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Journal of Japan Society of Immunology & Allergology in Otolaryngology最新文献

英文 中文
あとがき 后记
Pub Date : 1954-06-20 DOI: 10.2331/suisan.84.s270a
勤二 清水
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引用次数: 0
第5回日本耳鼻咽喉科免疫アレルギー学会奨励賞 第5回日本耳鼻喉科免疫过敏学会奖励奖
Pub Date : 1900-01-01 DOI: 10.5648/jjiao.38.s49
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引用次数: 0
Allergic rhinitis in infants: Current status and issues 婴儿变应性鼻炎:现状和问题
Pub Date : 1900-01-01 DOI: 10.5648/jjiao.37.17
S. Masuda
我が国でよく知られているのは,馬場らによる耳鼻咽 喉科医とその家族を対象にした 2008年のアレルギー性鼻 炎有症率の調査である。それによれば 0~4歳児の通年性 アレルギー性鼻炎の有症率は 4.0%,同じくスギ花粉症は 1.1%と決して高くはないが,その後 10歳代にかけて急速 に有症率が増加することが示されている。 低年齢児のアレルギー性鼻炎の疫学調査の難しさの一因 は,診断基準が確立していないことにある。2歳以下の有 症率は,「かぜをひいていないのに水ばなが出る」程度の 基準であれば 7~29%,医学的な診断基準を加えると 3~ 4%と報告されている。Herrらによる出生コホート研究 では,18ヵ月児の 9.1%に鼻汁,くしゃみ,鼻閉などのア レルギー性鼻炎様症状が認められた。Osawaらは本邦の 1歳 6ヵ月児健診で,1.5%の子どもが鼻症状,鼻汁中好酸 球陽性,ダニ・スギ・ネコいずれかの血清特異的 IgE抗体 陽性,の条件を満たしてアレルギー性鼻炎と診断されたと 報告している。
2008年马场等人以耳鼻咽喉科医生及其家人为对象进行的过敏性鼻炎患病率调查在日本广为人知。据此显示,0~4岁儿童的全年性过敏性鼻炎的患病率为4.0%,而杉树花粉症的患病率为1.1%,虽然并不高,但之后到10岁左右,患病率会急速增加。低龄儿童过敏性鼻炎的流行病学调查之所以困难,原因之一在于尚未确立诊断标准。2岁以下的患病率,以“没有感冒却有水垢”的程度为基准为7~29%,加上医学诊断基准为3~ 4%。Herr等人在出生队列研究中发现,9.1%的18个月婴儿出现流鼻涕、打喷嚏、鼻闭等过敏性鼻炎样症状。Osawa等人在本邦1岁6个月的儿童体检中,1.5%的孩子有鼻子症状,鼻涕中嗜酸性粒细胞阳性,螨虫、杉、猫血清特异性IgE抗体阳性,满足上述条件后被诊断为过敏性鼻炎。正在报告。
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引用次数: 0
Educational Lecture 教育讲座
Pub Date : 1900-01-01 DOI: 10.5648/jjiao.38.94
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引用次数: 0
Sick house syndrome 病宅综合症
Pub Date : 1900-01-01 DOI: 10.5648/jjiao.38.17
Hiroki Ikeda
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引用次数: 0
A case of pollen food allergy syndrome with anaphylaxis 花粉食物过敏综合征伴过敏反应1例
Pub Date : 1900-01-01 DOI: 10.5648/jjiao.37.257
T. Ishino
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引用次数: 0
第4回日本耳鼻咽喉科免疫アレルギー学会奨励賞 第4回日本耳鼻喉科免疫过敏学会奖励奖
Pub Date : 1900-01-01 DOI: 10.5648/jjiao.38.s48
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引用次数: 0
The present situation and prospect about pollen-food allergy syndrome 花粉食物过敏综合征的现状与展望
Pub Date : 1900-01-01 DOI: 10.5648/jjiao.38.43
Yoko Osawa
Pollen-food allergy syndrome (PFAS), also called “oral allergy syndrome”, is a class 2 food allergy that is induced by cross-reaction between pollen allergens and food allergens. It is mainly limited to the oral mucosa, but in some cases, PFAS induces allergic reactions in the nose, eyes, skin, and respiratory system, while also causing shock. PFAS is caused by allergens having high homology to various foods and pollen pan-allergens, and once it develops, it begins to induce allergic reactions to various foods and pollens. In Japan, PFAS is predominantly caused by pollens of the Betulaceae family, including PR-10/Bet v1 related proteins, and pollens of the Gramineae family, including profilins. However, single-sensitization to Japanese cedar pollen is not a risk factor for PFAS. Moreover, it is difficult to prove the existence of specific IgE causing food allergy in PFAS. Instead, it is recommended to measure pollen-specific IgE, which has cross-reactivity to food allergens. Although PFAS is generally prevented by avoiding eating foods that induce allergic reactions, in the case of PFAS caused by PR-10/Bet v1 related proteins and profilins, it is possible to eat cooked foods because heat deactivates antigenicity. However, precautions should be taken when PFAS is caused by foods containing Gly m 4 (soybean) and LTP because these allergens have heat-resistant properties. The measurement of Gly m 4 is already useful and applicable to insurance.
花粉食物过敏综合征(PFAS)又称“口腔过敏综合征”,是由花粉过敏原与食物过敏原交叉反应引起的二类食物过敏。它主要局限于口腔黏膜,但在某些情况下,PFAS会引起鼻子、眼睛、皮肤和呼吸系统的过敏反应,同时也会引起休克。PFAS是由与各种食物和花粉泛过敏原具有高度同源性的过敏原引起的,一旦发生,就开始诱发对各种食物和花粉的过敏反应。在日本,PFAS主要由桦树科花粉(包括PR-10/ betv1相关蛋白)和禾草科花粉(包括profilins)引起。然而,对杉木花粉的单一致敏不是PFAS的危险因素。此外,难以证明PFAS中存在引起食物过敏的特异性IgE。相反,建议测量花粉特异性IgE,它对食物过敏原有交叉反应。虽然PFAS通常是通过避免食用引起过敏反应的食物来预防的,但在由PR-10/ betv1相关蛋白和谱蛋白引起的PFAS的情况下,可以食用熟食,因为热量会使抗原性失活。然而,当PFAS是由含有Gly m1(大豆)和LTP的食物引起时,应采取预防措施,因为这些过敏原具有耐热性。Gly - m4的测量方法在保险中已经是有用的和适用的。
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引用次数: 0
Granulomatosis with polyangiitis 肉芽肿伴多血管炎
Pub Date : 1900-01-01 DOI: 10.5648/jjiao.37.265
J. Ohori
Granulomatosis with polyangiitis (GPA) is a condition that causes inflammation that primarily affects the respiratory tract (including the lungs and airways) and the kidneys. This disorder is formerly known as Wegener granulomatosis. A characteristic feature of GPA is inflammation of blood vessels (vasculitis), particularly the smalland mediumsized blood vessels in the lungs, nose, sinuses, windpipe, and kidneys, although vessels in any organ can be involved. Polyangiitis refers to the inflammation of multiple types of vessels, such as small arteries and veins. Vasculitis causes scarring and tissue death in the vessels and impedes blood flow to tissues and organs.
肉芽肿病伴多血管炎(GPA)是一种主要影响呼吸道(包括肺和气道)和肾脏的炎症。这种疾病以前被称为韦格纳肉芽肿病。GPA的一个特征是血管炎症(血管炎),尤其是肺、鼻、鼻窦、气管和肾脏的中小型血管,尽管任何器官的血管都可能受累。多血管炎是指多种血管的炎症,如小动脉和静脉。血管炎会在血管中造成疤痕和组织死亡,阻碍血液流向组织和器官。
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引用次数: 1
Special Lecture 特殊的讲座
Pub Date : 1900-01-01 DOI: 10.5648/jjiao.38.93
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引用次数: 0
期刊
Journal of Japan Society of Immunology & Allergology in Otolaryngology
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