 1 receptor agonists: are 2 drugs better than 1?
IF 14.8 1区 医学 Q1 UROLOGY & NEPHROLOGY
Kidney international
Pub Date : 2025-02-19 DOI: 10.1016/j.kint.2024.11.032
Talat Alp Ikizler
{"title":"The combination of SGLT2 inhibitors and glucagon-like peptide 1 receptor agonists: are 2 drugs better than 1?","authors":"Talat Alp Ikizler","doi":"10.1016/j.kint.2024.11.032","DOIUrl":"10.1016/j.kint.2024.11.032","url":null,"abstract":"","PeriodicalId":17801,"journal":{"name":"Kidney international","volume":"107 3","pages":"Pages 385-388"},"PeriodicalIF":14.8,"publicationDate":"2025-02-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143437637","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Kidney biopsy in acute kidney injury: increase insight or preserve the status quo?
IF 14.8 1区 医学 Q1 UROLOGY & NEPHROLOGY
Kidney international
Pub Date : 2025-02-19 DOI: 10.1016/j.kint.2024.09.022
Zoltán H. Endre
{"title":"Kidney biopsy in acute kidney injury: increase insight or preserve the status quo?","authors":"Zoltán H. Endre","doi":"10.1016/j.kint.2024.09.022","DOIUrl":"10.1016/j.kint.2024.09.022","url":null,"abstract":"","PeriodicalId":17801,"journal":{"name":"Kidney international","volume":"107 3","pages":"Pages 397-400"},"PeriodicalIF":14.8,"publicationDate":"2025-02-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143437641","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
"Star chain pattern" of focal emphysematous pyelonephritis in polycystic kidney disease
IF 14.8 1区 医学 Q1 UROLOGY & NEPHROLOGY
Kidney international
Pub Date : 2025-02-19 DOI: 10.1016/j.kint.2024.08.039
Xiaofeng Jiang , Ming Jin , Xi Meng , Jianghua Yang , Xiaoxing Liao
{"title":"\"Star chain pattern\" of focal emphysematous pyelonephritis in polycystic kidney disease","authors":"Xiaofeng Jiang ,&nbsp;Ming Jin ,&nbsp;Xi Meng ,&nbsp;Jianghua Yang ,&nbsp;Xiaoxing Liao","doi":"10.1016/j.kint.2024.08.039","DOIUrl":"10.1016/j.kint.2024.08.039","url":null,"abstract":"","PeriodicalId":17801,"journal":{"name":"Kidney international","volume":"107 3","pages":"Page 577"},"PeriodicalIF":14.8,"publicationDate":"2025-02-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143436788","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Mitochondrial ROS connects P2X7-mediated Ca2+ influx with IL-1α release by monocytes upon chronic tissue damage
IF 14.8 1区 医学 Q1 UROLOGY & NEPHROLOGY
Kidney international
Pub Date : 2025-02-19 DOI: 10.1016/j.kint.2024.12.002
Verónica Miguel
Whereas acute inflammation plays an important role in both tissue damage and repair, chronic inflammation favors tissue injury. Monocytes contribute to this deleterious effect by secreting proinflammatory cytokines. Amini et al. propose a novel mechanism involving adenosine triphosphate–induced purinergic P2X purinoceptor 7 receptor activation, which leads to calcium ion–dependent mitochondrial reactive oxygen species production, triggering interleukin-1α release by monocytes. This stimulates kidney immune cell infiltration and fibrosis in chronic kidney disease and promotes adverse cardiac remodeling following myocardial infarction.
{"title":"Mitochondrial ROS connects P2X7-mediated Ca2+ influx with IL-1α release by monocytes upon chronic tissue damage","authors":"Verónica Miguel","doi":"10.1016/j.kint.2024.12.002","DOIUrl":"10.1016/j.kint.2024.12.002","url":null,"abstract":"<div><div>Whereas acute inflammation plays an important role in both tissue damage and repair, chronic inflammation favors tissue injury. Monocytes contribute to this deleterious effect by secreting proinflammatory cytokines. Amini <em>et al.</em> propose a novel mechanism involving adenosine triphosphate–induced purinergic P2X purinoceptor 7 receptor activation, which leads to calcium ion–dependent mitochondrial reactive oxygen species production, triggering interleukin-1α release by monocytes. This stimulates kidney immune cell infiltration and fibrosis in chronic kidney disease and promotes adverse cardiac remodeling following myocardial infarction.</div></div>","PeriodicalId":17801,"journal":{"name":"Kidney international","volume":"107 3","pages":"Pages 389-391"},"PeriodicalIF":14.8,"publicationDate":"2025-02-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143437638","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Can we crystallize the role of urate-lowering treatment in chronic kidney disease?
IF 14.8 1区 医学 Q1 UROLOGY & NEPHROLOGY
Kidney international
Pub Date : 2025-02-19 DOI: 10.1016/j.kint.2024.11.025
Austin G. Stack
The therapeutic value of serum urate lowering in chronic kidney disease is questionable given the lack of clinical benefit from randomized clinical trials. Post hoc analysis of the CARES (Cardiovascular Safety of Febuxostat and Allopurinol in Patients with Gout and Cardiovascular Morbidities) trial suggests a protective effect of urate lowering in gout participants over 2.5 years of follow-up with the greatest benefit for those who maintained average urate levels of <6 mg/dl. A shift in research focus from asymptomatic hyperuricemia to symptomatic hyperuricemia may yet yield dividends.
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引用次数: 0
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